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1 sion of Egr1 while decreasing ATGL levels in epididymal fat.
2 showed significantly reduced body weight and epididymal fat.
3 dexamethasone prevented ATM accumulation in epididymal fat.
4 ependent and PPARalpha-independent manner in epididymal fat.
5 neal fat, but was without effect on GLUT4 in epididymal fat.
6 receptor type in islets, liver, kidney, and epididymal fat.
7 a, and an enhanced proinflammatory status of epididymal fat.
8 ce also exhibit adipocyte hypertrophy in the epididymal fat.
10 <0.01) less retroperitoneal, mesenteric, and epididymal fat accumulation, compared to their ob/ob cou
11 trophy and interlelukin-6 gene expression in epididymal fat, along with the splenic proinflammatory p
12 white adipose tissues including inguinal and epididymal fats and also in brown adipose tissue but not
13 ethylation of its promoter in progenitors of epididymal fat compared to Con offspring, which was corr
14 el was lower in human visceral fat and mouse epididymal fat compared with their subcutaneous fat.
16 leasable and extractable LPL activity in the epididymal fat decreased by 75-80% in the diabetic group
17 lation of Foxa3 have a selective decrease in epididymal fat depot and a cell-autonomous defect to ind
20 that regulates the selective enlargement of epididymal fat depots and suppresses energy expenditure
21 depot could affect vascular disease in mice, epididymal fat depots were transplanted into atheroscler
22 se in the diet, whereas [(3)H]NE turnover in epididymal fat did not respond to either monosaccharide.
23 oliferator-activated receptor (PPAR)gamma in epididymal fat; enzymes of fatty acid oxidation and thei
24 in GLUT4 (54 +/- 5% of high-carbohydrate) in epididymal fat from rats on the high-fat diet for 3 week
26 -induced glucose uptake in soleus muscle and epididymal fat; insulin inhibition of lipolysis was also
27 nce in skeletal muscle, whereas in liver and epididymal fat it was associated with increased expressi
30 NA methylation in the zfp423 promoter in the epididymal fat of OB/Obe offspring, which was correlated
31 in confined well-vascularized sites like the epididymal fat pad (EFP) improved graft outcomes, but on
32 Micro) in the confined and well-vascularized epididymal fat pad (EFP) site, a model of the human omen
33 ouse islets engrafted on the intra-abdominal epididymal fat pad ameliorated streptozotocin-induced hy
36 hich bioscaffolds were transplanted into the epididymal fat pad of diabetic mice, demonstrated that,
37 eeded scaffolds were then implanted onto the epididymal fat pad of syngeneic mice with streptozotocin
39 t affected until week 28 (decreased by 14%); epididymal fat pad weight also decreased (25%) at this t
40 yperleptinemia and reduction in food intake, epididymal fat pad weight declined 55% in wild-type but
41 air-fed group, a significant increase in the epididymal fat pad weight, BAT weight, and plasma leptin
43 ell as lower hepatic triglyceride levels and epididymal fat pad weights than in SHR harboring mutant
44 3) Does short-term elimination of T cells in epididymal fat pad without disturbing the systemic T cel
45 o the devices, which were implanted into the epididymal fat pad(s) of streptozocin diabetic mice.
46 er visceral adipose tissue mass estimated by epididymal fat pad, associated with iron accumulation in
47 w as 50 islets, either intraportal or in the epididymal fat pad, displayed similar glucose tolerance
48 ic mass of syngeneic islets implanted in the epididymal fat pad, followed by a subrenal capsular impl
49 that for M-CPT I in RNA isolated from whole epididymal fat pad, this was reversed in purified adipoc
50 mia and increased homocysteine levels in the epididymal fat pad, which was associated with decreased
52 cally, adiponectin KO mice possessed smaller epididymal fat pads and showed reduced body weight compa
55 have greater deposits of s.c. fat and larger epididymal fat pads in comparison with wild-type mice.
56 PPARgamma-regulated genes were higher in the epididymal fat pads of Adipoq-LPL mice than control mice
58 adipocytes from SPARC-null versus wild-type epididymal fat pads were 252 +/- 61 and 161 +/- 33 micro
59 stablish feasibility of fat transplantation, epididymal fat pads were harvested from wild-type C57BL/
61 ties, including low fertility, an absence of epididymal fat pads, and a tendency to develop blepharit
62 nd in primary adipocytes isolated from mouse epididymal fat pads, in response to acute activation of
63 ere found to have lower body weight, smaller epididymal fat pads, lower blood levels of nonesterified
65 that GSK-3 activity increased twofold in the epididymal fat tissue and remained unchanged in muscle a
66 ntrast, GSK-3 activity did not change in the epididymal fat tissue of A/J mice, regardless of the typ
67 th reduced levels of the pP65 protein in the epididymal fat tissue, suggesting less activation of the
68 1 protein levels were significantly lower in epididymal fat tissues from db/db and high fat diet-indu
70 The rate of triacylglycerol synthesis in epididymal fat was 4.2-fold greater in SED5 than in WL5,
73 t and GSK3 phosphorylation and activities in epididymal fat were opposite to those of brain after str