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1 fetal development but not postnatally in the epiphysis.
2 arrow and bone necrosis involving the entire epiphysis.
3 uding the articular cartilage, meniscus, and epiphysis.
4 hological fractures or slipped upper femoral epiphysis.
5 rain, spinal cord, tail epidermis, lens, and epiphysis.
6 elative trabecular bone volume in the tibial epiphysis.
7 ts with osteonecrosis of the capital femoral epiphysis.
8  the generation of neurones in the zebrafish epiphysis.
9 t requirements for ash1a and ngn1 within the epiphysis.
10  in the diagnosis of slipped capital femoral epiphysis.
11 ic chondrocytes to become osteoblasts at the epiphysis.
12  metaphysis and resulted in bone loss in the epiphysis.
13 ial cells (ECs), exclusively existing in the epiphysis.
14 cular thickness and number were lower in the epiphysis.
15 tomic sites), metaphysis (86%, 60 of 70) and epiphysis (67%, 47 of 70) were involved.
16             No bone loss was observed in the epiphysis, a site experiencing higher strain energy.
17                                Uniquely, the epiphysis also appears to employ CART as a neurotransmit
18 al disorders such as slipped capital femoral epiphysis and Blount disease is well reported.
19 was absent in the humeral shaft and proximal epiphysis and decreased in the proximal physis.
20 sed for diagnosis of slipped capital femoral epiphysis and in the assessment of pre-slips.
21      Additionally, mitfb is expressed in the epiphysis and olfactory bulb where mitfa is not, and whe
22 n lamb epiphyses (including cartilage of the epiphysis and physis, and bone of the secondary ossifica
23 ether with reduced bone volume in the tibial epiphysis and vertebrae detected by microcomputed tomogr
24 njuries includes injury to the bone, physis, epiphysis, and apophysis.
25                      Slipped capital femoral epiphysis appears to be associated with a variety of end
26 f the neurogenesis defects in the flh-mutant epiphysis but also shows that Flh has additional activit
27 es inhibits endochondral ossification at the epiphysis by suppressing HIF signaling pathways.
28  evolution of the vascular compromise of the epiphysis due to the metaphyseal osteomyelitis complicat
29 ion-recovery MR images of the distal femoral epiphysis in children aged 2 months to 5 years 5 months
30 emonstrated photopenia of the distal femoral epiphysis in the absence of infection of the epiphysis o
31        The pronounced uptake in the juvenile epiphysis indicates that the proliferating zone of the p
32         Osteonecrosis of the capital femoral epiphysis is a significant late toxicity of treatment fo
33  in the diagnosis of slipped capital femoral epiphysis is suspicion by the practitioner.
34 hrough the physis with resultant slip of the epiphysis, is the most common hip abnormality in adolesc
35  of an aberrant phenotypic definition of the epiphysis itself.
36  are seen to be more dramatic in the humeral epiphysis (larger marrow volume fraction) than in the fe
37 rized into locations: metaphysis--diaphysis--epiphysis (MDE), pelvis, ribs, spine, and others.
38 jection, the biodistribution of 153Sm in the epiphysis, metaphysis, diaphysis, and red marrow of the
39                 Mineralization at the distal epiphysis occurs in an osteoblast-rich secondary ossific
40                                          The epiphysis of developing bones is a cartilaginous structu
41 ge proportion of the osteogenic cells in the epiphysis of long bone carried the donor SP cell marker.
42  During fetal development, the cartilaginous epiphysis of the distal femur transformed from an oval t
43 ng signal intensity within the cartilaginous epiphysis of the distal femur.
44 ral content of the peripheral portion of the epiphysis of the suspended tibia was reduced by 0.89 +/-
45 /diaphysis, metaphysis only, diaphysis only, epiphysis only, and the entire bone.
46 in a region corresponding to the presumptive epiphysis or pineal organ.
47 epiphysis in the absence of infection of the epiphysis or the joint space.
48                      Slipped capital femoral epiphysis remains a diagnostic problem despite numerous
49                      Slipped capital femoral epiphysis (SCFE) is the most common adolescent hip condi
50                      Slipped capital femoral epiphysis (SCFE), a fracture through the physis with res
51 -years) of fracture, slipped capital femoral epiphysis (SCFE), and avascular necrosis/osteonecrosis (
52 bar vertebra and distal femur metaphysis and epiphysis; significant differences were not detected in
53                            They showed small epiphysis, slightly flared metaphysis of long bones and
54 s venous thrombosis, slipped capital femoral epiphysis, stroke, or testicular torsion.
55 es progression of the bone collar toward the epiphysis, suggesting a synergistic effect between ectop
56 stricted manner, including expression in the epiphysis, the olfactory bulb and placodes, the eyes, ea
57 Col2 expressing immature chondrocytes in the epiphysis transition into prehypertrophic and hypetrophi
58 turnover in the uncalcified cartilage of the epiphysis was decreased transiently in vivo, but this di
59                 The underlying cartilaginous epiphysis was deranged as well and displayed random patt
60  develop osteonecrosis of the distal femoral epiphysis when administered 4 to 8 mg/L dexamethasone in
61 ation of some cartilage in the metaphysis or epiphysis while growth continues.
62 ations in the shaft curvature and the distal epiphysis width.
63 A posterior displacement of the femoral head epiphysis with a physeal step was seen on the longitudin