ライフサイエンスコーパス: epithelial-mesenchymal transition

1 feration markers and proteins that drive the epithelial mesenchymal transition.

2 feration, differentiation, self-renewal, and epithelial-mesenchymal transition.

3 nhibition of renal fibroblast activation and epithelial-mesenchymal transition.

4 n inhibitory effect on the OPN-mediated cell epithelial-mesenchymal transition.

5 n regulating alternative splicing during the epithelial-mesenchymal transition.

6 athways including inflammation processes and epithelial-mesenchymal transition.

7 mesoderm and definitive endoderm, through an epithelial-mesenchymal transition.

8 led SLUG), implicating LGR4 in regulation of epithelial-mesenchymal transition.

9 s been shown to accelerate tumorigenesis and epithelial-mesenchymal transition.

10 tively correlated with genes associated with epithelial-mesenchymal transition.

11 Hippo signaling activity, nor does it induce epithelial-mesenchymal transition.

12 192 promoter methylation and expression with epithelial-mesenchymal transition.

13 Fringe, is specifically required for cardiac epithelial-mesenchymal transition.

14 and NCI-H1944 cells, accompanied by reduced epithelial-mesenchymal transition.

15 Smad pathways and preference of apoptosis to epithelial-mesenchymal transition.

16 metastatic behavior by triggering a partial epithelial-mesenchymal transition.

17 h promotes the expression of Tgfb1, enabling epithelial-mesenchymal transition.

18 d adenocarcinoma, characterized by extensive epithelial-mesenchymal transition.

19 involving resident stem/progenitor cells and epithelial-mesenchymal transitions.

20 s between inductions of partial and complete epithelial-mesenchymal transitions.

21 OXC2 is induced and necessary for successful epithelial-mesenchymal transition, a developmental progr

22 EZH2 has been involved in epithelial-mesenchymal transition, a key event in develo

23 Following epithelial-mesenchymal transition, acquisition of avian

24 to explore the influence of SHH/GLI1 axis on epithelial mesenchymal transition and invasion in breast

25 P compromised angiogenesis by downregulating epithelial-mesenchymal transition and angiogenic pathway

26 and stabilize SNAIL1, a key factor promoting epithelial-mesenchymal transition and breast cancer meta

27 or invasiveness and metastasis by increasing epithelial-mesenchymal transition and by enriching a can

28 istance can occur following the emergence of epithelial-mesenchymal transition and by reactivation of

29 leading to subsequent downregulation of the epithelial-mesenchymal transition and cancer stem-like c

30 e counterbalanced by its ability to suppress epithelial-mesenchymal transition and cell invasion.

31 thermore, BCL2L14-ETV6 fusions prime partial epithelial-mesenchymal transition and endow resistance t

32 tion of beta-catenin suppressed EGF-mediated epithelial-mesenchymal transition and facilitated epithe

33 Notch activation in Pten-null mice promoted epithelial-mesenchymal transition and FOXC2-dependent tu

34 Moreover, ILK inhibition blocked KRAS-driven epithelial-mesenchymal transition and growth factor-stim

35 Gene expression profiling revealed that epithelial-mesenchymal transition and hypoxia were the t

36 y EZH2, such as E-cadherin, which suppresses epithelial-mesenchymal transition and metastasis.

37 ing in promoting inflammation and hepatocyte epithelial-mesenchymal transition and migration, we used

38 controls cell migration in cSCC by blocking epithelial-mesenchymal transition and promoting prolifer

39 senescence, stress and interferon responses, epithelial-mesenchymal transition and protein metabolism

40 Resistin was found to promote epithelial-mesenchymal transition and stemness in breast

41 ssociated with stemness, chemoresistance and epithelial-mesenchymal transition and suppressed the pro

42 ticity (EMP), which collectively encompasses epithelial-mesenchymal transition and the reverse proces

43 ce of cell-cell junctions, regulation of the epithelial-mesenchymal transition and transcriptional si

44 infection in CCA development by induction of epithelial-mesenchymal transition and up-regulation of h

45 blocked tumor cell growth in vitro, reversed epithelial-mesenchymal transition, and abated tumor meta

46 ethionine restriction or sorafenib, promotes epithelial-mesenchymal transition, and induces cell migr

47 leads to activation of NF-kappaB signaling, epithelial-mesenchymal transition, and lung tumor metast

48 ial for primitive cardiac valve formation by epithelial-mesenchymal transition, and NOTCH1 mutations

49 ntified a potential role for circRNAs in the epithelial-mesenchymal transition, and provided new info

50 ed that PRSS8 targeted the Wnt/beta-catenin, epithelial-mesenchymal transition, and stem cell signali

51 Extracellular matrix genes involved in epithelial-mesenchymal transition are expressed most dif

52 The previous emphasis on the epithelial-mesenchymal transition as a driver of the mig

53 We report the epithelial to hybrid epithelial/mesenchymal transition as a result of decreas

54 dial cells were highly adipogenic through an epithelial-mesenchymal transition both in vitro and in v

55 ransforming growth factor beta signaling and epithelial-mesenchymal transition by cholesterol-lowerin

56 cells in vitro and in vivo and regulated the epithelial-mesenchymal transition by repressing AKT/glyc

57 atter stages of cancer, are known to promote epithelial-mesenchymal transition, cancer cell growth, s

58 17, and NANOG), and increased markers of the epithelial-mesenchymal transition (CDH1, SNAI2, TWIST1,

59 rucial events of cancer progression, such as epithelial-mesenchymal transition, cell migration, and c

60 ession of TAZ/YAP target genes implicated in epithelial-mesenchymal transition, cell migration, and i

61 silencing in lung epithelial cells promoted epithelial-mesenchymal transition, cell migration, tumor

62 te cancer progression phenomena, such as the epithelial-mesenchymal transition, cell motility, invasi

63 sis via altered gene expression, stimulating epithelial-mesenchymal transition, cellular migration/in

64 enhanced expression of epithelial genes and epithelial-mesenchymal transition downregulation signatu

65 regions, identifying E-box motifs common to epithelial-mesenchymal transition driver transcription f

66 In addition, Panx3 depletion reduced epithelial-mesenchymal transition during skin wound heal

67 (e.g., PUMA, BIK) and metastasis-associated epithelial-mesenchymal transition (e.g., CDH1), and by a

68 ss of its expression in association with the epithelial mesenchymal transition (EMT) occurs frequentl

69 pathy (PVR) is mediated by proliferation and epithelial mesenchymal transition (EMT) of retinal pigme

70 The epithelial mesenchymal transition (EMT) promotes tumor m

71 tissue types, specifically in regard to the epithelial mesenchymal transition (EMT) structural compo

72 /Twist has been implicated in the process of epithelial mesenchymal transition (EMT), an intermediate

73 of extracellular matrix (ECM) remodeling and epithelial mesenchymal transition (EMT), provide mechani

74 through lineage plasticity, characterized by epithelial-mesenchymal transition (EMT) and a basal-like

75 related gene networks of immune-suppression, epithelial-mesenchymal transition (EMT) and angiogenesis

76 survival, but also induced an AKT-dependent epithelial-mesenchymal transition (EMT) and beta-catenin

77 ne family and serves essential roles in both epithelial-mesenchymal transition (EMT) and breast cance

78 The epithelial-mesenchymal transition (EMT) and cancer stem

79 Twist-mediated oncogenic functions including epithelial-mesenchymal transition (EMT) and cancer stem

80 Hedgehog (Hh) pathway is involved in epithelial-mesenchymal transition (EMT) and cancer stem

81 les the phenotypic plasticity enabled by the epithelial-mesenchymal transition (EMT) and cell migrati

82 Furthermore, depletion of DLG5 induced epithelial-mesenchymal transition (EMT) and disrupted ep

83 mounts of IL-1beta and TNF which facilitated epithelial-mesenchymal transition (EMT) and epithelial c

84 ts extend the role of ZEB2 beyond regulating epithelial-mesenchymal transition (EMT) and establish ZE

85 induced expression of genes involved in the epithelial-mesenchymal transition (EMT) and increased tu

86 Elucidating the spectrum of epithelial-mesenchymal transition (EMT) and mesenchymal-

87 y are required in transition states, such as epithelial-mesenchymal transition (EMT) and mesenchymal-

88 y studied types of phenotypic plasticity are epithelial-mesenchymal transition (EMT) and mesenchymal-

89 hway plays critical roles during cancer cell epithelial-mesenchymal transition (EMT) and metastasis.

90 ion in the prostate epithelium promotes both epithelial-mesenchymal transition (EMT) and metastatic c

91 Lymphocyte conditioned media (CM) induced epithelial-mesenchymal transition (EMT) and migration in

92 e (RTK) AXL has been intrinsically linked to epithelial-mesenchymal transition (EMT) and promoting ce

93 eq analysis reveals that REST is involved in epithelial-mesenchymal transition (EMT) and stemness acq

94 wnstream target gene expression, inducing an epithelial-mesenchymal transition (EMT) and stemness fea

95 Our study showed that EGFL6 promoted epithelial-mesenchymal transition (EMT) and stemness of

96 erences between two histologic subtypes, the epithelial-mesenchymal transition (EMT) and the papillar

97 , PIPKIgamma, expression is upregulated upon epithelial-mesenchymal transition (EMT) and together wit

98 has been shown to causally contribute to the epithelial-mesenchymal transition (EMT) and tumor metast

99 e, we demonstrate that Notch1 activation and epithelial-mesenchymal transition (EMT) are coupled to p

100 Glioma stem cells (GSC) and epithelial-mesenchymal transition (EMT) are strongly ass

101 Albumin also stimulated an epithelial-mesenchymal transition (EMT) as indicated by

102 h in vitro and in vivo also induced HCC cell epithelial-mesenchymal transition (EMT) as well.

103 s of apical-basal polarity and activation of epithelial-mesenchymal transition (EMT) both contribute

104 hat metastases were characterized by reduced epithelial-mesenchymal transition (EMT) but increased MY

105 then found to result from TGFbeta1-triggered epithelial-mesenchymal transition (EMT) by beta-cells, r

106 Moreover, miR-644a modulates epithelial-mesenchymal transition (EMT) by directly targ

107 colleagues determined that IFNgamma induces epithelial-mesenchymal transition (EMT) by regulating th

108 ates is subject to intense discussion in the epithelial-mesenchymal transition (EMT) community.

109 esistance was associated with development of epithelial-mesenchymal transition (EMT) coordinated by i

110 Finally, mir-200 was able to antagonize the epithelial-mesenchymal transition (EMT) driven by mutant

111 Another application of CBNA is to discover epithelial-mesenchymal transition (EMT) drivers.

112 Epithelial-mesenchymal transition (EMT) encompasses dyna

113 During epithelial-mesenchymal transition (EMT) epithelial cells

114 the triggering mechanisms and regulation of epithelial-mesenchymal transition (EMT) factors in the c

115 nce of the phenotypic plasticity afforded by epithelial-mesenchymal transition (EMT) for cancer progr

116 Tumor cell plasticity exhibited as an epithelial-mesenchymal transition (EMT) has been identif

117 Epithelial-mesenchymal transition (EMT) has been linked

118 Epithelial-mesenchymal transition (EMT) has been recogni

119 and TGFbeta signaling pathways and a loss of epithelial-mesenchymal transition (EMT) in BVE(Cyp24a1-n

120 Functionally, RGC32 facilitated epithelial-mesenchymal transition (EMT) in CRC via the S

121 gated the mechanisms by which SPRY regulates epithelial-mesenchymal transition (EMT) in CRC.

122 Conversely, the introduction of inducers of epithelial-mesenchymal transition (EMT) in cystine-indep

123 ecular pathways, both cytokines also induced epithelial-mesenchymal transition (EMT) in lung cancer c

124 tamine deprivation leads to the induction of epithelial-mesenchymal transition (EMT) in pancreatic du

125 lture models, we demonstrate that ASC induce epithelial-mesenchymal transition (EMT) in prostate canc

126 to CD44(High)-CD24(-/Low) (CD44H) cells via epithelial-mesenchymal transition (EMT) in response to t

127 moresistance, and expressed genes related to epithelial-mesenchymal transition (EMT) in solid cancers

128 vation of renal interstitial fibroblasts and epithelial-mesenchymal transition (EMT) in vitro.

129 on the expression of the major regulators of epithelial-mesenchymal transition (EMT) including E-cadh

130 Here, we compare a reversible model of epithelial-mesenchymal transition (EMT) induced by TGFbe

131 related to cancer stem cells (CSCs) markers, epithelial-mesenchymal transition (EMT) inducers and bas

132 ion after PRP treatment and resulted in, (i) epithelial-mesenchymal transition (EMT) inhibition; (ii)

133 Epithelial-mesenchymal transition (EMT) is a critical pr

134 Epithelial-mesenchymal transition (EMT) is a development

135 Epithelial-mesenchymal transition (EMT) is a fundamental

136 Epithelial-mesenchymal transition (EMT) is a highly cons

137 Epithelial-mesenchymal transition (EMT) is a key event t

138 The epithelial-mesenchymal transition (EMT) is a process by

139 The epithelial-mesenchymal transition (EMT) is an embryonic

140 Epithelial-mesenchymal transition (EMT) is an important

141 The epithelial-mesenchymal transition (EMT) is an important

142 An epithelial-mesenchymal transition (EMT) is believed to b

143 The epithelial-mesenchymal transition (EMT) is considered es

144 Epithelial-mesenchymal transition (EMT) is induced by tr

145 Epithelial-mesenchymal transition (EMT) is prominent in

146 Epithelial-mesenchymal transition (EMT) is regarded as a

147 regulators (OCT4, SOX2 and NANOG) along with epithelial-mesenchymal transition (EMT) markers (Snail,

148 ealing correlated with altered expression of epithelial-mesenchymal transition (EMT) markers and redu

149 sed tumor sphere formation and expression of epithelial-mesenchymal transition (EMT) markers.

150 orming growth factor-beta (TGF-beta)-induced epithelial-mesenchymal transition (EMT) may contribute t

151 -cadherin expression in association with the epithelial-mesenchymal transition (EMT) occurs frequentl

152 demonstrate that loss of ARID1A promotes an epithelial-mesenchymal transition (EMT) phenotype and se

153 omic analyses revealed that Sfrp1 induced an epithelial-mesenchymal transition (EMT) phenotype in tum

154 cancer cells, implicating acquisition of an epithelial-mesenchymal transition (EMT) phenotype.

155 Epithelial-mesenchymal transition (EMT) plays a major ro

156 s, has been linked to the acquisition of the epithelial-mesenchymal transition (EMT) program and self

157 al and mesenchymal phenotypic states via the epithelial-mesenchymal transition (EMT) program can regu

158 al subtype, including core genes involved in epithelial-mesenchymal transition (EMT) program.

159 osed as critical regulatory molecules in the epithelial-mesenchymal transition (EMT) program.

160 ound that GPX8 expression was induced by the epithelial-mesenchymal transition (EMT) program.

161 The epithelial-mesenchymal transition (EMT) programs promote

162 MaSCs and MaTICs both use epithelial-mesenchymal transition (EMT) programs to acqu

163 We found that epithelial-mesenchymal transition (EMT) ranked first in

164 ific mechanism for HNSCC local invasion: the epithelial-mesenchymal transition (EMT) regulator Twist1

165 An epithelial-mesenchymal transition (EMT) represents a bas

166 sent our approach using published pan-cancer epithelial-mesenchymal transition (EMT) signatures as a

167 at hepatitis C virus (HCV) infection induces epithelial-mesenchymal transition (EMT) state and cancer

168 The induction of pathogenic epithelial-mesenchymal transition (EMT) through microRNA

169 Collagen was increased, and cancer cell epithelial-mesenchymal transition (EMT) was inhibited, a

170 tern blot results revealed that hallmarks of epithelial-mesenchymal transition (EMT) were altered as

171 The proteolytic profile and epithelial-mesenchymal transition (EMT) were investigate

172 Metastatic cancer cells undergo an epithelial-mesenchymal transition (EMT) where they lose

173 ession of the ESR1 Y537S mutation induced an epithelial-mesenchymal transition (EMT) with cells exhib

174 in gliomas, are associated with a process of epithelial-mesenchymal transition (EMT), a cellular alte

175 transcription factor known for participating epithelial-mesenchymal transition (EMT), a critical cell

176 c reprogramming is known to occur during the epithelial-mesenchymal transition (EMT), a process that

177 r suppressors to promote cell proliferation, epithelial-mesenchymal transition (EMT), and cancer prog

178 tumor edge, ceased to proliferate, underwent epithelial-mesenchymal transition (EMT), and expressed m

179 ssays showed that TBL1XR1 promoted stemness, epithelial-mesenchymal transition (EMT), and lung and ly

180 r localization of Twist and Slug, markers of epithelial-mesenchymal transition (EMT), but also consti

181 Upon the induction of epithelial-mesenchymal transition (EMT), EGFR diffusivit

182 We show that TGF-beta induces an epithelial-mesenchymal transition (EMT), generally consi

183 The process of epithelial-mesenchymal transition (EMT), in addition to

184 t example in human cancers of the concept of epithelial-mesenchymal transition (EMT), in which the tw

185 on signature similar to that observed in the epithelial-mesenchymal transition (EMT), including E-cad

186 Snail1, a key transcription factor of epithelial-mesenchymal transition (EMT), is subjected to

187 bilize Snail1 protein, a master regulator of epithelial-mesenchymal transition (EMT), leading to EMT

188 engulfment, Schwann cell (SC) demyelination, epithelial-mesenchymal transition (EMT), loss of sensati

189 We reveal that during epithelial-mesenchymal transition (EMT), one important s

190 TCF transcription induces epithelial-mesenchymal transition (EMT), promoting a tum

191 luding the inhibition of cell proliferation, epithelial-mesenchymal transition (EMT), the expression

192 diminished SAR1B expression could stimulate epithelial-mesenchymal transition (EMT), thereby promoti

193 AKT in LAD cells, although unable to induce epithelial-mesenchymal transition (EMT), VAL exerts pote

194 ncreasing appreciation of different roles of epithelial-mesenchymal transition (EMT), we address the

195 Because TWIST1 mediates epithelial-mesenchymal transition (EMT), we studied the

196 of a latent embryonic program, known as the epithelial-mesenchymal transition (EMT), which bestows s

197 Deregulation of TJ is a hallmark of epithelial-mesenchymal transition (EMT), which contribut

198 arcinoma cells undergo a partial or complete epithelial-mesenchymal transition (EMT), which contribut

199 These data demonstrate the induction of epithelial-mesenchymal transition (EMT), which included

200 Twist is a key transcription factor for Epithelial-mesenchymal transition (EMT), which is a cell

201 aB activation and (2) aberrant initiation of epithelial-mesenchymal transition (EMT), which together

202 Moreover, hnRNPF regulates an epithelial-mesenchymal transition (EMT)-associated CD44

203 rotein stimulates differential expression of epithelial-mesenchymal transition (EMT)-associated genes

204 Her2(+) eDCCs activated a Wnt-dependent epithelial-mesenchymal transition (EMT)-like disseminati

205 OE CMs present an epithelial-mesenchymal transition (EMT)-like regenerativ

206 Additionally, epithelial-mesenchymal transition (EMT)-related genes we

207 n, migration, invasion, drug resistance, and epithelial-mesenchymal transition (EMT).

208 proliferation (Ki67 expression), and induces epithelial-mesenchymal transition (EMT).

209 gration and molecular changes reminiscent of epithelial-mesenchymal transition (EMT).

210 FPR2 knock-down, implying the involvement of epithelial-mesenchymal transition (EMT).

211 g the process of cell invasion, adhesion and epithelial-mesenchymal transition (EMT).

212 es tumor invasion and metastasis by inducing epithelial-mesenchymal transition (EMT).

213 the endometrial adenocarcinoma (EAC) through epithelial-mesenchymal transition (EMT).

214 diated epigenetic reprogramming known as the epithelial-mesenchymal transition (EMT).

215 iferation, anchor-free colony formation, and epithelial-mesenchymal transition (EMT).

216 ells, consistent with a role in promoting an epithelial-mesenchymal transition (EMT).

217 a gene expression signature associated with epithelial-mesenchymal transition (EMT).

218 es mesoderm and endoderm germ layers through epithelial-mesenchymal transition (EMT).

219 GFbeta signaling and subsequent induction of epithelial-mesenchymal transition (EMT).

220 e cancer cells, as a model for physiological epithelial-mesenchymal transition (EMT).

221 consequently potentiates Pi triggered lethal epithelial-mesenchymal transition (EMT).

222 ophagitis (EoE) complication, is mediated by epithelial-mesenchymal transition (EMT).

223 characterized it to be a master regulator of epithelial-mesenchymal transition (EMT).

224 ecific 84-1 monoclonal antibody that detects epithelial-mesenchymal transitioned (EMT) CTCs.

225 homologous recombination (HR), and prevents epithelial-mesenchymal transitions (EMT) in an Ataxia-te

226 he development of renal fibrosis and partial epithelial-mesenchymal transitions (EMT).

227 ivation of peritoneal fibroblasts, mitigated epithelial-mesenchymal-transition (EMT), as well as enha

228 Metastasis is initiated after epithelial-mesenchymal-transition (EMT).

229 for disassembly of adherens junctions during epithelial-mesenchymal transitions (EMTs), loss of adher

230 nding sheet, triggering partial and complete epithelial-mesenchymal transitions (EMTs).

231 Notably, Jag1-ICD promoted tumor growth and epithelial-mesenchymal transition, enhancing colorectal

232 miR-299-3p overexpression also inhibited epithelial mesenchymal transition, expression of Slug, T

233 ear beta-catenin accumulation, and increased epithelial-mesenchymal transition factors and colon cell

234 ment architectures-such as those mandated by epithelial-mesenchymal transition, for example-or resett

235 vitro invasiveness and the expression of key epithelial-mesenchymal transition genes.

236 ams, and indirectly suppresses oncogenic and epithelial-mesenchymal transition genes.

237 ated transforming growth factor-beta-induced epithelial-mesenchymal transition in A549 cells and HCC8

238 f HNF4alpha combined with HF feeding induced epithelial-mesenchymal transition in an IL6-dependent ma

239 ignatures of receptor proteins associated to epithelial-mesenchymal transition in bladder and breast

240 ceptor expression signatures associated with epithelial-mesenchymal transition in bladder and breast

241 erized by high expression of ZEB1, including epithelial-mesenchymal transition in epithelial-derived

242 miR-194 stimulated migration, invasion, and epithelial-mesenchymal transition in human prostate canc

243 1/2 pathway to potentiate TGFbeta1-mediated epithelial-mesenchymal transition in normoglycemic condi

244 dies have demonstrated the importance of the epithelial-mesenchymal transition in promoting the cance

245 ; and, ultimately, (4) the activation of the epithelial-mesenchymal transition in the invasive stage

246 , and increased expression of markers of the epithelial-mesenchymal transition; inhibitors of STAT3 b

247 induced mammary tumorigenesis and suppresses epithelial-mesenchymal transition, intravasation, and me

248 of proliferation, cell cycle, self-renewal, epithelial-mesenchymal transition, invasion, and resista

249 een epithelial and mesenchymal tumors, since epithelial-mesenchymal transition is a key paradigm for

250 Whereas epithelial-mesenchymal transition is required for epithe

251 nds on whether tumor cells have gone through epithelial-mesenchymal transition, is determinant for th

252 nd reduced E-cadherin expression, indicating epithelial-mesenchymal transition-like abnormalities.

253 esponse results in a marked induction of the epithelial-mesenchymal transition marker MMP-1.

254 ferences were seen in melanoma stem cell and Epithelial-Mesenchymal Transition markers between GM2/GD

255 Analysis of epithelial-mesenchymal transition markers in U138 cells

256 However, no effects on epithelial-mesenchymal transition markers, proliferation

257 feration, survival, apoptosis, angiogenesis, epithelial-mesenchymal transition, metastasis, stemness,

258 egulated kinase (Erk) signaling and promotes epithelial-mesenchymal transition, migration/invasion in

259 RHAMM by the RB/E2F pathway was critical for epithelial-mesenchymal transition, motility, and invasio

260 ZL0454 prevents epithelial mesenchymal transition, myofibroblast expansi

261 ediated endocytosis, suppressed invasion and epithelial-mesenchymal transition of ovarian cancer cell

262 promoting invasion, Erk phosphorylation, and epithelial-mesenchymal transition of PNETs.

263 andscapes of target organs, and variation in epithelial-mesenchymal transitions open avenues for futu

264 heir reductions were insufficient to reverse epithelial-mesenchymal transition or TGFbeta-mediated an

265 Drastic changes of organization, such as epithelial-mesenchymal transitions or the formation of s

266 n translation impact on the configuration of epithelial mesenchymal transition pathways in tumor cell

267 e HDAC6i over the expression and function of epithelial-mesenchymal transition pathways and the invas

268 r epitranscriptome, regulating oncogenic and epithelial-mesenchymal transition pathways.

269 Epithelial-mesenchymal transitions play key roles in dev

270 ons; (ii) CRIS-B: TGF-beta pathway activity, epithelial-mesenchymal transition, poor prognosis; (iii)

271 epithelium, consistent with the onset of an epithelial-mesenchymal transition program.

272 tenin signaling in a manner dependent on the epithelial-mesenchymal transition program.

273 so induced the migration, invasion, and EMT (epithelial-mesenchymal transition) program of GBM cells

274 Although the epithelial-mesenchymal transition regulator Snail (Snai1

275 -beta transcriptional, tumor-suppressing and epithelial-mesenchymal transition responses.

276 llular clusters across varying states of the epithelial-mesenchymal transition, revealing a successiv

277 EMT (epithelial-mesenchymal-transition)-signaling regulates T

278 vasion, and promotes mesenchymal subtype and epithelial mesenchymal transition-specific gene expressi

279 n previously that HCV infection generates an epithelial-mesenchymal transition state and tumor-initia

280 verged to identify subsets with differential epithelial-mesenchymal transition status, carcinoma in s

281 y modulating cancer cell differentiation and epithelial-mesenchymal transition status.

282 s and for mRNAs encoding proteins related to epithelial-mesenchymal transition, stemness, and Wnt sig

283 SPINK1 elicits epithelial-mesenchymal-transition, stemness and cellular

284 )-DD, which promotes cellular proliferation, epithelial-mesenchymal transition, stromal reaction, and

285 22, phosphorylated STAT3, and markers of the epithelial-mesenchymal transition than nonneoplastic tis

286 omain they generate migratory progenitors by epithelial-mesenchymal transition that mature into neuro

287 d differentiation as well, but also promotes epithelial-mesenchymal transitions that lead to cataract

288 ity in melanoma bears a great resemblance to epithelial-mesenchymal transition, the lessons learned f

289 CRS pathophysiology: from the epithelium and epithelial-mesenchymal transition to innate and adaptive

290 ut of the periventricular zones, possibly by epithelial-mesenchymal transition, to colonize and maint

291 studies have suggested that the zinc-finger epithelial-mesenchymal transition transcription factor S

292 Epithelial-mesenchymal transition transcription factors

293 roliferation, invasion, as well as switch in epithelial-mesenchymal-transition transcription factor e

294 A protein essential for signaling in epithelial-mesenchymal transition, transforming growth f

295 h, clonal formation, migration and invasion, epithelial-mesenchymal transition, tumor sphere and tumo

296 cell that subsequently undergoes a prominent epithelial-mesenchymal transition underlying the attainm

297 Epithelial-mesenchymal transition was lower in BEV + RES

298 Morphologic correlates of epithelial-mesenchymal transition were associated with d

299 udding activity [BA] [potentially indicating epithelial-mesenchymal transition]) were evaluated in 66

300 all molecules that inhibit TGF-beta1-induced epithelial-mesenchymal transition without immediate TGF-