ライフサイエンスコーパス: epithelial-to-mesenchymal transition

1 We found that it induces reversion of epithelial to mesenchymal transition.

2 gulation of AKT/mTOR or in those involved in epithelial to mesenchymal transition.

3 ponents and their regulators associated with epithelial to mesenchymal transition.

4 a result SNAI1 is stabilized, triggering an epithelial to mesenchymal transition.

5 e and altered the expressions of markers for epithelial to mesenchymal transition.

6 factors best known for their role in driving epithelial to mesenchymal transition.

7 eat shock protein 27 (HSP27); and markers of epithelial-to-mesenchymal transition.

8 induces markers of the primitive streak and epithelial-to-mesenchymal transition.

9 enhancing expression of the genes related to epithelial-to-mesenchymal transition.

10 sed extracellular matrix, cell adhesion, and epithelial-to-mesenchymal transition.

11 ression of twist1b-a well-known regulator of epithelial-to-mesenchymal transition.

12 onize distant sites, including the so-called epithelial-to-mesenchymal transition.

13 , including cell cycle arrest, apoptosis and epithelial-to-mesenchymal transition.

14 poorly differentiated state with features of epithelial-to-mesenchymal transition.

15 (MUC1-CT) represent initiating steps in the epithelial-to-mesenchymal transition.

16 static potential through the upregulation of epithelial-to-mesenchymal transition.

17 arcinomas marked by poor differentiation and epithelial-to-mesenchymal transition.

18 as computationally designed and that impairs epithelial-to-mesenchymal transition.

19 n of a set of transcription factors to drive epithelial-to-mesenchymal transition.

20 proliferation, cancer stem cell biology, and epithelial-to-mesenchymal transition.

21 experimental metastases in fact undergo the epithelial-to-mesenchymal transition.

22 rd chemokine loop that further drives an IBC epithelial-to-mesenchymal transition.

23 re correct epithelial cell shape and prevent epithelial-to-mesenchymal transitions.

24 These tumors display features of epithelial-to-mesenchymal transition, a cellular state a

25 and gene expression analysis of oncogenesis, epithelial to mesenchymal transition and apoptosis pathw

26 ysis, angiogenesis, cell-matrix interaction, epithelial to mesenchymal transition and metastatic abil

27 catenin signaling acts as a switch to induce epithelial to mesenchymal transition and promote colorec

28 Given that DDR2 has a crucial role in the epithelial-to-mesenchymal transition and cancer progress

29 in tumor progression and resistance, such as epithelial-to-mesenchymal transition and cell adhesion p

30 itabine recovers FBXL7 expression and limits epithelial-to-mesenchymal transition and cell invasion i

31 ERK1/2 hyperactivation drives ZEB1-dependent epithelial-to-mesenchymal transition and chemoresistance

32 The C4S- and C6S-enhanced epithelial-to-mesenchymal transition and expression of M

33 es we show MBC-specific increases related to epithelial-to-mesenchymal transition and extracellular m

34 PEB2B isoform inhibited pathways driving the epithelial-to-mesenchymal transition and hypoxic respons

35 aling cascade culminating in Slug induction, epithelial-to-mesenchymal transition and increased invas

36 Mechanistically, STAT3 promotes epithelial-to-mesenchymal transition and increases mucin

37 the posterior side of the embryo undergo an epithelial-to-mesenchymal transition and ingress through

38 Vimentin is a classic marker of epithelial-to-mesenchymal transition and is therefore an

39 invasive abilities and higher expression of epithelial-to-mesenchymal transition and mammary stem ce

40 mals, various in vitro and in vivo models of epithelial-to-mesenchymal transition and metastasis, an

41 on protein products of genes instrumental to epithelial-to-mesenchymal transition and metastasis.

42 They undergo epithelial-to-mesenchymal transition and migrate through

43 knockdown enhanced the invasiveness, induced epithelial-to-mesenchymal transition and promoted metast

44 dergo an order-to-disorder transition via an epithelial-to-mesenchymal transition and sort symmetrica

45 te-induced airway remodeling by facilitating epithelial-to-mesenchymal transition and STAT3/NF-kappaB

46 henotypes were also affected at the level of epithelial-to-mesenchymal transition and the ERK1/2 sign

47 -8), real-time polymerase chain reaction for epithelial-to-mesenchymal transition and tight junction

48 ve enhanced glycocalyx height in response to epithelial-to-mesenchymal transition and to oncogenic KR

49 structure and gene expression suggestive of epithelial-to-mesenchymal transition and transdifferenti

50 ures and exhibited features of self-renewal, epithelial-to-mesenchymal transition and tumor initiatio

51 e report the novel functions of PHF8 in EMT (epithelial to mesenchymal transition) and breast cancer

52 ic cells were used to evaluate the CSC, EMT (epithelial-to-mesenchymal transition), and metabolic pro

53 issue remodeling, cytoskeletal organization, epithelial-to-mesenchymal transition, and cellular migra

54 including profibrotic genes, genes promoting epithelial-to-mesenchymal transition, and genes associat

55 point were associated with moderate hypoxia, epithelial-to-mesenchymal transition, and inflammatory r

56 XCL11 significantly induced tumor migration, epithelial-to-mesenchymal transition, and matrix remodel

57 minal layer, led epithelial cells to undergo epithelial-to-mesenchymal transition, and resulted in la

58 al and resistance to environmental stresses, epithelial-to-mesenchymal transition, and the emergence

59 regulation, extracellular matrix remodeling, epithelial-to-mesenchymal transition, and the enrichment

60 umor angiogenesis, cell adhesion, migration, epithelial-to-mesenchymal transition, and transendotheli

61 cal processes, including tissue homeostasis, epithelial-to-mesenchymal transition, and wound repair.

62 uire mobility and invasiveness by undergoing epithelial-to-mesenchymal transition, and yet most metas

63 s in altered expression of genes involved in epithelial-to-mesenchymal transition, angiogenesis, and

64 There was also an increase in epithelial-to-mesenchymal transition at acquired resista

65 t and -independent AR signaling mediated HCC epithelial-to-mesenchymal transition by regulating the t

66 nd metastasis, including analysis of partial epithelial-to-mesenchymal transition, cell isolation and

67 tes several key oncogenic processes, such as epithelial-to-mesenchymal transition, cellular migration

68 population of progenitor cells that undergo epithelial-to-mesenchymal transition displaying characte

69 6-E7 proteins augmented invasion and induced epithelial to mesenchymal transition (EMT) accompanied b

70 ODH promotes NSCLC tumorigenesis by inducing epithelial to mesenchymal transition (EMT) and IKKalpha-

71 l glycine t-butylester) abrogates GH-induced epithelial to mesenchymal transition (EMT) and is associ

72 (UTR) nucleic acid regulatory motif, driving epithelial to mesenchymal transition (EMT) and metastasi

73 Epithelial to mesenchymal transition (EMT) and wound vas

74 gests that cell-fate transitions such as the epithelial to mesenchymal transition (EMT) are associate

75 (miRNA-200) family have a regulatory role in epithelial to mesenchymal transition (EMT) by suppressin

76 tion with canonical Wnt signaling during the epithelial to mesenchymal transition (EMT) from NMP to m

77 Activation of the epithelial to mesenchymal transition (EMT) genetic progr

78 Previous studies have proposed that epithelial to mesenchymal transition (EMT) in breast can

79 Epithelial to mesenchymal transition (EMT) in cancer cel

80 icroenvironment and acts as a key inducer of epithelial to mesenchymal transition (EMT) in lung cance

81 ow that the initiation of limb formation, an epithelial to mesenchymal transition (EMT) in the latera

82 Epithelial to mesenchymal transition (EMT) is a dynamic

83 Epithelial to mesenchymal transition (EMT) is an essenti

84 detachment, misplaced retinal cells undergo epithelial to mesenchymal transition (EMT) to form contr

85 dherens junctions, dedifferentiation, and an epithelial to mesenchymal transition (EMT) transcription

86 Cells were analyzed for markers of the epithelial to mesenchymal transition (EMT) using flow cy

87 is study, we investigated ST6Gal-I's role in epithelial to mesenchymal transition (EMT) using the Sui

88 the chorioamniotic membranes consistent with epithelial to mesenchymal transition (EMT) with loss of

89 ) a hormone related subtype, associated with Epithelial to Mesenchymal Transition (EMT), and gain of

90 harbor its receptor, leads to occurrence of epithelial to mesenchymal transition (EMT), in time and

91 After epithelial to mesenchymal transition (EMT), mesenchymal

92 HCC pathology involves epithelial to mesenchymal transition (EMT), the basis of

93 ar event during embryonic development called epithelial to mesenchymal transition (EMT), we hypothesi

94 breast cancer with histological evidence of epithelial to mesenchymal transition (EMT).

95 Esrp1 also regulates splicing during the epithelial to mesenchymal transition (EMT).

96 , which is indicative of an induction of the epithelial to mesenchymal transition (EMT).

97 -dependent CRC epithelial cell migration and epithelial to mesenchymal transition (EMT).

98 activation of several genes involved in the epithelial to mesenchymal transition (EMT).

99 beta-catenin activates epithelial to mesenchymal transition (EMT).

100 trol of transcriptional programs such as the epithelial-to-mesenchymal transition (EMT) [7, 8].

101 nit, caused HME cells to undergo spontaneous epithelial-to-mesenchymal transition (EMT) and aberrant

102 a (GBM) is, in part, attributed to increased epithelial-to-mesenchymal transition (EMT) and enhanced

103 riptional factor is essential for triggering epithelial-to-mesenchymal transition (EMT) and inducing

104 lso play essential roles in the induction of epithelial-to-mesenchymal transition (EMT) and induction

105 Epithelial-to-mesenchymal transition (EMT) and its rever

106 We also highlight the epithelial-to-mesenchymal transition (EMT) and known mol

107 ognosis is potentially due to cancer-related epithelial-to-mesenchymal transition (EMT) and poor chem

108 ingle-cell population shifts toward invasive epithelial-to-mesenchymal transition (EMT) and prolifera

109 ant induction of apoptosis and inhibition of epithelial-to-mesenchymal transition (EMT) and stemness

110 ional reprogramming of the cells, leading to epithelial-to-mesenchymal transition (EMT) and stimulati

111 taRIII-SS in lung cancer cell models induces epithelial-to-mesenchymal transition (EMT) and that thes

112 The epithelial-to-mesenchymal transition (EMT) and the unjam

113 senchymal-to-epithelial transition (MET) and epithelial-to-mesenchymal transition (EMT) are important

114 uring implantation, cytotrophoblasts undergo epithelial-to-mesenchymal transition (EMT) as they diffe

115 Alu RNA induces epithelial-to-mesenchymal transition (EMT) by acting as

116 n-regulation of TRPS1 in TNBC cells promoted epithelial-to-mesenchymal transition (EMT) by deregulati

117 -3 supports the TIC/CSC state and induces an epithelial-to-mesenchymal transition (EMT) by driving ex

118 a master regulator of cellular identity and epithelial-to-mesenchymal transition (EMT) directly repr

119 can transcriptionally regulate SNAI1, a key epithelial-to-mesenchymal transition (EMT) driver, which

120 Epithelial-to-mesenchymal transition (EMT) enables scatt

121 man mammary epithelial cells that undergo an epithelial-to-mesenchymal transition (EMT) following tra

122 Transcriptome sequencing identified a strong epithelial-to-mesenchymal transition (EMT) gene signatur

123 Epithelial-to-mesenchymal transition (EMT) has been asso

124 Phenotypic alteration in epithelial-to-mesenchymal transition (EMT) has been link

125 Epithelial-to-mesenchymal transition (EMT) has been prop

126 urvival, and migration; however, its role in epithelial-to-mesenchymal transition (EMT) has been scar

127 d gene expression changes consistent with an epithelial-to-mesenchymal transition (EMT) have been ass

128 Modulators of epithelial-to-mesenchymal transition (EMT) have recently

129 translational regulatory programs underlying epithelial-to-mesenchymal transition (EMT) in breast epi

130 Reversing epithelial-to-mesenchymal transition (EMT) in cancer cel

131 ZEB1 is best known for driving an epithelial-to-mesenchymal transition (EMT) in cancer cel

132 Snail and Zeb transcription factors induce epithelial-to-mesenchymal transition (EMT) in embryonic

133 ession on the Wnt/beta-catenin signaling and epithelial-to-mesenchymal transition (EMT) in human CRC

134 Inducing epithelial-to-mesenchymal transition (EMT) in human ovar

135 The involvement of epithelial-to-mesenchymal transition (EMT) in metastasis

136 atic cancer, especially in the regulation of epithelial-to-mesenchymal transition (EMT) in Pancreatic

137 cent study has revealed that MT1-MMP induces epithelial-to-mesenchymal transition (EMT) in prostate a

138 prostate adenocarcinoma through induction of epithelial-to-mesenchymal transition (EMT) in Pten-null

139 ERK is a key coordinator of the epithelial-to-mesenchymal transition (EMT) in that a var

140 The contribution of epithelial-to-mesenchymal transition (EMT) in the establ

141 ce temporary or transient cell fate, such as epithelial-to-mesenchymal transition (EMT) in tumor meta

142 olarized epithelial phenotype, and ESRP1, an epithelial-to-mesenchymal transition (EMT) inhibitor.

143 The epithelial-to-mesenchymal transition (EMT) is a cell bio

144 The epithelial-to-mesenchymal transition (EMT) is a cellular

145 Epithelial-to-mesenchymal transition (EMT) is a core pro

146 Epithelial-to-mesenchymal transition (EMT) is a fundamen

147 Aberrant epithelial-to-mesenchymal transition (EMT) is a proposed

148 The epithelial-to-mesenchymal transition (EMT) is a reversib

149 The epithelial-to-mesenchymal transition (EMT) is an essenti

150 The epithelial-to-mesenchymal transition (EMT) is an essenti

151 In cancer cells, epithelial-to-mesenchymal transition (EMT) is controlled

152 Epithelial-to-mesenchymal transition (EMT) is critical f

153 Epithelial-to-mesenchymal transition (EMT) is implicated

154 The epithelial-to-mesenchymal transition (EMT) is involved i

155 Epithelial-to-mesenchymal transition (EMT) is orchestrat

156 Alterations of epithelial-to-mesenchymal transition (EMT) marker expres

157 red to maintain expression of metastasis and Epithelial-to-mesenchymal transition (EMT) markers and t

158 activities of lung cancer cells and reduced epithelial-to-mesenchymal transition (EMT) markers as N-

159 ssays together with qRT-PCR determination of epithelial-to-mesenchymal transition (EMT) markers were

160 n of the EOC cells and reduced expression of epithelial-to-mesenchymal transition (EMT) markers ZEB1,

161 of apical-basal polarity, and appearance of epithelial-to-mesenchymal transition (EMT) markers.

162 monstrate that LRIG1 is downregulated during epithelial-to-mesenchymal transition (EMT) of human mamm

163 Depletion of NatD suppresses the epithelial-to-mesenchymal transition (EMT) of lung cance

164 h STAT4 mediated EOC metastasis via inducing epithelial-to-mesenchymal transition (EMT) of ovarian ca

165 itary tumor transcriptome data revealed the "epithelial-to-mesenchymal transition (EMT) pathway" as o

166 stases requires cell dissemination utilizing epithelial-to-mesenchymal transition (EMT) program.

167 Metastatic lung cancer cells can undergo an epithelial-to-mesenchymal transition (EMT) regulated by

168 ells to undergo VM and that knockdown of the epithelial-to-mesenchymal transition (EMT) regulator, Zi

169 esicular trafficking that is associated with epithelial-to-mesenchymal transition (EMT) remains uncle

170 ased migration phenotype along with enhanced epithelial-to-mesenchymal transition (EMT) signature aft

171 confers distinct proliferative and invasive epithelial-to-mesenchymal transition (EMT) states in sub

172 HRAS-induced epithelial-to-mesenchymal transition (EMT) through activ

173 e of the neural tube, these cells undergo an epithelial-to-mesenchymal transition (EMT) to delaminate

174 The contribution of epithelial-to-mesenchymal transition (EMT) to the profib

175 were characterized by a higher expression of epithelial-to-mesenchymal transition (EMT) traits and by

176 cellular matrix organization, TGFbeta genes, epithelial-to-mesenchymal transition (EMT) transcription

177 In human breast carcinomas, epithelial-to-mesenchymal transition (EMT) upregulates L

178 omoter methylation of three genes regulating epithelial-to-mesenchymal transition (EMT), a key mechan

179 Here we show that execution of Epithelial-to-Mesenchymal Transition (EMT), a migratory

180 adherin to N-cadherin is associated with the epithelial-to-mesenchymal transition (EMT), a process re

181 cells is a bona fide physiological model of epithelial-to-mesenchymal transition (EMT), a process th

182 twork of transcription factors that controls epithelial-to-mesenchymal transition (EMT), a reversible

183 s lncRNA was proven a pivotal element of the epithelial-to-mesenchymal transition (EMT), a transdiffe

184 within primary breast cancers can undergo an epithelial-to-mesenchymal transition (EMT), although the

185 eir clusters and become motile by undergoing epithelial-to-mesenchymal transition (EMT), an essential

186 cells lose PDX1 expression while undergoing epithelial-to-mesenchymal transition (EMT), and PDX1 los

187 ast cancer, increased ECM stiffness promotes epithelial-to-mesenchymal transition (EMT), cell invasio

188 During epithelial-to-mesenchymal transition (EMT), cells lining

189 ion of a beta-catenin/TCF4-dependent partial epithelial-to-mesenchymal transition (EMT), followed by

190 RNAi-mediated silencing of RASSF1A induced epithelial-to-mesenchymal transition (EMT), fomenting a

191 applied to a proposed 22-gene network of the Epithelial-to-Mesenchymal Transition (EMT), from which w

192 asticity, including transdifferentiation and epithelial-to-mesenchymal transition (EMT), in the devel

193 oor prognosis owing to its role in promoting epithelial-to-mesenchymal transition (EMT), invasiveness

194 mnion membranes during labor were subject to epithelial-to-mesenchymal transition (EMT), mediated, in

195 altered level or activity of p53, markers of epithelial-to-mesenchymal transition (EMT), or MIR34A or

196 n hepatocytes is associated with HCV-induced epithelial-to-mesenchymal transition (EMT), providing an

197 Through investigating epithelial-to-mesenchymal transition (EMT), we develop a

198 h suggested PrP and ZIP6 are critical during epithelial-to-mesenchymal transition (EMT), we investiga

199 h factor-beta (TGF-beta) is major inducer of epithelial-to-mesenchymal transition (EMT), which associ

200 derstood about the contribution of lncRNA to epithelial-to-mesenchymal transition (EMT), which correl

201 pithelial ovarian cancer (EOC) cells undergo epithelial-to-mesenchymal transition (EMT), which influe

202 ny tissue types, metastasis is fueled by the epithelial-to-mesenchymal transition (EMT)-a dynamic pro

203 Epithelial-to-mesenchymal transition (EMT)-inducing tran

204 ithelial cell transformation by promoting an epithelial-to-mesenchymal transition (EMT)-like phenotyp

205 difficult to treat, especially the squamous/epithelial-to-mesenchymal transition (EMT)-like subtype.

206 Changes in the expression of epithelial-to-mesenchymal transition (EMT)-related molec

207 rogenitor cells emanating from epithelia via epithelial-to-mesenchymal transition (EMT).

208 romoter chromatin signatures associated with epithelial-to-mesenchymal transition (EMT).

209 to probe the genetic circuitry that controls epithelial-to-mesenchymal transition (EMT).

210 3K27me3), and inhibition of genes related to epithelial-to-mesenchymal transition (EMT).

211 the switch between E- and N-cadherins during epithelial-to-mesenchymal transition (EMT).

212 ing invasive migration, drug resistance, and epithelial-to-mesenchymal transition (EMT).

213 ated, and miRNA-depleted CDs undergo partial epithelial-to-mesenchymal transition (EMT).

214 y reactivating a latent embryonic programme: epithelial-to-mesenchymal transition (EMT).

215 ation of the mesoderm after the gastrulation epithelial-to-mesenchymal transition (EMT).

216 d altered the expression of genes related to epithelial-to-mesenchymal transition (EMT).

217 of TGFbeta by limiting its ability to induce epithelial-to-mesenchymal transition (EMT).

218 (Hh) and MET signaling cascades driving the epithelial-to-mesenchymal transition (EMT).

219 Pten deletion during cell transformation and epithelial-to-mesenchymal transition (EMT).

220 rior to EHT, in a striking parallel with the epithelial-to-mesenchymal transition (EMT).

221 sistent with a general function in promoting epithelial-to-mesenchymal transition (EMT).

222 IN, being this profile characteristic of the epithelial-to-mesenchymal transition (EMT).

223 anges of expression of genes associated with epithelial-to-mesenchymal transition (EMT).

224 e expression of Zeb1, a key regulator of the epithelial-to-mesenchymal transition (EMT).

225 due to its role as a transcription factor in epithelial-to-mesenchymal transition (EMT).

226 ays show that they act as suppressors of the epithelial-to-mesenchymal transition (EMT).

227 est cells arise from the head neural tube by epithelial-to-mesenchymal transition (EMT).

228 increased motility and invasiveness through epithelial-to-mesenchymal transition (EMT).

229 pithelium into a tube that will then undergo epithelial-to-mesenchymal transition (EMT).

230 disruption of acinar homeostasis, we induced epithelial-to-mesenchymal transition (EMT).

231 nd activation of the AXL tyrosine kinase and epithelial-to-mesenchymal transition (EMT).

232 e progression of metastatic cancer along the epithelial-to-mesenchymal transition (EMT).

233 which loss of the BRMS1 gene contributes to epithelial-to-mesenchymal transition (EMT).

234 microenvironment aid in the induction of the epithelial-to-mesenchymal transition (EMT).

235 additional oncogenic pathways and may affect epithelial-to-mesenchymal transition (EMT).

236 s multiple developmental roles, including in epithelial-to-mesenchymal transition (EMT).

237 nin nuclear accumulation, which up-regulates epithelial-to-mesenchymal transition (EMT).

238 0a suppressed multiple genes involved in the epithelial-to-mesenchymal transition (EMT).

239 is both in vitro and in vivo by inducing the epithelial-to-mesenchymal transition (EMT).

240 The FAM3C interleukin-like epithelial-to-mesenchymal-transition (EMT) inducer (ILEI

241 fumarate in mouse and human cells elicits an epithelial-to-mesenchymal-transition (EMT), a phenotypic

242 tion and tumor growth, but later it triggers epithelial-to-mesenchymal-transition (EMT).

243 Epithelial-to-mesenchymal transitions (EMTs) are phenoty

244 Epithelial-to-mesenchymal transitions (EMTs) require a c

245 During epithelial-to-mesenchymal transitions (EMTs), cells disa

246 shared features, including cells undergoing epithelial-to-mesenchymal transitions (EMTs), collective

247 The epithelial-to-mesenchymal transition enables carcinoma c

248 Moreover, it suppressed IR-induced epithelial-to-mesenchymal transition, expressions of NF-

249 cific long non-coding RNA that modulates the epithelial-to-mesenchymal transition, facilitates cell m

250 ed CD133-mediated cancer stemness and hybrid epithelial-to-mesenchymal transition features in advance

251 types and correlates with the expression of epithelial-to-mesenchymal transition gene signature, a f

252 ded by ALDH5A1), and with the presence of an epithelial-to-mesenchymal transition gene signature, imp

253 r, time to formation, proliferation, volume, epithelial to mesenchymal transition, gene expression, o

254 s developmental gene programs that stimulate epithelial-to-mesenchymal transition; however, it is not

255 rP(C)), a neuronal protein known to modulate epithelial-to-mesenchymal transition in a variety of cel

256 IL-6 activated epithelial-to-mesenchymal transition in cancer cells, wh

257 nsion of primary cancer cells, and induce an epithelial-to-mesenchymal transition in cancer cells.

258 Because HMGA1 promotes epithelial-to-mesenchymal transition in cancer, we hypot

259 how plastic CSCs are, and the importance of epithelial-to-mesenchymal transition in conferring CSC p

260 of IL-8 after 24 hours, but possibly induces epithelial-to-mesenchymal transition in epithelial cells

261 AGRN promoted epithelial-to-mesenchymal transition in primary tumors,

262 GFB in platelets led to enhanced hypoxia and epithelial-to-mesenchymal transition in the primary tumo

263 cally, we found that ABHD5 knockdown induces epithelial to mesenchymal transition, increasing aerobic

264 ZEB1, a master epithelial-to-mesenchymal transition-inducing transcript

265 Epithelial to mesenchymal transition is a common event d

266 The epithelial-to-mesenchymal transition is also regulated b

267 Epithelial-to-mesenchymal transition is implicated in me

268 , increased proliferation, and activation of epithelial-to-mesenchymal transition leading to increase

269 ial for stimulating stem cells to undergo an epithelial to mesenchymal transition-like process necess

270 d with cancer associated fibroblasts (CAFs), epithelial to mesenchymal transition, mesenchymal stem c

271 Concurrently, the proteins associated with epithelial-to-mesenchymal transition, N-cadherin and Vim

272 n of MRC-5 fibroblasts and TGF-beta-mediated epithelial-to-mesenchymal transition of A549 cells by in

273 n the proliferation, survival, migration and epithelial-to-mesenchymal transition of cancer cells.

274 is frequently citrullinated in cells during epithelial-to-mesenchymal transition of metastasizing ep

275 During the epithelial-to-mesenchymal transition of neural crest cel

276 and transforming growth factor beta-induced epithelial-to-mesenchymal transition of SKOV-3 cells.

277 endothelial cells and platelets and induced epithelial-to-mesenchymal transition of tumor cells, pro

278 Snai1 activities that promote an incomplete epithelial to mesenchymal transition on a subset of epib

279 ound to promote and/or maintain the state of epithelial to mesenchymal transition on CTCs through pla

280 to tumorigenesis including TGF-beta induced epithelial-to-mesenchymal transition on NMuMG cells and

281 typically resemble cells that have undergone epithelial-to-mesenchymal transition or EMT.

282 o a drug-tolerant state, for example through epithelial-to-mesenchymal transition or transition to a

283 xia, epithelial differentiation, and partial epithelial-to-mesenchymal transition (p-EMT).

284 ular carcinoma progression by regulating the epithelial-to-mesenchymal transition pathway.

285 7a increased expression of genes involved in epithelial-to-mesenchymal transition pathways, consisten

286 In the absence of USP9X, cells exhibited an epithelial-to-mesenchymal transition phenotype, acquired

287 d enhances cell migration especially in post-epithelial-to-mesenchymal transition (post-EMT) cancer c

288 eta-catenin and TGF-beta signalling, and pro-epithelial-to-mesenchymal transition/pro-metastatic prot

289 p53 inactivation activates a cell-autonomous epithelial-to-mesenchymal transition program leading to

290 proteins and with an increased expression of epithelial-to-mesenchymal transition-related genes.

291 n breast cancer cells increased MEK-EMT (MEK-epithelial-to-mesenchymal transition) signaling, transwe

292 tory: initiated in basal cells exhibiting an epithelial-to-mesenchymal transition signature, tumorige

293 late-stage cancer cells could facilitate the epithelial-to-mesenchymal transition, stemness, and mobi

294 C-like cells, with these cells undergoing an epithelial-to-mesenchymal transition to form clonogenic,

295 s the ability of cancer cells to undergo the epithelial-to-mesenchymal transition to invade and disse

296 YAP/TEAD engage the epithelial-to-mesenchymal transition transcription facto

297 uence hormone signaling, cell-cell adhesion, epithelial-to-mesenchymal transition, transforming growt

298 and differentiation, resistance to anoikis, epithelial-to-mesenchymal transition, tumor cell dormanc

299 Expression of genes involved in epithelial-to-mesenchymal transition was transiently enr

300 vitro-derived epicardial cells underwent an epithelial-to-mesenchymal transition when treated with P