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1 al cord and nodular enhancement of the cauda equina.
2  toxin isolated from the sea anemone Actinia equina.
3  and dopamine or by stimulation of the cauda equina.
4 lated through their motor roots in the cauda equina.
5  and removal of the sciatic nerves and cauda equina.
6 re giving rise to a long and extensive cauda equina.
7  at admission and 17.3% presented with cauda equina.
8 e of leptomeningeal enhancement in the cauda equina although CSF cytology was negative, and one patie
9 tudies demonstrated enhancement of the cauda equina and leptomeninges of the lower spinal cord.
10  metastatic cancer with spinal cord or cauda equina compression, life expectancy greater than 8 weeks
11 0.85-2.75; p=0.15), and spinal cord or cauda equina compressions were reported in seven (2%) of 425 v
12 shrimp, Caprella spp.; sea anemones, Actinia equina; cone snails, Conidae; male platypus, Ornithorhyn
13 hetized rats spinalized at T10-T11 and cauda equina cut).
14                Conus medullaris and/or cauda equina forms of spinal cord injury commonly result in a
15 t future repair of brachial plexus and cauda equina injuries will include spinal cord surgery to rest
16 vulsed roots after brachial plexus and cauda equina injuries.
17 nal outcomes after brachial plexus and cauda equina injuries.
18 al interest after conus medullaris and cauda equina injuries.
19 n in future attempts of treating acute cauda equina injuries.
20                   Here, we developed a cauda equina injury and repair rodent model to investigate whe
21  ventral root avulsion injury model of cauda equina injury to investigate the lesion effect in the sp
22 rsonality in the beadlet sea anemone Actinia equina is linked to genetically distinct morphotypes, wh
23 , they demonstrated enhancement of the cauda equina nerve roots, trigeminal nerve, and pachymeninges.
24                        Injuries to the cauda equina of the spinal cord result in autonomic and motor
25 individuals within a single population of A. equina, particularly those associated with the lower sho
26 a protein toxin from the sea anemone Actinia equina, readily creates pores in sphingomyelin-containin
27 nts with bowel dysfunction in complete cauda equina syndrome (CES).
28 city, including seizures, papilledema, cauda equina syndrome (n = 2), and encephalitis after a median
29            Compression associated with cauda equina syndrome must be reversed within 48 hours to pres
30 ented with meningitis alone (73%), the cauda equina syndrome or focal infection (15%), or posterior c
31 myelitis, cholecystitis, osteoporosis, cauda equina syndrome, and osseous defect), resulting in a fin
32  and brainstem, spinal cord, conus and cauda equina to disorders of peripheral innervation.
33 ral tail artery in rats that had their cauda equina transected to eliminate the somatic innervation o
34 pecies in situ, Anemonia viridis and Actinia equina using 16 S rDNA pyrosequencing.
35  of the 18 lesions, enhancement in the cauda equina was similar to that seen in Guillain-Barre syndro
36  conditions indicates that A. viridis and A. equina will be winners under future ocean acidification