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1 distinguishes O and P cell fates within this equivalence group.
2 respond to inductive signals, expanding the equivalence group.
3 echanism for singling out a SOP from a large equivalence group.
4 t cells that collectively constitute the O/P equivalence group.
5 t and appears to set up or limit the size of equivalence groups.
6 elve ventral epidermal cells form the 'vulva equivalence group'; although all six cells are competent
7 e played by cell interactions within the O/P equivalence group, and in the apparent significance of e
9 ells and support cells arise within a common equivalence group by cell-cell interactions mediated by
11 volved in cell fate specification of the O/P equivalence group differ among three laboratory colonies
14 at the developmental architecture of the O/P equivalence group has undergone evolutionary diversifica
15 of the sensory organ precursor (SOP) from an equivalence group in Drosophila is a paradigm for studyi
16 thelium appear to constitute a developmental equivalence group in which developing hair cells suppres
17 gest that cell fate specification in the O/P equivalence group is a complex process that involves mul
18 rolaimidae, the number of cells in the vulva equivalence group is limited by apoptosis and decreased
19 istles is a progressive process: each neural equivalence group is transiently defined by the expressi
20 Hair cells of the inner ear develop from an equivalence group marked by expression of the proneural
22 lineage of the rostral segments and the O-P equivalence group of the midbody and caudal segments con
23 ed root-mean-square deviation (RMSD) between equivalenced groups of amino acids is used as a measure
25 ferentiation, but its role in specifying the equivalence group (proneural function) has been question
26 e fate-determining interactions in this 'O-P equivalence group' take place between the equipotent cel
27 elegans male hook competence group (HCG), an equivalence group that has similar developmental origins
28 recursors are selected from a three-cell ;R8 equivalence group' through repression of ato by the home
29 lateral inhibition on the proneural cluster equivalence group, thus maintaining the persistent prone
30 ls must be achieved to enable one cell of an equivalence group to segregate as a progenitor while its