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1 but also removed by demethylases (the m(6)A 'erasers').
2 ymes, or so-called epigenetic 'writers' and 'erasers'.
3 on, and is removed by ADP-ribosylhydrolases (erasers).
4 log 5 (ALKBH5) as the responsible writer and eraser.
5 larization and creating a structured quantum eraser.
6  10 min for PVDF membrane with MemCode stain eraser.
7 DNA methylation, their readers, writers, and erasers.
8  methylation, i.e., the readers, writers and erasers.
9 stones, and epigenetic readers, writers, and erasers.
10 the expression of key epigenetic writers and erasers.
11     Current strategies to measure writer and eraser activities in living animals largely depend on in
12                 Here, we reported that m(6)A eraser AlkB homolog 5 (ALKBH5), but not FTO, maintains t
13                                    As an m6A eraser, ALKBH5 specifically removes m6A from target mRNA
14 s but is demethylated less efficiently by an eraser, ALKBH5.
15  found that TG2 additionally functions as an eraser and exchanger of H3 monoaminylations, including H
16 lly destructive sampling methods such as PVC eraser and polishing films for successful ZooMS identifi
17  data into four cohorts: the reader, writer, eraser and remodeler cohorts, together with curated intr
18 ists of three enzymatic components (writers, erasers and chromatin remodelers) as well as one non-enz
19  in expression profile of epigenetic writers/erasers and chromatin-binding of histone deacetylases (H
20 ng of H3K27me3 writers, activity of H3K27me3 erasers and deposition of a sperm-specific histone, H3.1
21 e H3 tails, recruiting catalytic writers and erasers and other components of the epigenetic machinery
22  chromatin binding of key epigenetic writers/erasers and PcG complexes to restrict tumor development.
23 scuss the functional roles of m(6)A writers, erasers and readers in cancer.
24 its associated machinery, including writers, erasers and readers, is frequently observed in various c
25  the activity of DNA methylation writers and erasers and the enrichment of a regulatory chromatin sta
26 thyltransferases ("writers"), demethylases ("erasers"), and m(6)A-binding proteins ("readers").
27 es (so-called "writers"), RNA demethylases ("erasers"), and RNA-binding proteins ("readers") that int
28 r RNA modification, modulated by 'writers', 'erasers' and 'readers' of this mark.
29  studies have discovered protein 'writers', 'erasers' and 'readers' of this RNA chemical mark, as wel
30 regulators, including 'writers', 'readers', 'erasers', and 'remodelers', is able to normalize the imp
31  methyltransferases (writers), demethylases (erasers), and m(6)A-binding proteins (readers) in cells.
32 s (writers), enzymes removing modifications (erasers), and readers of the histone code.
33                         Specific m6A writer, eraser, and reader proteins have been identified.
34 nd is substantially modulated by the writer, eraser, and reader proteins of N(6)-methyladenosine (m(6
35 s in biological processes, tRNA modification erasers, and human-associated bacteria.
36                  Various epigenetic writers, erasers, and modulators shape the perceived replicative
37  cell type-specific roles played by writers, erasers, and readers of chromatin modifications in the d
38 sses can broadly be classified into writers, erasers, and readers of covalent chromatin modifications
39                              As the writers, erasers, and readers of histone lysine methylation are e
40 tes and the characterization of the writers, erasers, and readers, the breadth of chromatin functions
41  inhibitors for other methyl-lysine writers, erasers, and readers.
42 mains function, respectively, as "writers," "erasers," and "readers" of phospho-tyrosine modification
43 ification modulators/regulators ("writers," "erasers," and "readers") drive the initiation and progre
44 erminology, have been designated "writers," "erasers," and "readers." Such modifications in the trans
45 were absent in cells treated with the miR-21 eraser, antagomiR-21, and in miR-21 knockout mice.
46 emerging roles of these H3K27me3 writers and erasers as central hubs in stress adaptation, highlighti
47 n microscopy structures of the glutamylation eraser CCP5 in complex with the microtubule, and X-ray s
48 ECM probe tip is used as an electrochemical "eraser" cleaning of the surface attached molecules and l
49 zed mammalian writer (methyltransferase) and eraser (demethylase) of the DNA N6-methyladenine (N6mA)
50   Whereas the writers (e.g. ARTD1/PARP1) and erasers (e.g. PARG, ARH3) of poly-ADP-ribosylation (PARy
51 ecific demethylase 1 (Lsd1) is an epigenetic eraser enzyme positively regulating differentiation and
52 ting the impact of multiple modifications on eraser enzyme processing and molecular recognition by a
53 ry (EPIDD) targeted at epigenetic writer and eraser enzymes as well as reader domains over the last 1
54                                   Writer and eraser enzymes establish and maintain the epigenetic cod
55                     The histone modification eraser enzymes lysine demethylase 1 (LSD1) and histone d
56 criptionally modified by dedicated writer or eraser enzymes that add or remove specific modifications
57 s that are altered dynamically by writer and eraser enzymes(1).
58 ated by silencing of the m(6)A writer or the eraser enzymes, which decreased or increased HIV-1 repli
59 tional modifications (PTMs) by "writer" and "eraser" enzymes, respectively.
60 y controlled by the balance of "writer" and "eraser" enzymes, which add or remove PTMs on proteins.
61 on is dynamically regulated by "writer" and "eraser" enzymes.
62 teomics approach to comprehensively profile 'eraser' enzymes that recognize a lysine-4 crotonylated h
63                   The delayed-choice quantum eraser experiment further demonstrates that complementar
64                    Here, we report a quantum eraser experiment in which, by enforcing Einstein locali
65                In an interferometric quantum eraser experiment, one can actively choose whether or no
66                                          The Eraser Extraction Method (EEM), first developed for ZooM
67 ro-striations associated with the use of the eraser for all bone specimens.
68 fused split O-GlcNAcase (OGA) as an O-GlcNAc eraser for selective deglycosylation of a target protein
69                                        While erasers for the MARylation of glutamate/aspartate and ar
70  targeting epigenetic "readers, writers, and erasers" for the treatment of cancer and other pathologi
71 us, mediates phosphorylation of the m6Am/m6A eraser FTO and that this correlates with a UL13- and FTO
72 s-induced transcripts by limiting the m(6)A 'eraser' FTO from demethylation.
73 is canonical posttranslational modification "eraser" function, SIRT6 can use NAD(+) instead to "write
74  the homeostasis of CD4(+) T cells, but the "erasers" functioning in T cells is poorly understood.
75 t the targeted recruitment of dCas13b-m(6) A eraser fusion protein and site-specific m(6) A erasing w
76 n (FTO), an RNA N(6)-methyladenosine (m(6)A) eraser, has been identified as an oncogenic factor in ac
77                               The epigenetic eraser histone deacetylase 2 (HDAC2) has been connected
78 expected specific role of ALKBH5 as an m(6)A eraser in controlling the pathogenicity of CD4(+) T cell
79  the protein expression of m(6)A writers and erasers in cells.
80 n be reversed by demethylases that serve as "erasers." In this review, we mainly summarize recent pro
81 s-producing cells, while silencing the m(6)A erasers increased Gag expression.
82  achieved through the balance of writers and erasers, is critical for normal cell function(3-9), and
83 e regulation of L-lactylation by writers and erasers, its readers and its cofactor L-lactyl-CoA.
84 e diurnal expression of genes coding for the eraser (JMJ14) directly and writer (SDG2) indirectly for
85 , the Polycomb Repressive Complex 2, and its erasers, Jumonji-class histone lysine demethylases.
86 , hypoxia decreased activity of the H3K36me2 eraser KDM2A, while on the other hand promoting stabiliz
87 evels, such as recruitment of m6A writers or erasers leading to either 'global' or 'site-specific' mo
88           We compare the effects of HDAC (an eraser of acetylation marks) and BET (a reader of acetyl
89  these data indicate that ARH3 can act as an eraser of ADP-ribose chromatin scars at sites of PARP ac
90 iscuss the hypothesis that the writer-reader-eraser of N6mA-containining ssDNA is associated with mai
91 epigenetic regulation, including writers and erasers of all major histone marks.
92 fications, and show that sirtuins can act as erasers of HibK modified proteins.
93  identify 65 predicted readers, writers, and erasers of histone acetylation and methylation encoded i
94 ave been made in identifying the writers and erasers of histone acetylation marks during learning.
95  Histone deacetylases (HDACs) are epigenetic erasers of lysine-acetyl marks.
96 ined the expression of writers, readers, and erasers of m6A and report that HNRNPA2/B1 expression is
97 adily adapted to investigate the readers and erasers of many proteins that undergo reversible ubiquit
98  regulators of RNA epigenetics by serving as erasers of one-carbon marks on RNA with release of forma
99  ADP-ribosylation signals by the readers and erasers of protein ADP-ribosylation, has been significan
100                     The writers, readers and erasers of such diverse modifications, which constitute
101 ge polyubiquitin at DNA damage sites and an "eraser" of the Lys11-linkage ubiquitination, indicating
102 e code," although the "readers, writers, and erasers" of the cytoskeleton and epigenome have heretofo
103 n association with epigenetic "writers" and "erasers" of the histone code.
104  remodeller genes, the 'readers, writers and erasers' of methyl marks on chromatin, as major contribu
105  charge generated by gentle rubbing of a PVC eraser on the membrane surface.
106 ssociated proteins ("reader", "writer", and "eraser" or RWE proteins) and alters the landscape of RNA
107  knockdown of miR-21 using a specific 'miRNA eraser' or overexpression of SPRY2 inhibited betaAR-indu
108 bacteria in the context of the writer-reader-eraser paradigm, and progress toward ascribing regulator
109 y, we showed that m(6)A demethylation by the eraser protein ALKBH5 occurs at active synaptic ribosome
110 ally induce the degradation of an epigenetic eraser protein.
111  HEK293T cells with the genes encoding m(6)A eraser proteins (i.e., ALKBH5, FTO) and the catalytic su
112  degraders of epigenetic writer, reader, and eraser proteins and their in vitro and in vivo effects.
113  evidence suggests m(6)A writer, reader, and eraser proteins are attractive therapeutic targets.
114 ons and their associated writer, reader, and eraser proteins in infection by diverse RNA and DNA viru
115  the YTHDF1 and YTHDF3 reader and the ALKBH5 eraser proteins increase in co-localisation to m(6)A-mod
116 rtance of specific m(6)A writer, reader, and eraser proteins remains unknown.
117 odulating the binding of writer, reader, and eraser proteins.
118 diated by specific m(6)A writer, reader, and eraser proteins.
119  their interactions with acetyl- and phospho-eraser proteins.
120               The nanobody-directed O-GlcNAc eraser provides a new strategy for the functional evalua
121 ghts key mechanisms that epigenetic writers, erasers, readers, and remodelers control, as well as the
122 onents of the epigenetic machinery (writers, erasers, readers, and remodelers) and are thus expected
123 discovery of epigenetic modulators (writers, erasers, readers, and remodelers) has shed light on prev
124 rect role in epigenetic regulation (writers, erasers, remodelers, readers).
125 n, and the associated epigenetic writers and erasers, represent potential targets for the treatment o
126 denosine (m(6)A) and its reader, writer, and eraser (RWE) proteins assume crucial roles in regulating
127        Epitranscriptomic reader, writer, and eraser (RWE) proteins recognize, install, and remove mod
128 of 154 epitranscriptomic reader, writer, and eraser (RWE) proteins using a scheduled liquid chromatog
129 d by a writer complex and can be reversed by erasers such as the fat mass and obesity-associated prot
130 l entanglement verification uses the quantum eraser technique, and demonstrates that a high degree of
131  with both the m5C writer TRDMT1 and the m5C eraser ten-eleventranslocation protein 1 (TET1).
132 roups of cellular proteins named writers and erasers that add or remove m(6)A, respectively.
133 tribution of phosphatases, defining them as "erasers" that serve merely to restore the system to its
134 ffectors," including enzymes ("writers" and "erasers") that alter the modification level and binding
135  Among epigenetic "writers", "readers", and "erasers", the lysine methyltransferases G9a and GLP, whi
136 d-lineage leukemia 1 (Mll1) (n = 26), and an eraser, the histone lysine (K)-specific demethylase 5C (
137  that our device can also act as a "Landauer eraser", using externally supplied work to remove inform
138              For the HM readers, writers and erasers, we provided further ChIP-Seq analysis data for
139 amine cleaning sponges (also known as "magic erasers") with different strut densities against metal s
140  ADP-ribose signaling (writers, readers, and erasers) with a focus on those involved in the maintenan

 
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