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1 es exhibited a clinical score of 1 (pinpoint erythema).
2 rized by epidermal hyperplasia, scaling, and erythema.
3 mall and medium joint pain, without edema or erythema.
4 ndoscopy showing areas of nonspecific patchy erythema.
5 AEs were application-site reactions, usually erythema.
6 nt is often initiated by symptoms or visible erythema.
7 , consisting mainly of infusion syndrome and erythema.
8 ced more TTs than UVA1 for the same level of erythema.
9 lmarked by undermined borders and peripheral erythema.
10 ft breast with no skin retraction, edema, or erythema.
11 eukocyte markers, while quantifying clinical erythema.
12 on site pain and tenderness, induration, and erythema.
13 delivery, but are sometimes associated with erythema.
14 pital because of cough, sputum, pruritus and erythema.
15 ility and reproducibility for both scale and erythema.
17 and as tenderness (33 [66%] of 50 [51-79]), erythema (20 [40%] of 50 [26-55]), and pruritus (41 [82%
18 local adverse events with imiquimod included erythema (27%), scabbing or crusting (21%), flaking (9%)
19 mon adverse events were again injection-site erythema (33 [8%] of 416 with 50 mg sirukumab every 4 we
20 the dupilumab group developed injection-site erythema (35% vs 8% in the placebo group) and nasopharyn
22 eported injection site pain (52% vs 17%) and erythema (73% vs 25%) more frequently than placebo recip
23 acterized by less transient and nontransient erythema, a more lateral distribution of erythema and te
24 int scale of plaque thickening, scaling, and erythema; a score of 0 indicates clear, 1 almost clear,
25 ter an immediate allergic reaction including erythema, abdominal pain, vomiting, and anaphylactic sho
26 , 28-53 years of age), the mean reduction in erythema across six doses of UVR (300-800 mJ/cm(2) in 10
28 inant, disorder characterized by generalized erythema and cutaneous blistering at birth followed by h
31 patients (50.2%), followed by injection site erythema and fatigue in 227 (25.6%) and 212 patients (23
33 in the 1 g idarucizumab group (infusion site erythema and hot flushes), one in the 5 g plus 2.5 g ida
34 enotype consisting of early-onset patches of erythema and hyperkeratosis, as well as SCA manifesting
35 ed cutaneous immunity based on the extent of erythema and induration after intradermal VZV antigen in
43 d on the day of treatment, and short-lasting erythema and pain at the site of application were common
46 ed to PCV13 (facial diplegia, injection-site erythema and pyrexia, autoimmune hemolytic anemia, and s
48 for association with skin cGVHD involvement (erythema and sclerosis, skin symptoms), lacrimal dysfunc
49 he spontaneous behaviors, in addition to the erythema and skin pathology, were markedly improved.
50 haracterized by intermittent flares of pain, erythema and swelling in and around the joints, which ar
53 ent erythema, a more lateral distribution of erythema and telangiectasia, less neurogenic mast cell a
55 the cessation of spread of infection-related erythema and the absence of fever at 48 to 72 hours.
56 esponse (healing of erosions, improvement in erythema, and alleviation of pain), adverse effects, and
57 dema, lupus erythematosus [occurring twice], erythema, and dermohypodermitis all experienced by one [
60 nces in vaccination take rates, lesion size, erythema, and induration or in serum neutralizing-antibo
69 ned as a >=20% reduction of lesion spread of erythema area at the primary infection site at 48 to 72
70 edema in mice, and reduced susceptibility to erythema arising from narrow-band 311-nm UVR in humans.
72 r the sunburn threshold or the resolution of erythema, as assessed by spectrophotometric hemoglobin i
73 cterized by keratinocyte hyperproliferation, erythema, as well as a form of pruritus, involving cutan
74 safely treat skin inflammation, scaling, and erythema associated with psoriasis while avoiding possib
75 dpoint was a >/=20% reduction in the area of erythema at 48-72 hours in the intent-to-treat populatio
77 uncommon but included discomfort, cutaneous erythema, blistering, eyelash loss, and floaters; these
79 tamin D(3) synthesis is small, compared with erythema, but that this difference may be sufficient to
81 rate cutaneous inflammation characterized by erythema, dermal infiltrates of CD45(+) leukocytes, and
84 ( Fig 1 ), there was generalized periareolar erythema, dimpling, firmness, and fixation involving the
85 irradiated with UVB (three times, 1 minimal erythema dose (80 mJ/cm(2)), weekly) for 10 weeks, and e
87 -UVB) regimens start with 70% of the minimal erythema dose (MED) with 20% increments at each treatmen
88 s that measured time-related UVR in standard erythema dose (SED) and corresponding sun diaries (mean,
89 rotein changes in the skin after one minimal erythema dose of spectrally pure UVA1 (50 J cm(-2)) and
90 in vivo by looking at changes in the minimal erythema dose with subsequent doses of UV radiation.
91 ion, median (IQR) sunburn threshold (minimal erythema dose) was 28 (20-28) and 20 (20-28) mJ/cm(2) in
92 during the study was 39.1 (30.9) as standard erythema dose, comparable to a quarter of the median sum
94 exposures ranging from 0.65 to 3.9 standard erythema doses (SEDs), which were equivalent to 15-90 mi
95 n between our subjects' UVB and UVA1 minimal erythema doses implies that UVA1 and UVB erythema occur
96 or imiquimod, reduced the inflammation from erythema doses of artificial sunlight, and lowered the t
98 nically important differences in tendency to erythema during a standard 70/20% NB-UVB twice-weekly re
99 Side effects, including the development of erythema during phototherapy, were similar for the two l
102 narof leads to compound-driven reductions in erythema, epidermal thickening, and tissue cytokine leve
105 trichomegaly, eyelash hypertrichosis, eyelid erythema, eyelid edema, eyelid hyperpigmentation, high u
107 common after intradermal administration (31% erythema for full subcutaneous dose and 77% for intrader
108 se events in this period were injection-site erythema (four [1%] with placebo, 22 [8%] with 50 mg sir
109 Pulsed-dye laser was used for pruritus and erythema; fractional CO2 laser was used for stiffness an
111 eria at follow-up visits: fever; increase in erythema (>25%), swelling, or tenderness (days 3-4); no
112 ommon adverse events were diarrhoea, rash or erythema, hepatic adverse events, and neutropenia (not r
115 Area Severity Index (IASI), which integrates erythema (IASI-E) and scaling (IASI-S); transepidermal w
116 ticipants (78%) in the umbrella group showed erythema in 1 or more sites vs 10 of the 40 participants
119 nvestigated UV-induced sunburn apoptosis and erythema in mouse skin as a function of circadian time.
120 GE(2), PGF(2alpha), and PGE(3) accompany the erythema in the first 24-48 h, associated with increased
121 ymptoms mimicking SCAR, including hair loss, erythema, increase of inflammatory lymphocytes in the sk
123 h no difference in AUC analysis for measured erythema index after a geometric series of 10 UVR doses.
124 xtensively evaluated in vivo as a measure of erythema induced in human skin and is expressed as Sun P
125 asured by dermal microdialysis and cutaneous erythema), induces an intense epidermal infiltrate of ma
127 of human diseases, including fifth disease (erythema infectiosum) in children and pure red cell apla
130 over 3 mo did not significantly reduce skin erythema, leukocyte infiltration, or eicosanoid response
131 aracterized by transient figurate patches of erythema, localized or generalized scaling, and frequent
132 lar pain (100%), sleep disturbances (75.3%), erythema migrans (59.7%), headache (46.8%), fatigue (44.
133 ri DNA in skin samples from 90 patients with erythema migrans (EM) and in synovial fluid (SF) from 63
134 fection causes an initial skin lesion called erythema migrans (EM) in human Lyme disease and in model
135 ent with early LD presenting with or without erythema migrans (EM) or an annular, expanding skin lesi
136 hese inflammatory responses in patients with erythema migrans (EM) or Lyme arthritis (LA) to elucidat
137 l manifestation of early Lyme disease is the erythema migrans (EM) skin lesion that develops at the t
138 urgdorferi genotypes have been isolated from erythema migrans (EM) skin lesions in patients with Lyme
139 bjective was to obtain data on patients with erythema migrans (EM) who have symptoms/signs suggesting
140 ltiplex assays in the serum of patients with erythema migrans (EM), joint fluid of patients with Lyme
141 stage LD but is insensitive in patients with erythema migrans (EM), the most common manifestation of
142 ) of 12 particular symptoms in patients with erythema migrans (n = 52) differed from matched control
143 symptoms was compared between patients with erythema migrans and 81 control subjects without a histo
144 confirmed early LD, based on the presence of erythema migrans and documentation of seroconversion or
145 course of oral doxycycline for treatment of erythema migrans and for a 14-day course for treatment o
146 5 consecutive patients with the diagnosis of erythema migrans and reported original data regarding th
147 variability in the clinical presentation of erythema migrans and the need to factor in multiple comp
152 from 17 patients who received a diagnosis of erythema migrans between 1991 and 2011 and who had 22 pa
153 ive for B. burgdorferi DNA in a patient with erythema migrans early during therapy and in a patient w
154 ypically at least 10% of adult patients with erythema migrans experience persistence of at least 1 su
155 ent evidence supports treating patients with erythema migrans for no longer than 10 days when doxycyc
157 0-day doxycycline treatment in patients with erythema migrans has been assessed in the United States
158 ents with early Lyme disease associated with erythema migrans have a positive blood culture based on
162 Lyme disease as either a clinician-diagnosed erythema migrans lesion or a positive standard 2-tiered
164 the frequency of nonspecific symptoms among erythema migrans patients was similar to that among cont
165 arious MTTT protocols in patients with acute erythema migrans ranged from 36% (95% confidence interva
166 teins shed in the urine of patients with (1) erythema migrans rash and acute symptoms, (2) post treat
170 9 (MMP-9) was selectively upregulated in the erythema migrans skin lesions of patients with acute Lym
171 afzelii, or Borrelia garinii recovered from erythema migrans skin lesions of patients with Lyme borr
172 patients with culture-confirmed episodes of erythema migrans to distinguish between relapse and rein
175 one of the 22 paired consecutive episodes of erythema migrans were associated with the same strain of
176 study assessed whether repeated episodes of erythema migrans were due to the same or different strai
178 Similarly, serum samples from patients with erythema migrans who were infected with the RST1 genotyp
180 s with probable LD, based on the presence of erythema migrans without documented seroconversion or of
181 86 antibiotic-treated European patients with erythema migrans, 45 with post-Lyme symptoms and 41 with
200 City with early Lyme disease associated with erythema migrans; it is the largest number of borrelial
201 determined using serum from 55 patients with erythema migrans; specificity was determined using serum
202 s were well tolerated in the skin, with mild erythema, minimal wheal formation and complete resolutio
203 -independent, cascade of events resulting in erythema, mixed dermal infiltrate, and epidermal hyperpl
205 pothyroidism; grade 3 iridocyclitis, grade 1 erythema multiforme, and grade 3 erythema; and grade 2 i
206 eumonitis, localized myositis, folliculitis, erythema multiforme, or ophthalmological manifestations.
207 n in other closely related syndromes such as erythema multiforme, Stevens-Johnson syndrome, and toxic
208 on-eczematous contact dermatitis include the erythema multiforme-like, the purpuric, the lichenoid, a
209 een administration routes for injection-site erythema (n=10 [12%] and n=0, respectively) and nausea (
210 ts in the vitespen group were injection-site erythema (n=158) and injection-site induration (n=153).
211 rface area and UVB exposure dose, related to erythema, necessary to achieve a given level of vitamin
212 nodes (3.7%-9.0%), hyperhidrosis (<2%), and erythema nodosum (<2%) were particularly suggestive of f
213 ed oral ulcers (100%), genital ulcers (62%), erythema nodosum (46%), and papulopustular lesions (54%)
215 ease course (Pcombined = 5.94 x 10(-7)), and erythema nodosum (Pcombined = 2.27 x 10(-6)), respective
216 osition -308 was found to be associated with erythema nodosum in Caucasian sarcoidosis patients (stud
217 a gene adjacent to TNF, was associated with erythema nodosum in female Caucasian sarcoidosis patient
220 nts when evaluated before the onset of acute erythema nodosum leprosum (ENL) and persistently elevate
224 associated with increased susceptibility to erythema nodosum leprosum in an allelic analysis, wherea
226 s "reactional states" (reversal reaction and erythema nodosum leprosum) that result in major clinical
227 wn role in inflammation were associated with erythema nodosum status in 659 sarcoidosis patients and
228 noted: fever, sialadenitis, lymphadenopathy, erythema nodosum, leukocytoclastic vasculitis, transient
231 ed erythema, the number of treatments before erythema occurred did not differ between skin types (P=0
232 e drug eruptions, viral exanthema, and toxic erythema of chemotherapy, Demodex folliculitis is a clin
233 tis was defined as either pus discharge with erythema of the abdominal skin or severe redness (>2 cm
234 d related to cholestatic jaundice, oedema or erythema of the extremity associated with desquamation o
237 ndice, bilateral laterocervical lymph nodes, erythema of the palms, and strikingly red lips and conju
239 (aOR 5.17, 95% CI 1.9-14.1), abdominal wall erythema or discolouration at presentation (aOR 2.51, 95
241 n with placebo included injection site pain, erythema, or both (21 [20%] of 107 vs seven [6%] of 110)
242 se (31% prevalence) was associated with skin erythema (P < 0.001); salivary dysfunction (11% prevalen
244 s caused by protease overactivity, including erythema, peeling, and exacerbation on water exposure.
246 r Scale (VSS), which documents pigmentation, erythema, pliability, and height, and (2) University of
248 rate transient injection-site reactions (eg, erythema, pruritus) were the most frequent IMA901-relate
249 nal treatment, presence of serous discharge, erythema, purulent exudate, separation of the deep tissu
251 an increase in body temperature (fever) and erythema (rash) in comparison with humanized mice inocul
252 l (ie, patch-site) adverse events (including erythema, rash, pruritus, hyperpigmentation, pain, hypop
254 ed skin blood flow (Doppler velocimeter) and erythema (reflectance colorimeter a*) following topical
258 the dermatological sum score (DSS) assessing erythema, scaling, and plaque elevation on a 4-point sca
260 assessed, showing nonerosive lesions such as erythema, swelling, and lymphoid hyperplasia in 8 patien
263 ng, or tenderness (days 3-4); no decrease in erythema, swelling, or tenderness (days 8-10); and more
265 ainly by epidermal hyperplasia, scaling, and erythema; T helper 17 cells have a role in its pathogene
268 Index for Ichthyosis Severity for scale and erythema that provides (1) written descriptions of the f
269 n skin disorder of follicular prominence and erythema that typically affects the proximal extremities
271 UV radiation were pigmentation, scaling, and erythema; the most frequent dermoscopic changes were inc
272 d, although more frequent infusion site pain/erythema/thrombophlebitis was seen with fosaprepitant re
274 rized as Kansas City classification: type 1: erythema; type 2: ulcers (2a: superficial ulcers; 2b: de
275 ation (18 [44%]), xerosis (8 [20%]), scrotal erythema/ulceration (6 [15%]), and nail splinter hemorrh
281 By contrast, the interrater reliability for erythema was higher during in-person validation compared
282 tential genetic basis for alcohol-associated erythema was investigated as the function of polymorphis
284 eactions occurred in most patients, but only erythema was significantly more common in the VGX-3100 g
288 ptosis, inflammatory cytokine induction, and erythema were maximal following an acute early-morning e
294 tly responsible for the necrolytic migratory erythema, which resolves after amino acid administration
295 was commensurate with development of palatal erythema, which suggests a role for biofilm in the infla
296 ts, one from each group, reported persistent erythema, which was considered to be possibly related to
297 signs are skin lesions (necrolytic migratory erythema), while in subjects with inactivating mutations
298 blood flow without erythema and PG increased erythema with decreased skin blood flow, all as a functi
299 ion of KLK-5 in SOD3 knockout mice exhibited erythema with increased epidermal thickness, mast cell a
300 oM) each produced perceptible induration and erythema with moderate cellular infiltration resolving w