ライフサイエンスコーパス: escape response

1 of the hindbrain circuitry that supports the escape response.

2 inct from those of individuals exhibiting an escape response.

3 per se or change in luminance, triggered the escape response.

4 it organization for execution of the rolling escape response.

5 t fish, well-known for their pectoral aerial escape response.

6 ts ovipositor, it initiates a characteristic escape response.

7 logic of the dynamical system underlying the escape response.

8 , looming stimulus), chicks displayed a fast escape response.

9 to changes in ambient light and mediates an escape response.

10 ical or electrical synapses causes defective escape response.

11 locomotion during the Caenorhabditis elegans escape response.

12 t for flies to efficiently initiate the loom escape response.

13 s in line with expectations for a last-ditch escape response.

14 movements that are critical for a C. elegans escape response.

15 st, heterozygous mutants show an exaggerated escape response.

16 1000 Hz) of flies and their need for a rapid escape response.

17 creen for defects in the acoustically evoked escape response.

18 r1 circuit during development suppresses the escape response.

19 ons in circuits similar to those in the fish escape response.

20 sb420 mutants were active during an elicited escape response.

21 s of the crayfish nerve cord drive tail-flip escape responses.

22 mission and disrupts Mauthner cell-initiated escape responses.

23 subsequently guide individual and collective escape responses.

24 s, acting as central circuit elements gating escape responses.

25 track prey movement within 50-200 ms of prey escape responses.

26 pulations that exert inhibitory control over escape responses.

27 e required for the speed and coordination of escape responses.

28 of slow swimming during stereotyped acoustic escape responses.

29 e and initiate diverse drought avoidance and escape responses.

30 he inability of the larvae to perform normal escape responses.

31 ease in release at the warning signal during escape responses.

32 the system rather than in acutely mediating escape responses.

33 range of nociceptive cues and signal robust escape responses.

34 volved in the organization of sensory-evoked escape responses.

35 quick enough to exceed the prey fish's rapid escape responses [4].

36 ese subunits were defective in their hypoxia escape response-a rapid cessation of feeding and withdra

37 stress-sensitive (Stay) to stress-resilient (Escape) responses, an effect that was mimicked by geneti

38 The escape response and rhythmic swimming in zebrafish are d

39 a combination of a bacterial respiration and escape response and the neutrophil respiratory burst but

40 effects of an acute stressor (restraint) on escape responses and lick/guard reflexes to stimulation

41 nished spontaneous contractions and abnormal escape response, and impaired excitation-contraction cou

42 it sluggishness, uncoordination, a defective escape response, and male sterility.

43 ior but can also inhibit ingestion and evoke escape responses, and both stimulate grooming.

44 agonists and antagonists in abdomen posture, escape responses, and fighting have led to the suggestio

45 ptogenetic stimulation of the vLGN abolishes escape responses, and suppressing its activity lowers th

46 or change to improve camouflage and predator escape responses are adversely affected by ship noise bu

47 ications of spontaneous swimming and tactile escape response, as well as measurements of axonal proje

48 presence of neighboring vegetation and evoke escape responses before canopy cover limits photosynthes

49 ted Tau and with recovery of the stereotypic escape response behavior.

50 In contrast, Wistar rats showed no initial escape response but a prolonged period of freezing that

51 of sst1.1 did not impact acousto-vestibular escape responses but led to abnormal exploration.

52 tshocks commenced, animals could initiate an escape response by pressing the lever, terminating foots

53 re enemies, taking advantage of fish C-start escape responses by startling fish toward their strike--

54 These nerves directly coordinate the rapid escape response bypassing the central integrative struct

55 endrite of the Mauthner (M)-cell triggers an escape response (C-start) in goldfish.

56 ell defined neural circuit that underlies an escape response can be habituated, providing for the fir

57 idance responses to the signal but preserves escape responses caused by presentation of the threat.

58 n the VNC, guiding a developmentally plastic escape response circuit.

59 ess Mauthner cells are incorporated into the escape-response circuit, but they divide their target te

60 nstrating plasticity in the formation of the escape-response circuit.

61 these extra cells are incorporated into the escape-response circuit.

62 tacognition in animals, one must ensure that escape responses do not increase the overall density of

63 Escape response (dropping/non-dropping off a plant upon

64 xtual memory, and a greater active avoidance escape response during the active phase.

65 kable level of recovery, exhibiting a robust escape response following developmental delay.

66 on response when nutrients run out, or as an escape response from harmful situations(12-16).

67 nal threat likely evolved to enable flexible escape responses from predators.

68 ns between sensory input and motor output in escape responses have suggested two alternative patterns

69 Touch elicits an escape response in Caenorhabditis elegans where the anim

70 ry and sufficient to trigger a stereotypical escape response in fish.

71 expression of BLINK1 reversibly inhibits the escape response in light-exposed zebrafish larvae.

72 However, the kinematic performance of the escape response in mutant larvae was very similar to wil

73 RpoS also regulates the mucosal escape response in pathogenic strains of V. cholerae.

74 e examined novel aspects of the touch-evoked escape response in techno trousers (tnt) mutant embryos,

75 elegans, anterior touch initiates a backward escape response in which lateral head movements are supp

76 he body of Caenorhabditis elegans elicits an escape response in which the animal quickly reverses and

77 relatively simple neural circuit driving the escape response in zebrafish offers an excellent opportu

78 neration and results in significantly faster escape responses in dystrophic embryos.

79 dressing the disparity between detection and escape responses in future research.

80 ively long delay between their foveation and escape responses in individuals obscured the relationshi

81 uatic annelid worms have some of the fastest escape responses in nature, the sensory networks that me

82 ours to partially override the light-induced escape responses in the male.

83 e interneurons in transgenic animals impairs escape responses, indicating their crucial role in survi

84 In Drosophila larvae, one type of escape response involves C-shaped bending and lateral ro

85 nt at an early age, whereas the speed of the escape response is paramount, and that directional respo

86 A key feature of escape responses is the fast translation of sensory info

87 estigated links between a personality trait (escape response), life-history and state variables (grow

88 derstand how stimuli evoke sudden, ballistic escape responses, like fish fast-starts, a precise pathw

89 When a zebrafish makes a fast escape response, Mauthner cells directly activate contra

90 synaptic transmission between neurons in the escape response neural circuit of adult flies.

91 rosophila giant fiber system (GFS), a simple escape response neuronal circuit, by increasing targetin

92 The neural pathway that governs an escape response of Drosophila to sudden changes in light

93 level of heat stimulus from the stereotyped escape response of individual nematodes Caenorhabditis e

94 s of ectopic Mauthner cells (M-cells) in the escape response of larval zebrafish.

95 TDT, or jump muscle), which functions in the escape response of the Drosophila adult.

96 dfish, Carassius auratus, triggers the rapid escape response of the fish in response to various stimu

97 the Giant Fiber circuit, which mediates the escape response of the fly.

98 n together, we show that hypoxia triggers an escape response of the primary root that is controlled b

99 Escape responses of cockroaches, Periplaneta americana,

100 detection enables crayfish to produce reflex escape responses only to very abrupt mechanical stimuli.

101 Their escape response provides one of most effective mechanism

102 k intensity (0.1-0.7 mA) and shock avoidance/escape response requirement (FR1-16) were also manipulat

103 hock intensity or increasing shock avoidance/escape response requirement failed to increase fentanyl

104 is thought to produce different forms of the escape response that fish use to avoid predators.

105 are recruited during different forms of the escape response that fish use to avoid predators.

106 at their head or tail, nematodes display an escape response that is mediated by bacterially produced

107 and a DeltaHP0102 mutant exhibited low acid-escape response that might account for the poor coloniza

108 Painful or threatening experiences trigger escape responses that are guided by nociceptive neuronal

109 ne which has its primary effect on the fly's escape response, the other on wing morphogenesis, are mu

110 decades on habituation of startle and other escape responses, the underlying neural mechanisms are s

111 ally coordinates the different phases of the escape response through the synaptic activation of the f

112 ious touch and temperature, with stereotyped escape responses through activation of multimodal nocice

113 The escape response to a visual threat is, however, flexible

114 nge in spinal cord development optimizes the escape response to gentle touch of animals raised in and

115 ctural plasticity, that in parallel controls escape response to noxious mechanosensory stimuli.

116 , activation of LiGluR reversibly blocks the escape response to touch.

117 ant fiber pathway mediates a jump-and-flight escape response to visual stimuli.

118 s eliminated short-latency, high-performance escape responses to both head- and tail-directed stimuli

119 ological control points in regulating stress-escape responses to different environmental stimuli.

120 ed molecular rhythms of the circadian clock, escape responses to irritants, and multi-parameter day-n

121 cific TRPV3 transgenic mice showed increased escape responses to noxious heat relative to their wild-

122 These inputs, which mediate escape responses to sudden stimuli, may be modulated by

123 In contrast, learned escape responses to the same thermal stimulus were signi

124 In behavioral tests, rats performed learned escape responses to thermal stimulation of the paws by 4

125 that photoinhibition of this input decreases escape responses to threatening stimuli.

126 that endogenous basal ABA inhibits a stress-escape response under nonstressed conditions, allowing p

127 The hypoxia escape response was restored by reintroducing either Gyc

128 As seen during escape responses, we observed a time-locked decrease in

129 uding scototaxis, activity, exploration, and escape response were assessed after 7 and 14 days.

130 ve, and that appears to specifically inhibit escape response when activated.

131 example, zebrafish manifest a stereotypical escape response when exposed to an alarm substance relea

132 spontaneous coiling of the trunk, diminished escape responses when touched, and an absence of swimmin

133 he head of Caenorhabditis elegans induces an escape response where the animal rapidly backs away from

134 Finally, dark shadows activate SC and drive escape responses, whereas vLGN prefers bright stimuli.

135 centration of calcium, a potential stress or escape response, while these larvae elongated when expos

136 icient fish exhibit an abnormal touch-evoked escape response with excessive body contractions and a p

137 es evoke slower, more kinematically variable escape responses with relatively long latencies as well