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1 t a useful predictor of pathologic nocturnal esophageal reflux.
2  after a meal can reduce acidic postprandial esophageal reflux.
3 beran) previously evaluated as a therapy for esophageal reflux.
4 ), acid suppression therapy for infants with esophageal reflux (3814 of 7507 of [50.8%]), and blood c
5 ophagus, an otherwise benign complication of esophageal reflux, affects approximately three million A
6 phageal motility abnormalities, and proximal esophageal reflux among patients with end-stage lung dis
7 ngeal reflux at least once a day and/or high esophageal reflux at least 5 times a day, subsequently u
8 sed as a pathophysiological factor in gastro-esophageal reflux disease (GERD) and as a target for GER
9 ty-four-hour pH monitoring documented Gastro-Esophageal Reflux Disease (GERD) in 53.4% of patients.
10                           Symptoms of gastro-esophageal reflux disease (GERD) in pregnancy are report
11                                       Gastro-esophageal reflux disease (GERD) is suggested to be asso
12                     The prevalence of gastro-esophageal reflux disease (GERD) varies widely around th
13 in esophageal biopsies and absence of gastro-esophageal reflux disease (GERD) were included.
14 ve time, complications, postoperative gastro-esophageal reflux disease (GERD).
15 of new histology-based definitions of gastro-esophageal reflux disease (GERD).
16 f comorbidities is similar except for gastro-esophageal reflux disease and dyslipidemia that appear t
17 able after both procedures except for gastro-esophageal reflux disease and dyslipidemia, which were m
18 gurgitated acid entering the mouth in gastro-esophageal reflux disease can cause dental erosion.
19                Antireflux surgery for gastro-esophageal reflux disease is followed by varying rates o
20 ale with a history of well-controlled gastro-esophageal reflux disease presented to the emergency roo
21 important causes for cough, including gastro-esophageal reflux disease, allergic rhinitis, hay fever,
22 rative complications or recurrence of gastro-esophageal reflux disease.
23  causes of heartburn in patients with gastro-esophageal reflux disease.
24 re insufficient weight loss (43%) and gastro-esophageal reflux disease/dysphagia (25%).
25 sensitive cough reflex (P = .03) but similar esophageal reflux exposure and erosive disease, together
26                            Background acidic esophageal reflux exposure appeared stable over time, wh
27 We have previously shown that gastroduodenal-esophageal reflux (GDER) together with N-methyl-N-benzyl
28                                       Gastro-esophageal reflux (GERD) post-SG is a critical issue due
29         The prevalence of pathologic 24-hour esophageal reflux in both studies was significantly high
30 ility that the high prevalence of pathologic esophageal reflux might simply have resulted from calcul
31  this correlates with reflux on conventional esophageal reflux monitoring studies is unknown.
32 correlate reflux during HREMI with reflux on esophageal reflux monitoring studies.
33 ccurring 1 or more times per day and/or high-esophageal reflux occurring 5 or more times per day.
34                                              Esophageal reflux of an admixture of gastric acid and du
35 flux episodes and the percentage of proximal esophageal reflux off-PPI did not change significantly a
36  to detect laryngopharyngeal reflux and high-esophageal reflux (reflux 2 cm distal to the upper esoph
37 fects, severe hypotonia, pathological gastro-esophageal reflux, retinal disease, and sinus-node dysfu
38 ist use appears to be partially mediated via esophageal reflux symptoms.
39  result in dysphagia, chest pain, and gastro-esophageal reflux symptoms.
40 may falsely attribute exertional dyspnea and esophageal reflux to asthma, leading to excess rescue me
41                                              Esophageal reflux was found only at the times of simulta