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1 nal hemorrhage, and five patients (6.0%) had esophageal stricture.
2 hy (CT) and endoscopy in predicting risks of esophageal stricture.
3 copic (Zargar) classifications in predicting esophageal stricture.
4 ts have manifestations of fibrosis and gross esophageal strictures.
5 t years behaviorally compensating for narrow esophageal strictures.
6 phagia, food impaction of the esophagus, and esophageal strictures.
7           Fifty-six (37%) patients developed esophageal strictures.
8 ce of gastroesophageal reflux disease (26%), esophageal stricture (39%), or both (15%) does not accou
9 ng problem that can be complicated by peptic esophageal stricture and adenocarcinoma of the esophagus
10 de of esophagitis, frequency of treatment of esophageal stricture and subsequent antireflux operation
11 acterized by tissue remodeling that leads to esophageal strictures and food impactions.
12 iated with an increased risk of esophagitis, esophageal strictures, Barrett esophagus, and esophageal
13 tion is a risk factor for the development of esophageal strictures, Barrett esophagus, and esophageal
14                            The prevalence of esophageal strictures correlates with the duration of un
15                                              Esophageal stricture (ES) and gastric outlet obstruction
16 gency CT outperforms endoscopy in predicting esophageal stricture formation after caustic ingestion.
17  bleeding using EL (42% vs. 0%; P =.027) and esophageal stricture formation in the ES-treated patient
18                  Long term sequelae included esophageal stricture formation requiring dilatation, per
19                                  The risk of esophageal stricture formation was 0%, 17%, and 83%, for
20 senting symptom is dysphagia with associated esophageal stricture formation.
21 s were uncommon: 1 (2%) patient developed an esophageal stricture (grade 2) and 1 (2%) grade 4 esopha
22 al Classification of Diseases code 530.1) or esophageal stricture (ICD code 530.3) who were discharge
23 our ability to study the long-term course of esophageal strictures in patients with EoE is hampered b
24                 Similarly, the prevalence of esophageal strictures increased with duration of diagnos
25                                              Esophageal strictures more commonly occurred in L-EoE (P
26           Late toxicity consisted of grade 2 esophageal stricture (n = 3), bronchial stenosis (n = 2)
27                  We noted high grade, benign esophageal strictures (n = 8). All 8 strictures resemble
28                             Late toxicity of esophageal stricture occurred in five patients.
29 ngs suggest that most patients with IEE have esophageal strictures, often with distinctive ringlike i
30 ients; vitamin B12 deficiency, two patients; esophageal stricture, one patient; urinary tract problem
31 OR 0.411, 95%-CI 0.203-0.835, P = 0.014) and esophageal stricture (OR 2.666, 95%-CI 1.259-5.645, P =
32  (OR 1.150, 95%-CI 0.4668-2.835, P = 0.761), esophageal stricture (OR 2.832, 95%-CI 1.508-5.321, P =
33                            Refractory benign esophageal strictures (RBESs) have been treated with the
34 e underwent stent placement, three underwent esophageal stricture resection, and four underwent esoph
35                      Erosive esophagitis and esophageal stricture were associated with sinusitis (odd
36 had undergone APC treatment developed a late esophageal stricture, which required endoscopic dilation
37                                   History of esophageal stricture who have recurrent symptoms of dysp