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1 ibility of gene therapy for the treatment of esophageal ulcers.
2 ion is important for healing of experimental esophageal ulcers.
3  one patient had moderate hemorrhage from an esophageal ulcer 2 weeks after surgery.
4 sophageal lesions including fistula and deep esophageal ulcers and abscesses.
5               The patient developed multiple esophageal ulcers and hemorrhage.
6 e associated with cytomegalovirus-associated esophageal ulcers and probably contribute to the inflamm
7 erosive esophagitis, Barrett's esophagus, or esophageal ulcer), and pH impedance testing on PPI thera
8 c biopsy of their cytomegalovirus-associated esophageal ulcers before and after ganciclovir therapy.
9 eatment with celecoxib significantly delayed esophageal ulcer healing and suppressed ulceration-trigg
10 the COX-2 selective inhibitor, celecoxib, on esophageal ulcer healing as well as on the cellular and
11 hese findings indicate that celecoxib delays esophageal ulcer healing by reducing ulceration-induced
12 chanism(s) regulating VEGF expression during esophageal ulcer healing remain unknown.
13                          Whether they affect esophageal ulcer healing remains unexplored.
14 n essential role of VEGF and angiogenesis in esophageal ulcer healing, and 3) demonstrate the feasibi
15 termine the efficacy of VEGF gene therapy in esophageal ulcer healing, we studied whether a single lo
16 cantly enhanced angiogenesis and accelerated esophageal ulcer healing.
17 issue destruction associated with idiopathic esophageal ulcers (IEUs) poses a diagnostic and therapeu
18     A solitary report of an idiopathic giant esophageal ulcer in an immunocompetent patient exists.
19 the endoscopic finding of a longitudinal mid-esophageal ulcer in the presence of proximal stricture m
20 ities in presentation and treatment to giant esophageal ulcers in human immunodeficiency virus infect
21                             Idiopathic giant esophageal ulcers in immunocompromised patients have bee
22 ding one esophago-pulmonary fistula and deep esophageal ulcers in the other animals.
23 ment, and postablation endoscopy documenting esophageal ulcer may identify patients at higher risk fo
24 ith a history of severe erosive esophagitis, esophageal ulcer, or peptic stricture, should generally
25                   One out of 10 postablation esophageal ulcers progressed to perforation, and no pati
26                                              Esophageal ulcer seems to precede AEF development, and p
27                                              Esophageal ulcers were also more common in oral opium us
28                                              Esophageal ulcers were induced in rats by focal applicat
29                                              Esophageal ulcers were noted in 3 evaluable subjects in
30                                    Rats with esophageal ulcers were treated intragastrically with eit