ライフサイエンスコーパス: esophagi

1 ragastric zinc compared with zinc-sufficient esophagi.

2 ning in ZD esophagi compared with that in ZS esophagi.

3 were detected in the suprabasal layers of ZD esophagi.

4 y resulting in aneurysmal aortas and ectatic esophagi.

5 mens, human esophageal cell lines, and mouse esophagi.

6 enes in the PEITC + NMBA versus NMBA-treated esophagi.

7 -deficient esophagus and in zinc-replenished esophagi after treatment with intragastric zinc compared

8 expressed in the NMBA-treated versus control esophagi and 1,936 genes in the PEITC + NMBA versus NMBA

9 cultured esophageal fibroblasts from normal esophagi and esophagi from patients with severe EoE on a

10 y that many laparoscopic patients with short esophagi are unrecognized and perhaps treated inappropri

11 d levels of DNA damage in reflux-exposed rat esophagi, as observed by reduced levels of phospho-H2A h

12 tly, tumors were consistently observed in ZD esophagi at very early time points.

13 hosphorylated forms of Rb was detected in ZD esophagi by immunoblotting.

14 ng cell nuclear antigen was characterized in esophagi by immunohistochemistry at 0, 24, and 48 h, and

15 6ink4a showed reduced nuclear staining in ZD esophagi compared with that in ZS esophagi.

16 cial and transient infections of tongues and esophagi (detected by histology) at 1 to 2 weeks after o

17 phageal fibroblasts from normal esophagi and esophagi from patients with severe EoE on autologous ver

18 ptosis accelerator, was markedly stronger in esophagi from Zn-/DFMO+ animals that showed increased ap

19 Esophagi were divided into 4 groups (4 esophagi/group): control, Group1 (G1), Group2 (G2), Grou

20 PC) was applied circumferentially in porcine esophagi in vivo.

21 Here we show that atretic esophagi lack Noggin (NOG) expression, resulting in imma

22 n vitro uniaxial stretching was performed on esophagi (n = 16) obtained from the abattoir within 4-6

23 Fifty one RNAs, comprising 24 normal esophagi (NE), 18 BEs, and nine EACs were hybridized to

24 ell nuclear antigen (PCNA) expression in the esophagi of NMBA treated cyclin D1 mice.

25 y identified 2,261 dysregulated genes in the esophagi of rats that received a 1-week exposure to the

26 n, morphometric changes were observed in the esophagi of rats treated with N-nitrosomethylbenzylamine

27 Lesions developed on the tongues and esophagi of the 4NQO-treated animals and included hyperk

28 udy evaluating the morphological features of esophagi resected for endstage achalasia showed marked d

29 this, we utilized full thickness human donor esophagi to create and validate the ex vivo function of

30 was 88% in ZD rats with highly proliferative esophagi versus 0% in ZS rats.

31 Esophagi were assessed by gross morphology, histopatholo

32 h keratinized and nonkeratinized surfaces of esophagi were colonized to a considerably greater extent

33 Esophagi were divided into 4 groups (4 esophagi/group):

34 re sacrificed 24 h after the last treatment; esophagi were excised and processed for histologic gradi

35 S) as well as the expression of c-Jun in the esophagi, were evaluated to investigate the mechanism(s)