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1 henol A (BPA) and the pharmaceutical 17alpha-ethinyl estradiol (EE) are synthetic chemicals with estr
2 ted the effectiveness of oral treatment with ethinyl estradiol (EE) on EAE and the mechanisms involve
3             Male deer mice exposed to BPA or ethinyl estradiol (EE) through maternal diet showed no c
4                                      17alpha-ethinyl estradiol (EE), a synthetic analog of 17beta-est
5  from livers of bile duct ligation (BDL)- or ethinyl estradiol (EE)-injected rats, were viewed and re
6 spectively); and diet 6 contained 0.1 mug of ethinyl estradiol (EE)/kg food.
7 amine the bioavailability and bioactivity of ethinyl estradiol (EE2) sorbed onto SWCNTs in a fish gas
8 xposed to BPA, vehicle, or positive control [ethinyl estradiol (EE2)] by oral gavage beginning on ges
9 om 74 participants receiving an etonogestrel/ethinyl estradiol (ENG/EE) intravaginal ring while on no
10                                 The doses of ethinyl estradiol (or placebo) were adjusted for chronol
11  subcutaneous injections of vehicle or 10 ug ethinyl estradiol + 20 ug levonorgestrel (HCs) throughou
12           Other estrogenic compounds such as ethinyl estradiol and estrone did not have any effect on
13  a modest synergy between childhood low-dose ethinyl estradiol and growth hormone.
14                                              Ethinyl estradiol at a dose of 1 microgram/kg per day in
15 o 1.7 with oral contraceptives that included ethinyl estradiol at a dose of 20 mug and by a factor of
16                                         With ethinyl estradiol at a dose of 20 mug, the corresponding
17 ent use of oral contraceptives that included ethinyl estradiol at a dose of 30 to 40 mug was associat
18 actor of 1.3 to 2.3 with those that included ethinyl estradiol at a dose of 30 to 40 mug, with relati
19 eceive either oral contraceptives (triphasic ethinyl estradiol at a dose of 35 microg plus norethindr
20  oral contraceptives (COC) or etonorgesterol/ethinyl estradiol combined contraceptive vaginal ring (C
21    Mice treated with 17beta-estradiol or 17a-ethinyl estradiol had a total uterine tumor incidence of
22                                              Ethinyl estradiol is a semisynthetic estrogen compound f
23              These results suggest that oral ethinyl estradiol might be a successful candidate as the
24 droxyestradiol, 17beta-estradiol, or 17alpha-ethinyl estradiol on days 1-5 of neonatal life (2 microg
25                                   Removal of ethinyl estradiol was >40% for two of the three sealant
26                                   Removal of ethinyl estradiol was >=90% for all sealants in the incr
27                                              Ethinyl estradiol was also detected in PC-SPES lots.
28 nicity of bisphenol A, triclosan and 17alpha-ethinyl estradiol without generating obviously toxic byp
29 ts of OCPs (0.15 mg levonorgestrel + 0.30 ug ethinyl estradiol) vs placebo (N = 26) on MRI measures o
30 and better tolerated than the Yuzpe regimen (ethinyl estradiol, 100 microg, and levonorgestrel, 0.5 m
31 glucuronidation activity toward lamotrigine, ethinyl estradiol, chenodeoxycholic acid, and lithocholi
32 with maximally efficacious doses of 17-alpha ethinyl estradiol, the benzothiophene SERM, raloxifene,
33    However, the synthetic estrogens, DES and ethinyl estradiol, were detected in various lots of PC-S
34 reatment groups received escalating doses of ethinyl estradiol.
35 treated male transgenic rabbits with 17alpha-ethinyl estradiol.
36  1:1:1 to 24 weeks of low-dose COCPs (20 mug ethinyl estradiol/0.15 mg desogestrol), metforminXR (2,0
37 nt at urogenital infection sites, as well as ethinyl estrogen, a component of some hormonal contracep
38 er feeding pregnant rats a high-fat (HF)- or ethinyl-oestradiol (EE2)-supplemented diet affects carci