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1 uce S-adenosylethionine (SAE) from substrate ethionine.
2 ed based on their resistance to selection by ethionine.
3 ne to generate the unconventional amino acid ethionine, a known immunomodulator.
4  reuteri 6475 gives rise to immunomodulatory ethionine, a source for histone ethylation.
5 GS-overexpressing seedlings are resistant to ethionine, a toxic Met analog.
6 in a manner similar to SAMe, confirming that ethionine also uses the same catalytic site to form the
7                                              Ethionine, an S-ethyl analog of the amino acid methionin
8                               Treatment with ethionine and seleno-D,L-ethionine, two inhibitors known
9  choline-deficient diet supplemented with DL-ethionine) and control mice.
10  rats exposed to a cyclic choline deficiency-ethionine (CDE) diet (2 weeks on, 1 week off) supports t
11  undescribed co-factor, carboxy-S-adenosyl-l-ethionine (cxSAE), thereby enabling the stereoselective
12 fed a choline-deficient diet containing 0.1% ethionine for 2 and 6 weeks, respectively.
13 holine-deficient diet supplemented with 0.5% ethionine for 24 hrs and then challenging the animals wi
14  of THP-1 histones revealed incorporation of ethionine instead of methionine into proteins, a reducti
15 at incorporating the more hydrophobic analog ethionine (instead of methionine) into nisin improves it
16 sing mice fed methionine-choline-deficient + ethionine (MCDE) diets with or without cyclopamine.
17 )), choline-deficient diet supplemented with ethionine, or 3,5-diethoxycarbonyl-1,4-dihydrocollidine
18 ent synthesis of the SAM analog S-adenosyl-l-ethionine (SAE) and show SAE is a mechanistically-equiva
19 ent synthesis of the SAM analog S-adenosyl-l-ethionine (SAE) and show SAE is a mechanistically-equiva
20           The results of experiments using L-ethionine suggest that SAM is involved in post-synthetic
21 yzed livers of rats fed a choline-deficient, ethionine-supplemented (CDE) diet for phospho-Smad2.
22 regeneration using the choline-deficient and ethionine-supplemented (CDE) diet model.
23 hronic liver disease: the choline-deficient, ethionine-supplemented (CDE) diet versus thioacetamide (
24 oung female mice fed a choline-deficient and ethionine-supplemented (CDE) diet.
25 e mice by placing them on choline-deficient, ethionine-supplemented (CDE) diets for 15 days; mice the
26 -collidin (DDC)-diet and a choline-deficient ethionine-supplemented (CDE)-diet.
27 s isolated from rats fed a choline-deficient ethionine-supplemented diet (CDE diet, a regimen commonl
28 7/ bI/6J mice by feeding a choline-deficient/ethionine-supplemented diet (n = 95).
29 rimental models of AP (ie, choline-deficient-ethionine-supplemented diet and cerulein injections), ST
30    AP was induced by a choline-deficient and ethionine-supplemented diet for 4 days in normal C57BL/6
31 tes, respectively, in the choline-deficient, ethionine-supplemented diet model of liver injury and re
32 ung female mice were fed a choline-deficient/ethionine-supplemented diet to induce AP and were treate
33 idine (DDC)-containing or choline-deficient, ethionine-supplemented diet.
34 gue cerulein or feeding a choline-deficient, ethionine-supplemented diet.
35 e by 10 days of feeding a choline-deficient, ethionine-supplemented diet.
36  oval cell activation, the choline deficient ethionine-supplemented dietary regime.
37                        The choline-deficient ethionine-supplemented mouse model of hepatocellular inj
38                            Choline-deficient ethionine-supplemented-induced DR expanded as biliary ep
39   When we treated monocytic THP-1 cells with ethionine, their transcription of TNF-alpha was inhibite
40 t that the microbiome can expose the host to ethionine through a novel 2-carbon transporting variant
41      Treatment with ethionine and seleno-D,L-ethionine, two inhibitors known to have I50 values 50 to