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1 o three intravascular fluid protocol groups: euvolemic (3 mLkg/hour isotonic crystalloid), hypervolem
2 Drug Administration for use in patients with euvolemic and hypervolemic hyponatremia.
3 nd 34 (9.1%) were classified as hypovolemic, euvolemic, and hypervolemic at Time zero .
4  or on minimal exertion, who were clinically euvolemic, and who had an ejection fraction of less than
5 sfusion of stored leukodepleted red cells to euvolemic, anemic, critically ill patients has no clinic
6 F activity, essentially at normal levels for euvolemic animals, persists during AT1 receptor blockade
7 y edema during the study were hypovolemic or euvolemic at the time pulmonary edema developed.
8  overall functional status, in particular in euvolemic cases.
9 ifically drives sodium appetite, even during euvolemic conditions.
10 e in modulation of renal hemodynamics in the euvolemic diabetic rat, in the absence of KKS stimulatio
11 5-h oral-glucose-tolerance test (OGTT) and a euvolemic, euenergetic protein challenge.
12                                       In the euvolemic group, prolonged CO2 pneumoperitoneum caused d
13 uid volume status (hypovolemic hyponatremia, euvolemic hyponatremia, or hypervolemic hyponatremia).
14                             In patients with euvolemic or hypervolemic hyponatremia, tolvaptan, an or
15  of tolvaptan was evaluated in patients with euvolemic or hypervolemic hyponatremia.
16  according to whether they have hypovolemic, euvolemic, or hypervolemic hyponatremia.
17                                           In euvolemic patients with symptoms at rest or on minimal e
18                 CPDSR is extrapolated to non-euvolemic populations as a diuretic resistance mechanism
19                                           In euvolemic rats, the dipsogenic hormone angiotensin II, b
20                             These results in euvolemic septic animals suggest that total body Vo2 may
21 merular feedback (TGF) activity in 7-wk-old, euvolemic spontaneously hypertensive rats (SHR) and in W
22                 The pH dependence of KCC in "euvolemic" SS RBCs treated with urea was similar to that
23 d moderate (i.e., >0.5) for DeltaMMSE in the euvolemic subgroup.
24 rtan, whereas TGF was basically unchanged in euvolemic WKY.
25 y were unchanged by AT1 receptor blockade in euvolemic WKY.