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1 hronic treatment and intravenously for acute exacerbations of asthma).
2  evidence linking many indoor factors to the exacerbation of asthma.
3  specific indoor environmental exposures and exacerbation of asthma.
4 cientific literature on indoor exposures and exacerbation of asthma.
5 echanistic links between viral infection and exacerbation of asthma.
6 viduals, and implicated in the initiation or exacerbation of asthma.
7 , such as Alternaria, and development and/or exacerbation of asthma.
8 rnaria, is implicated in the development and exacerbation of asthma.
9 eased airway obstruction in allergen-induced exacerbation of asthma.
10 ctions are associated with the induction and exacerbation of asthma.
11 elop therapeutics to treat the virus-induced exacerbation of asthma.
12  is involved in both the onset and the acute exacerbations of asthma.
13 ral respiratory infections and virus-induced exacerbations of asthma.
14 use acute wheezing illnesses in children and exacerbations of asthma.
15 iral respiratory illnesses and virus-induced exacerbations of asthma.
16       Rhinoviruses cause the common cold and exacerbations of asthma.
17 on to increase the probability of RV-induced exacerbations of asthma.
18 ed to neutrophilic inflammation during acute exacerbations of asthma.
19 ns can contribute to airway inflammation and exacerbations of asthma.
20 logic, and pathologic outcomes in RV-induced exacerbations of asthma.
21 ental risk factor for both the inception and exacerbations of asthma.
22 osure to air pollution might reduce allergic exacerbations of asthma.
23 e dysregulation and subsequent induction and exacerbations of asthma.
24  antioxidant responses may contribute to the exacerbations of asthma.
25 cacy of telithromycin in patients with acute exacerbations of asthma.
26                  Rhinovirus infections cause exacerbations of asthma.
27 [LPS]) can develop airway symptomatology and exacerbations of asthma.
28 airway responsiveness in the development and exacerbations of asthma.
29 l) or studied within 48 h of an acute severe exacerbation of asthma (13 ng/ml) than in subjects with
30 ifelong risk factors for the development and exacerbation of asthma and allergic diseases.
31  Human rhinovirus (HRV) replication triggers exacerbation of asthma and causes most acute respiratory
32 ith angiotensin II were infrequent; however, exacerbation of asthma and congestive heart failure and
33 luding TNF-alpha and MCP-1) in viral-induced exacerbation of asthma and suggest examination of these
34 pathways, in context of viral infections and exacerbations of asthma and allergic diseases.
35 or the majority of common colds and triggers exacerbations of asthma and chronic obstructive lung dis
36 ral respiratory health effects, specifically exacerbations of asthma and chronic obstructive pulmonar
37 f the excessive inflammation observed during exacerbations of asthma and chronic obstructive pulmonar
38 gulated by rhinovirus infection during acute exacerbations of asthma and chronic obstructive pulmonar
39  respiratory tract illnesses and precipitate exacerbations of asthma and chronic obstructive pulmonar
40        P2X(7) pore function protects against exacerbations of asthma and loss of control, independent
41     Respiratory syncytial virus (RSV) causes exacerbations of asthma and preschool wheeze (PSW).
42           Air pollution contributes to acute exacerbations of asthma and the development of asthma in
43 are strongly associated with development and exacerbations of asthma, and they pose an additional hea
44 ovirus (RV) infections are closely linked to exacerbations of asthma, and yet most RV infections of p
45                    Episodes of virus-induced exacerbations of asthma are accompanied by increased eos
46                                              Exacerbations of asthma are thought to be caused by airf
47                                              Exacerbations of asthma are thought to be strongly depen
48 ections contribute to the causation of acute exacerbations of asthma, but that additional cofactors a
49 Viruses are frequently associated with acute exacerbations of asthma, but the extent to which they co
50 he pulmonary function of patients with acute exacerbations of asthma, but their effect on hospitaliza
51 ify pregnancy-related risk factors for acute exacerbations of asthma during pregnancy.
52  viral infections have been shown to trigger exacerbations of asthma; however, the mechanism by which
53 ia in 8 percent, croup in 18 percent, and an exacerbation of asthma in 14 percent.
54 n affected primary viral infection and viral exacerbation of asthma in experimental models.
55 RV) are the viruses most often implicated in exacerbations of asthma in both children and adults, lit
56 ow recognized as an important cause of acute exacerbations of asthma in school-aged children.
57 omolyn confer significant protection against exacerbations of asthma leading to hospitalization.
58 recognized as critical in the initiation and exacerbation of asthma, one of the most common chronic c
59 rlapping risk factors and symptoms, an acute exacerbation of asthma or an episode of acute chest synd
60    Such a phenomenon does not translate into exacerbation of asthma or mast cell-dependent arthritis.
61 ia, tracheobronchitis, bronchiolitis, croup, exacerbations of asthma or chronic obstructive pulmonary
62 Hazard ratios for hospital admissions due to exacerbations of asthma or COPD were 39.48 (95% CI 25.93
63 as a decrease in the number of patients with exacerbations of asthma (p = 0.01; 95% CI, 4.4 to 52.7)
64 agents, merit increased attention to prevent exacerbation of asthma, possibly even in nonsensitized i
65 aused by A fumigatus and is characterized by exacerbations of asthma, recurrent transient chest radio
66 gE, particularly to dust mite, correlates to exacerbations of asthma related to rhinovirus infection.
67 although their contribution to the onset and exacerbation of asthma remains poorly understood.
68 were enrolled within 24 hours after an acute exacerbation of asthma requiring short-term medical care
69 innate type 2 immunity and contribute to the exacerbation of asthma responses.
70 ieved to increase the risk of having asthma, exacerbation of asthma symptoms, or both.
71 ded basis resulted in a lower risk of severe exacerbation of asthma than as-needed use of a short-act
72 t state of knowledge, it is wise to view all exacerbations of asthma that last longer than a few days
73                 Among children with a severe exacerbation of asthma, the addition of ipratropium brom
74 efit of telithromycin in patients with acute exacerbations of asthma; the mechanisms of benefit remai
75 ight, around 4:00 AM, uncovering a nocturnal exacerbation of asthma usually unnoticed or hidden by th
76 drin might also play a role in virus-induced exacerbations of asthma, we measured pendrin mRNA expres
77 ed from 2000 to 2013 on indoor exposures and exacerbation of asthma were identified through PubMed, f
78 ures in relation to frequency or severity of exacerbation of asthma were selected for review.
79          Allergen-sensitized mice undergoing exacerbations of asthma were protected from lung inflamm
80      The primary outcome measure was rate of exacerbations of asthma, with criteria based on data fro
81              The main outcome measure was an exacerbation of asthma within 72 h of injection (defined
82  Viral respiratory infections trigger severe exacerbations of asthma, worsen disease symptoms, and im