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1 uate sleep quantity, poor sleep quality, and excessive daytime sleepiness.
2  exhibit insomnia, delayed sleep habits, and excessive daytime sleepiness.
3 ) and without (n=144) complaints of frequent excessive daytime sleepiness.
4 ty to predict important consequences such as excessive daytime sleepiness.
5 rousals, could prove useful in prediction of excessive daytime sleepiness.
6 sturbance may reduce sleep fragmentation and excessive daytime sleepiness.
7  during sleep are a well recognized cause of excessive daytime sleepiness.
8  and cortical arousal, often associated with excessive daytime sleepiness.
9 atients showed high risk for OSAS, 46.3% had excessive daytime sleepiness, 41.5% were positive for bo
10 d suspicion of sleep-disordered breathing or excessive daytime sleepiness, a formal sleep assessment
11                                              Excessive daytime sleepiness affected 32% of the patient
12 chronic neurologic disorder characterized by excessive daytime sleepiness and abnormal manifestations
13 eep disorder marked by chronic, debilitating excessive daytime sleepiness and can be associated with
14 ored on diagnostic features of the disorder (excessive daytime sleepiness and cataplexy), effects on
15 ye movement (REM) sleep, is characterized by excessive daytime sleepiness and cataplexy, a loss of mu
16  Narcolepsy type 1 (NT1) is characterized by excessive daytime sleepiness and cataplexy, accompanied
17 sy, a debilitating disorder characterized by excessive daytime sleepiness and cataplexy.
18 sorder narcolepsy, which is characterized by excessive daytime sleepiness and cataplexy.
19 l of a human sleep disorder characterized by excessive daytime sleepiness and cataplexy.
20                    Therapeutic NCPAP reduces excessive daytime sleepiness and improves self-reported
21 0 x 10(-13)) and between increased levels of excessive daytime sleepiness and increased measures for
22 n SWS, reproducing, respectively, narcolepsy excessive daytime sleepiness and poor sleep quality.
23 r risk benefits, and substantially improving excessive daytime sleepiness and quality of life.
24 way resistance syndrome (UARS) is defined by excessive daytime sleepiness and tiredness, and is assoc
25 falling asleep, 1.47 (95% CI, 1.39-1.56) for excessive daytime sleepiness, and 1.44 (95% CI, 1.36-1.5
26          We evaluated sleep characteristics, excessive daytime sleepiness, and chronotype using the P
27 e, which summarized abnormal sleep duration, excessive daytime sleepiness, and insomnia complaints ([
28 fort to breathe, and cough or loud snoring), excessive daytime sleepiness, and reduced vigor during t
29 vements during sleep are not associated with excessive daytime sleepiness, and therefore appear unlik
30 nts experience increased nocturnal activity, excessive daytime sleepiness, and weight loss.
31 investigated (1) the prevalence of insomnia, excessive daytime sleepiness, anxiety and depression amo
32  central nervous system disorders, including excessive daytime sleepiness, attention deficit hyperact
33                                              Excessive daytime sleepiness can be evaluated with both
34 arcolepsy, a sleep disorder characterized by excessive daytime sleepiness, cataplexy, and other patho
35  is a neurological disorder characterized by excessive daytime sleepiness, cataplexy, hypnagonic hall
36 nd impair quality of life, include insomnia, excessive daytime sleepiness, circadian disorders, obstr
37                             The complaint of excessive daytime sleepiness, commonly encountered in ne
38                            The prevalence of excessive daytime sleepiness, defined as an ESS score >/
39                                              Excessive daytime sleepiness (EDS) affects 10-20% of the
40 erred for polysomnography with complaints of excessive daytime sleepiness (EDS) and clinically suspec
41 e, psychostimulants (e.g., modafinil) reduce excessive daytime sleepiness (EDS) and sodium oxybate (g
42 is was tested by exploring the links between excessive daytime sleepiness (EDS) and vulnerability to
43 lness for the treatment of disorders such as excessive daytime sleepiness (EDS) as well as other slee
44 o determine the safety and efficacy of LT on excessive daytime sleepiness (EDS) associated with PD.
45                          We assessed whether excessive daytime sleepiness (EDS) at baseline was assoc
46                                              Excessive daytime sleepiness (EDS) can be caused by insu
47                                              Excessive daytime sleepiness (EDS) is a public health is
48                                              Excessive daytime sleepiness (EDS) is common and disabli
49  obesity and snoring and between obesity and excessive daytime sleepiness (EDS), although for the mos
50 der of the sleep-wake cycle characterized by excessive daytime sleepiness (EDS), cataplexy, nighttime
51  excessive daytime sleepiness, patients with excessive daytime sleepiness (Epworth Sleepiness Scale s
52 asured stress (Perceived Stress Scale; PSS), excessive daytime sleepiness (Epworth Sleepiness Scale;
53 ple, 26% were habitual snorers, 18% reported excessive daytime sleepiness (ESS > or = 11), and 29% we
54 leep disorder narcolepsy is characterized by excessive daytime sleepiness, fragmentation of nighttime
55                                              Excessive daytime sleepiness has emerged as one of the m
56                                              Excessive daytime sleepiness in CD patients may be drive
57 here is a distinct differential diagnosis of excessive daytime sleepiness in older adults.
58 al study assessing multiple risk factors for excessive daytime sleepiness in older subjects (mean age
59  servoventilation produces an improvement in excessive daytime sleepiness in patients with Cheyne-Sto
60  sleep fragmentation (SF) appear to underlie excessive daytime sleepiness in patients with sleep apne
61                              The etiology of excessive daytime sleepiness in patients with sleep-diso
62 ported sleep duration, insomnia symptoms and excessive daytime sleepiness in the UK Biobank (n = 112,
63 the most common medical disorder that causes excessive daytime sleepiness, increasing the risk for dr
64 ular, eczema associated with atopy, fatigue, excessive daytime sleepiness, insomnia, and only 0 to 3
65 at often are seen by neurologists, including excessive daytime sleepiness, insomnia, narcolepsy, rapi
66                                   Rationale: Excessive daytime sleepiness is a common disabling sympt
67                                              Excessive daytime sleepiness is typically the most frequ
68                ESS score > 10, indicative of excessive daytime sleepiness, is an independent predicto
69 L1 and TGFBI in females and WDR27 in males), excessive daytime sleepiness (near AR-OPHN1) and a compo
70 88, 95% confidence interval (CI) 1.18-3.00), excessive daytime sleepiness (OR = 1.49, 95% CI 1.04-2.1
71 95% CI, 1.11-2.10) (P = .009, I2 = 74%), and excessive daytime sleepiness (OR, 2.27; 95% CI, 1.54-3.3
72 ion (adjusted OR, 2.65; P=0.023), those with excessive daytime sleepiness (OR, 2.51; P=0.037), and th
73 95% CI, 1.14-1.88) (P = .003, I2 = 76%), and excessive daytime sleepiness (OR, 2.72; 95% CI, 1.32-5.6
74 porter availability and fatigue, depression, excessive daytime sleepiness, or rapid eye movement slee
75            Compared with PD patients without excessive daytime sleepiness, patients with excessive da
76                               Test accuracy, excessive daytime sleepiness, sleep-related and general
77 uate sleep quantity, poor sleep quality, and excessive daytime sleepiness, studied according to an a
78                     Active treatment reduced excessive daytime sleepiness; the mean Osler change was
79 patic encephalopathy might be unable, due to excessive daytime sleepiness, to accumulate the need/abi
80 a [OSA], short and long sleep durations, and excessive daytime sleepiness) utilizing the Million Vete
81 rsomnolence disorder mainly characterised by excessive daytime sleepiness, with prolonged night-time