ライフサイエンスコーパス: exchanger

1 eres (containing solvatochromic dyes and ion exchanger).

2 ly host the mitochondrial NCLX (Na(+)/Ca(2+) exchanger).

3 ate the AtSOS1 plasma membrane sodium/proton exchanger.

4 sodium-driven reversal of the sodium/calcium exchanger.

5 selective activation of the sodium/hydrogen exchanger.

6 simulations, of both a cotransporter and an exchanger.

7 and by augmented levels of the Na(+)/Ca(2+) exchanger.

8 (Sc) sites in CLC-ec1, the Escherichia coli exchanger.

9 t that involved plasma membrane Na(+)/Ca(2+) exchanger.

10 sed by a loss of function of this Na(+)/H(+) exchanger.

11 l as the surface delivery of the AE1-4 anion exchanger.

12 rate that OAT7 is a very weak urate-butyrate exchanger.

13 k1, l-type calcium channel, and Na(+)/Ca(2+) exchanger.

14 iculum calcium ATPase 2 and the Na(+)/Ca(2+) exchanger.

15 the major intestinal brush-border Na(+)/H(+) exchanger.

16 ven in the absence of the Na(+)/Ca(2+), K(+) exchanger.

17 tory effect of the CAs on an endogenous Na-H exchanger.

18 te the retention of the isotopes on PCST ion exchanger.

19 se findings have implications also for other exchangers.

20 port and voltage-dependent gating in the CLC exchangers.

21 systems, including optimization of the heat exchangers.

22 ities (>5 W/m(2)) and with finite-sized heat exchangers.

23 carrier family 26 member A6 (SLC26A6) anion exchangers.

24 ion of PaNhaP and other microbial Na(+)/H(+) exchangers.

25 ters, which contains both cotransporters and exchangers.

26 actants, castor oil as plasticizers, and ion exchangers.

27 Anion exchanger 1 (AE1) is responsible for the exchange of bic

28 Anion exchanger 1 (AE1) mediates Cl(-)/HCO3(-) exchange in ery

29 Anion exchanger 1 (AE1), also known as band 3 or SLC4A1, plays

30 on inside red blood cells, and band 3 [anion exchanger 1 (AE1)] provides a passage for HCO3(-) flux a

31 The sodium (Na(+))-calcium (Ca(2+)) exchanger 1 (NCX1) is an important regulator of intracel

32 The Na(+)-Ca(2+) exchanger 1 (NCX1) is reduced in stroke by the RE1-silen

33 We demonstrated earlier that Na(+)/Ca(2+) exchanger 1 (NCX1), a major calcium exporter in renal ep

34 Exogenous Na(+) /H(+) exchanger 1 (NHE1) expression stimulated the collective

35 Here, we identify how the Na(+)/H(+)-exchanger 1 (NHE1), a key regulator of cellular pH homeo

36 The Na(+) /H(+) exchanger 1 (NHE1, SLC9A1) is an important regulator of

37 (3,4,5)-trisphosphate (PIP(3))-dependent Rac exchanger 1 (P-Rex1) catalyzes the exchange of GDP for G

38 (3,4,5) trisphosphate (PIP(3))-dependent Rac exchanger 1 (P-Rex1) is a Rho guanine-nucleotide exchang

39 inositol (3,4,5) trisphosphate-dependent Rac exchanger 1 (P-Rex1) pleckstrin homology domain has effe

40 PtdIns(3,4,5)P3-dependent Rac exchanger 1 (PREX1) is a Rac-guanine nucleotide exchange

41 Likewise, the tomato CATION EXCHANGER 1 and TWO-PORE CHANNEL 1 (SlTPC1), key genes f

42 1-induced phosphorylation of NHE1 (Na(+)/H(+)exchanger 1), which is involved in the regulation of int

43 and impaired activity of the sodium/hydrogen exchanger 1, a known regulator of skin pH.

44 Na(+)/Ca(2+) exchanger-1 (NCX1) is a major calcium extrusion mechanis

45 ession of the acid-loading transporter anion exchanger 2 (AE2) (SLC4A2 product) was detected in myofi

46 Anion exchanger 2 (AE2), the principal bicarbonate secretor in

47 cell lines, we report here that Na(+)/Ca(2+) exchanger 2 (NCX2) works in conjunction with transient r

48 Inhibition of sodium hydrogen exchanger 2 (NHE2) caused significant delay but did not

49 tol 3,4,5-trisphosphate (PIP3)-dependent Rac exchanger 2 (PREX2) is a guanine nucleotide exchange fac

50 Enzyme 4a) > Enox2 (Ecto-NOX Disulfide-Thiol Exchanger 2) > Ube2d2 (Ubiquitin-conjugating enzyme E2D

51 Sodium hydrogen exchanger 2, although essential for repair, acts downstr

52 ansmembrane conductance regulator, and anion exchanger 2.

53 basolateral Cl(-) /HCO3(-) exchanger (anion exchanger 2; AE2).

54 Na(+)/H(+) exchanger 3 (NHE3) is present in pkd1-knockout and norma

55 nhibitor of gastrointestinal sodium/hydrogen exchanger 3 (NHE3), reduces paracellular phosphate trans

56 Na(+)/H(+) exchanger 3 activity was similar in undifferentiated and

57 e and cortical expression of sodium/hydrogen exchanger 3 and attenuated the increased expression of u

58 ulator and partially dependent on Na(+)/H(+) exchanger 3 and Na(+)/K(+)/2Cl(-) cotransporter 1 inhibi

59 diffuse subapical levels of sodium hydrogen exchanger 3 and SGLT1, which regulate transport of sodiu

60 a-hydroxylase, and the intestinal Na(+)/H(+) exchanger 3, agonists of components of the angiotensin-c

61 to other transporters, including Na(+)/H(+) exchanger 3, and to signaling pathways, such as the A-ki

62 inhibitor of the intestinal sodium-hydrogen exchanger 3, is being evaluated in clinical trials for i

63 mbranes, internalizes/inactivates Na(+)-H(+)-exchanger-3 and Na(+)/K(+)ATPase, prevents Na(+) retenti

64 ated major RPT Na(+) transporters Na(+)-H(+)-exchanger-3 and Na(+)/K(+)ATPase.

65 ation/inactivation of NHE-3 (sodium-hydrogen exchanger-3) and Na(+)/K(+)ATPase (sodium-potassium-atpa

66 primarily by the K(+)-dependent Na(+)/Ca(2+) exchanger 4 (NCKX4), and the removal of Ca(2+) leads to

67 amily 9 member A6 (SLC9A6)/(Na(+),K(+))/H(+) exchanger 6 (NHE6) gene that cause Christianson syndrome

68 The sodium-hydrogen exchanger 6 (NHE6), a protein mainly expressed in early

69 al calcium uniporter and extrusion by cation exchangers across the inner mitochondrial membrane may d

70 ecretion is removed by the basolateral anion-exchanger AE1.

71 HCO(3) (-) efflux via the basolateral anion exchanger AE2; and (3) inhibits NaCl reabsorption by med

72 ABSTRACT: The anion exchanger AE3, expressed in hippocampal (HC) neurons but

73 The anion exchanger AE3, expressed in hippocampal (HC) neurons but

74 laries were coated with 65-nm-diameter anion exchanger (AEX) latex nanoparticles that attach electros

75 s and Ca extrusion due to the sodium-calcium exchanger after an altered spatial relationship between

76 rbed layer of quaternary ammonium type anion exchanger and a phenolic azo type proton chromoionophore

77 orter genes, including vacuole cation/proton exchanger and inositol transporter, were considered to p

78 porate an ionic solvatochromic dye (SD), ion exchanger and ionophore.

79 ivation of countertransporters (Na(+)/Ca(2+) exchanger and Na(+)/H(+) exchanger) coupled to the proto

80 y (Miptode 3), the optode containing the ion exchanger and the MIP particles (Miptode 2) showed impro

81 primarily by ion channels, pumps (ATPases), exchangers and Ca(2+)-binding proteins.

82 The CLC family comprises H(+)-coupled exchangers and Cl(-) channels, and mutations causing the

83 ally homologous transporters including anion exchangers and prestin.

84 rial-natriuretic-peptide and inhibiting Na-H exchangers and sympathetic tone.

85 Dinonylnaphthalene sulfonic acid (a cation exchanger) and chromoionophore I (a lipophilic optical p

86 he terminals, activated vesicular Na(+)/H(+) exchanger, and regulated glutamate loading as a function

87 polymerization, derivatized as strong anion exchangers, and used for lipoproteins enrichment.

88 e activity of the basolateral Cl(-) /HCO3(-) exchanger (anion exchanger 2; AE2).

89 The aspartate-glutamate exchanger Aralar/AGC1 (Slc25a12), a component of the mal

90 Although NCX Na(+)/Ca(2+) exchangers are classically believed to operate only at t

91 antial activity in conditions when wild-type exchangers are inactive) and do not promote cargo-depend

92 A suggest that both V-ATPases and Na(+)/H(+) exchangers are required for glycosomal pH regulation.

93 Our results indicate that Na(+) -H(+) exchangers are the principal source of synaptic cleft pr

94 her L-type calcium current or sodium-calcium exchanger as predominant phenotypes for VT/VF.

95 we identify NHE9, an endosomal cation/proton exchanger, as a novel regulator of this system.

96 ve the heat carrying liquid through the heat exchanger at desired flow rates and temperatures.

97 separation is performed over a strong anion exchanger at pH 7.5.

98 wed that Dube3a regulation of the Na(+)/K(+) exchanger ATPalpha (adenosine triphosphatase alpha) in g

99 zes an ectodomain of human erythrocyte anion-exchanger, band 3/AE1, as a host receptor.

100 continuously operating microfluidic solvent exchanger based on the principle of free-flow electropho

101 K(+)-dependent Na(+)/Ca(2+) exchangers belong to the solute carrier 24 (SLC24A1-5) g

102 , which was recently identified as a PI4P-PS exchanger between the ER and PM, showed PI4P-dependent m

103 e suppressor consists of a solid polymer ion exchanger block.

104 odulation and was abolished by Na(+) /Ca(2+) exchanger blockade.

105 Na(+) /Ca(2+) exchanger, but not TRP channels, can also facilitate STO

106 Inhibition of mitochondrial Na/Ca exchanger by CGP37157 ameliorates the metabolic changes.

107 stimulate non- HCO3- transporters (e.g. Na-H exchangers) by accelerating CO(2) / HCO3- -mediated buff

108 and required Src family kinases and the Rap1 exchanger C3G.

109 nal Na(+) and in mutants of the Na(+)/Ca(2+) exchanger, CalX, which we immuno-localized to ER membran

110 pounds, traditionally considered to be anion exchangers, can also be considered cation exchangers, wh

111 O3 is that starch may act as a weak acid ion exchanger capable of exchanging alcoholic group protons

112 the generator eluate is trapped on a cation exchanger cartridge (100 mg, approximately 8 mm long and

113 Here, we identify a vertebrate Ca(2+)/H(+)exchanger (CAX) as part of a widespread family of homolo

114 sue, Melchionda et al. identify a Ca(2+)/H(+)exchanger (CAX) that is required for Ca(2+)uptake and ce

115 CN4, which encodes the chloride/hydrogen ion exchanger CIC-4 prominently expressed in brain, were rec

116 ing a voltage-gated electrogenic nCl(-)/H(+) exchanger ClC-5.

117 LCN6, encoding the late endosomal Cl(-)/H(+)-exchanger ClC-6.

118 The chloride-proton exchanger CLC-7 plays critical roles in lysosomal homeos

119 M/MM simulations of the E. coli anion/proton exchanger ClC-ec1 and observed that fluoride binds incom

120 ounterion transporter such as the Cl(-)/H(+) exchanger, CLCN7 (ClC-7), encoded by CLCN7.

121 Among coupled exchangers, CLCs uniquely catalyze the exchange of oppos

122 ably, the addition of monensin, a Na(+)/H(+) exchanger, completely halts the infection.

123 The heat exchanger consists of polymer tubes wrapped around a pla

124 contributes to phase 1, whereas Na(+)-Ca(2+) exchanger contributes to phase 2.

125 PCST ion exchanger could separate (225)Ac from isotopes of Ra and

126 closilicate (ZS-9), a novel selective cation exchanger, could lower serum potassium levels in patient

127 rters (Na(+)/Ca(2+) exchanger and Na(+)/H(+) exchanger) coupled to the proton gradient, ultimately ma

128 pport a model in which the AE3 Cl(-)/HCO3(-) exchanger, coupled with parallel Cl(-) and H(+)-extrusio

129 We found that a dominance of inward Na-Ca exchanger current (I(NCX)) over Ca-dependent inactivatio

130 ico, L-type calcium current and Na(+)/Ca(2+) exchanger current determine RA human cell-to-cell differ

131 Large Na(+)/Ca(2+) exchanger current is the main driver in translating Ca(2

132 component was identified as an Na(+)-Ca(2+) exchanger current mediated by Ca(2+) released from the s

133 The decay of Na(+) /Ca(2+) exchanger current that followed a stimulation protocol w

134 VMs, whereas the density of the Na(+)/Ca(2+) exchanger current was not different between PCs and VMs.

135 L-type Ca(2+) current, and NCX (Na(+)/Ca(2+) exchanger) current are often measured in nonphysiologica

136 Varying concentration of added cation-exchanger demonstrates that it limits the ion transfer c

137 eading to changes in ionic channel, pump and exchanger densities and kinetics.

138 Our solvent exchanger device demonstrates the power of micro-free-fl

139 Knockdown of the mitochondrial Na(+) -Ca(2+) exchanger did not prevent the development of I(CRAC) in

140 Loss of the Drosophila Na(+)-H(+) exchanger DNhe2 lowers pHi in differentiating cells, imp

141 rt the crystal structure of the band 3 anion exchanger domain (AE1(CTD)) at 3.5 angstroms.

142 for ion-transfer voltammetry between an ion-exchanger doped and plasticized poly(vinyl chloride) (PV

143 ch reduced expression of the Cl(-) / HCO3(-) exchanger DRA (SLC26A3), via direct binding to the promo

144 mixing in the membrane module, and high heat exchanger efficiencies.

145 -sensitive fluorophores, ionophores, and ion-exchangers enable highly selective and rapid fluorescenc

146 ase is under the control of the Na(+)/Ca(2+) exchanger, encoded by the NCLX gene, and of a H(+)/Ca(2+

147 amily 8, member 1 (SLC8A1), a sodium/calcium exchanger encoding NCX1, were validated in an independen

148 )-dextran and its blockage by the Na(+)/H(+) exchanger ethylisopropyl amiloride (EIPA).

149 Pendrin (SLC26A4) is a Cl(-)/anion exchanger expressed in the epithelium of inflamed airway

150 SLC26A7 encodes a chloride/bicarbonate exchanger expressed in the renal outer medullary collect

151 Pendrin is a Cl(-)/HCO3(-) exchanger expressed in type B and non-A, non-B intercala

152 We have recently found that a Na(+)/H(+) exchanger expressed on synaptic vesicles promotes vesicl

153 due to a deficiency in a sodium calcium ion exchanger expressed specifically in the heart.

154 rters" for the constellation of ion channels/exchangers expressed in each sensory neuron.

155 tosolic Na(+) activated vesicular Na(+)/H(+) exchanger, facilitated glutamate loading into synaptic v

156 is the best studied member of the Na(+)/H(+) exchanger family and a model system for all related Na(+

157 l (3,4,5)-trisphosphate (PIP3)-dependent Rac exchanger) family (P-Rex1 and P-Rex2) of the Rho guanine

158 e nanospheres (containing the probes and ion exchanger) followed by measuring the pH and Na(+) respon

159 , the compatibility of the developed solvent exchanger for cell based downstream applications was pro

160 gurable, and self-sufficient convective heat exchanger for regulation of temperature in microfluidic

161 ization suggests that SLC9A9, an Na(+) -H(+) exchanger found in endosomes, appears to influence the d

162 mesophases, exemplified by the bacterial ClC exchanger from Escherichia coli (EcClC) as a model ion t

163 ryotic models, mainly on the NhaA Na(+)/H(+)-exchanger from Escherichia coli (EcNhaA).

164 On the other hand, sodium-calcium exchanger function and phosphorylation levels of calcium

165 2+) transients, upregulation of Na(+)/Ca(2+) exchanger function, reduction of Ca(2+) uptake to sarcop

166 Endolysosomal CLC Cl(-)/H(+) exchangers function as electric shunts for proton pumpin

167 ic tail derived from the chicken AE1-4 anion exchanger (G(AE)).

168 C>T:p.Leu515Phe) in the alkali cation/proton exchanger gene SLC9A7 (also commonly referred to as NHE7

169 remodelling of the ionic channels, pumps and exchangers gives rise to changes in the Ca(2+) transient

170 Poorly crystalline silicotitanate (PCST) ion exchanger had high selectivity for Ba, Ag and Th.

171 that the transport activity of this vacuolar exchanger has a profound impact on cytosolic homeostasis

172 by SLC26A3, a key intestinal chloride anion exchanger, has recently been identified as a novel susce

173 Many ion channels, transporters, and exchangers have been identified in the ES luminal epithe

174 Na(+)/Ca(2+) exchangers have two Ca(2+) sensor domains, CBD1 and CBD2

175 sient duration via accelerating Na(+)/Ca(2+) exchanger (I(NCX))-mediated Ca(2+) efflux from cytosol,

176 pport the existence of a countercurrent heat exchanger in the head of R. prolixus, which decreases th

177 Activation of the sodium-hydrogen exchanger in the heart and vasculature (NHE1 isoform) an

178 nd a model system for all related Na(+)/H(+) exchangers, including eukaryotic representatives.

179 The Na(+) /Ca(2+) exchanger inextricably couples the cycling of Ca(2+) and

180 nsor causes an adjacent segment, namely, the exchanger inhibitory peptide (XIP), to move toward the C

181 KX4-deficient mouse cones revealed that this exchanger is essential for the wide operating range and

182 The activity of sodium-hydrogen exchanger is markedly increased in patients with heart f

183 ion of Na(+) import through the Na(+)/Ca(2+) exchanger is sufficient to block this pathway, preventin

184 es had greater phosphorylation of Na(+)/H(+) exchanger isoform 3 (NHE3), distribution of NHE3 at the

185 ll molecule inhibitor of the sodium/hydrogen exchanger isoform 3 that functions in the gut to reduce

186 test whether the putative H(+)/Ca(2+)/Mn(2+) exchanger known as TMEM165 (transmembrane protein 165) p

187 simplicity, and self-sufficiency of the heat exchanger makes it suitable for various microfluidic bas

188 Therefore, the sodium-hydrogen exchanger may play a central role in the interplay of di

189 In placenta, these exchangers mediate placental uptake of substrates for oe

190 le and nonpolarizable Donnan exclusion anion-exchanger membrane reference/counter electrode.

191 Fabrication of an ion exchanger microchannel, capable of withstanding at least

192 hibition of the mitochondrial sodium-calcium exchanger (mNCE) resulted in a rise in [Ca(2+) ]m at bas

193 d lipid-binding properties of the Na(+)/H(+) exchanger NapA from Thermus thermophilus and compare thi

194 The family of K(+)-dependent Na(+)/Ca(2+)-exchangers, NCKX, are important mediators of cellular Ca

195 e-specific isoform of the Na(+)/Ca(2+), K(+) exchanger, NCKX2, is responsible for removing Ca(2+) fro

196 Here, we show that the Na(+)/Ca(2+), K(+) exchanger NCKX4 is expressed in zebrafish, mouse, and pr

197 sion of the mitochondrial Na(+)/Ca(2+)/Li(+) exchanger NCLX (SLC8B1) in human colorectal tumors and i

198 in activating the mitochondrial Na(+)/Ca(2+) exchanger (NCLX) causing enhanced mitochondrial Na(+) up

199 lc8b1 encodes the mitochondrial Na(+)/Ca(2+) exchanger (NCLX), which is proposed to be the primary me

200 er (MCU) channel or the Na(+) -Ca(2+) -Li(+) exchanger (NCLX).

201 l deletion of the mitochondrial Na(+)/Ca(2+) exchanger (NCLX, Slc8b1 gene) accelerated memory decline

202 extrusion via the mitochondrial Na(+)/Ca(2+) exchanger, NCLX.

203 urrents (~30%), and increased sodium/calcium exchanger (NCX) activity (~52%).

204 at accomplish this task are the Na(+)/Ca(2+) exchanger (NCX) and the Na(+)/K(+) pump (NKA).

205 icate T-type CaV channels and sodium-calcium exchanger (NCX) as predominant mechanisms of calcium inf

206 function by activation of electrogenic Na/Ca exchanger (NCX) during diastole.

207 In the atrial-specific Na(+) /Ca(2+) exchanger (NCX) knockout mouse, cellular Ca(2+) accumula

208 lar Ca(2+), and the activity of Na(+)/Ca(2+) exchanger (NCX) may be altered when the Na(+) gradient i

209 iomyocyte contractility via a sodium-calcium exchanger (NCX) mediated pathway.

210 The electrogenic Na/Ca exchanger (NCX) mediates bidirectional Ca movements that

211 The electrogenic sodium/calcium exchanger (NCX) mediates bidirectional calcium transport

212 Na(+)/Ca(2+) exchanger (NCX) proteins operate through the alternating

213 tribution of the reverse-mode sodium-calcium exchanger (NCX) to these responses during hyperglycemia.

214 orters, including the mammalian Na(+)/Ca(2+) exchanger (NCX), our study provides a regulatory mechani

215 mibefradil) and by blocking the Na(+)/Ca(2+) exchanger (NCX), suggesting an important contribution of

216 ) oscillations induced by the sodium/calcium exchanger NCX1/3 working in its reverse mode.

217 by sequential expression of the Na(+)-Ca(2+) exchanger (NCX1) and L-type Ca(2+) channel (LTCC).

218 erized cardiac promoters, the sodium calcium exchanger (Ncx1) and the brain natriuretic peptide (Bnp)

219 pendrin and the Na(+)-driven Cl(-)/2HCO3(-) exchanger (NDCBE) in beta-intercalated cells of the coll

220 nd the sodium-dependent chloride-bicarbonate exchanger (NDCBE).

221 he exchange activity of the human Na(+)/H(+) exchanger NHA2 (SLC9B2) is electroneutral, despite harbo

222 d selective up-regulation of the Na(+) /H(+) exchanger NHA2 (SLC9B2) was observed in cysts of patient

223 inhibited by EZA and blocked by EIPA, a Na-H exchanger (NHE) inhibitor.

224 cells and increase cleft acidity via Na+/H+ exchanger (NHE) proton extrusion, which results in inhib

225 ylate transporter (MCT), the sodium hydrogen exchanger (NHE), and V-Type ATPase mediate acid extrusio

226 with voltage-dependent neutral sodium-proton exchanger (NHE).

227 In human melanoma, the Na(+)/H(+) exchanger NHE1 is an important modifier of the tumour na

228 and impaired activity of the sodium/hydrogen exchanger NHE1, a known regulator of skin pH.

229 IgE activates macrophage Na(+)-H(+) exchanger (Nhe1) and induces extracellular acidification

230 The epithelial brush-border Na(+)/H(+) exchanger NHE3 is acutely inhibited by cGKII/cGMP, but h

231 Na(+) /H(+) exchanger NHE3 of human or primates differs from NHE3 of

232 We show that the neuron-enriched Na(+)/H(+) exchanger NHE5 is abundantly expressed in C6 glioma cell

233 The Na(+)/H(+) exchanger NHE6 provides a leak pathway for protons, limi

234 the predominantly Golgi-resident Na(+)/H(+) exchanger NHE8 localizes to the developing acrosome of s

235 s a leak pathway for protons, the Na(+)/H(+) exchanger NHE9 limits luminal acidification to circumven

236 Na(+) /H(+) exchangers (NHEs) are ancient membrane-bound nanomachine

237 utations in human endosomal Na(+)(K(+))/H(+) exchangers (NHEs) NHE6 and NHE9 are implicated in neurol

238 Mammalian Na(+)/H(+) exchangers (NHEs) regulate numerous physiological proces

239 acidic synaptic vesicles and (3) Na(+) /H(+) exchangers (NHEs).

240 A study of the sodium/calcium exchanger now suggests that secondary structure may hold

241 The Na(+)/Ca(2+) exchanger of Drosophila melanogaster, CALX, is the main

242 Sodium/proton exchangers of the SLC9 family mediate the transport of p

243 comparison to the optode containing the ion exchanger only (Miptode 3), the optode containing the io

244 the parallel operation of the Cl(-)/HCO3(-) exchanger pendrin and the Na(+)-driven Cl(-)/2HCO3(-) ex

245 s the intercalated cell chloride/bicarbonate exchanger pendrin is unclear, as are potassium's role in

246 trin, L-type calcium channel, sodium-calcium exchanger, phospholamban, calcineurin, and calcium/calmo

247 nterferes with purinergic stimulation of the exchanger, possibly by obscuring T331, a previously iden

248 activation of the mitochondrial Na(+)/Ca(2+) exchanger promotes the import of Na(+) into the matrix.

249 ng of the Na(+)-K(+) ATPase and Na(+)-Ca(2+) exchanger proteins within these structures, as reduction

250 m fluxes (calcium current and sodium/calcium exchanger), providing mechanisms for triggered activity.

251 ride Channel a) is a vacuolar NO(3) (-)/H(+) exchanger regulating stomata aperture in A thaliana Here

252 nding of the scaffolding proteins Na(+)/H(+) exchanger regulatory factor (NHERF) 1 and 2.

253 NHE3 directly binds Na(+)/H(+) exchanger regulatory factor (NHERF) family scaffolding p

254 In this study, Na(+)/H(+) exchanger regulatory factor (NHERF)-1 loss-of-function a

255 ther the scaffolding protein sodium-hydrogen exchanger regulatory factor 1 (NHERF1) interacts with th

256 previously demonstrated that the Na(+)/H(+) exchanger regulatory factor 1 (NHERF1) promotes C3aR fun

257 Na(+)/H(+) exchanger regulatory factor 1 (NHERF1; also known as ezr

258 okine receptor (CCR) 2, CCR5, the Na(+)/H(+) exchanger regulatory factor 1, CXCR3, alpha (1)-AR, and

259 Na(+)-H(+) exchanger regulatory factor-1 (NHERF1) is a PDZ protein

260 ; V(2)R) and a cytosolic protein (Na(+)/H(+) exchanger regulatory factor-1; NHERF1) as examples, we d

261 rters are multi-subunit Na(+) (or K(+))/H(+) exchangers representing an ancestor of many essential re

262 Na(+) compartmentalization (i.e. Na(+)/H(+) EXCHANGERs, SALT OVERLY SENSITIVE, HIGH-AFFINITY K+ TRAN

263 Ps, the levels of both the glutamate-cystine exchanger Sc7a11 and glutathione were increased; by cont

264 voltage-gated chloride flux through the ion exchanger SLC26A11, active KCC2-mediated chloride extrus

265 The chloride/bicarbonate exchanger SLC26A3 (downregulated in adenoma) is expresse

266 Loss of the AE3 Cl(-)/HCO3(-) exchanger (Slc4a3) in mice causes an impaired cardiac fo

267 the potential of targeting cystine/glutamate exchanger (SLC7A11/xCT), which contributes to the mainte

268 Band 3 (also known as the anion exchanger, SLCA1, AE1) constitutes the major attachment

269 transporter (NBC1) and apical Cl(-) /HCO3(-) exchanger (solute carrier family 26 member A6; SLC26A6),

270 These values are greater than current anion exchangers such as the resins Amberlite IRA-400 (249 mg/

271 one acetyltransferase NuA4 and histone H2A.Z exchanger SWR1.

272 ClC-4 is an intracellular Cl(-)/H(+) exchanger that is highly expressed in the brain and whos

273 This transporter is a Ca(2+)-H(+) exchanger that raises cell surface pH.

274 NCLX is a Na(+)/Ca(2+) exchanger that uses energy stored in the transmembrane s

275 However, exchangers that cannot be palmitoylated do not inactivat

276 ier (Slc) family 26A encodes different anion exchangers that exchange Cl(-)/HCO3 (-), including Slc26

277 a member of the SLC9B family of Na(+) /H(+) exchangers, that correlated with cyst size and disease s

278 aling through the reversal of sodium/calcium exchanger, thereby establishing a new pathway for the ge

279 ction of several classes of ion channels and exchangers, they could act as functional "reporters" for

280 erative Ca(2+) binding is key to enable this exchanger to efficiently respond to changes in the intra

281 Raising [Na(+)]i activates the Na(+)/Ca(2+) exchanger to operate in a reverse mode leading to Ca(2+)

282 ombination of ultrasound (Us) with a thermal exchanger to produce high quality extra virgin olive oil

283 he surface provide dual-functionality as ion-exchangers to the hybrid material, firstly used for the

284 al cellular receptor, the chicken Na(+)/H(+) exchanger type 1 (chNHE1).

285 aminoindanes as inhibitors of the Na(+)/H(+) exchanger type 3 (NHE3) are described based on a hit fro

286 of Epac1 and Epac2 decreases sodium-hydrogen exchanger type 3 expression in the proximal tubule, lead

287 Instead, sodium-hydrogen exchanger type 3 levels in the proximal tubule were dram

288 Chicken Na(+)/H(+) exchanger type I (chNHE1), a multispan transmembrane pro

289 successful application as industrial cation exchangers under near neutral conditions, their performa

290 The CLC exchangers, unlike canonical 'ping-pong' antiporters, si

291 A tubular thermal exchanger was used for a rapid cooling treatment (CT) of

292 Titanium based ion exchanger were evaluated for purification of (225)Ac.

293 NCX1 is a Na(+)/Ca(2+) exchanger, which is believed to provide a key route for

294 ctionally interacts with the sodium-hydrogen exchanger, which is responsible for the majority of sodi

295 Acute inhibition of the sodium-calcium exchanger, which targets the downstream effects of enhan

296 on exchangers, can also be considered cation exchangers, which opens new avenues for future scientifi

297 ed by changing the configuration of the heat exchanger with respect to the microfluidic structure.

298 oss through a series of counter-current heat exchangers within its gills.

299 n quantifying changes in channels, pumps and exchangers without quantitatively linking these changes

300 The cystine/glutamate exchanger xCT is essential for amino acid and redox home