ライフサイエンスコーパス: excitatory

1 acid (GABA) mediated) and bipolar disorder (excitatory).

2 signaling molecules by astrocytes having an excitatory action on the CNS sympathetic control circuit

3 Among the principal excitatory afferent pathways, we showed that ventral hip

4 Excitatory amino acid transporters (EAATs) are prototypi

5 DNQX, by blocking excitatory AMPA glutamate inputs, is hypothesized to pro

6 nce of global gene expression levels between excitatory AMPA receptors (AMPARs) and inhibitory GABA(A

7 To assess the time window and mechanism of excitatory and calcium-dependent refinements during late

8 As ipsilateral PDFMEs express excitatory and contralateral PDFMEs inhibitory PDF autor

9 c scaffolding protein that localizes to both excitatory and GABAergic synapses.

10 ajor depression (inhibitory), schizophrenia (excitatory and gamma-aminobutyric acid (GABA) mediated)

11 volunteers on a visual task and measured the excitatory and inhibitory (E/I) balance in early visual

12 Transient imbalances in excitatory and inhibitory activity may provide a general

13 nk between astrocytes and the development of excitatory and inhibitory balance in the mouse hippocamp

14 ting the slow response fluctuations of local excitatory and inhibitory cell populations.

15 The topographic organization of excitatory and inhibitory cell types may enable parallel

16 Across excitatory and inhibitory cell types, genes encoding vol

17 o spreads through GABAergic synapses to both excitatory and inhibitory cell types.

18 arises in the interplay between presynaptic excitatory and inhibitory circuit elements.

19 These findings demonstrate how excitatory and inhibitory circuits interact to mediate a

20 ndicate that pF(L) neurons receive extensive excitatory and inhibitory inputs from several respirator

21 he cortical connectivity, we built models of excitatory and inhibitory inputs onto a single neuron, t

22 ngage canonical neurocircuits that encompass excitatory and inhibitory interneurons, similarly to tho

23 ed in the dorsal spinal cord by a network of excitatory and inhibitory interneurons.

24 Here we show that the excitatory and inhibitory intracortical connections to a

25 er targeting distinct sets of genes in human excitatory and inhibitory neural progenitors and neurons

26 These studies revealed diverse types of excitatory and inhibitory neuron in each circumventricul

27 detection inside the RF selectively enhances excitatory and inhibitory neuron responses to task-irrel

28 tex, expression of risk genes is enriched in excitatory and inhibitory neuronal lineages, consistent

29 res from discrete subpopulations of cortical excitatory and inhibitory neurons are significantly enri

30 d monosynaptic tracing to identify inputs to excitatory and inhibitory neurons of the intermediate an

31 and RNAscope showed that oxytocin affects S1 excitatory and inhibitory neurons similarly, whereas in

32 Surprisingly, excitatory and inhibitory neurons were equally selective

33 The selectivity of excitatory and inhibitory neurons within decision circui

34 mical distribution, and are detected in both excitatory and inhibitory neurons.

35 computation depends on interactions between excitatory and inhibitory neurons.

36 rsal medial prefrontal cortex (dmPFC) across excitatory and inhibitory neurons.

37 th IPANs and have direct connections to both excitatory and inhibitory neurons.

38 are predominantly associated with projecting excitatory and inhibitory neurons.

39 uld reduce social deficits via modulation of excitatory and inhibitory neurotransmission in the NAc.

40 elated with the spatial displacement between excitatory and inhibitory presynaptic ensembles.

41 rL projectors causally regulated activity in excitatory and inhibitory PrL neurons during context fea

42 d by PNNs show higher density of perisomatic excitatory and inhibitory puncta, longer axonal initial

43 Abnormal excitatory and inhibitory synapse assembly and elevated

44 ty of astrocytic ephrin-B1 to influence both excitatory and inhibitory synapses during development ca

45 ty of astrocytic ephrin-B1 to influence both excitatory and inhibitory synapses during development ca

46 prominent pathway involves crosstalk between excitatory and inhibitory synapses whereby Ca(2+)-enteri

47 ynapses would permit rapid crosstalk between excitatory and inhibitory synapses.

48 d explanation of asynchronous firing is that excitatory and inhibitory synaptic inputs are precisely

49 Excitatory and inhibitory synaptic transmission in CA1 n

50 rate to decrease and potentiate the synaptic excitatory and inhibitory tone onto mutant SF1 neurons,

51 and more complex than expected interplay of excitatory and inhibitory transmitter systems in modulat

52 Here we investigated how excitatory and VIP inhibitory cells in layer 2/3 of mous

53 boosts glutamatergic transmission, which is excitatory, and suppresses long-term potentiation, a sur

54 on of sharp Purkinje cell zones and disrupts excitatory axon patterning.

55 induction of long-term potentiation (LTP) at excitatory, axospinous synapses.

56 ercoming inhibitory mechanisms that restrict excitatory bursts, and engaging wide areas of cortex.

57 revealed a distributed network of long-range excitatory but not inhibitory projections with subtype-s

58 e (MA) at the Neiguan (P6) acupoint inhibits excitatory cardiovascular reflex responses through modul

59 tivates mTORC1-4E-BP signalling in pyramidal excitatory cells of the cortex(8,14).

60 s sustained spectral surround suppression in excitatory cells, indicating a dominant causal role for

61 ey almost exclusively innervate dendrites of excitatory cells, preferentially targeting distal apical

62 Fs) of SDs are organized concentrically: the excitatory center (about 4.5 degrees ) is surrounded by

63 Our results indicate that local subicular excitatory circuits are connected in a cell type-specifi

64 intrinsic connectivity and whether its local excitatory circuits are sufficient to generate epileptif

65 vides a high-resolution description of local excitatory circuits of the subiculum and highlights thei

66 d the hypothesis that endocannabinoids endow excitatory circuits with pathway- and cell-specific plas

67 n optimise signal integration rules in local excitatory circuits.

68 le postsynaptic model neuron receiving tuned excitatory connections alongside inhibition from two pla

69 t local subicular events, purely mediated by excitatory connections, may underlie the pre-ictal disch

70 ons, despite the presence of highly specific excitatory connectivity, which was deemed to underlie fe

71 Excitatory cortical neurons from both patients had prolo

72 Structurally, geniculate axons innervated excitatory cortical targets in a preferential manner and

73 2 to synchronize differentiation of induced excitatory cortical-like neurons, we investigated networ

74 rent was more than threefold larger than the excitatory current.

75 Small changes in the kinetics of dendritic excitatory currents amplify when reaching the soma.

76 wn to be critical for elevated NMDA-mediated excitatory currents in sympathoexcitatory PVN neurons fo

77 edge TRN cells evoke slower, less depressing excitatory currents that drive more persistent spiking.

78 circuit exhibits a balance of inhibitory and excitatory currents that persists on stimulation.

79 response to the neurotransmitter GABA, from excitatory depolarization to inhibitory hyperpolarizatio

80 nous levels of spinal dopamine are low, this excitatory dopaminergic pathway is likely physiologicall

81 FAP promoter-driven expression of hM3Dq, the excitatory DREADDs (designer receptors exclusively activ

82 (A)Rs), which provides inhibitory balance to excitatory drive and controls neuronal output.

83 ased depressive-like behaviors and increased excitatory drive onto BNST-CRF cells.

84 he substantial energy requirements, a strong excitatory drive, and a unique developmental trajectory.

85 from stimulation-evoked, temporally dynamic excitatory (E) and inhibitory (I) activity.

86 gamma-aminobutyric acid (GABA) are the major excitatory (E) and inhibitory (I) neurotransmitters in t

87 lity cortical and spinal networks, having an excitatory effect at the spinal level and an inhibitory

88 nces cortical and spinal networks, having an excitatory effect at the spinal level and an inhibitory

89 atal and GPe neurons, including biphasic and excitatory effects, which our modeling shows can be expl

90 nses to these inputs are limited and include excitatory effects.

91 22qDS model mice exhibited CA1 excitatory ensemble hyperexcitability and concomitant be

92 hanism does not rely on persistent activity, excitatory feedback, or synaptic plasticity for storage.

93 lls stratifying at distinct depths relay the excitatory feedforward drive from photoreceptors to amac

94 poral lobes, with development of an abnormal excitatory fronto-temporal projection in the left cerebr

95 lpha(2a)-AR autoreceptors, it also activates excitatory G(i)-coupled heteroreceptors in the bed nucle

96 HA negatively regulates excitatory gain onto D1(+)-MSNs via presynaptic H(3) rec

97 des was supported by the interaction between excitatory gamma inputs and local inhibition.

98 lia depletion reduced inhibitory GABA(A) and excitatory glutamate receptor-mediated synaptic transmis

99 detected using two approaches: (1) increased excitatory glutamate transmission at mossy fibers (MF)-C

100 cate but poorly understood interplay between excitatory glutamatergic and inhibitory GABAergic recept

101 ory interneuron, an amacrine cell, receiving excitatory glutamatergic input exclusively from S-ON bip

102 gative modulation of both receptors enhanced excitatory glutamatergic input to mouse prefrontal corte

103 ither sex to demonstrate that in addition to excitatory glutamatergic synapses, MOC neurons receive i

104 orpus callosum and that their terminals form excitatory, glutamatergic synapses on host cortical neur

105 Unipolar brush cells (UBCs) are excitatory granular layer interneurons in the vestibuloc

106 nel inactivation in synaptic facilitation of excitatory hippocampal neurons.

107 Excitatory hM3Dq DREADD-mediated chemogenetic activation

108 related hyperexcitability and restoration of excitatory-inhibitory balance in perilesional ventral pr

109 on, which is required for maintaining proper excitatory-inhibitory balance in the PFC and its control

110 However, little is known about how excitatory-inhibitory balance is defined at most synapse

111 n onto pyramidal neurons, disrupt prefrontal excitatory-inhibitory balance, and alter processing of a

112 tively enhance inhibitory activity and alter excitatory-inhibitory balance, and may be useful for pre

113 xactly achieved by a network with reciprocal excitatory-inhibitory competitive dynamics, similar to i

114 neages, consistent with multiple paths to an excitatory-inhibitory imbalance underlying ASD.

115 ng-term plasticity calibrates populations of excitatory-inhibitory inputs onto mouse auditory cortica

116 for these phenomena by training a recurrent excitatory-inhibitory neural circuit model of a visual c

117 However, these changes may also perturb the excitatory-inhibitory neurotransmission balance and trig

118 r acetylcholine (ACh) is known to affect the excitatory/inhibitory (E/I) balance of primate visual co

119 x metalloprotease-9 may thus enhance overall excitatory/inhibitory balance and neuronal population dy

120 ribution of distinct alpha2delta subunits to excitatory/inhibitory imbalance and aberrant network con

121 njury-related hyperexcitability and restores excitatory:inhibitory balance in vPMC, thereby normalizi

122 tes, greater spike frequency adaptation, and excitatory:inhibitory ratio.

123 is strongly activated in microglia following excitatory injury elicited by status epilepticus.

124 Y treatment caused persistent attenuation of excitatory input and induced dendritic hypotrophy via Y(

125 ceptor activation; conversely, CRF increased excitatory input and induced hypertrophy of BLA principa

126 The vpoDNs receive excitatory input from auditory neurons (vpoENs), which a

127 iking inhibitory interneurons receive direct excitatory input from the intermediate/ventral hippocamp

128 ored plasticity of glutamate uncaging-evoked excitatory input patterns with various spatial distribut

129 th CT and PT neurons providing the strongest excitatory input to TC neurons that project back to S1.

130 on graft location, whereas inhibitory versus excitatory input was dictated by the identity of grafted

131 To understand how the dendrite integrates excitatory inputs as a whole, we combined anatomic quant

132 recruitment of fast-spiking interneurons by excitatory inputs during adolescence.

133 ing by setting the relative amount of distal excitatory inputs from the entorhinal cortex.SIGNIFICANC

134 factors, regulate the inhibitory outputs and excitatory inputs of interneurons in the mouse cerebral

135 narrow the coincidence detection window for excitatory inputs to CA1 pyramidal cells.

136 Here we describe a second class of local excitatory inputs to granule cells that are more powerfu

137 s the coincidence detection window of direct excitatory inputs to pyramidal cells whereas increasing

138 vation are unknown, as the LHb receives many excitatory inputs whose contributions to cocaine aversio

139 ates the refinement of calcium signaling and excitatory inputs without affecting biophysical membrane

140 rons, as well as map putative inhibitory and excitatory inputs.

141 he contribution that eight classes of dorsal excitatory INs make to sensorimotor reflex responses.

142 spinal ERalpha-positive neurons are largely excitatory interneurons and many coexpress substance P,

143 marker for a discrete subset of nociceptive, excitatory interneurons.

144 NMDA receptors are excitatory ion channels with fundamental roles in synapt

145 ated with intense synchronized activation of excitatory layer 2/3 pyramidal neurons (PNs) and to a le

146 eived 21% of their input from bipolar cells, excitatory local circuit neurons receiving input from co

147 tic plasticity that prevents accumulation of excitatory LTP.

148 l types were identified for risky behaviour (excitatory), major depression (inhibitory), schizophreni

149 predicted the neural responses than a purely excitatory model, suggesting an enhancement of the rate-

150 numerous MCHS-related phenotypes, including excitatory neuron and microglia gene expression changes.

151 g significant enrichments of autism risk and excitatory neuron genes.

152 lpha has traditionally been considered as an excitatory neuron marker, our single-cell sequencing res

153 addition our analysis allows an estimate of excitatory neuron-restricted precursors (about 10) that

154 ia adeno-associated viral vectors, driven by excitatory neuron-specific promoter elements, to manipul

155 ction through multiple cell types, including excitatory neuronal and neuroimmune populations.

156 atively with the inhibitory influence on the excitatory neuronal population in the dACC of subjects w

157 These findings indicate that excitatory neurons and astrocytes are organized into dis

158 ix metalloprotease-9, increases branching of excitatory neurons and concomitantly attenuates the peri

159 reatest dysregulation occurred in deep layer excitatory neurons and immature oligodendrocyte precurso

160 eriods and demostrate enriched expression in excitatory neurons and radial glias but depleted express

161 Thus, BLA excitatory neurons are a highly heterogenous collection

162 Excitatory neurons are derived from the dorsal telenceph

163 Connections between the two types of excitatory neurons are sparse, and local processing of h

164 ates that CaMKIIalpha is not as specific for excitatory neurons as commonly believed.

165 ediated chemogenetic activation of forebrain excitatory neurons during postnatal life (P2-14), but no

166 enhanced Gq-mediated signaling in forebrain excitatory neurons during postnatal life can evoke persi

167 q-signaling-mediated activation of forebrain excitatory neurons during the critical postnatal window

168 nia and bipolar disorder are concentrated in excitatory neurons in cortical layers II-III, IV, and V,

169 Deleting ngr1 in excitatory neurons in L4, but not in L2/3, L5, or L6, pr

170 sion of nrg1,2,3,4 and erbB1,2,3,4 in PV and excitatory neurons in mouse visual cortex.

171 ther, these results suggest that stimulating excitatory neurons in the fastigial nucleus may be a pro

172 on of eukaryotic initiation factor 2alpha in excitatory neurons in the LA enhanced memory strength bu

173 resentation, we performed calcium imaging of excitatory neurons in the primary visual cortex (V1) of

174 These results suggest that SIRT1 in mPFC excitatory neurons is required for normal neuronal excit

175 In contrast, nrg1 expression by excitatory neurons is unchanged at P28 and P104 followin

176 NMDAR PAM AGN-241751 and identify GluN2B on excitatory neurons of mPFC as initial cellular trigger u

177 Activation of monSTIM1 in either excitatory neurons or astrocytes of mice brain is able t

178 Chemogenetic inhibition of excitatory neurons or pharmacologically strengthening GA

179 onditional Mef2c heterozygosity in forebrain excitatory neurons reproduced a subset of the Mef2c-Het

180 sory deprivation, whereas nrg3 expression by excitatory neurons shows changes depending on the age an

181 or cell-specific manipulation, we discovered excitatory neurons that induce nausea-related behaviors,

182 However, a subpopulation of excitatory neurons thought to mediate behavioral output

183 ression changes in PV inhibitory neurons and excitatory neurons track with sensory perturbation.

184 cted precursors (about 10) that generate the excitatory neurons within a cortical column.

185 tivity of the drugs is mediated by 4E-BP2 in excitatory neurons, and 4E-BP1 and 4E-BP2 in inhibitory

186 radial glia, intermediate progenitor cells, excitatory neurons, and interneurons isolated from mid-g

187 evealed feature-specific suppression between excitatory neurons, despite the presence of highly speci

188 ypes and circuit diagram of clonally related excitatory neurons, we performed multi-cell patch clamp

189 to be expressed during mid-gestation and in excitatory neurons, whereas neurodegenerative-disorder r

190 PV interneuron activity and disinhibition of excitatory neurons, which are known hallmarks of cortica

191 composed of multiple types of inhibitory and excitatory neurons, which form sophisticated microcircui

192 ually antagonistic fbRF and ffRF, similar to excitatory neurons.

193 istence of selective pools of inhibitory and excitatory neurons.

194 n by the differential spatial recruitment of excitatory neurons.

195 rain regions, than the overall population of excitatory neurons.

196 ll layers of A25, mostly targeting spines of excitatory neurons.

197 Kcna1 (encoding Kv1.1) in mouse hippocampal excitatory neurons.

198 with maximal enrichment in cortical layer V excitatory neurons.

199 icity at different timescales in hippocampal excitatory neurons.

200 c nerve terminals corelease zinc to modulate excitatory neurotransmission and sensory responses.

201 hile reports indicate that alcohol increases excitatory neurotransmission exclusively on NAc D1-MSNs

202 We assessed TEP sensitivity to changes in excitatory neurotransmission mediated by n-methyl-d-aspa

203 the ketamine-induced increase in hippocampal excitatory neurotransmission, and this effect concurred

204 aMP6f in ICC to evaluate ICC-IM responses to excitatory neurotransmission.

205 metabolic demands of neuronal activity with excitatory neurotransmission.SIGNIFICANCE STATEMENT Syna

206 We probed the effects of excitatory neurotransmitter glutamate and inhibitory neu

207 D1 results in an imbalance of inhibitory and excitatory neurotransmitters, and as Gad1-/- mice die ne

208 s may favor temporal precision of cerebellar excitatory outputs associated with specific features of

209 at they interact with interneurons to act as excitatory pacemakers for the V1 gamma rhythm.

210 Thus, the BLA is the dominant excitatory pathway onto D1 MSNs (BLA > PFC = vHipp) whil

211 ereas CB1 receptors (CB1R) uniformly depress excitatory pathways regardless of MSNs identity, TRPV1 r

212 Relatedly, decreased frequency of miniature excitatory post-synaptic currents accompanied changes in

213 Specifically, spontaneous excitatory postsynaptic current amplitudes measured from

214 ubjects with ASD exhibited reduced miniature excitatory postsynaptic current frequency and N-methyl-D

215 AS exon 3 recapitulated diminished miniature excitatory postsynaptic current frequency, supporting a

216 basket cells (BCs), we found that classical excitatory postsynaptic currents (EPSCs) are followed by

217 rats revealed higher frequency of miniature excitatory postsynaptic currents (mEPSCs) immediately af

218 stsynaptic potentials (fEPSPs) and miniature excitatory postsynaptic currents (mEPSCs) in rat hippoca

219 t increased the amplitude of the spontaneous excitatory postsynaptic currents and decreased the frequ

220 rocytes reduced the frequency of spontaneous excitatory postsynaptic currents in the direct pathway M

221 ng SF1 projections to the PVT elicits direct excitatory postsynaptic currents.

222 We used PSD95-eGFP mice, to visualise excitatory postsynaptic densities (PSDs) using high-reso

223 (2R,6R)-HNK were examined by recording field excitatory postsynaptic potentials (fEPSPs) and miniatur

224 Unitary excitatory postsynaptic potentials (uEPSPs) revealed a h

225 RIIA) level and the frequency of spontaneous excitatory postsynaptic potentials.

226 genetic stimulation of the claustrum induced excitatory postsynaptic responses in most neocortical ne

227 y direct pathway component while sparing the excitatory, potentially indirect pathway component of so

228 The elaboration of excitatory projection neuron (PN) processes likely contr

229 resumably because RTN, unlike C1, has direct excitatory projections to abdominal premotor neurons.

230 ned, our results identify that activation of excitatory projections, but not inhibition-driven change

231 accumbens activated downstream of long-range excitatory projections.

232 s of other brain disorders or genes encoding excitatory PSD proteins.

233 amilial AD crossed with mouse lines labeling excitatory pyramidal cells (PCs) and inhibitory PV cells

234 Thus, sound-driven activity accelerates the excitatory refinement and shortens the period of activit

235 ystems, large amygdalar terminals innervated excitatory relay and inhibitory neurons in the MDmc that

236 This tuning emerged after the initial excitatory response to the face and was expressed as the

237 ophysiology, that, contrary to expectations, excitatory responses evoked by sensory and brainstem inp

238 transient cation current that mediates fast excitatory responses in peripheral and central nervous s

239 ) release and Ano1 activation contributes to excitatory responses of colonic muscles.

240 -aspartate (NMDA) receptors mediate synaptic excitatory signaling in the mammalian central nervous sy

241 R) are coexpressed within a subpopulation of excitatory superficial dorsal horn (SDH) neurons.

242 MNTB neurons through a single calyx of Held excitatory synapse arising from the cochlear nucleus.

243 s) with latrophilins (LPHNs/ADGRLs) promotes excitatory synapse formation when LPHNs simultaneously i

244 e addition of purified hyaluronan suppresses excitatory synapse formation.

245 nvolved a new ephaptic coupling, in which an excitatory synapse generated large negative extracellula

246 al of hyaluronan increases the expression of excitatory synapse markers and results in a correspondin

247 l activation, BDNF-dependent increase in the excitatory synapse markers synaptophysin and PSD-95, and

248 te these deletions in the neurophysiology of excitatory synapses and in ASD-associated synaptic impai

249 strength of inhibitory synapses relative to excitatory synapses can be tuned from weak to strong to

250 Severe loss of excitatory synapses in key brain regions is thought to b

251 iled and quantitative map of the features of excitatory synapses in the lumbar spinal cord, detailing

252 g fibers from the inferior olive make strong excitatory synapses onto cerebellar Purkinje cell (PC) d

253 We confirm that the AON forms glutamatergic excitatory synapses onto piriform pyramidal neurons; and

254 itory synapses as well as the development of excitatory synapses received by these cells.

255 ermine which factors impact the diversity of excitatory synapses throughout the lumbar spinal cord.

256 e encodes a postsynaptic scaffold protein in excitatory synapses, and its disruption is implicated in

257 At excitatory synapses, how endogenous AMPARs, NMDARs, and

258 at a diverse set of identified and putative excitatory synapses, including a pair of connected neuro

259 , we report that SPARCL1 selectively induces excitatory synapses, increases their efficacy, and enhan

260 an extracellular matrix surrounds developing excitatory synapses, where it critically regulates synap

261 ics, we studied AMPA receptor (AMPAR)-silent excitatory synapses, which are generated in the nucleus

262 a corresponding increase in the formation of excitatory synapses, while also decreasing inhibitory sy

263 ich resulted in >90% decrease in spontaneous excitatory synaptic activity.

264 IFICANCE STATEMENT Existing studies focus on excitatory synaptic changes after social stress, althoug

265 Overexpression of alpha2delta3 increases the excitatory synaptic density as well but also facilitates

266 logy and reveal that a selective decrease in excitatory synaptic drive to PV basket cells (PVBCs) lik

267 Alterations of excitatory synaptic function are the strongest correlate

268 ntegration determine plasticity of different excitatory synaptic input patterns in perisomatic dendri

269 utamate sensor in awake monkeys, and map the excitatory synaptic inputs on dendrites of individual V1

270 regated dendritic domains receiving distinct excitatory synaptic inputs with specific properties and

271 Cs develop smaller spines and receive weaker excitatory synaptic inputs.

272 as well as (2) cellular adaptations and (3) excitatory synaptic physiology in the basolateral amygda

273 hat in the nucleus accumbens, opioid-induced excitatory synaptic plasticity involves presynaptic and

274 synaptic plasticity occurs in parallel with excitatory synaptic plasticity, with the ensuing interru

275 amantadine enhances dendritic integration of excitatory synaptic potentials in SPNs and enhances - no

276 greater intrinsic excitability and recurrent excitatory synaptic strength, while E/I ratio, local out

277 x 10(-7)), which is implicated in regulating excitatory synaptic transmission and plasticity.

278 eases intrinsic excitability and spontaneous excitatory synaptic transmission in layer V pyramidal ne

279 Fast excitatory synaptic transmission in the mammalian brain

280 utamate-gated ion channels that mediate fast excitatory synaptic transmission in the nervous system.

281 these paralog proteins in neurodevelopment, excitatory synaptic transmission, and plasticity.

282 E STATEMENT Synaptic AMPARs are critical for excitatory synaptic transmission.

283 and are recognized as regulatory elements of excitatory synaptic transmission.

284 uced dendritic spine density, and diminished excitatory synaptic transmission.

285 NMDA receptors play crucial roles in excitatory synaptic transmission.

286 nged the electrical properties and increased excitatory synaptic transmission.

287 Thus, SPARCL1 selectively boosts excitatory synaptogenesis and synaptic transmission by a

288 the pore-forming alpha1 subunit and trigger excitatory synaptogenesis.

289 but instead correlated with the magnitude of excitatory synchrony of the hippocampal output.

290 ted in CA1, revealing that most of them were excitatory, targeting dendritic spines and displaying a

291 transformation involves interactions between excitatory thalamocortical neurons that carry data to th

292 Neuronal GABAergic responses switch from excitatory to inhibitory at an early postnatal period in

293 The GABA response switch from excitatory to inhibitory is a key event in neuronal matu

294 formation and the resulting balance between excitatory to inhibitory signaling.

295 reach a capacity of 0.5 for any fraction of excitatory to inhibitory weights and have a peculiar syn

296 ting a model by which theta oscillations are excitatory to neural activity, and alpha oscillations ar

297 ausal role and have a profound impact on the excitatory-to-inhibitory balance and network connectivit

298 on by inducing long-term depression (LTD) of excitatory transmission at hippocampal CA3-CA1 synapses,

299 ignaling, AMPAR transport, localization, and excitatory transmission.

300 dendritic inhibition balances the increased excitatory weights, such that place cells return to thei