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1 A1 inhibitory postsynaptic potential into an excitatory postsynaptic potential.
2 currents sum effectively and produce a large excitatory postsynaptic potential.
3 eceptors mediate the slower component of the excitatory postsynaptic potential.
4 ng spatial and temporal summation of smaller excitatory postsynaptic potentials.
5 ersibly increased the slope and amplitude of excitatory postsynaptic potentials.
6  inhibitory effect of ethanol on NMDAR field excitatory postsynaptic potentials.
7 ices during extracellular recording of field excitatory postsynaptic potentials.
8 tentials from basket interneurons (BAS) into excitatory postsynaptic potentials.
9 erm potentiation and long-term depression of excitatory postsynaptic potentials.
10 equency but not the amplitude of spontaneous excitatory postsynaptic potentials.
11 RIIA) level and the frequency of spontaneous excitatory postsynaptic potentials.
12 mplitude and frequency of glutamate-mediated excitatory postsynaptic potentials.
13 cted by a concomitant decrease in the evoked excitatory postsynaptic potentials.
14  spike, but not those that exhibit only fast excitatory postsynaptic potentials.
15 ion of ventral CA1 fibers, and this broadens excitatory postsynaptic potentials.
16 ilm oxide leading to responses that resemble excitatory postsynaptic potentials.
17 n net synaptic efficacy as measured by field excitatory postsynaptic potentials.
18  temporal summation of their thalamocortical excitatory postsynaptic potentials.
19 AC-induced depression of inhibitory, but not excitatory, postsynaptic potentials.
20 isted of two components: pEPSP1, (population excitatory postsynaptic potential 1) and pEPSP2.
21 ime-dependent manner, significantly reducing excitatory postsynaptic potentials after a >/=30-min app
22      A plot of impulse rates or amplitude of excitatory postsynaptic potentials against ITDs (ITD cur
23 pines compartmentalize voltage, specifically excitatory postsynaptic potentials, albeit critical, rem
24                                 We find that excitatory postsynaptic potential amplitudes are inverse
25               The slope and amplitude of the excitatory postsynaptic potential and the number of evok
26 ression of the simultaneously recorded field excitatory postsynaptic potential and was greatly reduce
27                              Uncaging-evoked excitatory postsynaptic potentials and Ca transients are
28 pagating action potentials, the amplitude of excitatory postsynaptic potentials and dendritic excitab
29 erm potentiation (LTP) of both intracellular excitatory postsynaptic potentials and evoked field pote
30 by tetrodotoxin, and all display spontaneous excitatory postsynaptic potentials and IPSPs that remain
31 simultaneously recording the uncaging-evoked excitatory postsynaptic potentials and local Ca2+ signal
32 on in FB-m1KO hippocampal slices resulted in excitatory postsynaptic potentials and long-term synapti
33                                 Granule cell excitatory postsynaptic potentials and mitral cell inhib
34                   ASIC null mice had reduced excitatory postsynaptic potentials and NMDA receptor act
35 ) using voltage-sensitive dye imaging, field excitatory postsynaptic potentials and whole cell patch
36  shape and duration of the vestibular-evoked excitatory postsynaptic potential, and the time of onset
37 y depends on the magnitude and timing of the excitatory postsynaptic potentials, and that blockade of
38  action potentials fully invade spines, that excitatory postsynaptic potentials are large in the spin
39 enhancement of the monosynaptic sensorimotor excitatory postsynaptic potential, as compared with the
40 owever, the initial increase in the slope of excitatory postsynaptic potentials, as well as the eleva
41              Amh exposure also increased the excitatory postsynaptic potential at CA1 synapses.
42     Consistent with that idea, PTP of evoked excitatory postsynaptic potentials at Aplysia sensory-mo
43 ow dynamic range (1 log unit intensity), and excitatory postsynaptic potentials at light on and light
44 ment of mGlu II-mediated depression of field excitatory postsynaptic potentials at mossy fiber-CA3 sy
45 oes the low input resistance make individual excitatory postsynaptic potentials brief so that they mu
46 s PF-PC excitatory postsynaptic currents and excitatory postsynaptic potentials by 15-20%.
47 measured as the change in layer II/III field excitatory postsynaptic potentials by a multielectrode a
48 he amplitude and sharpens the time course of excitatory postsynaptic potentials by reducing current s
49 ow that reliably timed action potentials (or excitatory postsynaptic potentials) can be observed up t
50 nterneurons to the extent that even a single excitatory postsynaptic potential could initiate spiking
51 other neurons in the ARC and that all evoked excitatory postsynaptic potentials could be blocked by t
52 ion to neurotransmitter release, feedforward excitatory postsynaptic potentials could spread through
53 uency sensitivity of NMDA receptor-dependent excitatory postsynaptic potentials differed significantl
54 quency voltage fluctuations (presumed "field excitatory postsynaptic potentials") during 89% of chron
55  CA1 are associated with an increase in both excitatory postsynaptic potential (EPSP) amplitude and a
56 an excitatory action while also reducing the excitatory postsynaptic potential (EPSP) amplitude throu
57 extracellular stimulation experiments to map excitatory postsynaptic potential (EPSP) amplitudes and
58 llaterals depressed the initial slope of the excitatory postsynaptic potential (EPSP) in aged but not
59  a small amplitude, short latency population excitatory postsynaptic potential (EPSP) in the PRh.
60 cal pyramidal neurons, we observed an evoked excitatory postsynaptic potential (EPSP) or current (EPS
61 c population spike (PS) and minor effects on excitatory postsynaptic potential (EPSP) slope amplitude
62 mplitude was by 72+/-17% of control, and the excitatory postsynaptic potential (EPSP) slope was decre
63  IPSP, the amplitude of the cholinergic fast excitatory postsynaptic potential (EPSP) was partially i
64 tic potential (IPSP) is abolished before the excitatory postsynaptic potential (EPSP) when the extrac
65 postsynaptic potential (IPSP) followed by an excitatory postsynaptic potential (EPSP) whereas CSS onl
66 increase in stimulus intensity evoked a slow excitatory postsynaptic potential (EPSP) which was assoc
67 rimary afferent fibers-mediated monosynaptic excitatory postsynaptic potential (EPSP), and long-lasti
68 edback loop within the spine head; during an excitatory postsynaptic potential (EPSP), Ca(2+) influx
69 eta more potently blocks the potentiation of excitatory postsynaptic potential (EPSP)-spike coupling
70 arization of summating the NMDA component of excitatory postsynaptic potential (EPSP).
71 solitarii (TS) stimulation with a monophasic excitatory postsynaptic potential (EPSP).
72  depression reported for the I a-motoneurone excitatory postsynaptic potential (EPSP).
73 ribute to hippocampal CA(1) stratum radiatum excitatory postsynaptic potentials (EPSP) is a matter of
74 -methyl-D-aspartate (NMDA) receptor-mediated excitatory postsynaptic potentials (EPSPAs and EPSPNs, r
75  facilitation' of the NMDA receptor-mediated excitatory postsynaptic potential (EPSPN).
76 at activation of K(v)1 channels by dendritic excitatory postsynaptic potentials (EPSPs) accelerated m
77     As a result, subthreshold parallel fiber excitatory postsynaptic potentials (EPSPs) activate Cav3
78 fects Ca(2+) elevations evoked by coincident excitatory postsynaptic potentials (EPSPs) and back-prop
79 s the integration time window of concomitant excitatory postsynaptic potentials (EPSPs) and dampens t
80 h that their activity modulates the shape of excitatory postsynaptic potentials (EPSPs) and increases
81 d temperature-insensitive neurones displayed excitatory postsynaptic potentials (EPSPs) and inhibitor
82 0 nM) increased the frequency of spontaneous excitatory postsynaptic potentials (EPSPs) and spontaneo
83 ked compound glutamatergic receptor-mediated excitatory postsynaptic potentials (EPSPs) and spontaneo
84 nd rat axons, but the forward propagation of excitatory postsynaptic potentials (EPSPs) and the backw
85 unisensory and multisensory stimulation with excitatory postsynaptic potentials (EPSPs) and/or action
86 uch a model: it is reversible, anti-hebbian (excitatory postsynaptic potentials (EPSPs) are depressed
87                                              Excitatory postsynaptic potentials (EPSPs) are greatly p
88 ification (increase in duration and area) of excitatory postsynaptic potentials (EPSPs) at depolarize
89                      After the depression of excitatory postsynaptic potentials (EPSPs) by 60 min of
90                      The depolarization from excitatory postsynaptic potentials (EPSPs) can inactivat
91 N-methyl-D-aspartate (NMDA) and compared the excitatory postsynaptic potentials (EPSPs) elicited by N
92  the observed EPSC changes, we also compared excitatory postsynaptic potentials (EPSPs) elicited by P
93                                              Excitatory postsynaptic potentials (EPSPs) elicited by s
94 cally connected cells were used to study the excitatory postsynaptic potentials (EPSPs) elicited in b
95 tion of oxytocin (1 and 10 microM) inhibited excitatory postsynaptic potentials (EPSPs) evoked by dor
96 upling determined the degree of summation of excitatory postsynaptic potentials (EPSPs) evoked by eac
97 ave performed a detailed quantal analysis of excitatory postsynaptic potentials (EPSPs) evoked by min
98                                              Excitatory postsynaptic potentials (EPSPs) evoked by pre
99 Specifically, we sought to determine whether excitatory postsynaptic potentials (EPSPs) evoked by sti
100             The width, area and rise time of excitatory postsynaptic potentials (EPSPs) evoked by sti
101 ibly decreased the amplitude of monosynaptic excitatory postsynaptic potentials (EPSPs) evoked in BA1
102 ne, but resembled asynchronous glutamatergic excitatory postsynaptic potentials (EPSPs) evoked in the
103 of the L-MAN and the HVc fiber tracts evoked excitatory postsynaptic potentials (EPSPs) from >70% of
104       Peripheral noxious stimulation induced excitatory postsynaptic potentials (EPSPs) in CA1 pyrami
105 vin-binding protein ApVAP33 inhibited evoked excitatory postsynaptic potentials (EPSPs) in cultured c
106 ulated VPM or POm axons, and recorded evoked excitatory postsynaptic potentials (EPSPs) in different
107 ge-gated A-type Kv4 channels shape dendritic excitatory postsynaptic potentials (EPSPs) in hippocampa
108 nels on dendritic processing of subthreshold excitatory postsynaptic potentials (EPSPs) in mouse CA1
109 on evoked faster rising and shorter duration excitatory postsynaptic potentials (EPSPs) in MSNs (n =
110  channels) modulate excitability and curtail excitatory postsynaptic potentials (EPSPs) in neuronal d
111  evoked abrupt increases in the frequency of excitatory postsynaptic potentials (EPSPs) in two thirds
112                            LTP occurred when excitatory postsynaptic potentials (EPSPs) led single po
113 lls, spikes evoked 5 ms after parallel-fiber excitatory postsynaptic potentials (EPSPs) led to long-t
114                                  We recorded excitatory postsynaptic potentials (EPSPs) of regular (n
115 te or pyramidal cells) (n = 6) produce large excitatory postsynaptic potentials (EPSPs) of up to 4 mV
116 ormed to investigate the interaction between excitatory postsynaptic potentials (EPSPs) or currents (
117 th the mean peak amplitudes of the resulting excitatory postsynaptic potentials (EPSPs) ranging betwe
118 es the amplitude of two-photon (2P) uncaging excitatory postsynaptic potentials (EPSPs) recorded at t
119                          The visually evoked excitatory postsynaptic potentials (EPSPs) recorded duri
120           Here, by monitoring spine size and excitatory postsynaptic potentials (EPSPs) simultaneousl
121 rm manner; 85% of these cells responded with excitatory postsynaptic potentials (EPSPs) that maintain
122 rong stimulus by increasing the frequency of excitatory postsynaptic potentials (EPSPs) to TC cells,
123   In CA3 neurons, the amplitude and slope of excitatory postsynaptic potentials (EPSPs) transiently d
124                       Whether single unitary excitatory postsynaptic potentials (EPSPs) trigger spike
125                               Simulations of excitatory postsynaptic potentials (EPSPs) were analysed
126                                              Excitatory postsynaptic potentials (EPSPs) were elicited
127                       Short-latency (< 5 ms) excitatory postsynaptic potentials (EPSPs) were evoked i
128                                              Excitatory postsynaptic potentials (EPSPs) were evoked u
129 4) rats, TBS failed to induce LTP unless the excitatory postsynaptic potentials (EPSPs) were of suffi
130                           When low-amplitude excitatory postsynaptic potentials (EPSPs) were paired w
131                      Pairing of subthreshold excitatory postsynaptic potentials (EPSPs) with back-pro
132 d excitatory postsynaptic currents and field excitatory postsynaptic potentials (EPSPs) with threshol
133 es (paired-pulse facilitation, small initial excitatory postsynaptic potentials (EPSPs), a graded act
134 the endogenous release of glutamate, mediate excitatory postsynaptic potentials (EPSPs), alter presyn
135 hose temporal periods, and time constants of excitatory postsynaptic potentials (EPSPs), both increas
136 ential discharges to previously subthreshold excitatory postsynaptic potentials (EPSPs), even though
137 ulation of forward input evoked monosynaptic excitatory postsynaptic potentials (EPSPs), followed by
138 tential (AP) output and synaptic inputs, via excitatory postsynaptic potentials (EPSPs).
139 mplitude of the second of two rapidly evoked excitatory postsynaptic potentials (EPSPs).
140 uctances have previously been shown to boost excitatory postsynaptic potentials (EPSPs).
141 n of cholinergic axons evoked nicotinic fast excitatory postsynaptic potentials (EPSPs).
142  the auditory nerve excited Golgi cells with excitatory postsynaptic potentials (EPSPs).
143            Each event was supported by giant excitatory postsynaptic potentials (EPSPs).
144 reatment had any significant effect on field excitatory postsynaptic potentials (EPSPs).
145 icators (GEVIs) is the reliable detection of excitatory postsynaptic potentials (EPSPs).
146 olarizations than do dendritic shafts during excitatory postsynaptic potentials (EPSPs).
147  evoked bAPs, some of which were paired with excitatory postsynaptic potentials (EPSPs).
148 A) caused a dose-related reduction in evoked excitatory postsynaptic potentials (EPSPs).
149 gically isolated N-methyl-d-aspartate (NMDA) excitatory postsynaptic potentials (EPSPs).
150 rate at which each source was bombarded with excitatory postsynaptic potentials (EPSPs); and (3) the
151 ort-term depression, where successive evoked excitatory postsynaptic potentials (EPSPs; >5 Hz) usuall
152 anism depends on a passively conducted giant excitatory postsynaptic potential evoked by a mossy fibe
153 indle afferent population was estimated from excitatory postsynaptic potentials evoked by muscle stre
154                            Analysis of field excitatory postsynaptic potentials evoked by stimulation
155 tocol altered both the [Zn2+]t and the field excitatory postsynaptic potential (fEPSP) coordinately,
156 mined the cornu ammonis region 1 (CA1) field excitatory postsynaptic potential (fEPSP) response to co
157              In this work, we recorded field excitatory postsynaptic potentials (fEPSP) in the CA1 re
158 tion had different actions on both the field excitatory postsynaptic potentials (fEPSPS) and LTP in t
159 (2R,6R)-HNK were examined by recording field excitatory postsynaptic potentials (fEPSPs) and miniatur
160 s of isoflurane were similar on evoked field excitatory postsynaptic potentials (fEPSPs) and paired p
161 P) acting at P2 receptors mediates some fast excitatory postsynaptic potentials (fEPSPs) in myenteric
162      Intracellular recordings of evoked fast excitatory postsynaptic potentials (fEPSPs) in myenteric
163  acts at P2X receptors to contribute to fast excitatory postsynaptic potentials (fEPSPs) in myenteric
164 lethality in mice, (ii) NMDAR-mediated field excitatory postsynaptic potentials (fEPSPs) in the CA1 f
165 ically isolated NMDA-receptor-mediated field excitatory postsynaptic potentials (fEPSPs) in the CA1 r
166                             Spontaneous fast excitatory postsynaptic potentials (FEPSPs) occurred in
167                                        Field excitatory postsynaptic potentials (fEPSPs) or populatio
168 red oxygen and the amplitude of evoked field excitatory postsynaptic potentials (fEPSPs) recorded fro
169 ted one or two populations of nicotinic fast excitatory postsynaptic potentials (fEPSPs) that were gr
170                                        Field excitatory postsynaptic potentials (fEPSPs) were recorde
171                                        Field excitatory postsynaptic potentials (fEPSPs) were recorde
172                                        Field excitatory postsynaptic potentials (fEPSPs) were recorde
173  while in one AH-cell, some spontaneous fast excitatory postsynaptic potentials (FEPSPs) were recorde
174 perpolarizing pulse and (v) spontaneous fast excitatory postsynaptic potentials (FEPSPs).
175 oved the recovery of Schaffer collateral-CA1 excitatory postsynaptic potentials following a 15 min hy
176 potential, as compared with the sensorimotor excitatory postsynaptic potential in preparations that r
177 gulation of a prominent thalamocortical NMDA excitatory postsynaptic potential in stellate cells regu
178                         The S cell-triggered excitatory postsynaptic potential in the R cell diminish
179 n fact, both NMDAR- and AMPAR-mediated field excitatory postsynaptic potentials in CA1 decrease with
180 rlier reversal to polarizing currents of ATD excitatory postsynaptic potentials in comparison to thos
181 y correlated with both the onset of compound excitatory postsynaptic potentials in fast-spiking inter
182 d the size (amplitude and duration) of field excitatory postsynaptic potentials in hippocampal slices
183              Stimulation of the pTRG induced excitatory postsynaptic potentials in ipsi- and contrala
184    Excitatory stimuli drove trains of single excitatory postsynaptic potentials in relay cells, but g
185                             The light-evoked excitatory postsynaptic potentials in some types were re
186 A1 pyramidal neuron selectively inhibits the excitatory postsynaptic potentials in that cell.
187  optic tract stimulation routinely evoked an excitatory postsynaptic potential/inhibitory postsynapti
188                                        Field excitatory postsynaptic potentials, input/output curves,
189                                              Excitatory postsynaptic potential-like voltage commands
190  cytosolic glutamate and decreased miniature excitatory postsynaptic potential (mEPSC) frequency.
191 e course of NMDA receptor-mediated miniature excitatory postsynaptic potentials (mEPSPs).
192 rkinje cell TRPC3 channels underlie the slow excitatory postsynaptic potential observed after paralle
193 uced a similar biphasic modulatory action on excitatory postsynaptic potentials or currents (EPSPs/EP
194 rated predominantly by synchronised cortical excitatory postsynaptic potentials oscillating at freque
195 of the rising phase of the evoked population excitatory postsynaptic potential (pEPSP).
196 pecific dissociation of synaptic [population excitatory postsynaptic potential (pEPSP)] and cellular
197 omega-conotoxin GVIA (CTX) on the population excitatory postsynaptic potentials (pEPSP) in stratum ra
198          Extracellularly-recorded population excitatory postsynaptic potentials (pEPSPs) in stratum r
199  synapse density), we determined the average excitatory postsynaptic potential per synapse.
200 reduces the amplitude of two-photon uncaging excitatory postsynaptic potentials recorded at the soma.
201                                         Fast excitatory postsynaptic potentials recorded from S neuro
202  contributions are reduced because the early excitatory postsynaptic potential retards the opening of
203               Repetitive stimuli evoked slow excitatory postsynaptic potentials (SEPSPs) in some toni
204        To address this question, spontaneous excitatory postsynaptic potentials (sEPSPs) were recorde
205 urone excitability and/or contribute to slow excitatory postsynaptic potentials (sEPSPs).
206 nule cells NMDA receptors are increased, and excitatory postsynaptic potentials should be strongly NM
207 onstrated that ethanol inhibited NMDAR field excitatory postsynaptic potential slope and amplitude to
208 from control mice and (ii) the plot of field excitatory postsynaptic potential slope versus the popul
209 l recording was used to study a type of slow excitatory postsynaptic potential (slow EPSP) that was m
210 hippocampal area CA3 sum distal and proximal excitatory postsynaptic potentials sublinearly and activ
211 aused a NMDA receptor-dependent, supralinear excitatory postsynaptic potential summation, indicating
212 oping nicotine-induced potentiation of field excitatory postsynaptic potential that appeared to be de
213 r and nonlinear mechanisms were caused by an excitatory postsynaptic potential that reversed near 0 m
214  to excitatory neurons, heterogeneity in the excitatory postsynaptic potentials that impinge on PV ne
215 lectrical stimulation generated monosynaptic excitatory postsynaptic potentials that were indistingui
216 d under minimal synaptic stimulation and the excitatory postsynaptic potentials they generate.
217 , and slow-wave sleep, produce a large field excitatory postsynaptic potential throughout stratum rad
218                       NMDA receptors mediate excitatory postsynaptic potentials throughout the brain
219            Simultaneously, we recorded field excitatory postsynaptic potentials to monitor changes in
220                   Furthermore, recordings of excitatory postsynaptic potential-to-spike coupling (E-S
221       This inhibitory postsynaptic potential-excitatory postsynaptic potential transformation was pre
222 dual spines while monitoring uncaging-evoked excitatory postsynaptic potentials (uEPSPs) and Ca trans
223                                      Unitary excitatory postsynaptic potentials (uEPSPs) revealed a h
224 nstant for STP of the AMPA receptor-mediated excitatory postsynaptic potential was approximately 6 mi
225  and ethanol's inhibition of the NMDAR field excitatory postsynaptic potential was attenuated by a br
226 s were deleted using CRISPR/SaCas9, the slow excitatory postsynaptic potential was eliminated.
227 at this time, indicating that the underlying excitatory postsynaptic potential was more probable, but
228 nstant for STP of the NMDA receptor-mediated excitatory postsynaptic potential was only 1 min.
229               The SK channel contribution to excitatory postsynaptic potentials was absent in SK2-S o
230    High-frequency summation of AMPA-mediated excitatory postsynaptic potentials was smaller in OT neu
231  their activation during simulated dendritic excitatory postsynaptic potential waveforms.
232       Spontaneous depolarizations resembling excitatory postsynaptic potentials were observed at E12.
233 lateral pathway was stimulated and the field excitatory postsynaptic potentials were recorded in the
234 in post-tetanization the slopes of the field excitatory postsynaptic potentials were significantly di
235 red synaptic inputs as the amplitude of fast excitatory postsynaptic potentials were significantly la
236 ion that increase the size of NMDAR-mediated excitatory postsynaptic potentials, whereas at high conc
237 acellular spike-triggered averaging revealed excitatory postsynaptic potentials, which confirmed thes
238 m-CPP enhances the occurrence of spontaneous excitatory postsynaptic potentials with no alteration in

 
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