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1 which appear to use glutamate as their fast excitatory transmitter.
2 the spinal interneurons coreleasing the two excitatory transmitters.
3 le substrate and may use inhibitory or other excitatory transmitters.
4 suggest that ischemia-evoked release of the excitatory transmitters amino acids is a result, in part
5 to an increased number of neurons expressing excitatory transmitters and a decreased number of neuron
6 by mechanisms requiring the dual actions of excitatory transmitters and NO derived either from inter
7 ia; moreover, the neuroprotective actions of excitatory transmitter antagonists are not limited to th
8 dpole provides a case where the two main CNS excitatory transmitters are released from single vesicle
10 modulation provides a mechanism by which an excitatory transmitter can induce a long-term increase i
11 ike older embryonic cord circuits, the major excitatory transmitter driving activity was not glutamat
13 hese data demonstrate exaggerated release of excitatory transmitter from primary afferents after inju
14 in the nerve endings caused by release of an excitatory transmitter from the glomus cell, which is a
16 set of mechanisms whereby the actions of two excitatory transmitters, GABA and glutamate, do not lead
17 ble for packaging the primary inhibitory and excitatory transmitters, gamma-aminobutyric acid (GABA)
19 Elevated extracellular concentrations of the excitatory transmitter glutamate are an important cause
20 er, synaptic vesicle uptake of the principal excitatory transmitter glutamate depends on the electric
21 owever, vesicular transport of the principal excitatory transmitter glutamate depends primarily on me
22 ble for vesicular transport of the principal excitatory transmitter glutamate has remained unknown.
23 ory information to the spinal cord using the excitatory transmitter glutamate, a process that depends
24 a (VTA) neurons corelease dopamine (DA), the excitatory transmitter glutamate, and the inhibitory tra
25 corticofugal synapses primarily release the excitatory transmitter glutamate, many physiology studie
26 e we have compared background release of the excitatory transmitter, glutamate, and the inhibitory tr
27 l-time extracellular recordings of the major excitatory transmitter, glutamate, in the BLA of rats pe
29 with evidence for corelease of the two major excitatory transmitters, glutamate and acetylcholine (AC
30 ry neurons in the ARC and the identity of an excitatory transmitter have not been investigated physio
33 he possibility that PACAP may function as an excitatory transmitter in lumbosacral parasympathetic re
34 eurotransmitter pool of glutamate, the major excitatory transmitter in the mammalian central nervous
36 Glutamate is probably the most important excitatory transmitter in the vertebrate central nervous
37 ketones acutely impacts main inhibitory and excitatory transmitters in the whole fasting cortex, com
39 ns, whereas acetylcholine is most likely the excitatory transmitter of nonspiking type IIa1 local int
42 inal level was explored by measuring evoked, excitatory transmitter release in tissues taken from ner
45 ased presynaptic AP duration and facilitated excitatory transmitter release; which in turn enhanced s
48 horn of the spinal cord, evoking release of excitatory transmitters such as glutamate onto postsynap
49 rties of glutamate, the other, more abundant excitatory transmitter, to determine its receptor subtyp
50 AMPA and kainate channels activated by their excitatory transmitter, we examined outside-out patches