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1 of the life cycle throughout encystation and excystation.
2          Calmodulin antagonists also blocked excystation.
3 he resumption of motility and cytokinesis in excystation.
4 te a central role for calcium homeostasis in excystation.
5 nophores, and thapsigargin) strongly inhibit excystation.
6 tative growth and the cellular activation of excystation.
7 ntially preserved as mRNA in preparation for excystation.
8 ar late in encystation, and during and after excystation.
9 es that release their contents just prior to excystation.
10 capsules throughout sporogony and sporozoite excystation.
11  the harsh environment of the gut to undergo excystation.
12 uring development, including encystation and excystation.
13                       Cryptosporidium parvum excystation and host cell invasion have been characteriz
14       Interestingly, it was unable to induce excystation and increased trophozoite sensitivity to APC
15 scribes our understanding of Cryptosporidium excystation and the events leading to host cell invasion
16 ot after, stage II was sufficient to inhibit excystation, and inhibition could be partially reversed
17                              Encystation and excystation are crucial processes for the survival and t
18 ogy, as high-grade regulated encystation and excystation are readily achievable.
19 requires sporozoite egress from the capsule (excystation), but this process remains poorly understood
20  or to wheat germ agglutinin (WGA) inhibited excystation by > 90%.
21  host cell invasion and egress, encystation, excystation, catabolism of host proteins, differentiatio
22 a(2+) responses increased with each stage of excystation, consistent with the kinetics of inhibition.
23                               Microscopy and excystation experiments were conducted to support the di
24 tion of macrophages in supporting sporozoite excystation following oocyst internalisation.
25 station induced by bile and alkaline pH, and excystation in response to gastric pH followed by alkali
26         We have been able to reliably induce excystation in vitro by mimicking cyst passage through t
27 uired for survival outside the host, whereas excystation initiates infection.
28 arasite development using in vitro models of excystation, invasion, proliferation, and egress.
29                                              Excystation is blocked by specific cysteine protease inh
30                                     Although excystation is crucial to the initiation of infection by
31       Antigenic variation during encystation-excystation may be a novel form of immune evasion that c
32                      Our findings support an excystation model based on local, rather than global, we
33 ands, which bind cyst wall epitopes, inhibit excystation, most likely by interfering with proteolysis
34 ite- and host-specific molecules involved in excystation, motility and host cell invasion have been p
35 endent of cellular stress and, except during excystation, occurs throughout the G. lamblia life cycle
36 late (NaTC) are routinely used to induce the excystation of Cryptosporidium oocysts.
37                                              Excystation of Giardia lamblia, which initiates infectio
38 xpression did not affect parasite viability, excystation, or invasion of target cells.
39 in morphological modifications and decreased excystation rates of sporozoites.
40 he sporocyst cavity of the oocysts and after excystation, secretion was detected in culture supernata
41 ts, but is downregulated during the stage of excystation that models cyst arrival in the small intest
42                    Immuno-pathogenesis after excystation was similarly well advanced by the use of a
43 est that protein kinase A signaling triggers excystation, whereas calcium signaling is mainly require
44 ylated PKA inhibitor, amide 14-22, inhibited excystation with an IC(50) of 3 microm, suggesting an im