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1 CI, -0.047 to -0.011; p = 0.002), attention/executive (-0.020; 95% CI, -0.037 to -0.004; p = 0.016),
3 white or gray matter changes in the frontal-executive and corticolimbic circuitries as those with aM
4 erations in white and gray matter of frontal-executive and corticolimbic circuitries in five groups o
5 al volumes and cortical thickness in frontal-executive and corticolimbic regions of interest (ROIs).
7 or phone meetings with >50 federal staff in executive and legislative roles, as well as with a varie
10 ng a multimodal approach should motivate all executives and health care providers to support further
12 mics of population activity in a high-level, executive area - dorsolateral prefrontal cortex (dlPFC)
13 etween tinnitus and sustained, selective and executive attentions as well as response inhibition.
14 structural and functional specialization of executive brain systems that mediates cognitive maturati
15 s identifying as veterans in the civilian US executive branch exceeds the proportion in the wider pop
17 filtering can involve distinct automatic and executive circuit mechanisms, however, developing circui
18 lic blood pressure was associated with worse executive cognitive function in mid-life (44-69 years),
20 can Association of Anthropological Genetics, Executive Committee of the American Association of Physi
24 n canonical networks implicated in cognitive/executive control (frontoparietal, medial frontal) and i
25 rocessing domains, including those linked to executive control (N2b responses) and stimulus classific
26 to performance monitoring, all reflective of executive control abilities, and our surrogate for atten
27 onstrate that performance trade-offs between executive control and long-term semantic knowledge are l
28 data indicate that increased recruitment of executive control areas in pediatric T1D may act to offs
29 behavioral repertoire of remarkably similar executive control generated by distinctly different brai
30 te functional connectivity (RSFC) within the executive control network (ECN) which has been implicate
31 the superior longitudinal fasciculus in the executive control network better explains executive dysf
32 P and Th in humans are part of a subcortical executive control network, differentially involved in mo
34 ulation of activity between default mode and executive control networks play a role in improvisationa
37 er, heteromodal sensorimotor processing, and executive control of motor commands in dystonia pathophy
38 goal-directed behavioural systems, and when executive control over this maladaptive behaviour is dim
39 n with T1D exhibited increased activation in executive control regions (e.g., dorsolateral prefrontal
40 with greater increases in hyperactivation of executive control regions (T1D: r = 0.284, 95% CI 0.08 t
45 egulation of cortical activity in attention, executive control, and perceptual decision-making, but t
46 tworks were identified supporting attention, executive control, motor, visual, and default-mode funct
47 primarily on semantic knowledge, rather than executive control, was linked to a neural functional org
48 presentation, rather than those dependent on executive control, were associated with patterns of thou
54 and gray matter density in areas involved in executive (cortical regions) and integrative (bilateral
55 o the midbrain from sensory, behavioral, and executive cortices.SIGNIFICANCE STATEMENT Making sense o
58 ng measure is sensitive for the detection of executive deficits since the early phases of the disease
61 nts completed a 23-item survey on memory and executive domains from the Everyday Cognition (ECog) sca
65 s associated with depressive-like behaviors, executive dysfunction and poor response to antidepressan
66 tter integrity (r = - 0.74, p < 0.001), than executive dysfunction and stroke severity (r = 0.22, p <
67 ns confirmed a stronger relationship between executive dysfunction and white matter integrity (r = -
70 groups, consisting of anxiety/depression and executive dysfunction symptoms, respectively, that corre
71 cerebrovascular risk factors better explain executive dysfunction than markers of stroke severity.
72 he executive control network better explains executive dysfunction than markers of stroke severity.
73 60 years and older with major depression and executive dysfunction were randomized to 12 weeks of eit
74 ical dysfunction, defined as the presence of executive dysfunction, impaired processing speed, person
76 psychotherapy for late-life depression with executive dysfunction; (2) examine if nonresponse by mid
78 PFC) and (ii) dorsal frontoparietal "central executive" (FPN) network anchored in right dorsolateral
79 memory (0.21 [95% CI 0.08-0.34], p = 0.001), executive function (0.21 [95% CI 0.06-0.35], p = 0.006),
80 Food insecurity was associated with poorer executive function (b = -1.45, SE = 0.58, P <= 0.01) and
86 and leg lean mass (LM), muscle strength, and executive function (multiple primary outcomes), as well
87 Symbol Digit Modalities Test (R(2)=0.31) and executive function (R(2)=0.36) test scores, independent
89 ; accuracy: P = 0.98, Cohen's d = 0.07), and executive function (speed: P = 0.60, Cohen's d = 0.31; a
90 Conner's Continuous Performance Test-II) and executive function (Trail Making Test Part B, Controlled
91 g and back muscle strength (26% to 40%), and executive function (z-score SD: 0.33 to 0.39), nor the s
96 -limbic circuits), cognitive deficits (e.g., executive function and memory impairments), affective in
98 ia leading to brain injury, further impaired executive function and mental health, which results in s
99 tical dopaminergic abnormalities in impaired executive function and relapse in cocaine dependence.
100 cleus and anterior putamen, overlapping with executive function and social/language regions of the st
101 across multiple cognitive domains, including executive function and speed of information processing.
103 ly to all brain areas (for example, motor or executive function areas) the success of NSC-based model
104 motional reactivity at 7, 15, and 24 months, executive function at 36, 48, and 60 months, and emotion
105 ce was attributable to less deterioration in executive function at 4 months (23.3% v 40.4%; P = .01)
106 l reactivity in toddlerhood, followed by low executive function at school entry and high emotional an
109 site of cognitive tests including memory and executive function differed between biomarker groups.
113 llocatequin-3-gallate (EGCG), which improves executive function in young adults with DS and Ts65Dn mi
114 will address the topic of sex differences in executive function including a discussion of differences
115 pause-related deficits in working memory, an executive function mediated by the dorsolateral prefront
117 ry (+ 9.5%), processing efficiency (+ 7.5%), executive function reaction time (- 4.8%) and fluid inte
118 E4 carriers, lower OEF correlated with lower executive function scores (b = 0.079 z score for each pe
120 inhibition, assessed using the Delis-Kaplan Executive Function System Color-Word Interference ["Stro
123 he complex cognitive processes of memory and executive function that deteriorate in Alzheimer's disea
125 cognitive domains of memory, attention, and executive function to classify whether participants had
127 hibited relative deficits in psychomotor and executive function with fewer deficits in memory and lea
128 for the Assessment of Neurologic Status and executive function with the Trail Making Test Part B.
130 was also associated with hippocampal volume, executive function, and age-at-onset only among males.
131 limited on how phthalates affect cognition, executive function, and behavioral function into adolesc
132 ng function), cognitive performance (memory, executive function, and processing speed), emotional wel
136 test battery, we assessed processing speed, executive function, anxiety, depression and disease seve
137 measure cortical activation during tasks of executive function, attention, social cognition, and lan
138 centive salience, negative emotionality, and executive function, could be identified through factor a
139 g domain-specific composite scores including executive function, episodic memory, visual-spatial proc
140 n, language, learning, memory, visuospatial, executive function, information processing, psychomotor
141 eterioration in functions such as memory and executive function, is faced by most older adults and af
142 (episodic memory, attention/working memory, executive function, language/semantic memory, and global
143 ience/habits, negative emotional states, and executive function, mediated by the basal ganglia, exten
144 eral cognitive performance, processing speed/executive function, memory) in later life (after age 55
145 ering domains of attention/processing speed, executive function, memory, language, and visuospatial f
146 d are associated with deficits in cognition, executive function, memory, vision, hearing, motor skill
147 ve neuropsychological test battery assessing executive function, processing speed, attention/working
149 essing and regulation, as well as memory and executive function, some of which show trends of associa
150 activation profiles in regions critical for executive function, which mirrored differences in cognit
151 illustrated the complex relationship between executive function, white matter integrity, stroke chara
167 tarted cART at an older age deviated more in executive functioning (-0.13 z score, 95% CI -0.24 to -0
170 tive control is fundamental to healthy human executive functioning (Miller and Cohen, 2001) and defic
172 and with better parent-reported measures of executive functioning (r values < -0.29, 95% CIs -0.47 t
174 This study examined the relationship between executive functioning and adaptive behaviour in adolesce
175 regions, support age-related improvements in executive functioning and self-regulatory capacities in
177 9 to -1.53]; p = 0.08) months and with worse executive functioning at 3 (-3.61 [-7.48 to 0.26]; p = 0
178 vidence suggesting impairments in aspects of executive functioning but not psychomotor vigilance.
180 individuals reporting higher chronic stress, executive functioning decreased after dynamic conditions
181 interval [CI] 1.5-10.50; P = .012), whereas executive functioning decreased significantly more (grou
182 ion of the default mode network (DMN) during executive functioning have been observed in healthy and
186 ome (memory, language, processing speed, and executive functioning tests) and Mini-Mental State Exami
189 g speed, working memory, verbal fluency, and executive functioning) to assess ante mortem performance
190 ention, working memory, verbal learning, and executive functioning), were generally small (30 [40%] o
193 brain regions are involved in attention and executive functioning, and that these non-language domai
194 cortical thickness in regions implicated in executive functioning, attention, and memory after accou
195 ient (IQ), processing speed, working memory, executive functioning, learning ability, and visual-moto
196 had worse performance on several measures of executive functioning, MATRICS processing speed and MATR
197 s between cortical inhibition/excitation and executive functioning, or between any neurophysiological
199 eased response times and negatively impacted executive functioning, spatial planning and mental rotat
201 the basis of relative performance in memory, executive functioning, visuospatial functioning, and lan
206 s of cortical physiology and two measures of executive functioning: cognitive inhibition, assessed us
209 communication of frontal regions engaged in executive functions and emotion regulation represent dep
210 erformance in the domains of working memory, executive functions and procedural learning in healthy y
211 ognitive performance in the cognitive domain executive functions and processing speed (p = 0.027).
214 itive impairment was shown in the domains of executive functions and speed/attention and to a minor d
216 psychologists and neuroscientists still see executive functions as independent, domain-general, supe
217 (ERD/ERS) analysis, we investigated cortical executive functions during a Go/NoGo task in PD patients
218 ctual abilities, such as the broadly defined executive functions including working memory, the core p
219 th hypoxia-ischemia on brain development and executive functions is moderated by genotypes associated
220 uild on associative learning, and argue that executive functions might be better understood as cultur
222 ally if there are additionally reductions in executive functions or specific inhibitory control.
223 itive outcomes reported as the Attention and Executive Functions subscore of the Developmental Neurop
224 of cortico-striatal circuit hyperactivity on executive functions subserved by these circuits is uncle
225 esource-saving tools for early evaluation of executive functions using an ecological approach are nee
227 ol of thoughts and behaviours-the so-called 'executive functions' (for example, attention, inhibitory
228 ct of ALIC-NAcc-DBS on personality traits or executive functions, and no potential outcome predictors
230 gnitive functions, we tested working memory, executive functions, and several sub-processes of proced
231 tive control, serving as a core construct of executive functions, contributes substantially to genera
232 schizophrenia and contributes to deficits in executive functions, including working memory, attention
234 sis (ALS) exhibit mild cognitive deficits in executive functions, language and fluency, without demen
235 on in brain regions specifically involved in executive functions, language functions and verbal fluen
237 nt, we assessed attention, processing speed, executive functions, memory, emotions, and behavior with
238 ed with standard neuropsychological tests of executive functions, PIT 360 degrees , and measures of u
249 lateral frontoparietal network implicated in executive functions: the multiple demand (MD) network.
255 arying engagement of the SN with the central executive network and default mode network is a clinical
256 sFC within an emotion regulation and central executive network and replicate these associations with
258 ate fMRI evidence of large-scale sensory and executive network disruptions in youth with 22q11DS.
261 ons among the salience network (SN), central executive network, and default mode network contribute t
262 s-network interactions among the SN, central executive network, and default mode network in 130 patie
263 y within the primary cortices and within the executive network, but increased connectivity between th
265 l thickness in socio-emotional and attention/executive networks and inattention symptoms comprised th
267 ding the salience, default mode, and central executive networks were reduced in association with low-
272 hite-collar crimes, and companies with chief executive officers (CEOs) or chief financial officers (C
273 ng mixed methods (archival analyses of chief executive officers, field surveys in large US companies,
274 Obama Administration recalled property under Executive Order 13688, which resulted in a forced demili
275 r repeal these regulations, as encouraged by executive order 13813, issued in October 2017 by the Tru
277 ated with lower cognitive performance in the executive (P=0.021), memory (P=0.015), and global domain
278 tios in the right parahippocampal gyrus, the executive part of both putamina, both thalami, and the c
279 ateral prefrontal cortex was associated with executive performance and partially mediated age-related
280 ervices offered by USNWR "best hospitals" in executive physical packages, daylong comprehensive evalu
282 emands.SIGNIFICANCE STATEMENT Domain-general executive processes, such as working memory and cognitiv
285 ce on control tasks assessing perceptual and executive response demands, patients with posterior cort
286 oaches to cognitive remediation (training of executive skills and training of perceptual skills) and
287 D-19 hospitalizations before and after state executive stay-at-home orders issued in March and April
288 olved the functional cohesiveness of central executive subnetworks anchored in the frontoparietal cor
293 ted during math, logic, problem solving, and executive tasks, and the language system, typically recr
298 their domains of intervention, although only executive training resulted in improvement in neurocogni
299 of Oxford, Principal Investigator and Chief Executive, UK Biobank, and the British Heart Foundation