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1 mental adaptations such as fasting, cold, or exercise.
2 eflex (CR) on the cardiovascular response to exercise.
3 ined protection against steatosis induced by exercise.
4 ta) genes, in comparison with the once-daily exercise.
5 rease in blood-muscle O(2) flux from rest to exercise.
6 d to males during single-limb and whole-body exercise.
7 ts associated with heart rate recovery after exercise.
8 d after a controlled bout of symptom-limited exercise.
9 re evaluated before and during recovery from exercise.
10 ffect both endurance and resistance modes of exercise.
11 nges to physiological homeostasis, including exercise.
12 ient to maximize MyoPS rates after endurance exercise.
13 sible through systemic interventions such as exercise.
14  during rest, as well as submaximal and peak exercise.
15 e syncopal episodes that were not related to exercise.
16 muscles during recovery from plantar flexion exercise.
17 healthy females completed bouts of treadmill exercise.
18 xhibited highest levels of self-efficacy and exercise.
19 diac dysfunction and dilation in response to exercise.
20 s sustaining a greater relative intensity of exercise.
21 nd autonomic recovery subsequent to moderate exercise.
22 nutes also at rest, during recovery from the exercise.
23  heart rate recovery (HRR) following maximal exercise.
24 al muscle-specific cytokines, in response to exercise.
25 gramming that drives adaptation to endurance exercise.
26 tical methods and may include vision therapy exercises.
27 c steatosis compared with changes induced by exercise; 2) the impact of physical activity, exercise,
28 y was: normal everyday activities, 44 (40%); exercising, 33 (30%); concurrent illness, 13 (12%); slee
29                                              Exercise activates adenosine monophosphate-activated kin
30 in the musculoskeletal system, how long-term exercise affects stem cell aging, which is typified by r
31 nflammatory drugs, protonic pump inhibitors, exercise, alcohol, and fasting) were considered.
32  delivery were greater for HFrEF during peak exercise (all, P < 0.01), but not control.
33 dipose Pgc-1alpha transcript levels, whereas exercise alone was incapable of elevating Pgc-1alpha lev
34 nts on vitamin D/calcium supplementation and exercise alone.
35 sically trained and untrained subjects under exercise and after a protein-rich diet.
36 core clock gene expression, demonstrating an exercise and clock interaction, providing insight into p
37 ing interleukin 6 (IL-6) levels surge during exercise and IL-6 favors exercise capacity.
38  is sufficient in most situations, including exercise and often acute illness.
39 ery blood flow increased by ~43% during both exercise and passive heat stress, with no change in inte
40 ntion; (b) PEMF treatment; (c) exercise; (d) exercise and PEMF treatment.
41 e core priority treatments for OA, including exercise and physical activity, weight-loss, education a
42             This information may inform safe exercise and recovery protocols in asymptomatic male run
43 tic basis of resting Tpe and Tpe response to exercise and recovery, unveiling plausible candidate gen
44 scular consequences of these reflexes during exercise and revealed various modes of interaction.
45 erebral blood flow) is preserved during both exercise and temperature-matched passive heat stress ABS
46 ovascular coupling during submaximal cycling exercise and temperature-matched passive heat stress dur
47 l PCO2 was held constant) Submaximal cycling exercise and temperature-matched passive heat stress pro
48  our understanding of the health benefits of exercise and to provide insight into how physical activi
49 ng exercises including jaw exercises, tongue exercises and swallowing maneuvers with assistance and g
50 hose identified during this horizon scanning exercise, and (b) use of this methodology across the oth
51 xercise; 2) the impact of physical activity, exercise, and aerobic capacity compared with caloric res
52 ich type 2 diabetes is managed through diet, exercise, and medications only.
53 able protective effects of voluntary aerobic exercise, and the underlying mechanisms.
54 beta-blocker therapy and who limit strenuous exercise, aortic risk remains low when maximal aortic di
55 LPL, MTHFR, PCSK9, PNPLA3, PPARgamma2), gene-exercise (APOA1, APOA2, LPL), gene-diet (APOA5, APOE, IN
56 se findings indicate that lifetime endurance exercise appears to be extremely effective at preserving
57 f fatigability during critical power-matched exercise are mediated by sex.
58 ations focused on healthy eating and regular exercise are the primary recommendations for patients wi
59 nstrate that muscle contractions, as part of exercise, are sufficient to shift the muscle circadian c
60 s of age-related heart failure and highlight exercise as a valuable experimental platform for the dis
61 conductance were evaluated before and during exercise, as well as during recovery.
62 proteins in 12 participants before and after exercise at two physiologically defined intensities (mod
63                                              Exercise at ZT17, middle of the dark phase, did not alte
64                                     Patients exercised at both absolute (0, 5, 10, 15 W) and relative
65                                              Exercise-based interventions have been shown to be effec
66 ed a three round international expert Delphi exercise between April and June 2019 using REDCap to ide
67                                       During exercise, blood flow to working skeletal muscle increase
68    In Protocol 2, participants performed two exercise bouts in which muscle fibre recruitment was man
69 r lactate and lower pH than controls for all exercise bouts.
70 ing of pulmonary gas exchange during hypoxic exercise but had greater lactate and lower pH than contr
71  that lack of social activities and physical exercise can enable a relapse.
72 n these parameters following stress, such as exercise, can yield important information about imbalanc
73 action (HFrEF) exhibit severe limitations in exercise capacity ( VO2 peak).
74  exercise results in substantial benefits in exercise capacity ( VO2max ), cardiovascular function at
75 theter VSD closure prevents deterioration in exercise capacity and promotes left ventricular reverse
76 moves ventilation as the major constraint to exercise capacity in COPD, allowing maximal muscle funct
77 tor only in osteoblasts exhibit a deficit in exercise capacity of similar severity to the one seen in
78 chronic kidney disease (CKD) exhibit reduced exercise capacity, poor physical function and symptoms o
79      The EP group had significantly impaired exercise capacity.
80 vidual noninvasive FFR data acutely improves exercise capacity.
81 oglobin (HAH) affects maximal and submaximal exercise capacity.
82 elivery, thereby impairing VO2 peak and thus exercise capacity.
83 levels surge during exercise and IL-6 favors exercise capacity.
84                                    Fatiguing exercise causes hydrolysis of phosphocreatine, increasin
85      Habitual mechanical loading or targeted exercise causes tendon cells to increase the stiffness o
86 eak) ) with moderate-intensity constant-load exercise (CLE) at 75% WR(peak) , which yielded the same
87                         We hypothesized that exercise combined with non-invasive stimulation targetin
88           Results were compared with matched exercise controls at 22.5 degrees C.
89 HS and as late as 30 days compared with sham exercise controls.
90 (a) no intervention; (b) PEMF treatment; (c) exercise; (d) exercise and PEMF treatment.
91 not attenuated by mild or moderate intensity exercise (DeltaFVC: 5% MVC: -30 +/- 9; 15% MVC: -33 +/-
92                    Stretching was matched to exercise dosing arms on the basis of location, frequency
93        Approximately 40% of patients in both exercise dosing regimens were classified as VO(2)peak re
94 le absolute values of task oxygen uptake and exercise duration were lower and higher, respectively, i
95 ences with respect to chest tightness during exercise, dyspnoea and gender.
96          While it is generally accepted that exercise effectively combats the deleterious effects of
97 ological and physiological responses mediate exercise-elicited metabolic adaptations that maximize mu
98                                      Aerobic exercise elicits increases in cerebral blood flow (CBF),
99 ple of community-dwelling individuals, acute exercise elicits widespread changes in the circulating m
100                                              Exercise endurance time and total work output were almos
101 rate less force ex vivo, and exhibit reduced exercise endurance, associated with increased adiposity
102 tile function, and following heavy intensity exercise, females experienced less reduction in voluntar
103                 The combination of PEMFs and exercise for 6 weeks enhanced running performance and up
104 we highlight the importance of acute aerobic exercise for children with ADHD as a potential means to
105 or the beneficial cardiometabolic effects of exercise for future study.
106                     Although the efficacy of exercises for primary convergence insufficiency is plaus
107 rmacologic, and other interventions, such as exercise, for cancer cachexia.
108 nce exercise on cardiovascular structure and exercise function have been largely established in men.
109 ary pressure relative to cardiac output with exercise &gt;3 mm Hg/l/min.
110                                              Exercise has a wide range of systemic effects.
111 -wide molecular response to an acute bout of exercise has not been fully characterized.
112 ence of temperature on CBF regulation during exercise has not been investigated The present study ass
113 c signaling pathways responsible for raising exercise heart rate are impaired in HFpEF is unknown.
114                                              Exercise hemodynamic or confrontational fluid challenge
115 cial impact on the functional capacity of an exercising human.
116 ion entails the most functional impact on an exercising human.
117                                Intradialytic exercise (ID) programs are effective and safe for hemodi
118  patients with COPD, we compared an interval exercise (IE) protocol (alternating 30 s at 100% peak wo
119 riment, we tested the effect of PCMS without exercise in 13 individuals with spinal cord injury with
120  uptake and catabolism into myofibers during exercise in an osteocalcin-dependent manner.
121 in regulating blood flow and pressure during exercise in health, little is known about the role of ET
122 ailed map of the metabolic response to acute exercise in humans and identify potential mechanisms res
123 le of ET-1 in the cardiovascular response to exercise in hypertension.
124 prognostic value of B-line assessment during exercise in patients with HFpEF.
125 ing the delivery of oropharyngeal swallowing exercises including jaw exercises, tongue exercises and
126 pillaries actively vasodilating from rest to exercise, increased blood-myocyte O(2) flux occurs predo
127 e (T(c) ; +0.7-0.8 degrees C); however, such exercise increases cerebral blood flow (CBF; +10-20%) me
128 at stress ABSTRACT: Acute moderate-intensity exercise increases core temperature (T(c) ; +0.7-0.8 deg
129 -ene reactions and condensation reactions-to exercise independent control over both the shape forming
130    Mice that lack TLR9 are deficient in both exercise-induced activation of AMPK and plasma membrane
131 9)(4), and its interaction with beclin 1, in exercise-induced activation of AMPK in skeletal muscle.
132 undamental tendon cell functions relevant to exercise-induced adaptation and mechanotherapy.
133      Recent evidence indicates Bcl2 mediates exercise-induced autophagy and skeletal muscle adaptions
134 er rested-state total RNA (8.8%) and greater exercise-induced c-Myc mRNA expression (25%; Week 2, all
135 g accelerated coronary artery calcification, exercise-induced cardiac biomarker release, myocardial f
136 d clinical manifestations when compared with exercise-induced changes.
137 9 +/- 87 ml/min; P = 0.03) and augmented the exercise-induced hyperaemia at most intensities (80% sal
138  degrees C water-perfused suit) to match the exercise-induced increases in T(c) (EX: Delta0.75 +/- 0.
139 apitulating a genetic response implicated in exercise-induced metabolic adaptations.
140                                              Exercise-induced metabolite changes were variably relate
141 ody disease should be considered in cases of exercise-induced muscle stiffness.
142 oviding insight into potential mechanisms of exercise-induced phase shifts.
143                                 Detection of exercise-induced pulmonary congestion by lung ultrasound
144 se stress echocardiography; however, whether exercise-induced pulmonary congestion carries prognostic
145 2005-2006 NHANES adult population, increased exercise intensity is associated with decreased odds of
146 ry molecular mechanisms and the influence of exercise intensity remain poorly understood.
147                   Finally, application of an exercise intervention to a subset of PolgA(mut/mut) mice
148 for assessing aerobic capacity in studies on exercise intervention with untrained male Wistar rats.
149 e was ranked as the most effective among all exercise interventions (surface under cumulative ranking
150  is unclear what the most effective types of exercise interventions are.
151 on might have decreased adaptive response to exercise interventions.
152  peripheral mechanisms suggested to underlie exercise intolerance in HFrEF is excessive locomotor mus
153                                              Exercise intolerance is common in chronic obstructive pu
154                                              Exercise intolerance is prevalent among childhood cancer
155                                              Exercise intolerance was defined as peak oxygen uptake <
156                                              Exercise intolerance was present in 63.8% (95% CI, 62.0%
157                                              Exercise intolerance, associated with heart failure and
158 as, K(ATP) channel inhibition may exacerbate exercise intolerance.
159 ; 95% CI, 1.23 to 1.82) were associated with exercise intolerance.
160 ing the O(2) deficit and sowing the seeds of exercise intolerance.
161                                     Physical exercise is a powerful modulator of learning and memory.
162                                          How exercise is able to promote such systemic benefits has r
163                                              Exercise is among the most effective interventions for a
164                              Whereas regular exercise is associated with lower risk of cardiovascular
165  time of day at which short-duration maximal exercise is conducted represents an important variable f
166                                      Regular exercise is crucial for maintaining cognitive health thr
167                                    Endurance exercise is the most powerful intervention for promoting
168 y all individuals with greater agency do not exercise it, suggesting that merely knowing that one cou
169  peak effort during single-leg knee-extensor exercise (KE), where ventilation is assumed to be submax
170 ovascular function at submaximal and maximal exercise, left ventricular mass and compliance, and bloo
171 ales that were symptom-limited at much lower exercise levels and heat loads compared to females.
172                        Sleep-wake cycles and exercise levels were held constant.
173 ifferent protocols involving step changes in exercise load and compared the results to laboratory-bas
174  The sensor results reflected the changes in exercise load in real time.
175 sidering hemodynamics over the full range of exercise loads, PCWP was significantly reduced (-2.40 mm
176 hese, we speculate about which mechanisms of exercise may be universal across age groups and populati
177                                     Maternal exercise (ME) during pregnancy has been shown to improve
178 ascular disease and mortality, mechanisms of exercise-mediated health benefits remain less clear.
179           In the event that a broad spectrum exercise mimetic might ever be developed, we discuss som
180 n, Delirium monitoring/management, and Early exercise/mobility (ABCDE) bundle processes as improving
181 (1.3) years) performed moderate- (3 sets per exercise, MOD) and low-volume (1 set, LOW) resistance tr
182                                              Exercise modulates metabolism and the gut microbiome.
183  African women (n = 45) were randomized into exercise (n = 23) or control (n = 22) groups.
184 ed at rest, followed by a submaximal aerobic exercise on a treadmill and then remaining seated for 60
185          MFMD did not enhance the effects of exercise on any measures of muscle strength, gait speed,
186  sustained or lifelong (>25 years) endurance exercise on cardiovascular structure and exercise functi
187 that the beneficial effects of acute aerobic exercise on inhibitory control are sustained for 60 min
188 ments are promoted to augment the effects of exercise on muscle mass and strength, but their effectiv
189               The etiology of the effects of exercise on tumor progression are unclear, as are the ce
190 sure (1.5 mT, 10 min/week), with and without exercise, on systemic metabolism and gut microbiome in f
191 ttent claudication, compared with supervised exercise only.
192 (CP) and speed of the VO2 kinetics following exercise onset.
193 ail, screening for fall risk, and a targeted exercise or multifactorial intervention to prevent falls
194 5), heart rate (HR) at rest (p = 0.003), and exercise (p < 0.04) increased, whereas HR variability (H
195 E/e', and BNP significantly increased during exercise (P<0.001 for all).
196                                     For each exercise, patients showed higher VE/VCO(2) ratio, percen
197        Time-of-day dependent fluctuations in exercise performance have been documented across differe
198 r, the time-of-day effects on short duration exercise performance may be minimized by the following f
199 entation with nitrate can improve submaximal exercise performance.
200 anges were variably related to the amount of exercise performed (peak workload), sex, and body mass i
201 protective metabolites in women despite less exercise performed.
202                        Immediately after the exercise, perfusion was elevated to 79.3 +/- 9 mL/100 g/
203 ons of the dynamical statistical analysis in exercise physiology and cardiology, and the presented me
204        Finally, given that some calls for an exercise pill stem from a response to the perceived fail
205 Effective treatments include weight loss and exercise, positive airway pressure, oral appliances that
206 to eliminate carbon dioxide, VE/VCO2) during exercise potently predicts outcomes in advanced heart fa
207 e investigated the interactive effect of the exercise pressor reflex (EPR) and the chemoreflex (CR) o
208 al role in modifying the exaggeration of the exercise pressor reflex in PAD and a reduction in the ac
209 T) has beneficial effects on the exaggerated exercise pressor reflex in rats with peripheral artery d
210 s transduce nociceptive signals and modulate exercise pressor reflexes (EPRs).
211                             Lifelong aerobic exercise prevented age- and WD-related vascular dysfunct
212  Then the subjects performed plantar flexion exercise producing a torque of ~8ft-lb.
213                   This study shows that this exercise program can maintain general cognitive function
214 fects of 16 weeks of ID exercise versus a HB exercise program for HD patients.
215 he feasibility and effectiveness of a 6-week exercise program in patients with cirrhotic liver diseas
216 patients awaiting LT in an intensive aerobic exercise program with a signal of improvement in fitness
217 mobilisation techniques and a graduated home exercise programme supplemented by a steroid injection.
218 4KO) and subjected them to an intensive swim exercise protocol as well as transverse aortic constrict
219                    The duration and specific exercises provided to patients were also recorded.
220                                              Exercise provides a robust physiological stimulus that e
221                      Unhealthy diet, lack of exercise, psychosocial stress, and insufficient sleep ar
222 e ensuing hyperaminoacidemia) and resistance exercise (RE).
223 more nights with awakenings and more days of exercise-related symptoms (both P < .05).
224 evidence suggests muscle contractions during exercise release factors into the blood which cross into
225 owing 12-weeks of exercise training implying exercise resistance in this cohort.
226 variants for resting Tpe and Tpe response to exercise, respectively, were formally replicated.
227 e to delineate the metabolic architecture of exercise response patterns in humans.
228     We identified 4 metabolite signatures of exercise response patterns that were then analyzed in a
229                                              Exercise restriction, beta-blockers, and surgical interv
230 tted (>=4 weekly exercise sessions) lifelong exercise results in substantial benefits in exercise cap
231                                              Exercise seems to enhance the beneficial effect of baria
232       All participants performed a 40-minute exercise session twice a week for 8 weeks.
233 findings indicate that committed (>=4 weekly exercise sessions) lifelong exercise results in substant
234 ermine the total force-producing capacity of exercising skeletal muscle are altered during OCC.
235 ermine the total force-producing capacity of exercising skeletal muscle are significantly altered dur
236 erated in the liver and lactate derived from exercising skeletal muscle can also become important ene
237  midlife, weight loss in late life, physical exercise, smoking, sleep, cerebrovascular disease, frail
238  Resting SNA (p = 0.002) and VNA (p = 0.04), exercise SNA (p = 0.01), SNA response to evoked PVCs (p
239          Our results suggest that early-life exercise, specifically during the 4(th) postnatal week,
240              A key regulator of insulin- and exercise-stimulated glucose uptake and GLUT4 trafficking
241                                          The exercise strategy was associated with small gains in hea
242 s with HFpEF develop B-lines upon submaximal exercise stress echocardiography; however, whether exerc
243                                              Exercise studies targeting postnatal periods of hippocam
244                                Transcriptome exercise studies using bulk tissue analysis do not resol
245 ort (MGH-ExS [Massachusetts General Hospital Exercise Study]) and in a large sample of community-dwel
246 ged 12 to 60 years underwent cardiopulmonary exercise test and echocardiography 1 day before transcat
247    The participants performed an incremental exercise test to volitional exhaustion to determine VO2
248 with SCA underwent CMR, echocardiography and exercise test.
249 t ventricle, during invasive cardiopulmonary exercise testing, demonstrates that that the right heart
250  (n = 29) underwent invasive cardiopulmonary exercise testing, echocardiography, and assessment of mi
251 < 85% predicted from maximal cardiopulmonary exercise testing; organ functions were ascertained with
252             These subjects performed maximal exercise tests in normoxia and hypoxia to determine how
253 l blood gases were collected throughout both exercise tests.
254 nonadherence to lifestyle requirements (diet/exercise) than the usual care group (P < 0.05).
255 ng the detailed molecular signals induced by exercise that benefits health and prevents disease.
256 therapists spent substantial time performing exercises that elicited little muscle activation.
257                              For single-limb exercise, the intensity-duration relationship is differe
258 KOA strategy may be efficient for delivering exercise therapies for knee osteoarthritis.
259  group) or best medical therapy + structured exercise therapy (the nonrevascularization group).
260 rization + best medical therapy + structured exercise therapy (the revascularization group) or best m
261  to starvation, exacerbating the dieting and exercising, thus further activating SIRT1.
262 his is not well defined and it is unknown if exercise timing induces directional shifts of the muscle
263 a must be allowed prior to the initiation of exercise to achieve optimal pain, functional and physiol
264  metabolite profiling before and after acute exercise to delineate the metabolic architecture of exer
265                             We used a Delphi exercise to prioritise and divide the items of the guide
266 red data from 31 novices during laparoscopic exercises to extract features based on cardiac and ocula
267 erobic capacity ( V O(2) max) and submaximal exercise tolerance (i.e. speed-duration relationship) du
268 ease co-morbidities, will assist recovery of exercise tolerance in a variety of conditions that limit
269  to restrict motor unit activation and limit exercise tolerance.
270 ack restrict motor unit activation and limit exercise tolerance.
271 iated with reduced systolic augmentation and exercise tolerance.
272 altered haemoglobin affinity impacts hypoxic exercise tolerance.
273 haryngeal swallowing exercises including jaw exercises, tongue exercises and swallowing maneuvers wit
274                                      Aerobic exercise training (AT) improves endothelial function.
275 lipids and steatosis risk; 3) the effects of exercise training (modalities, volume, intensity) for tr
276 erformance objectives and to provide optimal exercise training and nutritional support for the modern
277           Still, other important benefits of exercise training appeared in women with PCOS, including
278 ions were not restored following 12-weeks of exercise training implying exercise resistance in this c
279 scribes the effects of physical activity and exercise training on coronary atherosclerosis in athlete
280 tial adverse effects of the highest doses of exercise training on the coronary arteries.
281 red to clarify whether interventions such as exercise training to improve LV compliance may prevent t
282                    Here we show that aerobic exercise training up-regulates DICER in adipose tissue o
283  whole-body metabolic adaptations to aerobic exercise training, in part, by allowing controlled subst
284 chondrial activity similar to the effects of exercise training.
285                                We found that exercise twice-a-day increased the nuclear abundance of
286 ions of body size and the use of ISS-like CM exercise upon the provision of life-support during explo
287 tarctic experts undertook a horizon scanning exercise using expert opinion and consensus approaches t
288 anism for the salutary effects of resistance exercise, using AKG as a systemically derived molecule f
289 was to compare the effects of 16 weeks of ID exercise versus a HB exercise program for HD patients.
290    Recent studies have also shown that large exercise volumes and vigorous intensities are both assoc
291 level, marital status, leisure time physical exercise, walking/cycling, height, energy intake, smokin
292                              Lack of regular exercise was the most powerful factor to be associated w
293 satisfaction, good sleep quality and regular exercise were positively associated with compassion sati
294 tive training, cognitive rehabilitation, and exercise were the most effective interventions for adult
295 ng and excess of allergen, but not NSAID and exercise, were other relevant cofactors for systemic rea
296            Equine athletes have a pattern of exercise which is analogous to human athletes and the ca
297 d bioinformatic analysis, a molecular map of exercise will be established.
298 gnosed with hypertension were studied during exercise with either saline or BQ-123 (ET(A) receptor an
299  also showed higher motivation to repeat the exercise with the enhanced device.
300 ed acutely by skeletal muscle in response to exercise, yet chronically elevated with obesity and agin

 
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