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1 ac magnetic resonance imaging, or nonimaging exercise tolerance test.
2 d point but not after adjusting for positive exercise tolerance testing.
4 cumented coronary artery disease, a positive exercise tolerance test, and stable chronic angina pecto
5 nsional echocardiography, Holter monitoring, exercise tolerance testing, and ajmaline provocation.
6 ography, cardiac magnetic resonance imaging, exercise tolerance testing, and biomarker assessment.
8 principally in middle-aged men, suggest that exercise tolerance testing can provide independent progn
9 dulthood caused male mice to underperform at exercise tolerance tests compared to their control and f
14 a and enhances functional capacity during an exercise tolerance test (ETT) in patients with coronary
15 on or myocardial infarction (MI), with a pre-exercise tolerance test (ETT) likelihood of CAD > or =0.
16 postinfarction angina or a strongly positive exercise tolerance test (ETT) typically had cost-effecti
17 , exercise, and nuclear models by use of pre-exercise tolerance test (ETT), post-ETT, and nuclear inf
18 depression > or =1 mm from baseline) during exercise tolerance testing (ETT) was examined in patient
20 le cohort studies demonstrate that screening exercise tolerance testing identifies a small proportion
21 Efficacy was evaluated at 90 and 180 days by exercise tolerance test, myocardial nuclear perfusion im
26 to limiting angina during bicycle exercise (exercise tolerance tests), performed at trough of drug a
27 s were similar in the 2 approaches, with the exercise tolerance test result exerting the greatest lev
29 ta-blocker therapy underwent cardiopulmonary exercise tolerance testing under 2 conditions in random