ライフサイエンスコーパス: exercise training

1 Cox models and explored an interaction with exercise training.

2 or the prediction of the VO2 max response to exercise training.

3 scular dysfunction are reversed by late-life exercise training.

4 ronal precursor cell proliferation following exercise training.

5 d related signalling pathways in response to exercise training.

6 r treating multiple defects at once, as with exercise training.

7 ny of the adaptations that accompany chronic exercise training.

8 ameliorated by correcting anemia and through exercise training.

9 s a key factor in the beneficial outcomes of exercise training.

10 muscle or whole-body aerobic adaptations to exercise training.

11 ypically observed in sedentary subjects with exercise training.

12 genesis, exercise tolerance, and response to exercise training.

13 muscle or whole-body aerobic adaptations to exercise training.

14 imals also performed low intensity treadmill exercise training.

15 quired for metabolic adaptation to endurance exercise training.

16 modeling, and muscle strength in response to exercise training.

17 vements in insulin sensitivity after aerobic exercise training.

18 and no major adverse events were related to exercise training.

19 metabolism, and depot-specific responses to exercise training.

20 tolerate (and possibly even be improved by) exercise training.

21 ovements in glucose metabolism after aerobic exercise training.

22 raction </=35% to usual care with or without exercise training.

23 olic twist in an acute augmentation phase of exercise training.

24 LVCD may be possible with moderate lifelong exercise training.

25 by acute and chronic aerobic and resistance exercise training.

26 icular, for individuals involved in physical exercise training.

27 pleted 14 weeks of controlled and supervised exercise training.

28 rol and high fat diet groups with or without exercise training.

29 ly Study who completed 20 weeks of endurance exercise training.

30 chondrial activity similar to the effects of exercise training.

31 r athletes to perform and adapt optimally to exercise training.

32 reased during the second and third months of exercise training.

33 to 9, and also not in response to submaximal exercise training.

34 ng older adults undertaking resistance-based exercise training 3-d/wk, participants who consumed lean

35 ecutive function; (2) Cognitive and physical exercise training; (3) Cognitive training combined with

36 non-invasive brain stimulation and physical exercise training; (4) Active control training in adapti

37 Chronic exercise training (6 weeks) increased soleus muscle PGC-

38 Relative to exercise training alone, the multimodal fitness and nutr

39 Exercise training also increased the insulin activation

40 Exercise training also increases expression of the brown

41 Chronic exercise training also promoted NRG1 cleavage, resulting

42 Regular exercise training ameliorates age-related diastolic dysf

43 ural and functional adaptations to endurance exercise training among competitive male rowers (n=12; a

44 s controversial with the association between exercise training and collateral growth still unclear.

45 Before and after exercise training and detraining, 12 previously sedentar

46 Exercise training and energy restriction are first-line

47 ortance of lifestyle modification, including exercise training and energy restriction, in the regulat

48 Exercise training and home or housework were not associa

49 th SRVs in this study safely participated in exercise training and improved peak VO2.

50 se of VCO2 versus VO2 ) remains important in exercise training and in the clinic, but its conceptual

51 vely evaluated the effects of a home aerobic exercise training and maintenance program (EX) on aerobi

52 e induces physiological responses that mimic exercise training and may underlie the beneficial effect

53 Moderate intensity exercise training and modest energy restriction provided

54 We also show that both exercise training and muscle-specific transgenic express

55 erformance objectives and to provide optimal exercise training and nutritional support for the modern

56 d recipients and on the outcomes relevant to exercise training and physical function that should be e

57 current countermeasures including in-flight exercise training and volume resuscitation on return, no

58 enuated in healthy adults engaged in aerobic exercise training, and aerobic exercise interventions im

59 e benefits of weight loss, physical activity/exercise training, and increases in cardiorespiratory fi

60 computer-based cognitive training, physical exercise training, and non-invasive brain stimulation, a

61 mechanical changes may exist in response to exercise training, and provide evidence supporting a pot

62 n exercise bouts, to optimize adaptations to exercise training, and to stay healthy.

63 ) at baseline and in response to 12-weeks of exercise training; and examined depot-specific associati

64 Still, other important benefits of exercise training appeared in women with PCOS, including

65 s age-related microvascular dysfunction, and exercise training appears to be particularly effective i

66 Physical activity and exercise training are effective strategies for reducing

67 s to amygdala reactivity, and help establish exercise training as a form of anxiolytic therapy toward

68 Aerobic exercise training (AT) improves endothelial function.

69 We hypothesised that exercise training attenuates the changes in cutaneous va

70 Exercise training benefits many organ systems and offers

71 After 8 weeks of exercise training, blood pressure was reduced in all 3 g

72 increased muscle fibrosis, while obesity and exercise training both increased muscle adiposity.

73 In additional experiments, voluntary exercise training by wheel running for only 11 days resu

74 rodent exercise studies have indicated that exercise training can alter circulating adipokine concen

75 These findings suggest that structured exercise training can ameliorate striatal D2/D3 receptor

76 sfunction; these data suggest that late-life exercise training can be implemented to improve coronary

77 Exercise/exercise training can enhance insulin sensitivity throug

78 Endurance exercise training can increase the ability to perform pr

79 to exercise intolerance, and conversely how exercise training can potentially modulate aging phenoty

80 may help to pinpoint the mechanisms by which exercise training can reduce the risk of brain diseases,

81 Exercise training causes adaptations to scWAT that elici

82 he change in body composition in response to exercise training combined with calorie restriction in o

83 Aerobic exercise training combined with fibre-enriched diet can

84 All subjects performed resistance exercise training combined with high-intensity interval

85 estigated the tolerability and efficacy of 2 exercise training dose regimens on cardiorespiratory fit

86 one, with an additive benefit of surgery and exercise training (e.g., collagen I: RYGB -41% vs. RYGB

87 We determined whether endurance exercise training (EET) alters macrophage content and ch

88 Exercise training enhanced myofibre cross-sectional area

89 Exercise training enhances extracellular superoxide dism

90 Whether exercise training enhances or injures the SRV is unclear

91 Exercise training enhances physical performance and conf

92 Swim exercise training enhances whole-animal health parameter

93 The promotion of physical activity and exercise training (ET) leading to improved levels of car

94 e value of high levels of physical activity, exercise training (ET), and overall cardiorespiratory fi

95 om calorie restriction (CR) and/or endurance exercise training (EX) is cardioprotective.

96 years) were randomized to either 2 years of exercise training (exercise group: n = 34) or control/yo

97 lity and induces insulin resistance, whereas exercise training exerts positive effects on substrate h

98 3+/-5 years) were randomized to 10 months of exercise training followed by 14 months of maintenance e

99 is study was to determine whether real-world exercise training for a first-time marathon can reverse

100 (Exercise Training for Chronic Heart Failure [ExTraMATCH

101 l events, but not a differential response to exercise training for clinical outcomes or changes in ex

102 osocial factors may influence adherence with exercise training for heart failure (HF) patients.

103 ent advances in cognitive rehabilitation and exercise training for treating MS-related cognitive impa

104 to 2.21) mL/kg/min among participants in the exercise training group and +0.08 (95% CI, -0.62 to 0.79

105 cardiovascular death or HF hospitalization, exercise training had a greater impact on patients with

106 ata analysis, patients with HFPEF undergoing exercise training had significantly improved CRF (mL/kg

107 Resistance exercise training had smaller but qualitatively similar

108 Exercise training has been reported to ameliorate heart

109 Exercise training has been shown to be effective in impr

110 Exercise training has been shown to improve cardiorespir

111 Exercise training has long been known to promote mitocho

112 To date, cognitive rehabilitation and exercise training have been identified as possible candi

113 As physical fitness and exercise training have been shown to benefit multiple ar

114 Regular physical activity and exercise training have long been known to cause adaptati

115 A Controlled Trial Investigating Outcomes of Exercise Training (HF-ACTION), 2279 (97.8%) completed su

116 A Controlled Trial Investigating Outcomes of Exercise Training [HF-ACTION]; NCT00047437).

117 ity and acceptability of two common types of exercise training-high-intensity interval training (HIIT

118 ions were not restored following 12-weeks of exercise training implying exercise resistance in this c

119 Habitual physical activity and regular exercise training improve cardiovascular health and long

120 and arteriovenous oxygen difference, whereas exercise training improved cardiac output, citrate synth

121 Ts in adult SOT recipients demonstrated that exercise training improved exercise capacity, lower extr

122 Exercise training improved insulin sensitivity beyond su

123 iddle-aged adults, 2 years of high-intensity exercise training improved integrated cardiovascular reg

124 adults, a 2 year programme of high-intensity exercise training improved integrated cardiovascular reg

125 suggesting a cross-organ mechanism by which exercise training improves cardiac function in diabetes

126 To determine whether moderate-intensity exercise training improves exercise capacity in adults w

127 These findings suggest exercise training improves muscle morphology and satelli

128 Given that exercise training improves outcomes in HF patients, asse

129 Exercise training improves whole-body glucose homeostasi

130 To examine the effect of exercise training in a weight-loss program on asthma con

131 ased and expert-informed recommendations for exercise training in adult and children solid organ tran

132 affect the response to 3 weeks of endurance exercise training in CACs, muscle mitochondrial capacity

133 ll maintained during 3-month aerobic dynamic exercise training in children and young adults with repa

134 This study examined whether outcomes with exercise training in HF vary according to AF status.

135 the current study, we aimed to determine if exercise training in humans diminished P-CoA attenuation

136 Here, we determined the metabolic impact of exercise training in obese mice with cardiac and skeleta

137 CE-I, reduced the peak work rate response to exercise training in patients with chronic obstructive p

138 Exercise training in patients with HFPEF is associated w

139 inical trials that evaluated the efficacy of exercise training in patients with HFPEF were included i

140 95% confidence interval, -0.01 to 0.16) with exercise training in patients with HFPEF.

141 e aim to evaluate the efficacy and safety of exercise training in patients with pulmonary hypertensio

142 es that evaluated the efficacy and safety of exercise training in patients with pulmonary hypertensio

143 Exercise training in patients with pulmonary hypertensio

144 The coactivator PGC-1alpha1 is activated by exercise training in skeletal muscle and promotes fatigu

145 oward raising awareness of the importance of exercise training in SOT patients among transplant profe

146 Furthermore, we highlight the benefits of exercise training in stimulating positive adaptations in

147 aimed to assess the haemodynamic effects of exercise training in transposition of the great arteries

148 Exercise training, in contrast, does the opposite.

149 whole-body metabolic adaptations to aerobic exercise training, in part, by allowing controlled subst

150 In contrast, exercise training increased mitochondrial ADP sensitivit

151 stable HFPEF, caloric restriction or aerobic exercise training increased peak VO2, and the effects ma

152 Six weeks of endurance exercise training increased the transcriptional level of

153 Exercise training increased Vo2max (2.2 +/- 0.2 vs. 2.5

154 Six weeks of exercise training increased whole-body glucose homeostas

155 In rodent models, exercise training increases mitochondrial biogenesis and

156 We show that endurance exercise training increases multiple CAC types, an adapt

157 Exercise training increases overall miRNA expression in

158 Consistent with the increase in UCP1, exercise training increases the presence of brown-like a

159 Short-term exercise training, independently of dosing schedule, is

160 In conclusion, endurance exercise training induced increases in multiple CAC type

161 We propose that exercise training-induced DICER-miR-203-3p up-regulation

162 O2 supply, whereas, in trained subjects, an exercise training-induced mitochondrial reserve results

163 e corresponding ionic current, If, underlies exercise training-induced sinus bradycardia in rodents.

164 the effects of non-specific antioxidants on exercise training-induced vascular adaptations remain el

165 However, the effects of antioxidants on exercise training-induced vascular adaptations remain el

166 Endurance and resistance exercise training induces specific and profound changes

167 Endurance exercise training induces substantial adaptive cardiac m

168 In contrast, endurance exercise training (initiated 2 weeks after AAV9-R735X in

169 Exercise training, initiated at an advanced age, reverse

170 Importantly, aerobic exercise training, initiated even at an advanced age, re

171 The impact of an exercise training intervention on the physiological resp

172 consumption (VO(2)peak) to 1 of 2 supervised exercise training interventions delivered with a standar

173 behind these impairments and the benefits of exercise training interventions, our laboratory has rece

174 he inclusion of cognitive rehabilitation and exercise training into clinical practice within the next

175 Exercise training is an effective treatment for importan

176 Previous studies have shown that short term exercise training is feasible in TGA patients, but its e

177 After exercise training, IVRT, a measure of active ventricular

178 to increase with an additional 14 months of exercise training (LA volumes 55%; LV end diastolic volu

179 potential mechanism by which high intensity exercise training leads to atrial fibrillation.

180 These data indicate that exercise training leads to parallel reductions in hot fl

181 of other laboratories suggest that endurance exercise training leads to similar changes in sedentary

182 , the profound changes to WAT in response to exercise training may be part of the mechanism by which

183 ermine whether nutritional interventions and exercise training may preserve muscle area and thereby i

184 Exercise training-mediated mitochondrial biogenesis, run

185 re we felt it essential to determine whether exercise training might injure a systemic right ventricl

186 All 3 exercise training modalities produced similar improvemen

187 In conclusion, different exercise training modalities were similarly effective in

188 lipids and steatosis risk; 3) the effects of exercise training (modalities, volume, intensity) for tr

189 y assigned to 16 weeks of moderate-intensity exercise training (n = 67) or usual activity (n = 69).

190 ized to a group that received 1 h supervised exercise training (n=10) or one that received equal-time

191 a Controlled Trial Investigating Outcomes of Exercise Training; n=510).

192 Intense aerobic exercise training negatively affects iron status; howeve

193 proximately 25% kcal reduction vs. O-SED) or exercise training (O-EX; treadmill running 20 m min(-1)

194 Assessing the effects of exercise training on cardiovascular variability is chall

195 affect the response to 3 weeks of endurance exercise training on CD3(+) , CD3(+) /CD31(+) , CD14(+)

196 scribes the effects of physical activity and exercise training on coronary atherosclerosis in athlete

197 The restorative effect of exercise training on coronary microvascular function may

198 -analysis, we aim to evaluate the effects of exercise training on CRF, quality of life, and diastolic

199 Studies superimposing exercise training on hypoxic exposure demonstrate an inc

200 ined the effect of 2 years of high-intensity exercise training on integrated cardiovascular function,

201 n analysis to assess the effect of prolonged exercise training on integrated cardiovascular regulatio

202 d the effects of 24 months of high intensity exercise training on left atrial (LA) mechanical and ele

203 There was no interaction between AF and exercise training on measures of functional status or cl

204 tudies that have investigated the effects of exercise training on mitochondrial function, the "beigin

205 PSSS did not impact the effect of exercise training on outcomes.

206 ought to determine the beneficial effects of exercise training on oxidative stress and inflammation i

207 Effect of exercise training on quality of life (estimated using Mi

208 ed work examining the effects of obesity and exercise training on radiation-exposed bone marrow, we e

209 tial adverse effects of the highest doses of exercise training on the coronary arteries.

210 ations of endurance athletes are a result of exercise training or a genetically determined characteri

211 locity; PWV) were evaluated before and after exercise training or cage confinement.

212 were randomly assigned to either submaximal exercise training or no forced exercise (untrained).

213 Young and old rats underwent 10 weeks of exercise training or remained as sedentary, cage-control

214 +) number did not change in this region with exercise training or skeletal myofiber VEGF gene deletio

215 HF patients with ejection fraction </=35% to exercise training or usual care.

216 Hct normalization, exercise training, or the combination thereof significan

217 Endurance exercise training partially reversed these abnormalities

218 A total of 469 exercise-training participants enrolled in 16 separate t

219 The study design included a 6-month aerobic exercise training period followed by a 2-week detraining

220 We hypothesised that a short exercise-training period would affect muscle and bone ma

221 ompleted a crossover study consisting of two exercise training phases at two hematocrit (Hct) values:

222 In adults, exercise training pretransplant was safe, but there was

223 We conclude that endurance exercise training prevents symptomatic onset of FRDA in

224 A 12-week standardized aerobic dynamic exercise training program (3 one-hour sessions per week)

225 to either a combined aerobic and resistance exercise training program following RYGB (RYGB + ET) or

226 The moderate-intensity endurance exercise training program improved the muscle capillary

227 this study was to evaluate whether adding an exercise training program to an inpatient behavioral int

228 (Exercise Training Program to Improve Clinical Outcomes i

229 performed a pilot study to assess whether an exercise training program would result in adverse change

230 adults undergoing a 3-d/wk resistance-based exercise training program, we investigated whether those

231 with TGA and SRV were enrolled in a 12 week exercise training programme (moderate and high-intensity

232 ought to investigate the effect of a 12-week exercise-training programme on these aspects of mitochon

233 letal muscle responses to a short controlled exercise-training programme.

234 f brain diseases, inform the optimization of exercise training programmes and assist with the identif

235 d (and may be improved) in TGA patients with exercise training programmes that are typical of recreat

236 l variation even in response to standardized exercise training programmes.

237 Exercise training promotes EcSOD expression in skeletal

238 discuss the various mechanisms through which exercise training promotes mitochondrial quantity and qu

239 Mitochondrial adaptations to a treadmill exercise training protocol, in either low-fat or high-fa

240 A Controlled Trial Investigating Outcomes of Exercise Training) randomized 2,331 ambulatory HF patien

241 in fitness that are mediated by 16 weeks of exercise training reduce the severity of physiological s

242 In this study, exercise training reduced magnetic resonance imaging-det

243 In the absence of P-CoA, exercise training reduced mitochondrial ADP sensitivity.

244 Exercise training reduced the self-reported severity of

245 A period of preoperative supervised exercise training reduces postoperative cardiac, respira

246 Exercise training reduces self-reported hot flush severi

247 nate manner in response to a sprint interval exercise training regimen in humans and to denervation o

248 muscle-specific autophagy on the benefits of exercise training remains incompletely understood.

249 t physiological factors, such as obesity and exercise training remains unknown.

250 ovements in mitochondrial respiration due to exercise training required Bcl2-mediated autophagy using

251 lume reserve during exercise correlates with exercise training response in our TGA patients, identify

252 12-weeks of exercise training resulted in a significant increase in

253 Twenty-four months of high intensity exercise training resulted in LA greater than LV mechani

254 Long-term endurance exercise training results in a reduction in the rates of

255 s of fully supervised, whole-body resistance exercise training (RET) (72.8 +/- 1.4 years; BMI 26.3 +/

256 Resistance exercise training (RET) can rejuvenate limb blood flow r

257 Resistance exercise training (RET) has a beneficial effect on muscl

258 Resistance exercise training (RET) is one of the most effective str

259 Resistance exercise training (RET) is widely used to increase muscl

260 mpaired hypertrophic responses to resistance exercise training (RET).

261 Exercise training reversed obesity-related mitochondrial

262 In old rats, exercise training reversed the reduction in E/A, reduced

263 Exercise training reverses age-induced declines in diast

264 dysfunction, and (2) initiation of late-life exercise training reverses age-related diastolic and mic

265 h oxidative and glycolytic muscle, late-life exercise training reverses age-related microvascular dys

266 nor high-intensity intervals, nor short-term exercise training seem to injure the systemic right vent

267 Endurance exercise training should be considered a key strategy to

268 To obtain benefits, exercise training should be of moderate to vigorous-inte

269 We recommended that exercise training should be offered in the pre- and post

270 Also independent of MitoQ supplementation, exercise training significantly increased quadriceps mus

271 Exercise training significantly reduced both MRI-detecte

272 odiesterase 5 inhibitors, muscle weakness by exercise training, sodium retention by diuretics and mon

273 w through hippocampal regions independent of exercise training state.

274 ovascular blood flow, protein metabolism and exercise training status in older men.

275 hysiological system, regardless of endurance exercise training status.

276 stressors during acute bouts of exercise or exercise training stimulate enhancement of cellular stre

277 ngs by adjusting performance in response to "exercise" training stimuli.

278 lating oestrogens in transducing the aerobic exercise training 'stimulus'.

279 e analyzed public transcriptome data from an exercise training study and revealed significant changes

280 care such as adherence to medical treatment, exercise training, symptom monitoring and symptom manage

281 We identified candidates modulated by exercise training that may become therapeutic targets fo

282 itate individualized approaches to implement exercise training therapy in clinical practice.

283 skeletal muscle function and is improved by exercise training through both mitochondrial biogenesis

284 ulin resistance and highlight the ability of exercise training to diminish P-CoA attenuation in mitoc

285 ations of an erythroid-stimulating agent and exercise training to examine if and where limitation to

286 red to clarify whether interventions such as exercise training to improve LV compliance may prevent t

287 education, health behavior modification, and exercise training to improve secondary prevention outcom

288 Specificity is a core principle of exercise training to promote the desired adaptations for

289 A Controlled Trial Investigating Outcomes of Exercise Training) trial to advance precepts of CR are c

290 A Controlled Trial Investigating Outcomes of exercise traiNing) trial, 10 CPX test variables measured

291 Here we show that aerobic exercise training up-regulates DICER in adipose tissue o

292 tolic HF patients who had been randomized to exercise training versus usual care.

293 To investigate the effect of exercise training, VL biopsies were collected from n=17

294 In the pooled analysis, exercise training was associated with significant improv

295 n, 16 weeks of supervised moderate intensity exercise training was found to improve cardiorespiratory

296 Furthermore, exercise training was well tolerated with a low dropout

297 he chronic impacts of interventions, such as exercise training, which affect both sympathetic activit

298 A Controlled Trial Investigating Outcomes of Exercise Training, which randomized 2331 patients with e

299 marked energy deficit in addition to intense exercise training would affect changes in body compositi

300 othesis that, similar to humans and rodents, exercise training would enhance mitochondrial (Mt) bioge