ライフサイエンスコーパス: exercising muscle

1 sympathoexcitation in the vasculature of the exercising muscle.

2 xygen delivery and oxygen utilization in the exercising muscle.

3 ved fatty acids and their sparing for use by exercising muscle.

4 ical activity increases energy metabolism in exercising muscle.

5 rease in sympathetic vasoconstriction in the exercising muscles.

6 ercise capacity is the delivery of oxygen to exercising muscles.

7 ch between blood/oxygen demand and supply in exercising muscles.

8 glycogen and do not generate lactic acid in exercising muscles.

9 t is not dependent on movement feedback from exercising muscles.

10 because of decreased fatty acid delivery to exercising muscles.

11 hypothesis that lactic acid accumulation in exercising muscle acted on ASICs located on thin fibre m

12 ect on the distribution of cardiac output to exercising muscle and an age-related reduction in cardia

13 l administration of lactic acid (to simulate exercising muscle and evoke a pressor reflex), endomorph

14 nosine is released into the venous efflux of exercising muscle and that adenosine is responsible for

15 control, and thus the provision of oxygen to exercising muscles and vital organs, because of enhanced

16 ty of metabolically sensitive afferents from exercising muscle are the principal determinants of symp

17 vasoconstriction in the microcirculation of exercising muscle, because such modulation is abrogated

18 ntributors to disease-related impairments in exercising muscle blood flow and oxygen utilization in t

19 tributes to the age-associated impairment in exercising muscle blood flow and vascular tone in humans

20 r than the amount of O(2) in plasma, and ii) exercising muscle blood flow can almost double (from 260

21 n muscle SNA had no effect on oxygenation in exercising muscles but produced robust decreases in oxyg

22 c vasoconstriction is normally attenuated in exercising muscle, but this functional sympatholysis is

23 Thus, SNA can restrict blood flow to exercising muscle by constricting FA and 1A while dilata

24 e nNOS is proposed to regulate blood flow in exercising muscle by diffusing from the skeletal muscle

25 h-intensity exercise is reflected within the exercising muscle by its [PCr] response.

26 s, the high levels of PGC-1alpha in dark and exercising muscles can explain their resistance to atrop

27 Blood flow limitation to exercising muscles engages the muscle reflex during exer

28 ts were: limiting oxygen (O(2) ) delivery to exercising muscle->increased glycolysis, La(-) and H(+)

29 appear to subserve an obligatory role in the exercising muscle hyperaemic response in the rat.

30 ugh multiple metabolites are concentrated in exercising muscle in CHF, only prostaglandins correlated

31 sympathetic vasoconstriction is enhanced in exercising muscle in heart failure as a result of impair

32 part, for the reduced acidification seen in exercising muscle in myotonic dystrophy.

33 ts were: limiting oxygen (O(2) ) delivery to exercising muscle increased glycolysis, La(-) and H(+) p

34 sympatholysis) within the vasculature of the exercising muscle is impaired in patients with HFpEF.

35 cellular P(O(2)) regulates PCr hydrolysis in exercising muscle is not due to the initial kinetic fall

36 which the latter signals that blood flow to exercising muscle is not meeting its metabolic demand.

37 ion induces physiological adaptations in the exercising muscle, it should become more fatigue resista

38 In contrast, the induction of ANGPTL4 in exercising muscle likely is counteracted via AMP-activat

39 r to increase the delivery of glucose to the exercising muscle of GLUT4(-/-).

40 tion is not reduced, but may be augmented in exercising muscle of healthy older humans.

41 c vasoconstriction is normally attenuated in exercising muscles of young men and women.

42 central neural factors, afferent input from exercising muscle plays an important role in modulating

43 de, while extrapolation of these data to the exercising muscle predicts a significant role of Ca(2+)

44 exercise, central command and reflexes from exercising muscles produce the breathing stimulation req

45 rylation in vivo and can limit ATP supply in exercising muscle to below the mitochondrial capacity.

46 synapses is reminiscent of that occurring in exercising muscle to sustain cellular function and ident

47 etic vasoconstriction is markedly blunted in exercising muscles to optimize blood flow to the metabol

48 nalling pathways (i.e. communication between exercising muscles to vascular/cardiac tissue) and intra

49 ards more predominant local control over the exercising muscle vasculature, systemic arterial blood p

50 (BFRT) employs partial vascular occlusion of exercising muscles via inflation cuffs.

51 ted by recent (13)C and (31)P NMR studies of exercising muscle, we propose a model of the energetics

52 ytokine interleukin-13 (IL-13) is induced in exercising muscle, where it orchestrates metabolic repro