ライフサイエンスコーパス: exhaust

1 ompound (IVOC) emissions in gasoline vehicle exhaust.

2 pacity or mutagenicity) of biogas combustion exhaust.

3 ators compared to both air controls and ULSD exhaust.

4 0 nm, near-spherical, single crystals in the exhaust.

5 s as a result of in utero exposure to diesel exhaust.

6 d from the photochemical oxidation of diesel exhaust.

7 n the soot samples collected from the engine exhaust.

8 TEa cells in skin-graft recipients were not exhausted.

9 sub-classified into active, transitional or exhausted.

10 s should mostly proceed unless resources are exhausted.

11 ng towards menopause when the oocyte pool is exhausted.

12 ed proliferative activity before they become exhausted.

13 arly once opportunities for compensation are exhausted.

14 roughly 30% of the observed particles in the exhaust, a new surface coating formed, approximately 2-5

15 Moreover, diesel exhaust affected A. mellifera in a way that reduced thei

16 in selecting fuels, engine technologies, and exhaust aftertreatment (EAT).

17 ytic metabolism and mTOR activity within the exhausted Ag-expCD4(+) T cell population during infectio

18 In utero exposure to diesel exhaust air pollution has been associated with increased

19 Therefore a deforming network is expected to exhaust all possible bending-based modes before engaging

20 The patient, who had exhausted all other therapeutic options, was treated wit

21 med ductal pancreatic adenocarcinoma who had exhausted all other treatment options received (177)Lu-3

22 of Pb including industrial emission, vehicle exhaust and dust storm with the mean contributions of 47

23 c myocytes after in utero exposure to diesel exhaust and found that the promoter for Mir133a-2 is dif

24 ooled case-control analysis on diesel engine exhaust and lung cancer by including three additional st

25 -type mice and infiltrating T cells are less exhausted and more active and proliferative.

26 ignatures that shared features of terminally exhausted and progenitor-exhausted T cells, respectively

27 Exhausted and regulatory B cells were declined whereas m

28 Tumor-infiltrating T cells mainly displayed exhausted and regulatory T-cell features.

29 zed by the formation of Slamf6(+) progenitor exhausted and Tim-3(+) terminally exhausted subpopulatio

30 Laparoscopic surgery can be exhausting and frustrating, and the cognitive load exper

31 T cells in this functional state are termed "exhausted" and T cell exhaustion is associated with inef

32 mbustion processes, are present in vehicular exhaust, and persist in the environment for weeks and bi

33 ervices after all treatment options had been exhausted; and (3) care complexity: patients' complex ca

34 ibes for the first time the evolvement of an exhausted antigen-specific CD8(+) TCR repertoire under c

35 that most particles in the fresh ship engine exhaust are in ultrafine particle size range.

36 primary particle formation in dilute engine exhaust as low quantities were collected on unimpregnate

37 ational analysis of tumours has largely been exhausted as a strategy for the identification of new ca

38 ng reactions such that clinopyroxene was not exhausted at high degrees of melting and was retained in

39 ion-resistant prostate carcinoma and who had exhausted available therapy options for their disease.

40 When nutrients in their environment are exhausted, bacterial cells become arrested for growth.

41 that other nutrients, notably iron, will be exhausted before cobalt can be fully depleted, helping t

42 gines would produce benefits with respect to exhaust burden on air quality, and thus their utilizatio

43 t least in malaria, atypical MBCs may not be exhausted but rather may be functional, possibly even be

44 d the SOA yield from dilute gasoline vehicle exhaust by a factor of 8.

45 pture efficiency of UFPs from diesel vehicle exhaust by nine temperate-zone plant species, in wind tu

46 production wells was estimated to be nearly exhausted by the end of the test.

47 e checkpoint blockade triggered expansion of exhausted CAR T cells and concordantly lowered viral loa

48 C) and noble-metal efficiency of automotive exhaust catalysts has been a continuous effort to elimin

49 This is signified by increased numbers of exhausted CD21(neg) memory B cells, driven by continuous

50 ties of naive B cells in HIV-1 infection, as exhausted CD21(neg) naive B cells may severely impair in

51 ic profile in the lungs, with a depleted and exhausted CD4 and CD8 T-cell population that resides wit

52 containing protein 3 (Tim-3) is expressed on exhausted CD4(+) and CD8(+) T cells and is upregulated o

53 implications for therapeutic reactivation of exhausted CD4(+) T cells during malaria infection and ot

54 pansion of dedifferentiated monocytes and of exhausted CD4(+) T cells.

55 DAA cure does not promote resurrection of exhausted CD4(+) T-cell memory in chronic infection.

56 While exhausted CD4, CD8 T and NK cells are major proliferativ

57 l analysis reveals prominent interactions of exhausted CD8 + T-cells and PD-L1 + macrophages and PD-L

58 ne score as a surrogate of interferon-primed exhausted CD8 + T-cells in the tumor microenvironment.

59 s with PD-L1 + macrophages in the first, and exhausted CD8 + T-cells with cancer cells harboring geno

60 responders shows differential clustering of exhausted CD8 + T-cells with PD-L1 + macrophages in the

61 Exhausted CD8 T (Tex) cells are a distinct cell lineage

62 study also shows that the repertoire of the exhausted CD8 T cell subset is almost completely derived

63 found that >95% of the TCR repertoire of the exhausted CD8 T cell subset was shared with the stem-lik

64 is a key transcription factor in progenitor exhausted CD8 T cells (Tex).

65 n immune-suppressive phenotype with numerous exhausted CD8 T cells and resident macrophages; fewer eo

66 We demonstrate that exhausted CD8 T cells are the major cells that respond t

67 sponses were associated with accumulation of exhausted CD8 T cells in the tumor at 3 weeks, with rein

68 etic signature of stem-like CD8 T cells with exhausted CD8 T cells.

69 and also differentiating into the terminally exhausted CD8 T cells.

70 gher accessibility to ETS and RUNX motifs in exhausted CD8 T cells.

71 hereas Prdm1 and Id2 were more accessible in exhausted CD8 T cells.

72 the functional recovery of antigen-specific exhausted CD8 T cells.

73 gives rise to the terminally differentiated exhausted CD8 T cells.

74 Exhausted CD8(+) T (T(ex)) cells in chronic infections a

75 known about their multifaceted impact on the exhausted CD8(+) T cell receptor (TCR) repertoire.

76 kpoint inhibitors are effective in restoring exhausted CD8(+) T cell responses in persistent viral in

77 decreased total CD8(+) T cells and increased exhausted CD8(+) T cell subpopulations in murine and hum

78 Despite this, the developmental biology of exhausted CD8(+) T cells (Tex) remains poorly defined, r

79 nt of T cells was preferentially observed in exhausted CD8(+) T cells and evident in patients with ba

80 ls in the TME of SCMs, and a predominance of exhausted CD8(+) T cells and macrophages in the TME of C

81 Specific subsets such as exhausted CD8(+) T cells and Tregs are preferentially en

82 environment elicits a subset of functionally exhausted CD8(+) T cells by creating conditions that ind

83 HC tetramer-based approach was used to track exhausted CD8(+) T cells in a male-to-female skin transp

84 aches to expand the population of progenitor exhausted CD8(+) T cells might be an important component

85 CD8(+) T cells, decreasing the prevalence of exhausted CD8(+) T cells that express the transcriptiona

86 o identify recently characterized subsets of exhausted CD8(+) T cells that may help to predict patien

87 peutics to restore cancer- and virus-induced exhausted CD8(+) T cells, by enhancing the quality and s

88 rgeting the PD-1/PD-L1 pathway reinvigorates exhausted CD8(+) T cells, it fails to restore T cell rep

89 COVID-19 individuals, but no differences in exhausted CD8(+) T cells, nor in any of these CD4(+) T c

90 tory T cells, M2 (protumor) macrophages, and exhausted CD8(+) T cells.

91 ke CD8(+) T cells, and increasing progenitor exhausted CD8(+) T cells.

92 Consequently, progenitor exhausted CD8(+) TILs are better able to control tumor g

93 Exhausted CD8(+) TILs include a subpopulation of 'progen

94 therapy acts on a specific subpopulation of exhausted CD8(+) tumor-infiltrating lymphocytes (TILs).

95 vated, clonally expanded, but phenotypically exhausted CD8+ T cells in human melanoma.

96 ied a unique stem-like T-cell subset amongst exhausted CD8-positive T cells in chronic infection(1-3)

97 PD-1 identifies "exhausted" CD8 T cells with impaired HIV-specific effect

98 n lead to the emergence of dysfunctional or 'exhausted' CD8(+) T cells, and the restoration of their

99 Fumarate can complex with GSH and thereby exhaust cells of functional GSH capacity.

100 arkers commonly associated with effector and exhausted cells and were induced by IL-6 in a STAT1-depe

101 a who have a higher percentage of progenitor exhausted cells experience a longer duration of response

102 re 2B4(+)CD160(+)PD1(+), a characteristic of exhausted cells.

103 ells that is transcriptionally distinct from exhausted cells.

104 TILs include a subpopulation of 'progenitor exhausted' cells that retain polyfunctionality, persist

105 m the Galactic Centre 'chimneys', constitute exhaust channels through which energy and mass, injected

106 ociated with occupational exposure to diesel exhaust characterized by elemental carbon (EC) concentra

107 phatics) were calculated to characterize the exhaust chemical composition at different engine operati

108 Highly phenotypically exhausted cluster of differentiation 8 (CD8) T cells exp

109 ion 3D structure of MeT1 in complex with its exhausted cofactor, S-adenosyl-l-homocysteine, together

110 dy was to identify which of the major pellet exhaust components (including high nitrogen oxide (NOx),

111 formation by oxidizing dilute, ambient-level exhaust concentrations from a fleet of on-road gasoline

112 s as low as ~160 (o)C under simulated diesel exhaust conditions while using 5 times less Pt-group met

113 how acute exposure to a high-dose of diesel exhaust (containing 19.8 and 17.5 ppm of NO and NO(2), r

114 NG exhaust contains some unburned methane due to inevitable

115 sity in the stem-like CD8 T cells than their exhausted counterpart (~40 versus ~15 GP33(+) clonotypes

116 ys-Arg-chloromethylketone repressed PD-1 and exhausted CTLs via induction of T-cell proliferation and

117 ) were exposed to 150 or 500 mug/m(3) diesel exhaust (DE) or filtered air (FA).

118 Rationale: Diesel exhaust (DE), an established model of traffic-related ai

119 However, these cells are not exhausted, despite high PD-1 expression.

120 t phases were concurrently measured from the exhaust duct.

121 g acute LCMV infection and are predominantly exhausted during chronic infection.

122 However, T cells that become fully exhausted during prolonged antigen exposure remain refra

123 An analysis of the exhaust effluent from the membranes indicates N(2)O as a

124 llowing sources of Fe-bearing nanoparticles: exhaust emissions (both diesel and gasoline), brake wear

125 Diluted exhaust emissions from the entire LA92 cycle were introd

126 The effects of photochemical aging on exhaust emissions from two light-duty vehicles with gaso

127 tertreatment technologies may affect vehicle exhaust emissions.

128 ta) cells, Th9(IL-4+IL-1beta) cells are less exhausted, exhibit cytotoxic T effector gene signature a

129 , which demonstrated that only in the diesel exhaust exposed honey bees was there a significant posit

130 Diesel exhaust exposure decreased honey bees' ability to learn

131 s the indoor-outdoor pressure difference (or exhaust fan flow rate), CPM test duration, exhaust fan l

132 Although exhaust fan intake sampling is sufficient to provide cri

133 r exhaust fan flow rate), CPM test duration, exhaust fan location, and air sampling location(s) and c

134 ential reductions result from reduced diesel exhaust fluid (DEF) usage due to lower NO(x) emissions.

135 Here, we detail a self-exhausting form of CRISPR-based drive system comprising

136 ic hydrocarbons (PAHs))-were measured in the exhaust from 26 stove/fuel combinations.

137 campaign measuring methane concentrations in exhaust from residential NG appliances was conducted in

138 and products are continuously introduced and exhausted from the chamber.

139 ory and on-board measurements of ship engine exhaust, fuel-specific particle number (PN) emissions fo

140 duction furnace, the flow characteristics of exhaust gas and silicon particles were analyzed.

141 The flow model and erosion model of exhaust gas and silicon particles were established based

142 act of formaldehyde (HCHO, formed in vehicle exhaust gases by incomplete combustion of fuel) on the p

143 he gPAD was applied to detect NOx in air and exhaust gases from cars.

144 vironmental problems, removal of NO (x) from exhaust gases is necessary.

145 The filters used for sampling exhaust gases tend to lighten with an increase in engine

146 dren, we compared the effects of exposure to exhaust generated by a diesel engine with Euro V/VI emis

147 lthough the carcinogenicity of diesel engine exhaust has been demonstrated in multiple studies, littl

148 dogenous non-HBV-specific T cells, bypassing exhausted HBV-specific T cells.

149 In the present study, we found that exhausted HSV-specific CD8(+) T cells, with elevated exp

150 deficiency in c-Jun mediates dysfunction in exhausted human T cells, and that engineering CAR T cell

151 cell phenotype was skewed toward a defective/exhausted immune profile with decreased frequencies of c

152 The exhausted immune response subclass expressed many genes

153 urable clinical responses by reactivating an exhausted immune response.

154 d 2 subclasses, characterized by adaptive or exhausted immune responses.

155 state on a per-cell basis, reflecting a less exhausted immune status in the LTB state.

156 Despite their normal, 'non-exhausted' immunophenotype, Tox-deleted TST cells remain

157 rolled exposure study to allergen and diesel exhaust in humans, and measured single-site (CpG) resolu

158 y the flow and erosion behavior of gas-solid exhaust in the polysilicon reduction furnace, the flow c

159 ly all adoptively transferred TEa cells were exhausted in CB6F1 mice.

160 Current methods for treating combustion exhaust include the catalytic converter in conjunction w

161 st mating-induced death until self-sperm are exhausted, increasing the chances that mothers will surv

162 in addition to engaging specific subsets of exhausted-like CD8 T cells.

163 and TIGIT were committed to a dysfunctional, exhausted-like lineage.

164 Finally, these 'exhausted-like' ILC2s are unable to induce type 2 immune

165 t inhibitors to restore the functionality of exhausted lymphocytes, very little is known about the fa

166 TCR repertoire response against a massively exhausted lymphocytic choriomeningitis virus (LCMV) epit

167 Exposure to diesel exhaust may play a role in the development and progressi

168 HIV-specific CTL responses and reversed the exhausted memory phenotype from a T-bet(low)/Eomes(+) to

169 hat comprised of pro-oxidant constituents of exhaust, namely, carbon dioxide (13%), carbon monoxide (

170 , tobacco, ozone, particulate matter, diesel exhaust, nanoparticles, and microplastic on the integrit

171 boratory experiments, the emission factor of exhaust NCA varied from a relatively low value of 1.6.10

172 ns for cancer immunotherapy and suggest that exhausted NK cells could be targeted with inhibitory che

173 ticulate samples taken from the cylinder and exhaust of a diesel engine during combustion of fossil d

174 ate and vapor phase emissions in the diluted exhaust of a light-duty diesel engine designed for Euro

175 between honey bees exposed to either diesel exhaust or clean air across the entire duration of the e

176 ilin as part of a molecular pathway in which exhausted or "dysfunctional" CD8+ T cells enhance cellul

177 ligible for the trial, patients have to have exhausted or declined standard therapies, and have malig

178 her frequencies of CD39(+)CD8(+) T cells and exhausted or dying T cells.

179 T cells, with limited overlap with those of exhausted or tolerant T cells; accordingly, CD8(+) T cel

180 to 8-wk intervals after the patients either exhausted or were considered unfit for all approved conv

181 n part because they enter a hyporesponsive ('exhausted' or 'dysfunctional') state(6-9) triggered by c

182 culate matter, including diesel or biodiesel exhausts, or wood smoke, all complex environmental mixtu

183 Board Haagen-Smit Laboratory to characterize exhaust organics from 20 gasoline vehicles recruited fro

184 The exhaust output of biodiesel was found to contain signifi

185 treatment but also with aftertreatment by an exhaust oxidation catalyst and particle filter.

186 s impact on the abundance and composition of exhaust PAH.

187 waste sites, and mobile sources (automobile exhaust); paints, paint products, and thinners; and seco

188 established asthma in the context of diesel exhaust particle (DEP) exposure.

189 dvanced EAT decreased the emissions of fresh exhaust particle mass as much as 98% (from 44.7 to 0.73

190 .7 to 0.73 mg/kWh) and the formation of aged exhaust particle mass ~100% (from 106.2 to ~0 mg/kWh).

191 e we showed that maternal exposure to diesel exhaust particles (DEP) predisposed offspring to allergi

192 xposure to traffic pollution, notably diesel exhaust particles (DEP), increases risk for asthma and a

193 Diesel exhaust particles (DEPs) are major air pollutants that l

194 els of airborne particulates, such as diesel exhaust particles (DEPs).

195 ophilic pollutants benzo[a]pyrene and diesel exhaust particles impact on the activation of lipid-spec

196 Similarly, diesel exhaust particles showed a marginal inhibitory effect.

197 The size distribution of fresh exhaust particles was bimodal for all the fuels, the nuc

198 typically dominated the composition of fresh exhaust particles, aged particles contained more sulfate

199 e potential health impact of exposure to the exhaust, particularly in regard to young children who ar

200 he pathophysiology due to exposure to diesel exhaust particulate matter (DEP).

201 In utero exposure to diesel exhaust particulates is associated with an altered cardi

202 eposited on the leaves after exposure to the exhaust particulates.

203 hibitory molecules-double-positive, severely exhausted PD-1(+)CD8(+) T cells, leading to reduced tumo

204 approach was associated with the absence of exhausted PD-1hi T cells in treated and distant tumors,

205 High levels of exhausted (PD-1+) and senescent (CD28null) CD4+ and CD8+

206 acteristics: T cells with a regulatory or an exhausted phenotype and macrophages with an M2 phenotype

207 HIV-specific CD8(+) T cells demonstrate an exhausted phenotype associated with increased expression

208 eptor and correlates with the presence of an exhausted phenotype during chronic infections with lymph

209 nal flow cytometry, we demonstrate that this exhausted phenotype is also prevalent among peripheral a

210 or-infiltrating T cells exhibit a terminally exhausted phenotype, marked by a loss of self-renewal ca

211 d decreased PD-1 and LAG3, suggesting a less exhausted phenotype.

212 islet-specific CD8(+) T cells expressing an 'exhausted' phenotype.

213 despite the reduction of their emissions at exhaust pipe over the last two decades.

214 ereas cases of intermediate severity show an exhausted platelet and hyporeactive neutrophil phenotype

215 The emission factors were compared to ship exhaust plume observations and, furthermore, exploited i

216 of antitumor recognition, but they are also exhausted, preventing an effective antitumor immune resp

217 rack [2] and control [3] prey, as well as to exhaust prey by causing involuntary fatigue through remo

218 asmablasts, while negatively associated with exhausted/regulatory B cells.

219 ve patients, are replenished with fresh, non-exhausted replacement cells from sites outside the tumou

220 Exposure to biodiesel exhaust resulted in significantly greater cell death and

221 Experiments consisted of 60 exhaust samples for seven engine types (used in commerci

222 ur level liquid fuels, natural gas (NG), and exhaust scrubbers on particulate mass (PM) and nonvolati

223 e HC emissions first indicated that roadside exhaust sensors could detect the presence of evaporative

224 ike organic aerosol (typically fresh traffic exhaust) sorbs DEHP more efficiently than aged organic a

225 We explicitly model exhaust stack CO(2) emissions, production costs, health

226 h low clonal expansion, unlike the classical exhausted state observed in primary HCC.

227 node in our network drives cells towards an exhausted state.

228 mechanisms by which these ligands affect the exhausted states of immune cells, however, are largely u

229 rrelated with PD-1 expression, indicating an exhausted status of SATB1-negative malignant T cells.

230 otential to produce olfactory cells; and (3) exhausted stem cells alter the local retinoic acid metab

231 In-use exhaust stream CH(4) emissions from two dual fuel marine

232 increase fuel economy, was captured from the exhaust stream of a diesel engine and was characterized

233 sensor was subsequently used to measure the exhaust-stream CH(4) concentration from two diesel pilot

234 tor of the differentiation of the terminally exhausted subpopulation that functions by attenuating ty

235 and their presence correlated with multiple exhausted subpopulations of T cells.

236 progenitor exhausted and Tim-3(+) terminally exhausted subpopulations through unknown mechanisms.

237 data suggest that dominant clonotypes in the exhausted subset are derived from a diverse pool of stem

238 Further, the exhausted subset was composed of dominant clonotypes tha

239 ubset; ~10-fold GP33(+) and ~5-fold GP276(+) exhausted subset).

240 timately converted into a fourth, terminally exhausted subset.

241 TCR repertoires of stem-like and terminally exhausted subsets generated during chronic LCMV infectio

242 Exhausted T and NK cells, regulatory T cells (Tregs), al

243 eal a link between Trib1 and effector versus exhausted T cell differentiation that can be targeted to

244 h PD-1 was associated with a more terminally exhausted T cell phenotype in HIV-1 patients.

245 al-9(-) T cells, which is consistent with an exhausted T cell phenotype.

246 otype, with the unhelped set showing a more "exhausted T cell" transcriptional profile.

247 n immune cells by enhancing IFNG produced by exhausted T cells (T(EX)).

248 xidase, suggesting activated and potentially exhausted T cells and activated neutrophils, respectivel

249 s reduced the proportion of T-regulatory and exhausted T cells and decreased T-cell expression of GAT

250 ession patterns, strong correlations between exhausted T cells and SLAMF7(+) tumor-associated macroph

251 TAMs showed the strongest correlations with exhausted T cells and were an independent prognostic fac

252 of T cell dysfunction and the maintenance of exhausted T cells during chronic infection, and provide

253 dysfunctional TST cells from tumours and in exhausted T cells during chronic viral infection.

254 he balance between terminally and progenitor-exhausted T cells favoring the latter.

255 oint-blockade (ICB)-mediated rejuvenation of exhausted T cells has emerged as a promising approach fo

256 evelopment and maintenance of populations of exhausted T cells in mice requires the thymocyte selecti

257 s reduces the proportion of T-regulatory and exhausted T cells in pancreatic tumors and increases num

258 ze the phenotypic and functional features of exhausted T cells remain poorly understood(9-12).

259 dichotomy between terminally differentiated exhausted T cells that are TCF1(-) and the self-renewing

260 he infection, chronic infection may generate exhausted T cells that could in fact facilitate transpla

261 Despite the similarities with exhausted T cells we speculate that at least in malaria,

262 s unique in its ability to cause the loss of exhausted T cells within tumors while sparing nonmaligna

263 been implicated in mediating dysfunction in exhausted T cells(7-10).

264 on increases T cell infiltration, diminishes exhausted T cells, and abrogates resistance to anti-PD-L

265 tures of terminally exhausted and progenitor-exhausted T cells, respectively.

266 f the CD44(+)PD1(+)TCF1(+)TIM3(-) progenitor exhausted T cells, which was associated with their reduc

267 to control tumor growth than are terminally exhausted T cells.

268 and epigenetic characteristics of terminally exhausted T cells.

269 continuous progression from pre-exhausted to exhausted T cells.

270 ressed and co-regulated on dysfunctional or 'exhausted' T cells in chronic viral infections and cance

271 Immune checkpoint therapy, which activates 'exhausted' T cells, has been found to be highly effectiv

272 Those exhausted TEa cells lost ability to reject Balb/c skins

273 ot reduce SOA production at idle loads where exhaust temperatures were low enough to limit removal of

274 a modern aftertreatment system (ATS) at low exhaust temperatures.

275 he model used a simulated mixture of vehicle exhaust that comprised of pro-oxidant constituents of ex

276 even preloading times of several days do not exhaust the structural dynamics under load.

277 est natural enzymes, suggesting that we have exhausted the potential of MtCM.

278 eural intermediate progenitor cells, thereby exhausting the hippocampal neural stem cell pool.

279 rs a mechanism by which genotoxic Salmonella exhausts the RPA response by inducing ssDNA formation, d

280 lied to PM samples from woodsmoke and diesel exhaust, the model accurately predicts HMW PAH concentra

281 Despite considerable resources it exhausts, the disease remains very prevalent, and the in

282 epeated injuries activate the stem cells and exhaust their potential to produce olfactory cells; and

283 Progenitor exhausted TILs can respond to anti-PD-1 therapy, but ter

284 respond to anti-PD-1 therapy, but terminally exhausted TILs cannot.

285 long term and differentiate into 'terminally exhausted' TILs.

286 T cells show continuous progression from pre-exhausted to exhausted T cells.

287 es that are 100 degrees C lower than current exhaust-treatment catalysts.

288 by reinvigorating ("rebooting") pre-existing exhausted tumor-infiltrating ilymphocytes (TILs).

289 Appliance exhaust typically exhibits a brief methane concentration

290 the residential coal combustion and vehicle exhaust under poor winter dispersion conditions.

291 ration is mainly due to an expansion of the "exhausted," virus-specific B cells, i.e., activated memo

292 content resulting into an upgrading of the "exhausted waste oils" and ii) the production of a bio-ba

293 uild a connection to the host vasculature to exhaust water, solutes and carbohydrates.

294 etical Ni sources (industrial and automobile exhausts) were evaluated, demonstrating the health benef

295 hen freshly harvested from mice, but rapidly exhausted when reintroduced in culture.

296 abundant ultrafine particles in the aviation exhaust with diameters less than 100 nm may penetrate de

297 ells are more differentiated, activated, and exhausted, with reduced killing capacity in vitro than b

298 e modes, and samples were collected from the exhaust without aftertreatment but also with aftertreatm

299 To determine whether exposure to diesel exhaust would alter their tolerance to a subsequent abio

300 Ignoring secondary chemistry from diesel exhaust would lead to underestimates of both organic aer