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1 t beta-catenin is required qualitatively for exocrine acinar but not endocrine development, precisely
2 ever, mice with beta1 syntrophin deletion in exocrine acinar cells exhibit increased severity of ceru
3 oform blocked the irreversible transition of exocrine acinar cells into pancreatic preneoplastic duct
4  and (4) aberrantly differentiated endocrine exocrine (ADEX) that correlate with histopathological ch
5                 We found that, compared with exocrine and beta cells, differentiated alpha cells exhi
6                   Within the human pancreas, exocrine and endocrine cells control secretion of digest
7  which are aliphatic amines, are reported in exocrine and endocrine cells, with insulin-producing bet
8 ong-term survival, quality-of-life pain, and exocrine and endocrine function.
9 d vertebrates, is a compound organ with both exocrine and endocrine functions.
10 ent of a periampullary tumor and can lead to exocrine and endocrine insufficiency such as malnutritio
11 llow-up of 36 months, the rates of new-onset exocrine and endocrine insufficiency were 6%and 2%, resp
12 igated the heretofore unknown role of Ogt in exocrine and endocrine islet development.
13                                   Pancreatic exocrine and endocrine lineages arise from multipotent p
14 radiographs did not discriminate between the exocrine and endocrine pancreas in control animals, wher
15 humans resulted in a marked expansion of the exocrine and endocrine pancreatic compartments, the form
16  acinar-specific CEL-MUT protein causes both exocrine and endocrine pancreatic disease.
17 ssue replacement, resulting in chronic pain, exocrine and endocrine pancreatic insufficiency, reduced
18  mild exocrine pancreas disease, progressive exocrine and endocrine pancreatic loss during the first
19                                              Exocrine and endocrine pancreatic secretions are regulat
20 letion attenuated the loss of PERK-deficient exocrine and endocrine pancreatic tissues and prevented
21 rmones and neural inputs regulate pancreatic exocrine and endocrine secretion.
22 ficient embryonic pancreas, defects occur in exocrine and endocrine tissues shortly after differentia
23  an indispensible role in the development of exocrine and ocular surface disease in Aire knockout mic
24 he presence of periepithelial infiltrates in exocrine and parenchymal organs or resulting from immuno
25               Metformin decreased pancreatic exocrine and trended to decrease endocrine apoptosis in
26 ing from abdominal pain, acute pancreatitis, exocrine and/or endocrine dysfunction, and pancreatic ca
27 ue constellation of neurological, endocrine, exocrine, and haematological findings that is caused by
28 oride channel (CaCC) important for neuronal, exocrine, and smooth muscle functions.
29  when administered concurrently, and reduces exocrine apoptosis (reducing the impact on potential isl
30 ibed in the literature, with an incidence of exocrine atrophy/inflammation seen in S-D (42-72%), HIP
31 -1R knockout mouse model to demonstrate that exocrine binding of exendin is exclusively via GLP-1R (~
32 l that central D2Rs mediate a neuroendocrine-exocrine cascade that controls the maturation of the GH
33 s new insights into pancreatic endocrine and exocrine cell differentiation landscapes and identifies
34          Using these reagents, the timing of exocrine cell fate commitment in the developing pancreas
35 eas development is critical to endocrine and exocrine cell fate.
36  the 52-week study showed an increase in the exocrine cell mass in liraglutide-dosed animals, with no
37                                   Pancreatic exocrine cell plasticity can be observed during developm
38 d with incretin therapy, with both increased exocrine cell proliferation (P < 0.0001) and dysplasia (
39 non-endocrine cells that resemble pancreatic exocrine cells and a previously unreported population th
40 4 may play a role in the efflux of cAMP from exocrine cells and neurokinin receptors are important in
41 acute inflammatory stress by locking damaged exocrine cells in a permanently de-differentiated state.
42                            The endocrine and exocrine cells in the adult pancreas are not static, but
43                  Reprogramming of pancreatic exocrine cells into cells resembling beta cells may prov
44  cells (hPSCs) into pancreatic endocrine and exocrine cells through intermediates resembling in vivo
45 he nongenetic conversion of human pancreatic exocrine cells to endocrine cells is novel and represent
46  development, in the progression from normal exocrine cells to metastatic PDA.
47                                       In the exocrine cells, a rapid efflux of radioactivity was obse
48 ecent studies report low levels of GLP-1R on exocrine cells, complicating beta-cell mass quantificati
49 protein content), as well as degeneration of exocrine cells, decreased zymogen granules, and alterati
50  (INS-1 and human islets), but not PANC1 and exocrine cells, was mediated specifically by intracellul
51 ntling of the secretory apparatus of diverse exocrine cells.
52 ndscape of human pancreatic alpha, beta, and exocrine cells.
53 soforms play a regulatory role in pancreatic exocrine cells.
54 ha, 105 beta, 6 delta endocrine cells and 47 exocrine cells.
55 ls, with normal composition of endocrine and exocrine cellular compartments.
56 lear form of Yap are apparent throughout the exocrine compartment and correlate with increases in lev
57                                          The exocrine compartment makes and secretes digestive enzyme
58                  No changes were seen in the exocrine compartment of the pancreas, and the mice had o
59 s1 leads to the impaired regeneration of the exocrine compartment, accelerated fatty metaplasia, and
60 ptophysin dual positive cells arise from the exocrine compartment.
61 t the islet is completely separated from the exocrine compartment.
62 n show elevated levels of CD8 T cells in the exocrine compartment.
63 ontent or the number of Ki67(+) cells in the exocrine compartment; however, pancreatic protein conten
64 blocked differentiation of the endocrine and exocrine compartments, whereas loss of a single Yap alle
65 We found that serpin B13 is expressed in the exocrine component of the mouse pancreas, including the
66                                          The exocrine congenital defects of Prox1(DeltaPanc) pancreat
67 rom beta-cell injury due in part to pancreas exocrine damage and lipofibrosis.
68                                   Along with exocrine defects, including impaired acinar cell maturat
69 reatic findings in all groups included focal exocrine degeneration, atrophy, inflammation, ductular c
70  in transcriptional regulators of pancreatic exocrine differentiation have been implicated in pancrea
71  and disruptions in pancreatic branching and exocrine differentiation.
72 as, as well as genetic forms of diabetes and exocrine diseases.
73 ators Neurog3, MafA, Pdx1 and Pax6 converted exocrine duct cells into endocrine progeny with hallmark
74  a monogenic form of diabetes and pancreatic exocrine dysfunction due to mutations in the CEL gene en
75 s with severe developmental delay, endo- and exocrine dysfunction, impairment of the sensory and auto
76 the exocrine pancreas and, in the context of exocrine dysplasia, may accelerate formation of neoplast
77 minations and quantifications indicated that exocrine, endocrine and ductal compartments retained the
78                                          The exocrine-enriched cells rapidly dedifferentiated in cult
79 his study was to determine whether the human exocrine-enriched fraction from the islet isolation proc
80        In conclusion, reprogramming of human exocrine-enriched tissue can be best achieved using fres
81 , most AMY(-) clusters were positive for the exocrine enzymes lipase and trypsinogen.
82  controls the decision between endocrine and exocrine fates of multipotent progenitors in the develop
83 functions (including water/salt homeostasis, exocrine fluid secretion, and epidermal hydration) and h
84  per gram at 15 min), most likely due to the exocrine function and high protein turnover within the p
85 e high viability and preserved endocrine and exocrine function in HPS for at least 10 days after sect
86 rfusion, injury, as measured by amylase, and exocrine function of human pancreases using EVNP and dem
87 , quality of life [QoL]), medical (endo- and exocrine function), and clinical (reoperation) results r
88  gonadal testosterone production, as well as exocrine function, with spermatogenesis.
89  noninvasive method for assessing pancreatic exocrine function.
90 lop a noninvasive imaging test of pancreatic exocrine function.
91 ic tissue with differentiated morphology and exocrine function.
92 pment and support a rationale for performing exocrine functional tests for patients with IRF6-damagin
93 nced by preservation of pancreatic endocrine/exocrine functions and a low rate of reoperation for tum
94 w-up of 50 months, both pancreatic endocrine/exocrine functions were preserved in 92% of patients.
95 as controls metabolism through endocrine and exocrine functions.
96 nvolved in the generation and maintenance of exocrine gland acinar cells have not yet been establishe
97  Although IgG autoantibodies against various exocrine gland antigens have been described in IgG4-RD,
98 ion, our data report a novel role of Irf6 in exocrine gland development and support a rationale for p
99 ver 3 wk post-AdV infection, consistent with exocrine gland dysfunction as a consequence of the infla
100 lling, smooth muscle contraction, airway and exocrine gland secretion, and rhythmic movements of the
101 S. invicta venom, N. fulva applies abdominal exocrine gland secretions to its cuticle.
102  Characterizations of the myriad neuropteran exocrine gland systems, including prothoracic, metathora
103 s similar to CpMRP and assume therefore that exocrine gland-based defensive strategies, evolved by th
104  defined circuit by which a single compound, exocrine gland-secreted peptide-1 (ESP1), enhances repro
105 roduced by young mice before puberty, termed exocrine-gland secreting peptide 22 (ESP22).
106                         The morphology of 21 exocrine glands and 13 supposedly exocrine structures re
107 helial mononuclear cell infiltrates, both in exocrine glands and in other parenchymal organs (kidney,
108      In primary Sjogren's syndrome (pSS) the exocrine glands become infiltrated with lymphocytes inst
109 likely calcium-dependent chloride channel of exocrine glands has led to a more detailed understanding
110 HAT-L4 was expressed in epithelial cells and exocrine glands in tissues including skin, esophagus, tr
111 ale detail of dynamic actomyosin networks in exocrine glands of live mice.
112 oimmune disease involving salivary and other exocrine glands that leads to progressive lymphocytic in
113  lymphocytic infiltration and destruction of exocrine glands, followed by systemic organ damage and B
114 characterized by lymphocytic infiltration of exocrine glands, mainly salivary and lacrimal, resulting
115 -Higueras et al., 2014], suggesting that the exocrine glands, similar to the endocrine, develop from
116  identify the role of IRF6 in development of exocrine glands, specifically the major salivary glands.
117 ch as the secretions of airway epithelia and exocrine glands, the contraction of smooth muscles, and
118 c autoimmune disorder that typically affects exocrine glands--mainly labial and lacrimal--leading to
119 ized by diminished secretory function of the exocrine glands.
120 secretion is utilized by epithelial cells of exocrine glands.
121  by a progressive chronic attack against the exocrine glands.
122 e (SjS), an autoimmune disease targeting the exocrine glands.
123 ent of salivary glands and potentially other exocrine glands.
124 y and autonomic nervous system and endo- and exocrine glands.
125 eaf clipping, and application of fluids from exocrine glands.
126 ophilic Cy-7 exendin to selectively preblock exocrine GLP-1R in healthy and streptozotocin-induced di
127 ort the idea that pocket shark's pockets are exocrine holocrine glands capable of discharging a biolu
128 ke peptide 1 (GLP-1) receptor agonist, is an exocrine hormone, which has potent insulinotropic action
129 l hernias (23% vs 53%; P = .004), pancreatic exocrine insufficiency (29% vs 56%; P = .03), or endocri
130                                   Pancreatic exocrine insufficiency (PEI) reduces pancreatic secretio
131 left-sided resection, the risk of developing exocrine insufficiency after surgery was higher after dr
132  sequelae include diabetes in 38% to 40% and exocrine insufficiency in 30% to 48%.
133               Two minipigs failed to develop exocrine insufficiency most likely due to undetected acc
134 ts with cystic fibrosis (CF) have pancreatic exocrine insufficiency that results in nutrient malabsor
135                                  Subclinical exocrine insufficiency was demonstrated by low fecal ela
136 fficiency was present in 57% of patients and exocrine insufficiency was present in 77%.
137 mplications such as malnutrition, pancreatic exocrine insufficiency, and diabetes; refer to a special
138                             We also measured exocrine insufficiency, quality of life (using the Short
139 adult-onset diabetes, with subclinical or no exocrine insufficiency, suggesting an important role for
140  or childhood-onset diabetes with or without exocrine insufficiency.
141 evention and control of diabetes and perhaps exocrine insufficiency.
142  adult-onset diabetes with subclinical or no exocrine insufficiency.
143 ent-labeled red blood cells at the endocrine-exocrine interface revealed bidirectional blood flow, wi
144 apillaries were, however, found at the islet-exocrine interface, frequently located along blood capil
145 ted in the insulitic lesion in the endocrine-exocrine interface.
146 trates, independently of the severity of the exocrine lesion and in the absence of amyloid deposits.
147 ination to inactivate Pdx1 in the pancreatic exocrine lineage during embryonic stages.
148 st that POSTN is a crucial factor for proper exocrine lineage-specific regeneration after severe acut
149 s were located in fibrotic septa between the exocrine lobules and adjacent to the ductal system of th
150 stinct from the actin web that surrounds the exocrine lumen.
151 gulate networks involved in KRAS activation, exocrine (NR5A2 and RBPJL), and endocrine differentiatio
152                          MNH is emitted from exocrine opisthonotal oil glands, which are potent organ
153 ations such as pancreaticocutaneous fistula, exocrine or endocrine pancreatic insufficiency, need for
154 osin lattice that regulates secretion in the exocrine organs of live animals.
155  essential role in the secretory function of exocrine organs.
156 lated rapidly in both endocrine CM cells and exocrine PANC-1 cells.
157 inhibition of polyamine biosynthesis reduces exocrine pancreas and beta cell mass, and that these red
158 s a lethal autoimmune syndrome involving the exocrine pancreas and other abdominal organs.
159 herapy may induce focal proliferation in the exocrine pancreas and, in the context of exocrine dyspla
160                              Diseases of the exocrine pancreas are often associated with perturbed di
161  we report an unexpected role for SHP in the exocrine pancreas as a modulator of the endoplasmic reti
162 creatitis is an inflammatory disorder of the exocrine pancreas associated with tissue injury and necr
163             A genuine understanding of human exocrine pancreas biology and pathobiology has been hamp
164                                          The exocrine pancreas can give rise to endocrine insulin-pro
165                                          The exocrine pancreas can undergo acinar-to-ductal metaplasi
166 Expression of the homeobox gene Prox1 in the exocrine pancreas changes throughout development in mice
167 and that maintained ductal fluid flow in the exocrine pancreas could delay the onset of CFRD.
168 netic factors likely influence the extent of exocrine pancreas disease in CF ferrets and have implica
169     Although CF kits are born with only mild exocrine pancreas disease, progressive exocrine and endo
170 raumatic brain injury or cardiac arrest; and exocrine pancreas DNA was identified in patients with pa
171 eatorrhea was induced by embolization of the exocrine pancreas gland and pancreatic duct ligation in
172 pletion or inhibition of 12-LOX impairs both exocrine pancreas growth and unexpectedly, the generatio
173 dence of cellular heterogeneity in the human exocrine pancreas has not been yet established because o
174                   To date, the endocrine and exocrine pancreas have been studied separately by differ
175                            The endocrine and exocrine pancreas have been studied separately by endocr
176 e direct evidence that Men1 is essential for exocrine pancreas homeostasis in response to inflammatio
177 ated the role of Prox1 in development of the exocrine pancreas in mice.
178 sizes an underappreciated involvement of the exocrine pancreas in the natural course of type 1 diabet
179 ment and underscore a potential role for the exocrine pancreas in the pathogenesis of type 1 diabetes
180  increased density of leukocytes) within the exocrine pancreas in this disease, but the mechanisms un
181 cer (11)C-hydroxytryptophan in endocrine and exocrine pancreas in vitro and in vivo.
182 t collateral damage from inflammation in the exocrine pancreas is not a likely cause of DM in these d
183                                          The exocrine pancreas of adult mice can be remodeled by re-e
184 y, we report that CD8 T cells infiltrate the exocrine pancreas of diabetic subjects in high numbers a
185 copy and immunohistochemical analysis in the exocrine pancreas of multiorgan donors with T1D (both at
186  the effects of exenatide (EXE) treatment on exocrine pancreas of nonhuman primates.
187 lete neoplastic transformation of the entire exocrine pancreas shortly after birth.
188 ounts and chemotaxis as well as a diminished exocrine pancreas size in a SRP54-knockdown zebrafish mo
189 ctrum of fibro-inflammatory disorders of the exocrine pancreas that includes calcifying, obstructive,
190 ype 1 diabetes includes abnormalities in the exocrine pancreas that may induce endocrine cellular str
191 on and pancreatitis, and inflammation of the exocrine pancreas that promotes development of pancreati
192 ancreatitis is a debilitating disease of the exocrine pancreas that, under chronic conditions, is a m
193 n order to reveal a possible requirement for exocrine pancreas tissue in endocrine development and/or
194                                Endocrine and exocrine pancreas tissues are both derived from the post
195  not suffer from DM links the disease in the exocrine pancreas to a pathological process in the endoc
196  that actin-coated secretory vesicles of the exocrine pancreas travel this distance over bundles of s
197 istorically, diabetes due to diseases of the exocrine pancreas was described as pancreatogenic or pan
198 cute inflammatory phase, the recovery of the exocrine pancreas was massively impaired in Postn-defici
199 ities, including extensive fibrosis, loss of exocrine pancreas, and islet disorganization.
200 s, dynamic topographies in the endocrine and exocrine pancreas, and principles of morphologic organiz
201 eatment did not exert any negative effect on exocrine pancreas, by inducing either pancreatic inflamm
202 itis is caused by inflammatory injury to the exocrine pancreas, from which both humans and animal mod
203 in cey mutants, including HSPCs, the retina, exocrine pancreas, intestine, and jaw cartilage.
204              They are deeply embedded in the exocrine pancreas, limiting their accessibility for func
205 r tissues, including the heart, vasculature, exocrine pancreas, liver, and central nervous system.
206 ctional blood flow between the endocrine and exocrine pancreas, not necessarily a unidirectional bloo
207 P) is a painful inflammatory disorder of the exocrine pancreas, ranking as the most common gastrointe
208        This reduces enzyme production by the exocrine pancreas, resulting in digestive insufficiencie
209 activity and duct cells within the liver and exocrine pancreas, whereas hepatocyte and acinar pancrea
210 were continuously integrated to those in the exocrine pancreas, which made the islet circulation rath
211 atic capillaries were regularly found in the exocrine pancreas, with small lymphatic vessels located
212 nse hyperactivation and tissue injury of the exocrine pancreas.
213 of other diseases, including diseases of the exocrine pancreas.
214  capillary network between the endocrine and exocrine pancreas.
215 that is distinct from those that inhabit the exocrine pancreas.
216 s that are scattered unevenly throughout the exocrine pancreas.
217 t data suggest MEN1 may also function in the exocrine pancreas.
218  the pancreatic islets while being absent in exocrine pancreas.
219 nd complete neoplastic transformation of the exocrine pancreas.
220 ntracellular cyclic AMP (cAMP) levels in the exocrine pancreas.
221 atic progenitors and derived lineages of the exocrine pancreas.
222 stigated the role of Bmi1 in regeneration of exocrine pancreas.
223  syntrophin specifically in the endocrine or exocrine pancreas.
224      IL-1betaAb treatment also protected the exocrine pancreas; the number of infiltrating macrophage
225  and KCR8-16 mice developed dedifferentiated exocrine pancreata within 2 months of age and became mal
226 ficient hESCs and iPSCs manifest deficits in exocrine pancreatic and hematopoietic differentiation in
227 e of 51 y at baseline were included, and 865 exocrine pancreatic cancers were diagnosed after a media
228 yptophan is trapped in beta-cells but not in exocrine pancreatic cells.
229 imilar to those in people with CF, including exocrine pancreatic destruction, focal biliary cirrhosis
230 noreactive trypsinogen (IRT), a biomarker of exocrine pancreatic disease in cystic fibrosis (CF), is
231 le entity; it occurs because of a variety of exocrine pancreatic diseases with varying mechanisms of
232 , autophagy, and obesity interact to promote exocrine pancreatic disorders.
233 zed by bone marrow failure with neutropenia, exocrine pancreatic dysfunction and skeletal abnormaliti
234  autosomal dominantly inherited diabetes and exocrine pancreatic dysfunction.
235                               We studied how exocrine pancreatic function affects intestinal microbio
236 ancreatic cancer, which affect endocrine and exocrine pancreatic function and impose a substantial di
237          No correlation between endocrine or exocrine pancreatic function and pain was found, whereas
238                              Associations of exocrine pancreatic function with microbial diversity or
239  accessory ducts will lead to maintenance of exocrine pancreatic function.
240 ic duct ligation in a minipig model leads to exocrine pancreatic insufficiency (EPI).
241 utosomal-recessive disorder characterized by exocrine pancreatic insufficiency and hematopoietic dysf
242  the presence of epilepsy and the absence of exocrine pancreatic insufficiency and hypoplasia of nasa
243 perioperative management, a determination of exocrine pancreatic insufficiency are scarce in the lite
244 s with insulin-treated neonatal diabetes and exocrine pancreatic insufficiency requiring enzyme repla
245                 Two of the four patients had exocrine pancreatic insufficiency requiring replacement
246 an experimental approach to the induction of exocrine pancreatic insufficiency via pancreatic duct li
247                                              Exocrine pancreatic insufficiency was successfully induc
248 ve ribosome assembly and are associated with exocrine pancreatic insufficiency, chronic neutropenia a
249  minipigs is an excellent method of inducing exocrine pancreatic insufficiency.
250  via midline laparotomy for the induction of exocrine pancreatic insufficiency.
251 s primarily characterized by neutropenia and exocrine pancreatic insufficiency.
252           Patients who underwent surgery for exocrine pancreatic malignancy with curative intent were
253    Secretin is a hormone that stimulates the exocrine pancreatic secretion of bicarbonate-rich fluid
254 evidence to support a causal contribution of exocrine pancreatic status on CFRD risk.
255 fat infiltration (P < 0.05) was noted in the exocrine parenchyma of IL-1betaAb-treated CDs-HSD rats c
256 dual pancreatic lipase knockouts are lethal, exocrine parotid acini lacking lipases were used to veri
257 on model physically links both endocrine and exocrine parts of the pancreas as a single organ through
258  GATA4 gene who have diabetes and a variable exocrine phenotype.
259 eatic oncogenesis and the maintenance of the exocrine phenotype.
260                                          The exocrine portion of the pancreas functions in digestion
261 o (INS-1 and PANC1 cells and human islet and exocrine preparations) and in vivo (nonhuman primates an
262 analyzed the function of POSTN in pancreatic exocrine regeneration after the induction of a severe ac
263            Unraveling the molecular bases of exocrine regeneration may identify new therapeutic targe
264                                  Fourth, the exocrine scent glands express vvl and are regulated by H
265 t preparation was used to measure pancreatic exocrine secretion (PES) and plasma insulin following mi
266                           The final stage in exocrine secretion involves translocation of vesicles fr
267 i-organ manifestations, including pancreatic exocrine secretion.
268    Recent studies have shown that pancreatic exocrine secretions (PES) are modulated by dorsal motor
269 roader binding profile, decreases pancreatic exocrine secretions and may prevent postoperative pancre
270 ood, as well as stimulation of endocrine and exocrine secretions from a wide range of organs.
271         We show here novel effects of insect exocrine secretions produced by caterpillars in modulati
272                          Bladder drainage of exocrine secretions was protective (HR 0.54, p = 0.002).
273 ial benefit elsewhere as lacritin flows from exocrine secretory cells.
274                        Salivary glands exert exocrine secretory function to provide saliva for lubric
275 mor microenvironment and implicate endocrine-exocrine signaling beyond insulin in PDAC development.
276 es (sweat chloride concentration, pancreatic exocrine status, and lung function) to develop benchmark
277 logy of 21 exocrine glands and 13 supposedly exocrine structures recorded for lepidopteran larvae is
278 To do so, we quantified pancreatic islet and exocrine sympathetic nerve fiber area from autopsy sampl
279                          There is no loss of exocrine sympathetic nerves in either type 1 or type 2 d
280    Importantly, low-purity (30:70% endocrine:exocrine) syngeneic rat islet preparations displayed fun
281       Mantled islets are those surrounded by exocrine tissue and are difficult to purify by density g
282                          However, the mutant exocrine tissue displays extensive disorganization leadi
283 ilar expression of HLA class I in islets and exocrine tissue in subjects with recent-onset type 1 dia
284 tudy, we generated mutant mice, in which the exocrine tissue is hypoplastic, in order to reveal a pos
285 nown but essential factor(s) in the adjacent exocrine tissue that regulates proper formation of endoc
286 ass I expression was found between islet and exocrine tissue using Western blot, flow cytometry, real
287 f insulin secretion, cold ischemia time, and exocrine tissue volume transplanted, accounted for 43% o
288                    Proliferation rate of the exocrine tissue was low and comparable between groups.
289   In all subjects, HLA class I expression on exocrine tissue was low.
290 gnificantly higher CD8 T cell density in the exocrine tissue without the presence of prominent insuli
291 spontaneous inflammatory infiltration of the exocrine tissue, common to both T1D and T2D subjects.
292  CD4(+) and CD11c(+) cells were found in the exocrine tissue.
293  underlying causes of inflammation at source exocrine tissue.
294 hich are essentially mini-organs embedded in exocrine tissue.
295 d 4946 (4009) IEQ/kg, with 59.1% embedded in exocrine tissue.
296 regulated independently from the surrounding exocrine tissues.
297 acinar cell carcinoma (ACC) is an aggressive exocrine tumor with largely unknown biology.
298 of (67)Cu-CuSarTATE in AR42J (rat pancreatic exocrine) tumor-bearing mice was compared with (177)Lu-L
299 sight into the origins of neuroendocrine and exocrine tumors.
300 lls' containing granules resembling those of exocrine zymogen and endocrine hormone secreting cells;

 
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