ライフサイエンスコーパス: explain

1 is one of the most challenging phenomena to explain.

2 ying this 'molecular diversity' remain to be explained.

3 e and its metabolites have been structurally explained.

4 tial increase from the ~20 to 30% previously explained.

5 uted 56% of the immune variation, whereas MS explained 1 to 2% of the immune variance.

6 h univariable and multivariable analyses and explained 13% variance.

7 ng was a negative factor; these five factors explained 25.7% of the total variance.

8 19 LCVs, the first principal component (PC1) explained 27.7% of the total variance, and the PC1 score

9 Together the 14 putatively causal SNPs explained ~38% of NMR variation, a substantial increase

10 and MTX systemic clearance (P = 4.4 x 10-2) explained 42% of the variation in MTXPG accumulation (P

11 ferences between the term and preterm groups explained 5.7% (q = 0.024) of the variance in microbial

12 ely, sex ratio (SR) and sex-specific density explained 52.8% (males) and 91.0% (females) of variance

13 Anatomical site explained 54% (q = 0.0004) of the variance and differenc

14 Our multiple linear regression model explained 61.1% of the variance in 2017 aggregate scores

15 Adjustment for the age distribution of cases explains 66% of the variation across countries, with a r

16 PCA was applied (explaining 68.43% of the total variance), and the higher

17 ixture of the identified six compounds could explain 79% of the observed effect, with albendazole and

18 We project that sample sizes required to explain 80% of GWAS heritability vary from 60,000 cases

19 eviously characterized and that collectively explain a diverse range of cannabis terpenes.

20 that TE-derived CTCF binding divergence may explain a large fraction of variable loops.

21 e arising metastable point defect population explains a common root cause of performance losses.

22 Our modeling framework recovers and explains a large diversity of shell forms and highlights

23 the additive effect of many common variants explains a larger proportion of genetic susceptibility t

24 plasma, is a plausible pathogenic mechanism explaining adverse obstetrical outcomes in antiphospholi

25 f B cell activation has long been invoked to explain alternate B cell recruitment into immune respons

26 ntal and computational work to characterize, explain, and make predictions about these pathways.

27 ely high fluid delta(30)Si values, which can explain anomalies in the marine Si budget like in the Ca

28 A functional perspective helps explain asymmetries in blame and praise: we propose that

29 Additive genetic factors explained between 49.1% and 62.7% of variation in caries

30 Results provide testable hypotheses explaining both intermittent and regular patterns of spo

31 i at which gene expression could potentially explain breast cancer risk phenotypes.

32 ts on olfactory decision tasks could be best explained by a Bayesian model that combines reinforcemen

33 tients with early COVID-19 can be reasonably explained by a combination of pulmonary embolism, ventil

34 We found that the dynamics of eS6-P can be explained by a feedforward circuit with inputs from both

35 terogeneity of oil concentration, 89% can be explained by additional linked mean-effects genetic vari

36 While some discordances can be explained by additional records of deportations within t

37 Variances explained by adult-based GRSs for disease-related traits

38 that the longest codon dwell times could be explained by aminoacylation levels or high codon usage r

39 rger basal ganglia and internal capsule, not explained by antipsychotic dose.

40 9% of the shared variants are not explained by any model.

41 sults suggest that aHC BOLD signal is better explained by avoidance behavior than by threat features

42 served increases in gonorrhea rates could be explained by both increases in screening and the prevale

43 These effects are not explained by changes in metabolic rate, nor CO(2) , and

44 Age disparities in observed cases could be explained by children having lower susceptibility to inf

45 This pattern is explained by color variation within families: Chick colo

46 These associations may be explained by defective clearance of intraorgan microthro

47 Poor photoproduct yields are explained by donor-independent, fast charge recombinatio

48 at enhancers in PD neurons, which is partly explained by elevated hydroxymethylation levels.

49 deficits in task performance were plausibly explained by elevated sensory noise.

50 we demonstrate that anthropophagy was better explained by extrinsic factors (namely, local host avail

51 This heterogeneity may be explained by genetic variation within the fatty acid des

52 We show that this can be explained by genuine Neanderthal ancestry due to migrati

53 tively), while that of the abundant was more explained by geospatial distance (22.1%) than local fact

54 MSM with >10 recent sex partners, and partly explained by higher CMV seroprevalence in MSM.

55 ulated that such paradoxical findings may be explained by impaired hypoxic pulmonary vasoconstriction

56 genotype-derived heritability that cannot be explained by inclusion of milder cases and a higher prop

57 ory effects, which our modeling shows can be explained by intracellular chloride processing.

58 ominance of DNRA over denitrification is not explained by kinetics or thermodynamics.

59 urs in all individuals with SCA6, and can be explained by lesser temporal variability.

60 The phylogeny of rare bacteria was equally explained by local factors (soil and vegetation) and geo

61 hey reveal postdepositional U redistribution explained by major U speciation changes due to redox cyc

62 xcessive alcohol consumption could be partly explained by metabolic shifts.

63 These relationships could not be explained by other functional annotations known to be as

64 rigated maize fields in the US Corn Belt was explained by persistent factors and identified the under

65 ditions; 81.7% of edaphic variation could be explained by polyploidisation.

66 systemic therapy, a difference not entirely explained by posttreatment IOP elevation.

67 ss likely to co-express, co-localize, and be explained by simple mass action kinetics, and more likel

68 stability of the 5Ni/3Mg-ZrO(2) catalyst was explained by the ability of CO(2) to partially oxidize t

69 is imprecise, and these imprecisions can be explained by the combined influences of random diffusive

70 The bulk Earth's carbon/sulfur ratio is best explained by the delivery of most of Earth's volatiles f

71 We found that confidence reports are best explained by the difference between the posterior probab

72 reast cancer and schizophrenia may partly be explained by the genetic overlap between the two phenoty

73 ts treated with belatacept and whether it is explained by the higher risk of CMV reactivation and inf

74 Having SAM among under-five children was explained by the individual-, household- and neighbourho

75 The mechanism inducing the enhancement is explained by the initially reparable pores generated by

76 % of the variance in chi trends across years explained by the model.

77 This can be explained by the observation that, on the Au electrode,

78 ive activities of genomic sequences are best explained by the predicted occupancy of binding sites, r

79 e mechanism underlying these symptoms can be explained by the presence of nonfiring pacemaker cells.

80 (i.e., the number of cusps, crests), can be explained by the same developmental model.

81 s, the proportion of variance for SUA levels explained by the variants was greater in the Korean popu

82 e fraction of feeding study intake variation explained by these regression models.

83 d natural variation in chorismate metabolism explained by transcriptional reprogramming of specific s

84 XRD and FTIR analyses were used to explain changes in protein structure and the presence of

85 ive lability, aggressive or impulsive traits explain childhood trauma's effects on SI variability and

86 we outline a unified mechanistic scheme that explains competing processes under varying photochemical

87 ndent habitat selection patterns may help to explain complex predator-prey dynamics and cascading ind

88 proposed, based on advances in immunology to explain conflicting data.

89 m fundamental demographic choices while also explaining consistent variations.

90 Here, we propose a model that explains cophylogenetic patterns without relying on co-e

91 ians in informed risk-benefit discussions to explain COVID-19 risk and enable an evidenced-based appr

92 lexity of its infection process and may help explain current challenges in controlling it.

93 ct emissions, is an overlooked mechanism for explaining decadal-scale changes in the land carbon sink

94 SNPeffect is used to explain differential growth rate and metabolite accumula

95 These differences are sufficient to explain differential recruitment of diacylglycerol bindi

96 The possibility that such processes can explain experimental live-cell imaging data measuring di

97 The dehydration sequence of saccharides was explained from the study of saccharide/cation interactio

98 altered repair of damaged telomeres, can be explained from this viewpoint.

99 Here we examine which traits best explain fungal decomposition ability by combining detail

100 d height-for-age z score (HAZ) decomposition explained >100% of predicted change in mean HAZ between

101 on environment, for which our model can only explain half of our observation.

102 These findings explain how a distinctive ubiquitin-like protein alters

103 P signaling in insect antiviral immunity and explain how a family of cGAS-STING evasion enzymes evolv

104 In this Perspective we explain how executable computational models meet this ne

105 anistic models at the psychological level to explain how humans plan, execute, and consolidate skille

106 ts provide a general conceptual framework to explain how internal signals can be integrated into our

107 This helps to explain how mass cell suicide can evolve, as any small b

108 Collectively, we derive a molecular model to explain how Microprocessor recognizes a pri-miRNA and ac

109 ings are noteworthy because they may in part explain how obesity drives colon cancer progression.

110 approach reproduce the spectroscopy data and explain how periodic microstructures play a critical rol

111 We applied machine learning to explain how specific interactions controlled the promote

112 cross disparate but linked brain regions may explain how stereotyped patterns of neurodegeneration ar

113 Our models explain how the activation of pyramidal cells or PV(+) c

114 To explain how these problems develop, the neuroimmune netw

115 Here, we explain how these remarkable birds can help us uncover t

116 ential key checkpoint of cambial activities, explaining how cambium-driven growth is altered in respo

117 newly-discovered NSC-BV communication route explains how circulatory neurogenic mediators are 'sense

118 Our analysis explains how mechanical force applied to alphaE-catenin

119 Our structure explains how up-stream effectors, including DOCK2 and EL

120 assic model of vision, including ffCNNs, can explain human global shape processing.

121 had the strongest predictive power, and each explained hundreds of metabolites-in some cases, explain

122 tes that individual differences in behaviour explain important differences in survival but not in the

123 -of-the-art RBC-mediated delivery systems is explained in detail to inspire the design and implementa

124 Mechanistically, this was explained in neonatal mice, in which long-chain, but not

125 The metastability is explained in terms of the intrinsic conformations of the

126 nt focal epilepsy were instructed to overtly explain, in a sentence, 'what is in the image (subject)'

127 boxylate oxidase 4 (ACO4), which may in part explain increased ethylene production and signaling in R

128 o be expensive and prone to failure, in part explaining its low rates of implementation compared with

129 itionally, we find that larval tolerance can explain large-scale biogeographic patterns for this spec

130 logy of schizophrenia, most of the currently explained liability is within common variation, suggesti

131 These findings may help to explain many of the biogeographic patterns and connectio

132 A reaction pathway that explains MeIQ formation is proposed.

133 ce of 79% of infant-colonizing microbes, but explain microbial prevalence less well in adults from ri

134 ained hundreds of metabolites-in some cases, explaining more than 50% of the observed variance.

135 and that ideological self-selection does not explain most of that coexposure.

136 diated regulation of gene expression did not explain most of the effects.

137 overs to crossover-specific strand exchange, explaining Mph1's apparent anti-crossover function.

138 nd climate zones, with local characteristics explaining much of the variability.

139 The identified SNPs explained nearly all the heritability expected in each s

140 microsite heterogeneity is often invoked to explain non-normal distributions of greenhouse gas (GHG)

141 perimental P(ion) values were high enough to explain observed cellular uptake by passive diffusion wi

142 ound traits of tropical tree seedlings could explain observed occurrence along gradients of resources

143 on-colonization dynamics within species that explains observed patterns of geographical range loss an

144 The detected 290 chemicals explained only a small fraction (<8%) of the measured ef

145 enable to ssNMR, thus appears to be a key to explaining other macroscopic properties, such as hardnes

146 gene-centered view of this kind can help to explain otherwise puzzling aspects of biology, including

147 Deterministic trends with dynamic covariates explain over 35% of the variability in DO.

148 at dilution benefits alone are not enough to explain patterns in interspecific sociality.

149 This framework will be important for explaining patterns of extant floral diversity and exami

150 at protein tissue-enrichment information can explain phenotypes of genetic diseases, which cannot be

151 s as viral uptake inhibitors in COVID-19 and explained possible mechanisms of actions and the therape

152 oduced by posttetanic twitches, structurally explaining posttetanic potentiation.

153 ial expression of transcripts only partially explains protein abundance.

154 r-limited natural food sources in cities-can explain reduced avian reproductive success in urban area

155 e 'pursed' lip embouchures, which may partly explain reduced consumption from these glasses.

156 xpression, specifically miR-326, may in part explain regional differences in inflammatory cells.

157 -induced calcium signaling upon injury might explain results from large, randomized, controlled trial

158 n, soil pH and plant community richness both explained significant variation in Streptomyces beta div

159 ifying dynamic bonding model for LnB(6) that explains simultaneously EuB(6) (possessing an anomalous

160 that emerge by young adulthood and may help explain social disparities in the development of chronic

161 en more commonly admitted to ICU than can be explained solely by the demographic shift.

162 l updating into grasp plans, and may help to explain some of the more complex symptoms associated wit

163 e functional differences between species can explain species richness effects, empirical evidence reg

164 nological models to improve how these models explain T cell sensitivity and antigenic specificity.

165 Our results explain that the ostensibly equal susceptibility of NMR

166 This pattern helps to explain the absence of population-level Allee effects in

167 le the presence of garlic in the mixture can explain the activity against planktonic cultures, garlic

168 Mechanistic models have been proposed to explain the association of AF and dementia.

169 itively regulating TNF signaling may help to explain the association of PACRG and PRKN polymorphisms

170 To explain the basis for the extraordinarily high recurrenc

171 , whereas DFT studies have been used to help explain the bonding and electronic structure in these un

172 Here, we explain the challenges of sample preconcentration for CE

173 ion, we demonstrate how 'Arctic Dimming' can explain the circumpolar 'Divergence Problem', and discus

174 l Least Squares regression model was able to explain the combination of genes that contributed to inc

175 The model and its extensions parsimoniously explain the cooperative nature of the social learning ad

176 s on the ability of a computational model to explain the data, quantified in modern statistical appro

177 nstrate that resource-dependent behavior can explain the density distribution observed in locust hopp

178 ian turtles and Hawaiian turtles, may partly explain the differential dynamics of FP between the two

179 ral alternative interpretations put forth to explain the effect.

180 Another goal is to explain the enormous differences in species richness amo

181 els of conformist and anticonformist bias to explain the evolution of large-scale cooperation, and su

182 These findings help explain the flexibility of leatherwood and also call int

183 The identified electronic states explain the formation of the very large ordered magnetic

184 uctures revealed some differences that might explain the gain of potency at alpha3beta2 nAChRs.

185 lied to detect downstream pathways that best explain the gene expression data.

186 unknown but likely of critical importance to explain the geochemical behavior of U.

187 values of D(b) > 10(-10) cm(2) s(-1) fail to explain the growth of the Aitken mode.

188 rectal cancer molecular subtypes potentially explain the inconsistent findings between dietary fruit

189 frican ancestry-predominant alleles may help explain the increased frequency and severity of SSc amon

190 ption/regeneration mechanism was proposed to explain the increasing adsorption efficiency after elect

191 and extend our previous model of SZ and can explain the lack of full efficacy of conventional antips

192 I argue that these differences are unable to explain the lack of replication in Markel, 2020.

193 sensor, targeting of these pathways does not explain the lethality of anthrax toxin(1,2).

194 While atomic tunneling has been invoked to explain the low-temperature thermal conductivity of soli

195 who received FMC63-28Z T cells, which could explain the lower level of neurologic toxicity associate

196 In this article, we explain the main concepts of such analyses and provide a

197 mental dependency and genetic redundancy can explain the maintenance of atavistic traits under domest

198 ry ago(2), several efforts have been made to explain the many unusual properties of dense hydrogen, i

199 ) cross-loop interaction but failed to fully explain the means by which s(2)C(32) restricts I(34) wob

200 -derived DCs and purified MLN DCs, which may explain the mechanism underlying the impairment of DCs i

201 ith adduct formation in high-salt conditions explain the molecular species observed.

202 AMOC during the penultimate deglaciation can explain the more-rapid sea-level rise compared with the

203 However, this hypothesis cannot explain the multi-Myr lag between the delta(13)C excursi

204 The structures explain the nucleotide dependence of mHsp60 ring formati

205 g myosin interaction with actin, which could explain the observed delays in the onset of relaxation.

206 ent diversity of spatial signaling states to explain the observed shapes.

207 orphological convergence is also proposed to explain the occurrence of nonmonophyletic MIE species.

208 mes identified in the TLR pathways that also explain the operation of other immune signaling pathways

209 We explain the optimizability of DNNs by characterizing the

210 ion at unprecedented high frequency, here we explain the origin of secondary microseism Love waves.

211 The need to regulate these structures may explain the parallel expansion of the number of Nek fami

212 These findings may explain the persistent failure of NK cell therapy in pat

213 ediated Wnt signaling inhibition may help to explain the phenomenon of bone loss and increased adipog

214 sembly, we find that neutral processes alone explain the prevalence of 79% of infant-colonizing micro

215 boration (E&E) document, which serves (1) to explain the rationale behind each item in the guidelines

216 nd Elaboration document, which serves (1) to explain the rationale behind each item in the guidelines

217 theory (DFT) calculations were performed to explain the reactivity differences between indazole and

218 These processes explain the relationship between an ice-free Arctic and

219 Two-population models cannot explain the results.

220 much larger hatchlings in most nautilids may explain the selective extinction of ammonoids as a conse

221 olves differential motility is sufficient to explain the spontaneous patterning of the cells upon con

222 The mechanisms proposed to explain the stabilization involve either mesoscale eddy

223 ke structure of templated CMP pairs helps to explain the success of sticky-ended CMP association and

224 he downstream dynamics and can independently explain the typical decaying envelope and the progressiv

225 These findings may help explain the underrepresentation of women in critical car

226 HD genes and demonstrate that these nonamers explain the vast majority of tandem fusions in human rep

227 t the corresponding RRVs, which consequently explained the outcome of glycosylations for the synthesi

228 an indication of increased river discharge, explained the second-most variation in PicoP abundance (

229 wground productivity, one possible mechanism explaining the "shrubification" of northern peatlands in

230 Potential mechanisms explaining the cumulative glycemic effect are also brief

231 ry genes in innate immune cells, potentially explaining the link between mutations and increased card

232 Thus, explaining the North Atlantic temperature trends and par

233 ese two asthma phenotypes and the mechanisms explaining the paradox of severe asthmatic children, who

234 ention for expectant fathers that focused on explaining the ramifications of smoking on maternal and

235 er, very few review works are reported which explains the application of in situ nasal gel for brain

236 iently reduced following PAR2 activation and explains the changes in PTEN activity.

237 Sinking into oxygen depleted waters explains the exceptional soft tissue preservation.

238 n biotin levels between humans and mice, and explains the failure of potent biotin biosynthesis inhib

239 A reaction pathway that both explains the formation of MeIQx and is valid for the for

240 rylation of the newly introduced Thr residue explains the functional change.

241 metabolism, and inhibited quiescence, which explains the life span shortening.

242 RFT explains the morphology and waveform-dependent performan

243 The model explains the observed low dip angle of the slab and pred

244 organogenesis process across all plants then explains the prevalence of certain patterns in plants, i

245 This new model explains the recent ultrafast dynamics in flavodoxin and

246 erminus of CTCF interacts with cohesin which explains the requirement for convergent CTCF binding sit

247 Predictive modeling explains the sorption data in considering that only weak

248 sto-hydrodynamic origin of the fragmentation explains the strong acceleration of platelet biogenesis

249 enrich for p38-YAP-TEAD interactions, which explains the topography-dependent diversity in fibroblas

250 The model explains the trade-off among chemical design parameters

251 rtially unoccupied perovskite lattice, which explains the unusual regenerative properties of these ma

252 iotic factors (e.g., competition) may better explain their extreme distributions.

253 aptive, with several competing hypotheses to explain their function.

254 ike proteases are actually active proteases, explaining their capacity to undergo processes such as a

255 nce above intrusive magma bodies, which best explains their regionality and the enormity of the appar

256 f cytoskeleton dynamics have been invoked to explain these dynamical features.

257 o describe adhesive properties are unable to explain these experimental observations, instead underes

258 Three key hypotheses have been used to explain these macroecological patterns: the thermal mela

259 We explain these observations with the Texas A&M Oil spill/

260 We explain these observations, which should be readily exte

261 Two primary hypotheses have been proposed to explain these patterns.

262 d substrates of the redirected ligase do not explain these phenotypes.

263 To explain these results, we explore two mechanisms: 1) a g

264 We explain this behavior in terms of how the concentration

265 ther patterns of local charge of H1N1 HA can explain this discrepancy and thus further associate elec

266 What computations explain this hierarchical temporal organization?

267 We explain this phenomenon, which we call the colossal flex

268 olutionary processes and alternative drivers explaining this pattern remain elusive.

269 ving simulator crash risk, though mechanisms explaining this relationship remain unknown.

270 The experimental results are well explained through analytical and atomistic modeling.

271 cognitive and computational mechanisms that explain treatment response remain elusive.

272 ies but not in pulmonary arteries, which may explain TRPV4(EC) -IK/SK channel coupling in mesenteric

273 We apply this framework and explain underlying fundamental concepts through experime

274 vidence of association with uncaring scores (explaining up to 1.59% of variance; self-contained Q = 0

275 ngement in the process XS -> XS(-) cannot be explained using the values of the electron detachment en

276 Soil substrate explained variation, with deeper roots on sandy, nutrien

277 We then test whether life history explains variation in species' niches and niche-distribu

278 urements of the root systems cannot entirely explain variations in citrate-enhanced uptake.

279 he reliance of RDH expression on CDK11 could explain why CDK11 is essential for the growth of many ca

280 This may explain why DCs respond faster and more vigorously to TL

281 We use canard theory to explain why enlarging the size of the window region elic

282 Our results help explain why eukaryotic cells possess multiple resection

283 l ties, and alterations in this map may help explain why lonely individuals endorse statements such a

284 ibition levels at median exposure levels may explain why no relationship between exposure to TTR-bind

285 These data likely explain why patients with PMNs are frequently resistant

286 hibitor of NLRP3 inflammasome activation and explain why patients with XLA are prone to develop Crohn

287 Here we propose that Gestalt theory may explain why rodent islet architecture has historically b

288 tually exclusive competing interactions, may explain why some proteins are dynamic while others are r

289 lved in plant-pathogen response, which could explain why the 20rDNA line is hyper-resistant to both b

290 nzyme less sensitive to feedback inhibition, explaining why assembly occurs under physiological condi

291 d-coil 1B (CC1B) domain of p150(Glued), thus explaining why H2 is necessary for tight binding.

292 -apoptotic proteins to MCL-1 and vice versa, explaining why in vivo combinations of BCL-2 and MCL-1 a

293 ptible strain under EPS inhibitor treatment, explaining why resistance does not evolve.

294 nsulin stimulus to cause insulin resistance, explaining why short-term, insulin-dependent glucose uti

295 ions tended to be weakly oncogenic, probably explaining why they did not give rise to discernible les

296 Such functionality plausibly explains why DNA methylation, a well-known mutagen, has

297 are favored in arcs with thick crust, which explains why magmatism and differentiation in continenta

298 ovel antithrombotic strategies, this article explains why safer anticoagulants are needed, provides t

299 exceed historically observed tolerances help explain widespread bumble bee species decline.

300 for achieving accumulated temperature can be explained with the variations in the daily temperature p