ライフサイエンスコーパス: explanation

1 dimensional data analysis that required much explanation.

2 traction and muscle stretch that defy simple explanation.

3 ss consistent with the data than an adaptive explanation.

4 into account by a complete resource-bounded explanation.

5 y differentiated populations is one proposed explanation.

6 cohorts, makes measurement error an unlikely explanation.

7 ection from this disease, contradicting this explanation.

8 esults question the general validity of this explanation.

9 ve some value, but it is far from a complete explanation.

10 ut the scope and power of the group identity explanation.

11 and exhaustion paradigms offer only partial explanations.

12 depression, they say little about potential explanations.

13 d to contradictory results with the existing explanations.

14 allenged such findings, offering alternative explanations.

15 versity patterns and their macroevolutionary explanations.

16 topes, or differences in procedure as likely explanations.

17 We tested these three concurrent explanations across five experiments.

18 power that is beyond that of the biophysical explanation alone while also uncovering a common mechani

19 ables and examination of several alternative explanations, although the generality of the successful

20 ed by permutation tests and Shapely Additive Explanations analysis.

21 We have also developed the accompanying Explanation and Elaboration (E&E) document, which serves

22 This explanation and elaboration document describes the inten

23 This explanation and elaboration document was developed as pa

24 The PATH Statement, together with its explanation and elaboration document, may guide future a

25 We have also developed the accompanying Explanation and Elaboration document, which serves (1) t

26 onsists of 54 items and is accompanied by an explanation and elaboration document.

27 em in epilepsy that lacks pathophysiological explanation and treatment.

28 of non-equilibrium physical chemistry, whose explanation and understanding still capture the imaginat

29 Mechanistic explanations are provided for many common challenges ass

30 A processing and 60S generation and offer an explanation as to why a subtle dysfunction of BCCIP can

31 during physical therapy provides a potential explanation as to why physical therapy may often be inef

32 These findings provide a mechanistic explanation as to why the deletion of CFHR3 and CFHR1 is

33 y values, or dimensions along which we judge explanations as better or worse.

34 tography design have provided an alternative explanation behind some methods failing when in the QC l

35 hich lineage-specific genes call for special explanations beyond homology detection failure, highligh

36 e bed (< 10 mm) scale that has so far defied explanation by a single formative process.

37 Explanations capitalizing on either the delocalization o

38 One explanation could be that we have not examined gene expr

39 virology for which no consensus evolutionary explanation exists.

40 Here, we provide a mechanistic explanation for aberrant hell ant morphology through phy

41 nd epigenetic spaces has been proposed as an explanation for aging.

42 ines of evidence making this the most likely explanation for changing incidence among n = 513 wolves'

43 Their results provide one explanation for differential insulinotropic potential of

44 Taken together, we unravel a technical explanation for discrepancies in the published literatur

45 ger synaptic terminals, providing a putative explanation for functional divergence.

46 fic protein aggregation, providing molecular explanation for general cellular toxicity.

47 st that this model may possibly be a general explanation for global diversity patterns, unconstrained

48 hanism of HERV-K expression and a structural explanation for HIV-1 Rev-mediated expression of HERV-K

49 st body temperature, and provide a potential explanation for host shifts being less likely between ge

50 h a multimodal assembly platform provides an explanation for how cells build a powerful yet transient

51 a-2, and provide a comprehensive mechanistic explanation for how functional synaptic phenotypes contr

52 tibody was employed to provide a mechanistic explanation for how fundamental alloantibody biology inf

53 These data provide an explanation for how low levels of Beclin 1 facilitate tu

54 These findings provide one explanation for how obesity worsens cancer outcomes and

55 hoto-alteration of DOM provide a mechanistic explanation for how sunlight exposure of terrigenous DOM

56 This study provides a molecular explanation for how Twist1-induced dissemination works a

57 Our results provide a long-awaited explanation for how Ubx controls morphogenetic transform

58 Our results provide a theoretical explanation for how virus antigenic evolution can be hig

59 e-8-dependent pathway provides a mechanistic explanation for IL-1beta production and its consequences

60 podocytes and evaluated INF2 cleavage as an explanation for immunoblot findings.

61 ntegration during inflammation; and offer an explanation for immunodeficiency caused by mutations of

62 ther, StanDep also provided a transcriptomic explanation for inclusion of lowly expressed reactions t

63 As a potential explanation for increased hepatic IR injury, fecal short

64 plant immune signaling, providing a probable explanation for iron-immunity cross-talk.

65 0-dihydroxystearate, which presents a likely explanation for its accumulation to ~0.7% in castor oil,

66 a form of camouflage, providing an adaptive explanation for its taxonomically widespread occurrence.

67 vantageous mutants and provide a mechanistic explanation for Lon's widespread impact.

68 Here, we propose an explanation for lower than expected skew in low CO regio

69 nanoparticles provides for the first time an explanation for Marks decahedron truncation.

70 control GAS disease potential and provide an explanation for observed strain- and serotype-specific v

71 lusions from observational data, a plausible explanation for our findings is that, during lockdown, i

72 ns of lipid bilayer elasticity and offers an explanation for previous reports of prolonged hormone re

73 ken together, our study provides a molecular explanation for root epidermal cell fate switch in respo

74 ubstrate specificity of hGGT1 and provide an explanation for seemingly disparate observations regardi

75 hanism for distal organ damage as well as an explanation for sepsis-associated acute respiratory dist

76 erence between DNMT3A and DNMT3B provides an explanation for site-specific epigenomic alterations see

77 yield N(2)O.This mechanism could provide an explanation for sulfate formation under some winter haze

78 ts own gene expression, providing a possible explanation for survival differences based on HIF1A CNV

79 trachain symmetry that provides a structural explanation for the ability of the MS-ring to function a

80 ed in a GCC acetic acid system, providing an explanation for the absence of a cyclic voltammetry peak

81 These findings provide a mechanistic explanation for the acute O(2) regulation of breathing,

82 member of this population offers a possible explanation for the anomalous properties of GW170817, in

83 ship than previously suggested, providing an explanation for the apparent absence of universal prosoc

84 A satisfactory explanation for the ASRR has been elusive for more than

85 necessarily detrimental in LT and provide an explanation for the association between HCV eradication

86 This study provides a rational explanation for the autoimmunity of patients with missen

87 These results provide a neural explanation for the behavioural dissociation of acoustic

88 plasticity-related recovery) and provides an explanation for the bilateral yet asymmetric lateralizat

89 host antimicrobial defense, offer a possible explanation for the clinical observation that coagulase-

90 against infection and provide a mechanistic explanation for the clinically observed resistance of in

91 ved phenomenology and provides a theoretical explanation for the coexistence and interaction of patte

92 lines with H3.3K27M mutations, providing an explanation for the common co-occurrence of alterations

93 nsumer distribution, providing a mechanistic explanation for the context-dependent outcomes often rep

94 bosome loss in aneuploid cells, providing an explanation for the decreased cellular density of aneupl

95 One potential explanation for the differential cellular response to th

96 Our model provides a mechanistic explanation for the differential effects of habitat on s

97 ct with the lipid or detergent, providing an explanation for the distinct activation kinetics that ma

98 data instead support a largely host-neutral explanation for the distribution of human-infecting viru

99 cal weakness of the stem was ruled out as an explanation for the downward bending.

100 aldwin effect offers a plausible and elegant explanation for the emergence of complex behavioural tra

101 However, so far no mechanistic explanation for the emergence of typically observed lami

102 ichomes of N. tabacum, providing a plausible explanation for the enhanced biosynthesis of secondary m

103 sicle release sites, providing a morphologic explanation for the enhanced glutamate release.

104 e light-adapted state, providing a plausible explanation for the experimental observation of faster p

105 ipheral clock, which may provide a molecular explanation for the extensive associations between seaso

106 rd-generation ABCB1 inhibition, providing an explanation for the failure of ABCB1 inhibitors in clini

107 Our findings present an alternative two-step explanation for the formation of actin bundles due to me

108 ssical plasma membrane suggesting a physical explanation for the formation of nuclear membrane invagi

109 ess values, hence supporting a biomechanical explanation for the fusion as a functional adaptation fo

110 substrate specificity providing a potential explanation for the genetic repertoire of epoxide hydrol

111 in the same processes, offering one possible explanation for the high comorbidity rate of the two dis

112 These findings may provide an explanation for the higher missed diagnosis rate in youn

113 n bias provides a more plausible, sufficient explanation for the improved safety.

114 omplex with LDLa repeats on LRP1 provided an explanation for the increased affinity of uPA:PAI-1 comp

115 Thus, our results offer an explanation for the increased invasiveness of serotype 1

116 Our study provides a mechanistic explanation for the increased sensitivity of TNBC to Hsp

117 on factor via SLC15A4 provides a mechanistic explanation for the involvement of these proteins in sys

118 t auxin transport and a logical parsimonious explanation for the known physiological effects of NPA o

119 in COVID-19 disease, providing a mechanistic explanation for the limited durability of antibody respo

120 These results provide a possible explanation for the limited effectiveness of birch polle

121 discoveries provide a plausible mechanistic explanation for the long-standing appreciation that RAAS

122 idation, while volcanism remains an adequate explanation for the longer-term geochemical changes.

123 The most likely explanation for the markedly divergent VE estimate from

124 lex with LXE408, which provides a compelling explanation for the noncompetitive mode of binding of th

125 e heterochiral complex provides a definitive explanation for the nonlinear behaviour of this system.

126 This provides a quantitative mechanistic explanation for the observed benefits of SGLT2i in HF-rE

127 gen molecule activation), thus providing one explanation for the observed photothermal activity.

128 in in the active site, offering a structural explanation for the observed reactivity.

129 These findings provide a possible explanation for the observed sex biases in COVID-19, and

130 energy gain by sulfate limitation offers an explanation for the occurrence of naphthalene-derived me

131 However, we miss an explanation for the onset of the NDs.

132 The model provides a mechanistic explanation for the origins of multicellular hyphal orga

133 Shared genetic etiology is a plausible explanation for the overlap, and in this study we tested

134 Our data thus provide an explanation for the paradox surrounding IFN-I and TH17 i

135 Collectively, our findings provide an explanation for the paradoxical clinical-outcomes after

136 vide an alternative, although complementary, explanation for the phenotype observed.

137 As an explanation for the poor cell uptake of Ru360, we show t

138 A classic explanation for the prevalence of such warfare in some h

139 These findings provide both an explanation for the protective effect of Dantu, and fres

140 Our study provides an explanation for the puzzling dissimilarity of ARF6 to ot

141 ic social dynamics, and offer an alternative explanation for the rise of "fake news" as a societal ph

142 S) constriction, thus providing a structural explanation for the role of the His-Gly dipeptide in the

143 synthesizing nutrients may provide a unified explanation for the sequence of gene losses that occur d

144 We also provide a possible explanation for the sex-specific differences in survival

145 ciple kinetic derivation may thus provide an explanation for the slower-than-adiabatic temperature de

146 In place of Tomasello's explanation for the source of moral obligation, we sugge

147 This mechanism provides an explanation for the specific role of CaNAbeta in hypertr

148 cross a wide age range, and hence provide an explanation for the survival of the APOE epsilon4 allele

149 We provide an explanation for the variation in vaccine effectiveness b

150 herms, we suggest that this may be a general explanation for the weak synchrony observed at large dis

151 tin fibrillogenesis, providing a mechanistic explanation for the well-known widespread accumulation o

152 We develop an explanation for their evolution drawing from cultural ev

153 differences among HSCs, providing a possible explanation for their functional heterogeneity(12-17).

154 ndividuals with dyspepsia have no structural explanation for their symptoms and have functional dyspe

155 ny people who have them will have no organic explanation for their symptoms.

156 Here we propose an explanation for these differences based on new theory ab

157 The long-standing mechanistic explanation for this adaptive behaviour is that neural a

158 e and human NaCTs provide at least a partial explanation for this conundrum, but a structural basis f

159 One possible explanation for this difference is that children cannot

160 A possible explanation for this dissociation in DP could be that th

161 However, no clear mechanistic explanation for this entropy increase has been put forwa

162 Although a detailed mechanistic explanation for this increase is lacking, recent evidenc

163 scription Start Sites, providing a molecular explanation for this interplay.

164 ation Database (gnomAD)(1), we show that one explanation for this paradox involves alternative splici

165 cell wall loosening, providing a mechanistic explanation for this phenotype and a previously undescri

166 One explanation for this variation is that individuals face

167 re is currently no functional or mechanistic explanation for this.

168 An explanation for two-thirds of the worldwide ambient wave

169 ses SLC30A8 function, which is the canonical explanation for type 2 diabetes risk association.

170 respective EC tissues, providing a possible explanation for TZ tissue being the primary target for H

171 Although this model provides an explanation for variation in molar proportions, what rem

172 ansporter activity, and provide a structural explanation for varied substrate specificity and ion tra

173 immune escape variants, provides a molecular explanation for why immunotherapy for HBV infection may

174 iently in the presence of Lis1, providing an explanation for why Lis1 is a required cofactor for most

175 nhibited state, which provides a mechanistic explanation for why Lis1 is required for efficient trans

176 sical principle, this provides a theoretical explanation for why noninteger dimensions are useful in

177 by food odors, providing a possible unifying explanation for why sea turtles interact with marine pla

178 Our study advances an adaptive explanation for why the placenta evolves by arguing that

179 Our results suggest a mechanistic explanation for why the selective fitness advantage intr

180 ay redundant roles in synapsis, providing an explanation for why these genes have evaded previous gen

181 Whilst explanations for anomalously fast diffusion have been pr

182 rtantly, the model provides viable molecular explanations for decoupling: mechanosensing mechanisms a

183 and validate it using computer simulations; explanations for higher skew with higher CO rates, as in

184 he Antarctic green algae and offer potential explanations for how green plants adapt to extreme envir

185 Evolutionary explanations for mammalian sociality typically center on

186 One of the explanations for mechanosensitivity, a lipid-bilayer mod

187 etabolite multifunctionality can provide new explanations for ontogenetic patterns of defense product

188 naptic plasticity, which provide mechanistic explanations for opposing experimental findings.

189 We provided possible explanations for our results and suggested further resea

190 Furthermore, we present possible explanations for pregnancy as a risk factor for an invas

191 mechanisms (e.g., social support) hold up as explanations for religion-health associations now that m

192 f subducted oceanic crust could provide good explanations for some lower-mantle seismic heterogeneiti

193 ting strong priors by generating alternative explanations for surprising information involves more me

194 lity in numerical skill and discuss possible explanations for the absence of an effect on spatial ski

195 n alfa); (3) drugs with alternative biologic explanations for the association (e.g., sevelamer); and

196 functions of the Hippo pathway and potential explanations for the cancer-restricted mutation profile

197 Here, we propose several potential explanations for the disparate nature of our findings fr

198 this new sarcomeric protein, some alternate explanations for the effects of KBTBD13R408C, and the ad

199 Our results constrain theoretical explanations for the emergence of superconductivity in T

200 ems in organic solvents-furnish quantitative explanations for the empirically unpredictable aggregati

201 ersity has been dominated by studies seeking explanations for the equator-to-poles decline in richnes

202 lished, leading to controversies surrounding explanations for the evolution of altruism.

203 Identifying developmental explanations for the evolution of complex structures lik

204 Here, we test mechanistic and ecological explanations for the evolution of developmental duration

205 Finally, we explored possible explanations for the experimental discrepancy.

206 h influenced by beating and provide possible explanations for the experimental observation that a bea

207 ic and bivalent regions and provide possible explanations for the genomic instability, as well as the

208 ontrol trials allow us to reject alternative explanations for the link between observed aggression an

209 d socioeconomic status have been traditional explanations for the observed racial disparities in HF o

210 ted disease phenotypes and discuss potential explanations for the observed tissue specificity.

211 Our results provide explanations for the puzzling fact that growth of B. sub

212 We advocate for a re-evaluation of adaptive explanations for the rapid diversification of reproducti

213 ted mutation positions, leading to plausible explanations for the relationship between nuclear export

214 Explanations for the stall have focused on rising drug-r

215 ve been precluded by the lack of mechanistic explanations for the variety of its desirable/undesirabl

216 an uses rationalization to refer to people's explanations for their own actions.

217 membrane fusion process to provide plausible explanations for these observations.

218 While explanations for this gender pay gap vary, they have ten

219 sis regulation and then we consider possible explanations for why MCU-deficient mice are spared from

220 Different explanations have been proposed ranging from differentia

221 To date both explanations have relied on poorly constrained delta(13)

222 As an alternative explanation, here we report 2-methylhopanoid production

223 ts, only achieved a satisfactory biochemical explanation in the last 20 years of the 20th century.

224 Possible explanations include mild wave or atmospheric climates a

225 A number of explanations-including three-site exchange magnetization

226 e inertia in our experiments, an alternative explanation is needed for this OCTP profile.

227 A theoretical explanation is provided, as well as numerical simulation

228 A parsimonious explanation is that coordinated CD4(+) T cell, CD8(+) T

229 One possible explanation is that habitat selection is context-depende

230 An alternative explanation is that multimers could become entrenched if

231 The most plausible explanation is that p represents an index of impairment

232 One possible explanation is the mutagenic activity of these foods, pe

233 h this gradient is that it conflates two key explanations, namely biome stability (age and area) and

234 Arguing against a motor explanation, no other cerebellar area exhibited stimulus

235 lar dynamics simulations provided structural explanation of accessibility of the RNA cleavage sites t

236 We show that a quantitative and coherent explanation of all observables is possible within the fr

237 A commonly hypothesized explanation of asynchronous firing is that excitatory an

238 e-scale delivery to surfaces (removal) as an explanation of contrasting pathogen prevalence in granul

239 discussing the role of neutral models in the explanation of diversity.

240 Our results give a biophysically plausible explanation of how a class of prefrontal cortical pyrami

241 wth, do not fit into a single, comprehensive explanation of how auxin controls cell expansion, and wh

242 Self-organized criticality is an elegant explanation of how complex structures emerge and persist

243 The self-categorization explanation of influence grounded in group norms, modera

244 glycosylation with weak nucleophiles for the explanation of O-glycosylation.

245 Recognition and explanation of such differences in multimorbidities info

246 hypothesis, have come a long way towards an explanation of the basic features of retinal organizatio

247 This gives a unified microscopic explanation of the dispersion of confined states in the

248 aining, scalable inference and interpretable explanation of the DNA repair outcome following a Cas9 c

249 ileptogenic networks, provides a mechanistic explanation of the epileptic processes during the interi

250 An explanation of the KLR results is that in the prediction

251 n of the visible light spectrum, no adequate explanation of the mechanism has been given.

252 hand, the 'criticality' hypothesis posits an explanation of the more coordinated state that also requ

253 /closing of the salivarium, with a plausible explanation of the movement of saliva within the salivar

254 We obtain a comprehensive explanation of the observed experimental signatures from

255 ttempts at functional analysis and prevented explanation of the origin of cyclic dinucleotide signall

256 These findings provide further explanation of the refractory nature of respiratory illn

257 (2) Together, these data provide a molecular explanation of the role of ROS in pathogenesis of plaque

258 A comprehensive explanation of these effects on the transport of magneti

259 rate, and criticality do not provide a full explanation of this dichotomy.

260 ead to ventricular arrhythmias as a possible explanation of this increased mortality.

261 One possible explanation of this lack of effect is that well-practice

262 Here we offer an explanation of this puzzling observation based on the st

263 xplored providers' confidence in counseling, explanation of VUSs, topics covered before and after gen

264 question about the target article's proposed explanation of what goes on when we think through other

265 has been linked to voluntary action, but an explanation of why decisions to act emerge at particular

266 Our studies provide a structural explanation of why TAT-SF1 must be displaced before the

267 rating testable hypotheses about mechanistic explanations of brain function, behavior, and mind based

268 reproductive success, and supports adaptive explanations of divorce as a strategy to improve individ

269 s will improve by developing mechanism-based explanations of elevated levels that can be expanded and

270 Explanations of how the brain makes successful predictio

271 s compared and contrasted to other normative explanations of influence, notably the concept of injunc

272 bridge the cognitive-sociocultural divide in explanations of the discovery, production, and cultural

273 Thus, our model provides explanations of the pathophysiology of hemoglobinopathie

274 Providing explanations of the procedure developed including the fo

275 ata were obtained or adapted thereafter; and explanations of whether any outliers (data points or ent

276 ing, and exploratory experiments or studies; explanations of whether key elements of study design, su

277 The prevailing explanation points to the interplay between predisposing

278 Each dataset is accompanied by an explanation regarding the nature of the collected data.

279 l interpretation framework, SHapley Additive exPlanations (SHAP), which locally assigns feature impor

280 results from NN, we employ SHapley Additive exPlanations (SHAP), which reveals that the influence of

281 tlas, we show that cluster analysis on model explanations substantially outperforms the classical app

282 One controversial explanation suggests seamount-induced chlorophyll enhanc

283 Our data suggest a clear explanation that is based on the classic biochemical pri

284 ay help promote cooperation, the alternative explanation that they facilitate coordination has gained

285 They dismiss theoretical explanations that include scene construction [2,3], and

286 erimental evidence, supported by theoretical explanation, that the B-site cation stoichiometry is cru

287 nd provide insight into why people adopt the explanations they do.

288 To tease apart possible explanations through a comparative approach, we assessed

289 the detection of GORD, providing a possible explanation to discrepancies in AET and impedance interp

290 ical studies fall short in giving a thorough explanation to particle equilibrium states.

291 ic Migration (EVM) theory to give a unifying explanation to particle migration in Newtonian and visco

292 his new magnetic field provides a nontrivial explanation to the puzzling observation of a difference

293 This article provides an explanation to this phenomenon, as well as a set of ther

294 Here, we propose a mechanistic explanation to understand these differences.

295 cover subtypes via cluster analysis on model explanations to classical cluster analysis on the origin

296 But Bratman's makes the explanation too difficult to succeed, and Gilbert's make

297 Moreover, we provide a structural explanation underlying the distinct CD8(+) T cell reacti

298 imental observations that lack a mechanistic explanation: variability in the firing rate of single gr

299 They also provide a possible explanation why such connectivity and activity patterns

300 Our data provide a mechanistic explanation why the same proteins can act either as tole