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1 -4-CLP36 complex in podocytes and provide an explanation as to how alpha-actinin-4 deficiency or muta
2 he HIV capsid in nuclear entry and offers an explanation as to how an exogenous entity much larger th
3 ace of the T3 channel, providing a plausible explanation as to how BCL9 co-factors binding to Pygo PH
4                    This study also offers an explanation as to how CK2alpha exclusion mutations (S120
5 ivity and might begin to provide a molecular explanation as to how E2 modulates dendritic spine plast
6 l transcriptomic vulnerability, providing an explanation as to how heterogenous pathological entities
7                  Our results provide a novel explanation as to how ICH in newborns results in seizure
8 rogen bonding in the active site suggests an explanation as to how in the wild-type and H98N mutated
9         These findings offer a network-level explanation as to how inhibition regulates the experienc
10  by membrane lipid composition, providing an explanation as to how its membrane-stressing activity is
11 s of intracellular Na+ and hence provides an explanation as to how KNa channel can control normal neu
12             This mechanism provides a likely explanation as to how Mn combats superoxide stress in ce
13         These findings provide a mechanistic explanation as to how Rb regulates cell division and apo
14          Furthermore, this study provides an explanation as to how Salmonella evades activation of au
15 experimental evidence for its presence or an explanation as to how such a polar molecule could exit t
16 methylation in cis and provide a mechanistic explanation as to how these loci impact upon OA suscepti
17 ing pathway, and provide a first mechanistic explanation as to how upregulation of CD44 may constitut
18           These findings provide a metabolic explanation as to how VPA-mediated inositol depletion ca
19 stal structures and, in many cases, detailed explanations as to how and why organisms synthesis and m
20 for clinical application, allowing for clear explanations as to how the predictions are produced, how
21 tion domains, our results provide a possible explanation as to the reason that RUNX1 is recurrently f
22                        There are no definite explanations as to the variable sensitivity to progeria
23 A processing and 60S generation and offer an explanation as to why a subtle dysfunction of BCCIP can
24       This finding also offers a mechanistic explanation as to why a tumor suppressor gene, such as R
25 dy to cause pathogenicity and may provide an explanation as to why apparently similar auto-antibodies
26  p53 to maintain hepatic CSCs and provide an explanation as to why autophagy is required to promote h
27 ipose tissue and also represents a plausible explanation as to why brown adipocytes ultimately specia
28      These observations suggest a structural explanation as to why certain serine lipases are resista
29                                We provide an explanation as to why DD can characterize structural het
30              These findings offer a possible explanation as to why DNA repair was implicated in SCA1
31                      Results herein offer an explanation as to why EGFR inhibitors failed TNBC patien
32                 Our results offer a possible explanation as to why Env immunogens have been ineffecti
33 ng the optic nerves, could be an alternative explanation as to why glaucoma develops in patients with
34                       Our results provide an explanation as to why GPER is not readily detected on th
35 W loci in (NZB x NZW)F1 mice and provides an explanation as to why H2(d/z) heterozygosity is required
36                        These data provide an explanation as to why high trkA levels are associated wi
37               This could serve as a possible explanation as to why HS-deficient cells showed signific
38                These results also provide an explanation as to why IFN-gamma may play both pathogenic
39 f Slc30a8 deletion, though the physiological explanation as to why impaired insulin secretion is not
40 ith that of a group, and is the current best explanation as to why individuals become empowered to ac
41          Our data also provide an additional explanation as to why malignant melanocytes lose cKit ex
42 l expansion of this population, providing an explanation as to why Mir155 deletion impairs affinity m
43 ery common, but to date there is no complete explanation as to why one out of two people systematical
44             This scenario leads to a natural explanation as to why only oscillations from a single el
45 une evasion of PR-positive tumors may be one explanation as to why over 65% of breast cancers are PR
46 urthermore, our findings provide a plausible explanation as to why P[6] RV strains are more common in
47              Our results provide a normative explanation as to why PFC implements an adaptive, minima
48 during physical therapy provides a potential explanation as to why physical therapy may often be inef
49         These advantages provide a plausible explanation as to why plants have evolved so many anthoc
50              The present findings provide an explanation as to why previous whole-cell Ca2+ currents
51          These results provide a mechanistic explanation as to why rpaA-null mutants die in the dark,
52                     This finding provides an explanation as to why so many large proteins, without a
53            As of yet, there is no systematic explanation as to why some static images are likely to p
54 tractility, and ECM stiffness and provide an explanation as to why stiffer environments result in enh
55         Altogether, these results provide an explanation as to why Th17 cells are highly susceptible
56  of the R(61,2)C adduct provides a potential explanation as to why that adduct does not induce A -->
57           Our results provide a quantitative explanation as to why the costs of reproduction are not
58         These findings provide a mechanistic explanation as to why the deletion of CFHR3 and CFHR1 is
59 vivo, and they provide a potential molecular explanation as to why the developmental impact of these
60                     This provides a possible explanation as to why the Hb dynamics are robust under p
61 perimental regioselectivities and provide an explanation as to why the hydrogen is transferred from t
62                 These results may provide an explanation as to why the multiantennary structure is ub
63 eported high-resolution structure provide an explanation as to why the mutation reduces ATP sensitivi
64 y Hsp70/40 chaperones, providing a molecular explanation as to why the N domain is dispensable for pr
65                   This could offer a partial explanation as to why the T-cell response is impaired in
66                          It also provides an explanation as to why the unfolded protein response may
67       This analysis also provides a possible explanation as to why these marker genes can be promiscu
68 ural communication signals, but also a novel explanation as to why these preferentially occur on top
69 heir bacterial homologues, thus providing an explanation as to why this novel chloroplast SRP pathway
70 mine transporter, and provides an intriguing explanation as to why use of these compounds, unlike coc
71                                We provide an explanation as to why ZMapp cocktail has higher efficacy
72 of myelin to CNS plasticity provide possible explanations as to why almost all patients with hypomyel
73                                      Several explanations as to why converging processes may drive dr
74    These experimental results offer possible explanations as to why Juliette is perceived to have a s
75 g merlin, Falco columbarius, and alternative explanations as to why observed behaviours should differ
76                          We discuss possible explanations as to why sequence-profile and contact-pote
77 d the acquisition/loss of mutations, to give explanations as to why there is a discrepancy in pharmac
78 e in migraine prevalence over time, offering explanations as to why this may be illusory.