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1 1.01-1.25] per 10-mug/m(3) increase in NO(2) exposure).
2 f infection with NiV Bangladesh 3 days after exposure.
3 and of shortest latency with 10 mg/kg/d GenX exposure.
4  largely unaltered in children with in utero exposure.
5 an endogenous IgG response within 20 days of exposure.
6 e reached high oral bioavailability and oral exposure.
7 trometry, SEM, optical microscopy, and light exposure.
8 the need for additional imaging or radiation exposure.
9 children and young adults with long-term PGA exposure.
10 ces in the natural history of IM after e-HRT exposure.
11 g altered membrane integrity after the field exposure.
12 icity responses representative of real-world exposure.
13  which were significantly reduced after heme exposure.
14 glaucoma patients (n = 26) without senolytic exposure.
15 FGFR) 2 in urothelium after cyclophosphamide exposure.
16 dem, could strongly compound population heat exposure.
17 wash and lavage were obtained 24 hours after exposure.
18 ns rapidly respond to intravenous antibiotic exposure.
19  2 weeks, and then weekly until week 8 after exposure.
20 ad severe PAP after long-term unilateral PGA exposure.
21 tions of the 2 components of the risk-factor exposure.
22 preserve benefit while minimizing antibiotic exposure.
23 ly understood, particularly during long-term exposure.
24      A similar effect was observed upon NaCl exposure.
25 exposure was significantly elevated after PM-exposure.
26 compared to those that survived insecticidal exposure.
27       We used blood fructose as a validation exposure.
28 ementing the aftereffect following prolonged exposure.
29 breeding numbers at environmentally relevant exposures.
30 ut that more work is required to assess InHg exposures.
31 times with intervals of 3-4 days between UVR exposures.
32  factors, with lagged and unlagged pesticide exposures.
33  and intense heatwaves projected to increase exposures.
34 o, it may not detect relevant, lesser known, exposures.
35 e properties and compared them to mass-based exposures.
36                                    At longer exposures (25 min) the s-MNPs/SOD1 dispersion destabiliz
37                 The surge in population heat exposure across the Sunbelt is driven by concurrent GHG-
38 amine how trait impulsivity and acute stress exposure affect participants' choice behavior and decisi
39 les, chromatin structures, and environmental exposures, all of which impact the resulting distributio
40 ces between types and levels of occupational exposures among women and men and can affect prevalence
41 o examine associations between antenatal IPT exposure and adverse pregnancy outcomes, maternal TB, al
42 aluate relationships between classroom NO(2) exposure and asthma symptoms and morbidity by body mass
43 s no dose-dependent relationship between PPS exposure and diagnosis of any maculopathy.
44 ent [CCT] scenarios), EPA's Stochastic Human Exposure and Dose Simulation (SHEDS)-Multimedia/Integrat
45 ubentonii were sampled for evidence of virus exposure and excretion during a prospective nine year se
46 M), while the association between high PM2.5 exposure and GDM risk has not been well studied.
47  therapy for cancer, but is limited by short exposure and high toxicities.
48 oonotic pathogens use information about host exposure and infection rather than competence, defined a
49 s that have adapted to survive environmental exposure and perform the tasks necessary for respiration
50 t of UV-induced DNA damage as a dosimeter of exposure and predictor of skin cancer risk has been prop
51 istic insights into how both chronic alcohol exposure and protracted withdrawal dysregulate 5-HT sign
52 te connections, have elevated presynaptic PS exposure and reduced PS engulfment by microglia.
53 is known of the mechanisms underlying opioid exposure and sleep.
54  of E. dermatitidis to acute gamma-radiation exposure and the major mechanisms it uses to recover fro
55 f the nuc-aAST after 15-30 min of antibiotic exposure and the results of the gold-standard culture-ba
56 worse among those who are more vulnerable to exposure and this can be enhanced by communities' vulner
57 s mediate associations between prenatal PFAS exposures and child health outcomes.
58 e relationships between rain-related climate exposures and diarrheal disease.
59 ere no clear associations between any of the exposures and either of the outcomes.
60 nmental fluctuation due to longer periods of exposure, and animals adapted to live in these environme
61 c Heart Failure score, genotype, level of BB exposure, and BB propensity score.
62  benefits during 1 and 6 months of continued exposure, and further displayed growth benefits to at le
63 ion, but whether they alter TFV-DP or 3TC-TP exposure, and therefore compromise prevention efficacy,
64 ).Conclusions: Rare and common environmental exposures are independent risk factors for radiologic ab
65 lial colonization; and cigarette smoke toxin exposure; as well as to elucidate genes required for iro
66 eport, we present a comprehensive inhalation exposure assessment of indoor microenvironments at six c
67                                              Exposure-based treatments share the aim of facilitating
68 ensory trial, both trial-wise and cumulative exposure bias the encoding of the acoustic information,
69  T cell immunological memory of past antigen exposure can confer long-lived protection against infect
70 Our results demonstrate that decreases in Pb exposure can result in large benefits for the adult popu
71                                        These exposures can not only cause genetic and epigenetic alte
72  eliminated within 24 h after the waterborne exposure ceased, reflecting the desorption of weakly bou
73 lergen challenge (NAC) and the environmental exposure chamber (EEC) are 2 methods of conducting contr
74 th HO-1 and P450s are affected by xenobiotic exposure, changes in HO-1 expression can potentially aff
75  8-, and 13-different classes of steroids in exposure concentrations of 10-10,000 ng/L.
76 tative method will enable the procurement of exposure concentrations of airborne microplastics guidin
77 le tissue damage under dysregulated or acute exposure conditions.
78  model that will facilitate the inclusion of exposure data in computational analysis of human disease
79         PL levels measured before and during exposure demonstrate distinct responses to the four test
80 p = 0.39 in individuals not reporting trauma exposure, difference p = 2.3 x 10(-4)).
81                                       Median exposure (dose area product/kg) was decreased by 30% for
82    LTBI prevalence increased with increasing exposure duration, with an incremental prevalence increa
83  was to quantify the effect of maternal heat exposure during early-mid gestation, when pig placentae
84 ay lessen the risk of PTD associated with PM exposure during pregnancy among primiparas of Han ethnic
85                                        PM2.5 exposure during pregnancy for each participant was estim
86                                      Smoking exposure during the prenatal period was directly associa
87  active ingredients and how quickly systemic exposure exceeds 0.5 ng/mL as recommended by the US Food
88                   Subsequent shipboard light exposure experiments showed ultrastructural changes in t
89                       Health care access and exposure factors may underlie the observed disparities m
90                                Fetal alcohol exposure (FAE) is the leading preventable developmental
91 ere obtained at baseline, every 3 days after exposure for 2 weeks, and then weekly until week 8 after
92 ome of veterinary students with occupational exposure for 3 months.
93              We estimated occupational noise exposure for each case and control using a job-exposure
94 ned at recruitment, as a biomarker for metal exposure from any source.
95 rials, but the quantity of early-life gluten exposure has been a major focus of prevention efforts.
96                                Environmental exposures, health history, and clinical outcomes were pr
97 pidome, reflects gene regulation and dietary exposures, heralding the development of islet autoantibo
98 potential can be greatly altered by chemical exposure history and NP surface transformations.
99 remental prevalence increase of 8.2% per 250 exposure hours (P < .0001).
100 he proinflammatory immune response to trauma exposure immediately after trauma exposure, in the emerg
101 drug to discern whether blood-stage parasite exposure impacts protection against homologous controlle
102  the vanishing potential for incidental news exposure in digital media environments.
103 at DBS is a suitable tool for assessing MeHg exposure in human population studies, but that more work
104  and diet, and fewer studies have focused on exposure in the indoor environment.
105 y assessed indoor and personal air pollution exposures in this area; however, the influence of indoor
106  to trauma exposure immediately after trauma exposure, in the emergency department, may help identify
107 k has established that risk factors for lead exposure include race/ethnicity, poverty, Medicaid enrol
108                                 Brief female exposure induces a persistent increase in male aggressiv
109  cation channel; while repeated or prolonged exposure induces formation of a transmembrane pore.
110                         The duration of VEGF exposure influenced both barrier relaxation and anti-VEG
111                       When the environmental exposure is a binary variable, analyses from exposure-st
112 in PCB levels in food indicates that dietary exposure is comparable to PCB inhalation exposures repor
113 ndings imply that the behavior concerning UV exposure is complicated by a genetic predisposition shar
114 ophrenia in vivo and that antipsychotic drug exposure is unlikely to account for them.
115 sensitizer in regions with high olive pollen exposure, leading to the peach nsLTP sensitization.
116 ations of atezolizumab were above the target exposure level in all patients.
117 ly low estimated inhibition levels at median exposure levels may explain why no relationship between
118 reductions of >50% in breeding numbers under exposure levels otherwise associated with <20% reduction
119 posure for each case and control using a job-exposure matrix.
120          Reduced transpiration upon seawater exposure may contribute to controlling the movement of t
121  an understanding of patterns in and between exposure metrics that are based on specific hazards of t
122                                              Exposure mixtures frequently occur in data across many d
123                                   Upon laser exposure, MOF crystals shrank while metal oxide nanopart
124  and hepatitis B virus coinfection, group of exposure, nadir CD4 count, CD4:CD8 ratio, and last CD4 l
125 latedness and traits known to promote vector exposure neither predicted infection probability nor par
126 vidence of racial/ethnic disparities in PBDE exposures (Non-Hispanic Black > Latina/Hispanic > Non-Hi
127                                     A 10-min exposure of A549 human lung cancer cells to sequential 5
128 arallel studies, and to assess the potential exposure of ABX to the brain, we quantified the levels o
129 ed microalgal species were cultivated during exposure of artificial UVB.
130  simulations, that represent the wide-spread exposure of bare soil in the 1930s, suggest human activi
131 d compounds that do not reflect the complete exposure of chemicals to biota and do not provide the co
132                        Pulsed magnetic field exposure of human breast cancer cells that express a sia
133                                         Upon exposure of human cells to DNA-damaging agents, NUCKS1 c
134      Results of this study show that an 18 h exposure of human pulmonary artery endothelial cells to
135                                          The exposure of interest was PPV23 vaccination at any time p
136                                 We show that exposure of leukemia cells to daunorubicin activated an
137                Consistent with this, ex vivo exposure of LSVs to acute arterial WSS promoted monocyte
138  the inactivation of MNV was governed by the exposure of microorganisms to PFA, i.e., the integral of
139 gmn evokes pain-like behavior through PAR(2) Exposure of pain-sensing neurons to Lgmn decreased the c
140 o promote tumor retention and limit systemic exposure of potent intratumoral therapies.
141                                     Lifelong exposure of rats to low-dose BPA at 25 and 250 mug/kg BW
142                                              Exposure of the enzyme decorated biochip to glyphosate c
143                            Here we show that exposure of the skin to the experimental contact allerge
144  of millions of years, is entrenched because exposure of this interface to solvent reduces protein st
145                                 Experimental exposure of White Ibises (Eudocimus albus) to MeHg also
146 lutionary consequences of decreased parasite exposure on an immune system are not well understood.
147  to assess the impact of neonatal anesthesia exposure on behavioral learning in adolescent subjects,
148 y, we examined the effects of early ketamine exposure on brain structure and function.
149  mediating the effects of parental Holocaust exposure on C-reactive protein.
150 h has targeted the effect of oil and hypoxia exposure on developing immune systems.
151 importance that the effects of air pollutant exposure on human skin be investigated.
152 rm the effects of ionizing radiation chronic exposure on nonhuman species, we investigated the transp
153 ed with both maternal and paternal Holocaust exposure or associated with both maternal and paternal a
154 y be due to differences in abundance, recent exposure, or space weathering.
155   Slower plasma clearance and increased drug exposure over time of ALDC1 were observed compared to AL
156     BCG protection decreased with increasing exposure (P = .05) and increasing age (P = .004).
157 d trauma-induced by supraphysiological shear exposure-paradoxically increases the deformability of RB
158                      Twenty-four hours after exposure, participants underwent bronchoscopy to collect
159                    Furthermore, prolonged RA exposure phenocopies LSD1 inhibition, suggesting that LS
160  in reproductive timing in response to light exposure, potentially creating phenological mismatches(7
161 revention and the impact of doxycycline post-exposure prophylaxis (PEP).
162 erventions, such as antiretrovirals, for pre-exposure prophylaxis.
163  significant in individuals reporting trauma exposure (r(g) = 0.24, p = 1.8 x 10(-7) versus r(g) = -0
164                                         PFOS exposure recapitulated the reduction in length but could
165 atform for drug assays employing cyclic drug exposure regimens.
166                             Sustained gluten exposure reliably induces histologic changes but is burd
167      In an adjusted Cox model, brincidofovir exposure remained associated with a lower hazard for HHV
168                          The mechanism of PS exposure remains uncertain but it can follow increased i
169 ary exposure is comparable to PCB inhalation exposures reported for the same study population.
170        Differences in health care access and exposure risk may be driving higher infection and mortal
171 from 1 day to 6 months after a synaptopathic exposure, shows dramatic synaptic re-organization during
172  showed correlation with other environmental exposure signatures and a field effect in NATs.
173 tress-related stimuli following acute stress exposure.SIGNIFICANCE STATEMENT This study in rats shows
174 exposure is a binary variable, analyses from exposure-stratified models which consist of estimating g
175 ulin-resistant KKAy mice were used for ozone exposure studies.
176 matter (PM) using mass-based measurements as exposure surrogates.
177 velengths required significantly lower laser exposure than near infrared wavelengths to disable subje
178 ils with "hidden hearing loss" through noise exposure that increased hearing thresholds only temporar
179 ses are strongly influenced by environmental exposures that stimulate innate immune pathways capable
180  in a randomized clinical trial of prolonged exposure therapy (n = 36) versus treatment waiting list
181                                              Exposure to 10 or 25 uM ivabradine-an open channel block
182  measured in livers of fathead minnows after exposure to 38 different conditions.
183                                              Exposure to 5000 mug/L fluoxetine resulted in elevated 7
184 t of severe corneal epithelial disease after exposure to a dry environment.
185 he improvement in clinical disease following exposure to a lethal dose of LPS.
186                             During prolonged exposure to Ags, such as chronic viral infections, susta
187 counts for a large part of students' diurnal exposure to air pollution, especially in cities in devel
188    The study results suggest that short-term exposure to ambient air pollution is associated with an
189                                        Brief exposure to ATP induces the opening of a nonselective ca
190                                      Whether exposure to BPA or its structural analogs bisphenol S (B
191 d with unnecessary antibiotic escalation and exposure to broader-spectrum antibiotics than needed.
192 expression levels were reduced after 2 hours exposure to catechins but increased after 24 hours.
193 f corals to environmental stressors and that exposure to chemical dispersants may pose a greater thre
194 and TLR4(-/-) RAGE(-/-) mice following acute exposure to cigarette smoke (CS).
195  barrier dysfunction could facilitate T cell exposure to commensal/pathogenic microbes.
196 ially leading to bioaccumulation in fish and exposure to communities downstream.
197                         The cycA mutation or exposure to DCS were both associated with the accumulati
198     Here we evaluate the effects of repeated exposure to Delta9-tetrahydrocannabinol (THC) in a mouse
199  translational study indicate that low level exposure to diethyl phthalate results in measurable geno
200 for purification and formulation of rNDV, as exposure to different salt concentrations may be needed.
201        In terms of cardiac physiology, acute exposure to e-cigarette aerosols in human subjects led t
202 investigated the immunoregulatory effects of exposure to EE, especially in autoimmunity.
203 sponsiveness, a characteristic of asthma, on exposure to electronic cigarettes, across mouse strains,
204 hich revealed a local leaflet expansion upon exposure to elevated concentrations of a long-chain AC,
205 natal models of BPD induced by intraamniotic exposure to endotoxin (ETX) or sFlt-1 (soluble fms-like
206  sources of nutrients and water, high pH and exposure to extreme variations in temperature, humidity
207                           We find that local exposure to fatal school shootings increases youth antid
208 ding the relationship between relatively low exposure to fine particulate matter (particulate matter
209                                              Exposure to flame retardants (FRs) is associated with ad
210                                              Exposure to GenX or PFOA resulted in increased GWG, with
211                                     Patients exposure to harmful treatment practices, such as shackli
212 related HCC can be prevented by reducing the exposure to HBV by vaccination or by treatment of CHB in
213                Viruses can be inactivated by exposure to heat or bleach, but poliovirus, coxsackievir
214 ogen activator inhibitor-1, was depressed by exposure to high glucose, with the reduction in nitric o
215 eating behavior were also normal, even after exposure to high-fat diet.
216  lower local flows speeds, they can tolerate exposure to high-flow speeds of up to 114 cm s(-1), the
217 or dissociated, state of the dye cargo after exposure to human biologic fluids.
218         Sexual contact carries some risk for exposure to infection with severe acute respiratory synd
219 enic afterdepolarizations, particularly upon exposure to inflammatory mediators.
220  procedural serious adverse events (SAE) and exposure to ionizing radiation.
221     Efficient base editing requires cellular exposure to levels of base editors that can be difficult
222 3 in macrophages safeguards mice from lethal exposure to lipopolysaccharides, but this protection is
223    Our results show that population-weighted exposure to locally defined extreme heat (i.e., "populat
224 500 sequencer on the strain with and without exposure to meropenem.
225 ans in both their developmental timeline and exposure to microorganisms.
226 ersity, yet stands to lose both from present exposure to natural hazards, and future climate-driven s
227  of oxidation of the Cu surface prior to the exposure to negative potentials.
228 about the current communication environment, exposure to news, and ongoing attempts to limit the effe
229 nserved in human islets and is stimulated by exposure to nonesterified free fatty acids at concentrat
230 l intubation group included children without exposure to noninvasive ventilation within 6 hours befor
231 ip in both treatments declining in continued exposure to OA.
232            These data suggest that timing of exposure to oil and/or hypoxia matters, and underscores
233             There is concern about potential exposure to opportunistic pathogens when reopening build
234 ears following DWH, more recent increases in exposure to PAHs in some species suggest a complex inter
235                                      Chronic exposure to particulate matter < 2.5u (PM(2.5)) has been
236 opionic acid (AMPA) receptors in vitro after exposure to patients' CSF antibodies or SSM5, we used a
237 ccumulation of hydroxylated metabolites upon exposure to PCB-153, leading to oxidative stress, hemoly
238 this study was to investigate the effects of exposure to PCB-153, the most prevalent PCB congener in
239 lence of ADHD in association with early life exposure to PFOS and PFOA.
240 asures to control dust may reduce children's exposure to phthalates found in building materials and h
241 uce the risk of PTD associated with maternal exposure to PM in ambient air during pregnancy.
242 als life that may include sex differences in exposure to predators, immune capacity and cost of repro
243                      In contrast, continuous exposure to rec-TF up to 14 days, resulted in lower PTEN
244               Here we examined the effect of exposure to repeated multiple concurrent stressors (RMS)
245 tial adverse neurobehavioral consequences of exposure to saccharin alone or saccharin and nicotine fo
246 ficant metabolic derangements, hypoxemia, or exposure to sedating medications that may have contribut
247  well as staff and subsequent family members exposure to severe acute respiratory syndrome coronaviru
248 ion observed during hypercapnia or after the exposure to short-term sustained hypoxia.
249 to address the relationship between in utero exposure to smoking and the heightened risks for the sub
250 sient rhinitis was associated with antenatal exposure to smoking, higher maternal education levels, a
251 R(ER) activation and extended longevity, and exposure to stress led to splicing and activation of xbp
252 evidence of TB infection by TST and no known exposure to TB were used as controls to establish a thre
253                                              Exposure to thalidomide during a critical window of deve
254                               As a result of exposure to the alkaloids, the parasites became swollen
255 cisely at the time of birth, coinciding with exposure to the external environment.
256 is shift occurred spontaneously on prolonged exposure to the Lewis acidic reaction conditions.
257 upport the hypothesis of a potential role of exposure to the plume from Mount Etna in determining the
258                               In particular, exposure to the poly(allylamine hydrochloride)-coated pa
259                 We quantified the population exposure to these detailed particle properties and compa
260 increase in P. aeruginosa TOL-TAZ MICs after exposure to TOL-TAZ.
261                                              Exposure to traffic noise is associated with stress and
262 vels may explain why no relationship between exposure to TTR-binding compounds and circulating T4 lev
263 nd increasing oxygen demand, high and rising exposure to ultraviolet radiation, low ionic strength, a
264  be physiologically threatened by short-term exposure to warm temperatures and that longer-term physi
265 buting to stationary phase cell death during exposure to weak acid stress.
266    Pneumococcal carriage was associated with exposure to young children (OR:2.71, 95%CI 1.51-5.02, p<
267  surface to investigate whether PTSD and ELT exposure uniquely affect CT, controlling for scanner sit
268 ose Simulation (SHEDS)-Multimedia/Integrated Exposure Uptake and Biokinetic (IEUBK) model was applied
269 s in subjects with significant tobacco smoke exposure using deep gene resequencing and alpha-1 antitr
270 sociations with, SAE and high-dose radiation exposure using large-scale registry data.
271             If both between- and within-area exposure variability are exploited, RF may be preferred.
272 studies should use standard, clearly defined exposure variables to strengthen understanding of the re
273 e to short active (SA; 19:5 L:D) photoperiod exposure versus their wildtype (WT) littermates.
274 r mechanisms of PR and the role of polymyxin exposure versus transmission in PR emergence.
275                 Research has focused on PFAS exposure via drinking water and diet, and fewer studies
276 cles per cubic centimeter (cc)] relative UFP exposure was 1.04 [95% confidence interval (CI): 1.02, 1
277  For apixaban 2.5 mg twice daily, the median exposure was 2780 ng/(mL.h) for patients with CrCl 25 to
278 A expression obtained before and after ozone exposure was also used to identify changes associated wi
279                                          The exposure was APT preceding ICH diagnosis.
280                                     Firework exposure was not associated with an elevated rate of oth
281 ac standard uptake value and volume 1-M post-exposure was significantly elevated after PM-exposure.
282                                      The key exposure was the historical opioid-prescribing pattern o
283                                Following ddC exposures, we measured cell viability, mtDNA copy number
284    Moreover, increases in smoking-related Cd exposure were associated with differential DNAm.
285  the DEGs associated with parental Holocaust exposure were enriched for glucocorticoid-regulated gene
286  both maternal and paternal age at Holocaust exposure were in the same direction.
287                   Changes in outdoor PM(2.5) exposure were observed between 2012 and 2016 with highes
288 hodilator lung function, atopy, and cleaning exposures were assessed in 425 cleaners and 281 referenc
289                                       Target exposures were reached and antiviral activity was observ
290 mbination of genetic and early environmental exposures, whereas late transient rhinitis may relate to
291 nditions for photolysis would maximize light exposure, which may differ from those for nutrients.
292 ngiography decreased participants' radiation exposure while preserving diagnostic image quality.
293 h high ESA doses and decrease the cumulative exposure-while reducing the need for red blood cell tran
294 vestigate the association of long-term PM2.5 exposure with total mortality and cause-specific (cardio
295 We studied the association of Cd and smoking exposures with human blood DNA methylation (DNAm) profil
296 luated, the method detected 19 distinct drug exposures with statistically significant, large relative
297 evalence studies may enable investigation of exposure within dialysis units and hence, assessment of
298 with EVALI can provide direct information on exposure within the lung.
299 nclusions about the relative toxicity of the exposures within each case study.
300 defined extreme heat (i.e., "population heat exposure") would increase by a factor of 12.7-29.5 under

 
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