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1 s and civilian trauma victims with traumatic exsanguination.
2 CA for cardiac tamponade and 5 minutes after exsanguination.
3 a now do so as a result of causes other than exsanguination.
4  of cardiac tamponade (105 patients, 17.5%), exsanguination (418 patients, 69.6%), and exsanguination
5 later period, as did the rate of deaths from exsanguination (9% to 1%), multiple organ failure (12% t
6       Seven of 19 affected male mice died of exsanguination after tail snipping, and two affected mic
7 s appear to be in reducing early deaths from exsanguination and late deaths from multiple organ failu
8 ar to those of today, rapidly preventing any exsanguination and the breakdown of osmoregulation of th
9 iac tamponade, 8 of 418 patients (1.9%) with exsanguination, and none of the 72 patients with combine
10 emostasis and fewer experienced death due to exsanguination by 24 hours.
11 the 1980s in dog outcome models of prolonged exsanguination cardiac arrest has culminated in brain an
12 ation (EPR) of trauma victims who experience exsanguination cardiac arrest may allow survival from ot
13                      Suspended animation for exsanguination cardiac arrest of trauma victims would ha
14  have developed and used novel dog models of exsanguination cardiac arrest to explore suspended anima
15 mprise a novel approach for resuscitation of exsanguination cardiac arrest victims.
16 ats showing no neurologic injury, despite an exsanguination cardiac arrest, followed by 20 mins of EP
17 f cold saline immediately after the start of exsanguination cardiac arrest-which rarely can be resusc
18 y) cannot be resuscitated, such as traumatic exsanguination cardiac arrest.
19 tion) induced immediately after the start of exsanguination cardiac arrest.
20 ), exsanguination (418 patients, 69.6%), and exsanguination combined with cardiac tamponade (72 patie
21 decreased to 40 mm Hg during 60 minutes with exsanguination from the 30th to the 60th minute to a mea
22 P] of approximately 40 mm Hg) was induced by exsanguination into a reservoir.
23                                              Exsanguination is still the principal cause of early dea
24 etabolic effects of SAAP with HBOC-201 in an exsanguination model of cardiac arrest.
25                                        After exsanguination of the rats 2, 4, 6, 12, 24, or 72 hours
26 in mean arterial pressure induced by partial exsanguination or blood transfusion.
27                          After sacrifice via exsanguination, the following serum levels were determin
28                                    Following exsanguination under anaesthesia, fetal hearts were moun
29               The animals were sacrificed by exsanguination under general anesthesia and then perfuse
30                                              Exsanguination, which was the predominant cause of death
31 ontraumatic (e.g., ruptured aortic aneurysm) exsanguination, without severe brain trauma, by enabling