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1 he developmental and evolutionary origins of external genitalia.
2 es, brachydactyly, seizures, and hypoplastic external genitalia.
3 ation, outgrowth or normal patterning of the external genitalia.
4 lved in the normal development of gonads and external genitalia.
5 es showed strong contrast enhancement of the external genitalia.
6 sinus, and clitoral hypertrophy or ambiguous external genitalia.
7  normal development of the male internal and external genitalia.
8 es, except that males and females had female external genitalia.
9 te markers of primordia for the hindlimb and external genitalia.
10 tand diseases associated with defects in the external genitalia.
11 eam targets of the androgen receptor (AR) in external genitalia.
12 as 6.7% in the oral cavity and 16.9% for the external genitalia.
13 critical structures such as the scalp, face, external genitalia, acral, periumbilical, and perineal a
14 nd dominate them in social encounters; their external genitalia also are highly masculinized.
15 ions for the evolutionary diversification of external genitalia and for the association between exter
16                       Shh knockout mice lack external genitalia and have a persistent cloaca.
17 tissue layers and cell types within the LUT, external genitalia and lower reproductive structures.
18  (Ihh) as a novel downstream target of AR in external genitalia and show that conditional deletion of
19 e at 9 weeks of age displayed underdeveloped external genitalia and uteri.
20 lays down the primordia for the hindlimb and external genitalia, and switches from the epiblast/primi
21 ng, urorectal septation, and modeling of the external genitalia; and internally, the emergence of bas
22                              Male and female external genitalia appear identical early in gestation.
23                         Malformations of the external genitalia are among the most common congenital
24                         Birth defects of the external genitalia are among the most common in the worl
25                                              External genitalia are body appendages specialized for i
26       We found that overall, male and female external genitalia are largely composed of the same core
27                                          The external genitalia are some of the most rapidly evolving
28  support a limb-like developmental origin of external genitalia as the ancestral condition.
29 gene cause incomplete masculinization of the external genitalia by disrupting AR function in males wi
30        In mammalian embryos, male and female external genitalia develop from the genital tubercle.
31 humans, the genetic pathways governing early external genitalia development and urethra formation are
32 e dynamic changes in cell populations in the external genitalia during the critical morphogenetic win
33 s study, we show that inactivation of Shh in external genitalia extends the cell cycle from 8.5 to 14
34 ere we show that the developmental origin of external genitalia has shifted through evolution, and in
35                       Masculinization of the external genitalia in humans is dependent on formation o
36                               Development of external genitalia in mammalian embryos requires tight c
37 as led to an altered developmental route for external genitalia in mammals, while preserving parts of
38 egulated during embryonic development of the external genitalia in mice, and that this regulation is
39 gnaling, during embryonic development of the external genitalia in mice.
40                 While most birds do not have external genitalia, in a small number of species the mal
41                                  The size of external genitalia increased in both sexes, but they rem
42                 We propose that induction of external genitalia involves an epithelial-epithelial int
43                 Development of the mammalian external genitalia is controlled by a network of signali
44 wever, the origin of cells that give rise to external genitalia is unknown.
45                          Although congenital external genitalia malformations represent the second mo
46 6 variants prevalence and persistence in the external genitalia of men enrolled in the prospective HP
47          Circulating levels of testosterone, external genitalia, or fertility were not altered in pes
48 haracterized by necrotizing infection of the external genitalia, perineum, and perianal region.
49                      Proper formation of the external genitalia requires a highly orchestrated proces
50 ence of HPV-16 variants observed at the male external genitalia suggest differences in the natural hi
51 ovide insights into the morphogenesis of the external genitalia that could be used to understand dise
52    Hypospadias is a congenital defect of the external genitalia that results from failure of urethral
53  In the early phase, the embryonic anlage of external genitalia, the genital tubercle (GT), is morpho
54 limbs and an altered Hoxd gene regulation in external genitalia, the limb-associated bimodal HoxD chr
55 nal cord conveying information from the male external genitalia to MRF, and (2) ascending bilateral p
56                     Serial MR imaging of the external genitalia was performed in 12 healthy sexually