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1 duced by transforming growth factor-beta1 or extracellular matrix protein.
2 Dentin sialoprotein (DSP) is a dentin extracellular matrix protein.
3 f different integrin subtypes and sensing of extracellular matrix proteins.
4 trix, involved in the degradation of various extracellular matrix proteins.
5 oduction of collagen type I (COL1) and other extracellular matrix proteins.
6 occal adhesins interacts with a multitude of extracellular matrix proteins.
7 cells resulted in a decrease in adhesion to extracellular matrix proteins.
8 erentiation into myofibroblasts that secrete extracellular matrix proteins.
9 cretion of extracellular vesicles containing extracellular matrix proteins.
10 s and to RGD motifs present in loops in many extracellular matrix proteins.
11 ps was assessed after treatment with various extracellular matrix proteins.
12 y stabilizing fibrin clots and cross-linking extracellular matrix proteins.
13 s endothelial function via interactions with extracellular matrix proteins.
14 pression of bone-related gene regulators and extracellular matrix proteins.
15 ofibroblasts, which secrete large amounts of extracellular matrix proteins.
16 oepicardial organ and aberrant deposition of extracellular matrix proteins.
17 tory proteins and enhances the production of extracellular matrix proteins.
18 f SMC contractile genes and up-regulation of extracellular matrix proteins.
19 differentiation with resulting deposition of extracellular matrix proteins.
20 st-derived MVs including calcium-binding and extracellular matrix proteins.
21 x metalloproteinase (MMP) degradation of the extracellular matrix proteins.
22 l cell surface where it promotes adhesion to extracellular matrix proteins.
23 control their proliferation and secretion of extracellular matrix proteins.
24 integrin receptors and subsequently degrades extracellular matrix proteins.
25 by an extensive network of stromal cells and extracellular matrix proteins.
26 ired cell spreading and adhesion to selected extracellular matrix proteins.
27 MicroRNA29 (miR29) targets extracellular matrix proteins.
28 collagen IX-deficient cartilage included 15 extracellular matrix proteins.
29 ion with adenosine deaminase (ADA) and other extracellular matrix proteins.
30 tes, endothelial and neuronal cells, and the extracellular matrix proteins.
31 ase-beta expression, indicative of increased extracellular matrix proteins.
32 mineralization process that is controlled by extracellular matrix proteins.
33 roteomics revealed a significant increase in extracellular matrix proteins.
34 metalloproteinase) family, known to process extracellular matrix proteins.
35 ng its ability to function as a receptor for extracellular matrix proteins.
36 kinase (AKT) signaling and up-regulation of extracellular matrix proteins.
37 (63%) were plasma membrane, cell surface or extracellular matrix proteins.
38 PSC) and is characterized by accumulation of extracellular matrix proteins.
39 so increasing expression of cartilage tissue extracellular matrix proteins.
40 esembles structures found in many eukaryotic extracellular-matrix proteins.
42 345W mutation in EGF-containing fibulin-like extracellular matrix protein 1 (EFEMP1) to investigate t
44 pha/beta integrins, osteopontin, and related extracellular matrix proteins; (2) clastic cell fusion a
45 duced fibrogenic gene expression, release of extracellular matrix proteins, activation of hepatic myo
46 d phenotypes, including enhanced adhesion to extracellular matrix proteins, activation of intracellul
47 ur study unveils netrin-4 as a non-enzymatic extracellular matrix protein actively disrupting pre-exi
49 e previously reported that a fragment of the extracellular matrix protein agrin promotes cardiac rege
50 l density, and enhanced glycosylation of the extracellular matrix protein, alpha-dystroglycan, all co
51 anced adherence to human enterocytes through extracellular matrix protein and bacterial aggregation.
52 livery of a bispecific molecule targeting an extracellular matrix protein and delivering a TGF-beta m
53 tent TGF-beta binding protein 1 (LTBP-1), an extracellular matrix protein and key regulator of TGF-be
54 re muscle centric followed by cell adhesion, extracellular matrix proteins and a few pro-inflammatory
55 ssion of alpha-smooth muscle actin (SMA) and extracellular matrix proteins and activated AMP-activate
56 g to an increased level of cell-junction and extracellular matrix proteins and an altered cytokine se
57 ucine-rich proteoglycans interact with other extracellular matrix proteins and are important regulato
59 dystroglycan that is responsible for binding extracellular matrix proteins and certain arenaviruses.
60 nned by a network of stromal cells producing extracellular matrix proteins and chemokines, enabling l
61 ent mesangial cells, increased production of extracellular matrix proteins and cytokines, and ultimat
62 cruited and activated to lay a foundation of extracellular matrix proteins and glycosaminoglycans tha
63 eric antigen receptor (CAR) T cells, reduced extracellular matrix proteins and glycosaminoglycans.
64 taneous wound healing with discussion on how extracellular matrix proteins and hypoxia can be utilize
65 new approaches to MSC transplantation using extracellular matrix proteins and hypoxia preconditionin
66 ing, and the effect of collagen I and fibrin extracellular matrix proteins and insulin-like growth fa
67 expression of alpha-smooth muscle actin and extracellular matrix proteins and is dependent on metabo
68 roglycan (DG) is a cell surface receptor for extracellular matrix proteins and is involved in cell po
69 oglycan functions as a receptor for multiple extracellular matrix proteins and its dysfunction leads
70 r observed that Pneumocystis binding to host extracellular matrix proteins and lung epithelial cells
71 le pro-fibrotic molecules, including several extracellular matrix proteins and myofibroblast and cell
72 n, inflammatory cytokines, immune mediators, extracellular matrix proteins and oncogene expression.
73 omic analysis shows extensive alterations in extracellular matrix proteins and pathways related to fi
74 cle cell migration and adhesion to different extracellular matrix proteins and regulate cellular stif
75 lysaccharide that decorates cell surface and extracellular matrix proteins and regulates the biologic
76 and characterized by increased deposition of extracellular matrix proteins and scar formation in the
77 t, through proper assembly of RGD-containing extracellular matrix proteins and the correct incorporat
78 ed silica nanoparticle monolayer coated with extracellular matrix proteins and the desired siRNA.
79 sulted in 2-4 folds increase in secretion of extracellular matrix proteins and the reorganization of
80 ion of inert heat-derived HSA hydrogels with extracellular matrix proteins and these may be used as a
81 olved in the synaptic plasticity by cleaving extracellular matrix proteins and, thus, is associated w
82 oto-crosslinkable formulation of native ECM (extracellular matrix) proteins and used this bioink to 3
83 sections with collagen-I, fibronectin (major extracellular-matrix proteins), and alpha-SMA (well-char
84 rs, potentiation of growth factor signals by extracellular matrix proteins, and activation of self-re
85 HTRA1 has the capacity to degrade numerous extracellular matrix proteins, and as such, its potentia
86 actor beta-1-SMAD3 activation, production of extracellular matrix proteins, and cell contractility.
87 sm, unfolded protein responses, secretion of extracellular matrix proteins, and cell proliferation.
88 d chemokines, enzymes and enzyme inhibitors, extracellular matrix proteins, and membrane receptors, b
91 wound healing, where excessive collagen and extracellular matrix proteins are deposited within the w
92 des that cell surface, soluble-secreted, and extracellular matrix proteins are generally rich in disu
93 ngs demonstrate the direct involvement of an extracellular matrix protein as a receptor for TeNT at t
95 nt TGF-beta-binding protein-2 (LTBP-2) is an extracellular matrix protein associated with microfibril
96 novel candidates were identified, including extracellular matrix proteins associated with the baseme
97 MV receptor integrin beta 1 dissociates from extracellular matrix proteins, becoming internalized wit
98 repeating disaccharide that is necessary for extracellular matrix protein binding to O-mannosylated a
99 pport cells, gap junctions, soluble factors, extracellular matrix proteins, blood vessels and neural
100 and acute inflammation not only by degrading extracellular matrix proteins but also by controlling th
101 ations in the primary amino acid sequence of extracellular matrix proteins can have profound effects
103 stribution was dependent on induction of the extracellular matrix protein collagen type V alpha 1 (co
106 stronger response to TGF-beta1 in producing extracellular matrix protein (collagen and fibronectin)
107 lasts correlated with gene expression of the extracellular matrix proteins, collagen (Col)1a1, Col1a2
108 h Muscle Actin, Vimentin, and beta-catenin), extracellular matrix proteins (Collagens, Fibronectin, a
110 tions in eyes shut homolog (EYS), a secreted extracellular matrix protein containing multiple laminin
112 thermore, we found that CD157 binds to other extracellular matrix proteins containing heparin-binding
114 cers more invasive by increasing proteolytic extracellular matrix protein degradation fostering colle
115 minantly responsible for A549 cell rounding, extracellular matrix protein degradation, and IL-8 degra
117 hage activation to decrease TGF-beta-induced extracellular matrix protein deposition in the kidney in
118 ion of candidate molecules revealed that the extracellular matrix protein dermatopontin (Dpt) is invo
120 roperties in vitro Thus, Fgfbp1 is a crucial extracellular matrix protein during BBB maturation that
122 , which execute tissue fibrosis by producing extracellular matrix proteins, efficiently engulf dead c
123 simple coacervation of proteins, such as the extracellular matrix protein elastin, have not been repo
124 ted interaction between cancer cells and the extracellular matrix proteins, enhancing cell detachment
125 exhibited defective adhesion to a variety of extracellular matrix proteins, especially laminin-1 and
126 Our findings unravel a mechanism by which an extracellular matrix protein evokes stress signaling in
128 h factor beta-induced protein (TGFBIp) is an extracellular matrix protein expressed in several cell t
131 Punctin/MADD-4, a member of the ADAMTSL extracellular matrix protein family, was identified as a
132 materials that mimic the nanoscale order of extracellular matrix protein fibers and yield suitable e
136 mponent, and, separately have shown that the extracellular matrix protein fibronectin (FN) contribute
139 e this vasodilatation, we show here that the extracellular matrix protein fibronectin also contribute
140 des are known to mimic the binding domain of extracellular matrix protein fibronectin and selectively
141 served that proteolytic cleavage of the host extracellular matrix protein fibronectin by peritoneal c
142 s an environmental factor, aggregates of the extracellular matrix protein fibronectin perturb the mat
145 adhesion of vascular smooth muscle cells to extracellular matrix proteins fibronectin and collagen.
146 ced mesangial expansion, accumulation of the extracellular matrix proteins fibronectin and type IV co
147 of these peptides further delineated how the extracellular matrix protein FN can support cell surviva
149 ed by endogenous alarmins such as fragmented extracellular matrix proteins found in degenerating disc
152 ltistep process in which adhesion molecules, extracellular matrix proteins, growth factors, and their
153 ions, such as collagenous and noncollagenous extracellular matrix proteins, growth factors, proteases
157 s growth factors, cytokines, chemokines, and extracellular matrix proteins have been limited yet prov
159 factor-binding domains identified in various extracellular matrix proteins have been shown to regulat
160 Dentin sialophosphoprotein (DSPP) is an extracellular matrix protein highly expressed by odontob
162 type 1 collagen expression (COL1A1), a major extracellular matrix protein important in liver fibrosis
163 ATEMENT Emerging evidence supports roles for extracellular matrix proteins in boosting synapse format
164 merging evidence supports roles for secreted extracellular matrix proteins in boosting synaptogenesis
165 en also identified widespread involvement of extracellular matrix proteins in DBL-1 regulation of bod
167 tion upon stimulation with growth factors or extracellular matrix proteins in different tumor cells.
168 ue culture, TNC was superior amongst several extracellular matrix proteins in enhancing the sphere-fo
170 the potential role of fibronectin and other extracellular matrix proteins in HIV-1 biology.IMPORTANC
171 ofibroblast proliferation and mRNA levels of extracellular matrix proteins in mice and attenuated myo
173 l junction-related proteins in squamous, and extracellular matrix proteins in sarcomatoid subtypes.
176 atment altered the expression of a subset of extracellular matrix proteins in the skin, including upr
179 We investigated the role of periostin, an extracellular matrix protein, in the pathophysiology of
180 models to explain the observed enrichment of extracellular matrix proteins include both increased pro
181 , we identify copy number alterations in key extracellular matrix proteins including collagen 1 type
182 develops in a microenvironment enriched with extracellular matrix proteins including laminin (Ln)-332
184 decreased the TGFbeta-dependent induction of extracellular matrix proteins, including collagen Ialpha
185 on, revealed that miR-195 targets a cadre of extracellular matrix proteins, including collagens, prot
186 cells failed to express normal levels of key extracellular matrix proteins, including laminin alpha2.
187 e thought to be linked by several additional extracellular matrix proteins, including nidogen and per
190 ngth and diminishes the capacity of specific extracellular matrix proteins-including collagen I, coll
191 Neutrophil activation by FN, but not other extracellular matrix proteins, induces the release of th
192 four cell polarity, cell junction and apical extracellular matrix proteins involved in tracheal tube
193 rneuron-derived Collagen XIX, a synaptogenic extracellular matrix protein, is required for the format
195 Animal studies showed COL13A1, a synaptic extracellular-matrix protein, is involved in the formati
196 tin is one of the most highly phosphorylated extracellular matrix proteins known in nature with uniqu
197 tem provides a mechanism for the delivery of extracellular matrix proteins known to be important for
198 ts a new perspective on how mutations in the extracellular matrix protein laminin cause severe conseq
200 inoglycans such as heparan sulfate (HS), the extracellular matrix protein laminin, and/or integrin be
201 sclA allele restored the ability to bind the extracellular matrix proteins laminin and cellular fibro
202 ecular-weight kininogen (HK), as well as the extracellular matrix proteins laminin and collagen V.
203 ecrosis, and apoptosis and the generation of extracellular matrix proteins leading to fibrosis/cirrho
206 ifferentiation, and deposit large amounts of extracellular matrix proteins maintaining the structural
207 e used the EAE model and determined that the extracellular matrix protein matrilin-2 (MATN2) is an en
208 of the matrix metalloprotease MMP-10 and the extracellular matrix protein mindin (encoded by Spon2) i
209 at catalyzes the formation of cross-links in extracellular matrix proteins, namely, collagen and elas
213 ut did not significantly drive production of extracellular matrix proteins or alpha-smooth muscle act
217 ted migration signaling pathways and reduced extracellular matrix protein production, and blocked myo
218 processes, such as mucus hypersecretion and extracellular matrix protein production, are also direct
219 naling enhanced fibroblast proliferation and extracellular matrix protein production, effects relevan
220 e provide evidence that tenascin-C (TNC), an extracellular matrix protein prominent in malignant glio
224 e cellular and molecular mechanisms by which extracellular matrix proteins regulate left-right organ
225 ombospondin repeats)-like family, a class of extracellular matrix proteins related to the ADAM protea
226 nt HSCs to HSC myofibroblasts, which secrete extracellular matrix proteins responsible for the fibrot
227 blast-like cells that secrete high levels of extracellular matrix proteins, resulting in fibrosis.
228 E-glycan of dystroglycan serves as a tunable extracellular matrix protein scaffold, the extension of
230 c carboxypeptidase-like protein (ACLP) is an extracellular matrix protein secreted by fibroblasts and
232 a multifunctional scaffold that coordinates extracellular matrix proteins, secreted cues, and transm
235 mbinant WxL proteins from locus A bind human extracellular matrix proteins, specifically type I colla
236 amatically reduces lymphoma cell adhesion to extracellular matrix proteins, subcutaneous tumor size i
237 ustained fibrosis, excessive accumulation of extracellular matrix proteins substantially dampens the
240 r the interaction of alpha-dystroglycan with extracellular matrix proteins such as laminin-alpha2.
241 ils were activated in vitro with immobilized extracellular matrix proteins, such as fibronectin (FN),
244 n with integrin beta1 were also required for extracellular matrix protein synthesis in response to HG
246 imulate fibroblast activation, inducing ECM (extracellular matrix) protein synthesis and promoting fi
248 , diminishing expression of the desmoplastic extracellular matrix protein tenascin C, suppressing tum
249 SC derived from either PDLN or mPIN used the extracellular matrix protein Tenascin-C (TNC) to inhibit
251 ed injury and attenuated upregulation of the extracellular matrix protein, tenascin C, which affords
256 h factor-beta-induced protein (TGFBIp) is an extracellular matrix protein that has a role in a wide r
257 n-2 (MMRN2) is a unique endothelial specific extracellular matrix protein that has been implicated in
258 se-like protein (ACLP) is a collagen-binding extracellular matrix protein that has important roles in
259 treatment reduced fibronectin expression, an extracellular matrix protein that is increased in diabet
260 entin sialophosphoprotein (DSPP) is a dentin extracellular matrix protein that is processed into dent
261 ility to angiogenic diseases and identify an extracellular matrix protein that is regulated by melano
263 gamma90 and the secretion of fibronectin, an extracellular matrix protein that regulates cell migrati
266 ause senescent erythrocytes to interact with extracellular matrix proteins that are exposed within th
267 mation of mineralized tissues is governed by extracellular matrix proteins that assemble into a 3D or
268 ortant sources of inflammatory molecules and extracellular matrix proteins that contribute to disease
269 binant gp120 produced fibronectins and other extracellular matrix proteins that copurified with gp120
270 In many cases, natural aGPCR ligands are extracellular matrix proteins that dissociate the NTF to
272 evelop contractile functions and secrete the extracellular matrix proteins that form this fibrous sca
273 s regulated by factors including enzymes and extracellular matrix proteins that promote or inhibit hy
275 d virus infectivity in vitro by proteolyzing extracellular matrix proteins, thereby increasing viral
276 selective peptides have been identified from extracellular matrix proteins; these peptides have prove
277 e of marked downregulation of genes encoding extracellular matrix proteins, those involving matrix de
279 or-1 alpha (HIF-1alpha) and the synthesis of extracellular matrix proteins through a mechanical mecha
280 ated myofibroblasts that excessively deposit extracellular matrix proteins, thus compromising lung ar
281 Given the perceived difficulty in modifying extracellular matrix proteins to create aGPCR probes, we
282 gated in 96-well plates in medium containing extracellular matrix proteins to promote epithelializati
283 basis included enzymes, regulatory proteins, extracellular matrix proteins, transcription factors, an
285 Further, MMP12-mediated degradation of the extracellular matrix proteins tropoelastin and fibronect
286 irulence factor interacting with laminin, an extracellular matrix protein ubiquitously expressed in t
287 phil adhesion and migration on periostin, an extracellular matrix protein upregulated in asthma by ty
289 ance of the interaction between uPAR and the extracellular matrix protein vitronectin (VN) for the si
291 approach to pattern the presentation of the extracellular matrix protein vitronectin, we accomplishe
292 n the levels of cytoskeletal, signaling, and extracellular matrix proteins were associated with CXCL8
294 s as well as the expression of cytokines and extracellular matrix proteins were evaluated by immunofl
295 Collagen and fibronectin (Fn) are two key extracellular matrix proteins, which are known to intera
296 PK increased the expression of alpha-SMA and extracellular matrix proteins, while inhibition of AMPK
299 ions in fibrillin-1 (FBN1), which encodes an extracellular matrix protein with homology to latent TGF