ライフサイエンスコーパス: extracellular matrix remodeling

1 anges are likely to be the result of scleral extracellular matrix remodeling.

2 by this enzyme family is critical for normal extracellular matrix remodeling.

3 d whether ONOO- and its metabolites modulate extracellular matrix remodeling.

4 s involved in oncogenesis, angiogenesis, and extracellular matrix remodeling.

5 e internal elastic lamina fragmentation, and extracellular matrix remodeling.

6 gulation in RCC and a potential regulator of extracellular matrix remodeling.

7 tes cell proliferation, differentiation, and extracellular matrix remodeling.

8 x metalloproteinases, critical regulators of extracellular matrix remodeling.

9 PARC on beta-catenin accumulation as well as extracellular matrix remodeling.

10 -encoded mitochondrial genes, and increasing extracellular matrix remodeling.

11 consistent with its role in angiogenesis and extracellular matrix remodeling.

12 sive maspin inhibits tumor cell invasion and extracellular matrix remodeling.

13 me course, consistent with a net increase in extracellular matrix remodeling.

14 nal transduction, metabolism, transport, and extracellular matrix remodeling.

15 angiogenesis, immune cell infiltration, and extracellular matrix remodeling.

16 response, cell cycle arrest, apoptosis, and extracellular matrix remodeling.

17 rix turnover is a novel mechanism regulating extracellular matrix remodeling.

18 in regulating physiological and pathological extracellular matrix remodeling.

19 ntegrin, that are required for smooth muscle extracellular matrix remodeling.

20 n that IR can also induce growth factors and extracellular matrix remodeling.

21 e base of the follicle, differentiation, and extracellular matrix remodeling.

22 cal and pathological events characterized by extracellular matrix remodeling.

23 icated in both intracellular proteolysis and extracellular matrix remodeling.

24 in convertase-MT1-MMP axis that can regulate extracellular matrix remodeling.

25 sm whereby cytokines or Ox-LDL may influence extracellular matrix remodeling.

26 ents during the menstrual cycle that require extracellular matrix remodeling.

27 etalloproteinases (MMPs) both participate in extracellular matrix remodeling.

28 sion of genes engaged in AMD, autophagy, and extracellular matrix remodeling.

29 d by immune activation, oxidative stress and extracellular matrix remodeling.

30 es associated with collagen biosynthesis and extracellular matrix remodeling.

31 nt in cell cycle regulation, DNA repair, and extracellular matrix remodeling.

32 adation, highlighting the effects of IL-9 on extracellular matrix remodeling.

33 tion, eosinophilic inflammation, and altered extracellular matrix remodeling.

34 enes and downregulation of genes involved in extracellular matrix remodeling.

35 scle injury, inflammation, regeneration, and extracellular matrix remodeling.

36 psular fibrosis by inducing inflammation and extracellular matrix remodeling.

37 ordinating stress responses and dysregulated extracellular matrix remodeling.

38 kinase) pathways, along with glycocalyx and extracellular matrix remodeling.

39 unctions, including immunity, signaling, and extracellular matrix remodeling.

40 vating to myofibroblasts that drive aberrant extracellular matrix remodeling.

41 signature, particularly in genes involved in extracellular matrix remodeling.

42 vasion, angiogenesis, lymphangiogenesis, and extracellular matrix remodeling.

43 kine secretion, immune cell recruitment, and extracellular matrix remodeling.

44 gulating inflammation, immune responses, and extracellular matrix remodeling.

45 through its correlation with MMP-9-mediated extracellular matrix remodeling.

46 ent, epithelial structure, inflammation, and extracellular matrix remodeling.

47 ways, epithelial-mesenchymal transition, and extracellular matrix remodeling.

48 meostatic processes, including cell size and extracellular matrix remodeling.

49 in part, from ongoing inflammation and poor extracellular matrix remodeling.

50 OXL2 independent of its conventional role in extracellular matrix remodeling.

51 P-10 may be implicated in H. pylori-mediated extracellular matrix remodeling.

52 ression of genes related to inflammation and extracellular matrix remodeling.

53 th G-protein-mediated muscle contraction and extracellular matrix remodeling.

54 to regulate cytoskeleton, cell adhesion, and extracellular matrix remodeling.

55 Wnt signaling, cell adhesion, migration, and extracellular matrix remodeling.

56 ar cells, cytokine/chemokine expression, and extracellular matrix remodeling.

57 13, which are critical modulators of stromal extracellular matrix remodeling.

58 inflammation, cellular stress response, and extracellular matrix remodeling across species.

59 ously unappreciated variation of hormone and extracellular matrix remodeling activities.

60 icular (ESLV) wall stress is associated with extracellular matrix remodeling activity after myocardia

61 ere was an enrichment in those implicated in extracellular matrix remodeling activity, cytoskeletal n

62 zymogen, MT1-MMP plays an essential role in extracellular matrix remodeling after an undefined proce

63 suggest that MMP-9 plays a prominent role in extracellular matrix remodeling after MI.

64 RATIONALE: Cardiac fibroblasts (CFs) drive extracellular matrix remodeling after pressure overload,

65 inhibited the gene program for fibrosis and extracellular matrix remodeling, although deletion of Tg

66 cerbating local inflammation, and increasing extracellular matrix remodeling, an environment conduciv

67 ort growing evidence that TGFbeta(2) induces extracellular matrix remodeling and abnormal cytoskeleta

68 IPF is characterized by extracellular matrix remodeling and accumulation of acti

69 matrix, and a second phase, characterized by extracellular matrix remodeling and altered mesenchymal-

70 MP-9), which has critical roles in promoting extracellular matrix remodeling and angiogenesis during

71 filtrating the onplants were associated with extracellular matrix remodeling and angiogenesis, in par

72 scriptional regulation of genes critical for extracellular matrix remodeling and cell cycle progressi

73 s) have been shown to be key players in both extracellular matrix remodeling and cell migration durin

74 red in collagen-rich mesenchymal tissues for extracellular matrix remodeling and cell proliferation d

75 ast and cervical cancer cells by controlling extracellular matrix remodeling and cellular contractili

76 re cytoskeletal-based structures involved in extracellular matrix remodeling and cellular motility.

77 ent for transcriptional pathways controlling extracellular matrix remodeling and chemotaxis in the lu

78 K/IKKalpha contributes to collagenase-driven extracellular matrix remodeling and chondrocyte hypertro

79 the secondary outcomes, HA seems to enhance extracellular matrix remodeling and collagen maturation,

80 owed that ML290 treatment primarily affected extracellular matrix remodeling and cytokine signaling,

81 rix metalloproteinases (MMPs) participate in extracellular matrix remodeling and degradation and have

82 erized by the concomitant hyperactivation of extracellular matrix remodeling and dsRNA-IFN signaling

83 Inflammation, immunomodulation, extracellular matrix remodeling and endoplasmic reticulu

84 cell and cell-matrix interactions, affecting extracellular matrix remodeling and endothelial cell mig

85 deposition, and significant upregulation of extracellular matrix remodeling and fibroblast activatio

86 oteomics from WS revealed an upregulation of extracellular matrix remodeling and focal adhesion proce

87 atrix metalloprotease-9, a known mediator of extracellular matrix remodeling and growth factor activa

88 oteinases (MMPs) are key enzymes involved in extracellular matrix remodeling and have been implicated

89 oproteinases (TIMPs) are involved in scleral extracellular matrix remodeling and have shown different

90 going inflammation, where it participates in extracellular matrix remodeling and healing.

91 pathways related to lipid metabolism as well extracellular matrix remodeling and immunosuppression wa

92 w that Ambra1 deficiency in melanoma impacts extracellular matrix remodeling and induces hyperactivat

93 1/2 directly regulate Mmp9, which integrates extracellular matrix remodeling and inflammation.

94 simultaneous assessment of collagen-related extracellular matrix remodeling and inflammatory activit

95 immune response resolution may contribute to extracellular matrix remodeling and interstitial myocard

96 at3 regulates angiogenesis, axon growth, and extracellular matrix remodeling and is essential for lon

97 serine proteases known to be responsible for extracellular matrix remodeling and metastatic dissemina

98 oproteinase inhibitor, TIMP1, which controls extracellular matrix remodeling and neutrophil function.

99 cellular adhesion, proliferation, branching, extracellular matrix remodeling and organ development.

100 data support a role for Tbx20 in repressing extracellular matrix remodeling and promoting cell proli

101 ronic acid production, as well as associated extracellular matrix remodeling and represents pharmacol

102 role and high energy requirements of hGFs in extracellular matrix remodeling and response to inflamma

103 th tumor microenvironment factors, including extracellular matrix remodeling and signaling pathways i

104 c subset of proteins to the cell surface for extracellular matrix remodeling and signaling.

105 human polymorphisms in proteins involved in extracellular matrix remodeling and the immune response

106 ions into therapeutics of diseases involving extracellular matrix remodeling and the immune response.

107 ed atherosclerotic plaques may contribute to extracellular matrix remodeling and the onset of acute t

108 al programs but also in subtypes involved in extracellular matrix remodeling and vascularization.

109 rse metabolic abnormalities in biomarkers of extracellular matrix remodeling and/or aging, pentoses/p

110 To test whether CTGF is required for extracellular matrix remodeling and/or angiogenesis duri

111 in increasing trabecular meshwork outflow by extracellular matrix remodeling and/or by modulation of

112 with activated marker expression, increased extracellular matrix remodeling, and acquisition of a pr

113 ycle re-entry, loss of contractile elements, extracellular matrix remodeling, and altered signaling b

114 tasis, promotes chronic inflammation, drives extracellular matrix remodeling, and alters hepatocyte c

115 aged in senescence, inflammation, autophagy, extracellular matrix remodeling, and AMD.

116 munity, stem cell activation and regulation, extracellular matrix remodeling, and angiogenesis.

117 ycation of proteins, alternative metabolism, extracellular matrix remodeling, and angiogenesis.

118 ed in cell signaling, immune system/defense, extracellular matrix remodeling, and cell cycle regulati

119 tivation, M2 macrophages, adaptive immunity, extracellular matrix remodeling, and cell proliferation.

120 encompass inflammation, hormonal signaling, extracellular matrix remodeling, and compensatory adapta

121 cholangiocyte and hepatocyte proliferation, extracellular matrix remodeling, and fibrosis.

122 intercellular interactions, cytoskeletal and extracellular matrix remodeling, and gradients of agonis

123 ure, improved re-epithelialization, enhanced extracellular matrix remodeling, and greater nerve reinn

124 tive response, including cell proliferation, extracellular matrix remodeling, and hair follicle neoge

125 change, with proteins related to metabolism, extracellular matrix remodeling, and immune function pat

126 ched for genes involved in lipid metabolism, extracellular matrix remodeling, and immunosuppression.

127 e severity of pulmonary fibrotic lesions and extracellular matrix remodeling, and improved pulmonary

128 mune avoidance, increased lymphatic network, extracellular matrix remodeling, and increased seeding t

129 proteins (involved in cardiomyocyte stretch, extracellular matrix remodeling, and inflammation) that

130 tion of hematopoietic stem cell development, extracellular matrix remodeling, and inflammatory cytoki

131 g-GLI signaling and of proteases involved in extracellular matrix remodeling, and matricellular prote

132 ration, stem cell maintenance, angiogenesis, extracellular matrix remodeling, and metastasis.

133 th smooth muscle cells (SMCs), inflammation, extracellular matrix remodeling, and mitogens.

134 ement in lesions, pruritus, stabilization of extracellular matrix remodeling, and processes associate

135 angiogenesis, blood coagulation, apoptosis, extracellular matrix remodeling, and regulation of infla

136 n the pathogenesis of collagen accumulation, extracellular matrix remodeling, and renal and cardiac f

137 he immune response, cell-cell communication, extracellular matrix remodeling, and the aryl hydrocarbo

138 motes tumor progression, including fibrosis, extracellular matrix remodeling, and the metabolic/catab

139 d in pathways that regulate inflammation and extracellular matrix remodeling, and they include matrix

140 The effects of Cav1 on lipid trafficking, extracellular matrix remodeling, and vascular inflammati

141 enes essential for mesenchyme proliferation, extracellular matrix remodeling, and vasculogenesis.

142 variety of growth factor, immunomodulatory, extracellular matrix-remodeling, and stress response gen

143 nsight into the effects of hydroxytyrosol on extracellular matrix remodeling; and ii) test whether hy

144 of chondrocyte death, chondroclast function, extracellular matrix remodeling, angiogenesis and bone f

145 (tumor necrosis factor)-family members; (2) extracellular matrix remodeling, angiogenesis and growth

146 on of mesenchymal transition, cell adhesion, extracellular matrix remodeling, angiogenesis, and wound

147 Biomarkers of extracellular matrix remodeling are associated with prev

148 inner choroid, degranulation, and subsequent extracellular matrix remodeling are early events in AMD

149 everse existing DD and establish blockade of extracellular matrix remodeling as a second mechanism by

150 es related to inflammation (CHI3L1, IL6) and extracellular matrix remodeling (ASPN), validating our p

151 ellular matrix architecture, and ameliorated extracellular matrix remodeling at gene expression level

152 s" AMR, which guides membrane deposition and extracellular matrix remodeling at the division site.

153 and show that macrophages are essential for extracellular matrix remodeling at the wound border zone

154 differences suggest disparate mechanisms of extracellular matrix remodeling between these 2 groups o

155 th and survival of cardiomyocytes as well as extracellular matrix remodeling by cardiac fibroblasts.

156 hat CTGF is a crucial regulator of cartilage extracellular matrix remodeling by generating Ctgf(-/-)

157 Nuclear ANKRD1 appears to modulate extracellular matrix remodeling by MMPs.

158 astin-derived peptides are released from the extracellular matrix remodeling by numerous proteases an

159 moved under perfusion and that inhibition of extracellular matrix remodeling causes mESCs to differen

160 actin-positive blood vessels, and 5) of key extracellular matrix remodeling (CD44, Col1a1, integrins

161 icated alterations in the pathways involving extracellular matrix remodeling, cell adhesion, and cell

162 he tumor microenvironment and is involved in extracellular matrix remodeling, cell proliferation and

163 sociated matrix metalloproteinase (MMP)-2 in extracellular matrix remodeling compared with that of th

164 nvolved in vascular permeability, secretion, extracellular matrix remodeling, cytoskeleton reorganiza

165 minal aorta, due to chronic inflammation and extracellular matrix remodeling/degradation within the v

166 of pathways involved in muscle function and extracellular matrix remodeling/deposition.

167 der-appreciated role for the muscle fiber in extracellular matrix remodeling during adaptation, along

168 egulates the secretion of MMPs to facilitate extracellular matrix remodeling during cell migration.

169 d in MR-null myoblasts, suggesting a role in extracellular matrix remodeling during cell motility.

170 Lysyl oxidases (LOXs) play a central role in extracellular matrix remodeling during development and t

171 ch biological processes as wound closure and extracellular matrix remodeling during tissue developmen

172 portance has been ascribed to this enzyme in extracellular matrix remodeling during tumoral, inflamma

173 es genes involved in cell growth control and extracellular matrix remodeling [e.g., plasminogen activ

174 in, and away from the tumor, associated with extracellular matrix remodeling (eg, granzyme B and fibr

175 osomes and metalloproteases is essential for extracellular matrix remodeling, enabling migration thro

176 bolic remodeling, mitochondrial dysfunction, extracellular matrix remodeling, endoplasmic reticulum s

177 ulation, gut leak and dysbiosis, cell death, extracellular matrix remodeling, endoplasmic reticulum s

178 d that WNT2-mediated fibroblast motility and extracellular matrix remodeling enhanced cancer cell inv

179 ulation in lysyl oxidase (LOX) expression,an extracellular matrix remodeling enzyme, in a highly inva

180 t cg25354716, annotated to CRTAP, a critical extracellular matrix remodeling enzyme.

181 othelial cells, with decreased expression of extracellular matrix remodeling-enzyme coding genes and

182 aled suppression of contractile SMC markers, extracellular matrix remodeling enzymes, and cytokines/r

183 ased lactate secretion, enhanced activity of extracellular matrix remodeling enzymes, and impaired cl

184 te to angiogenesis, metabolic reprogramming, extracellular matrix remodeling, epithelial-mesenchymal

185 pathways, principally cell-cycle regulation, extracellular matrix remodeling, epithelial-to-mesenchym

186 cal injury processes of the immune response, extracellular matrix remodeling, epithelial-to-mesenchym

187 metalloproteinases (MMPs) play a key role in extracellular matrix remodeling events associated with h

188 ersatile MT-MMP playing an important role in extracellular matrix remodeling events in the brain and

189 during acute RSV infection could potentiate extracellular matrix remodeling, facilitating conditions

190 ironment and secrete angiogenic, growth, and extracellular matrix remodeling factors, cytokines and c

191 nd extracellular pH in cancer contributes to extracellular matrix remodeling, favors cell migration,

192 itors of metalloproteinases (TIMPs) modulate extracellular matrix remodeling for maintaining homeosta

193 luded elevated inflammation and deteriorated extracellular matrix remodeling function.

194 tokines, yet their role in the regulation of extracellular matrix remodeling genes remains largely un

195 Interferon (IFN)-stimulated genes (ISGs) and extracellular matrix remodeling genes were suppressed, t

196 Micu2(-/-) mice had increased expression of extracellular matrix remodeling genes, while single-cell

197 regulation of SIK1 induces the expression of extracellular matrix remodeling genes.

198 arrest; but, it induced transcription of the extracellular matrix-remodeling genes MMP1 and PAI2, whi

199 Focal brain ischemia induces inflammation, extracellular matrix remodeling, gliosis, and neovascula

200 motility, epithelial-mesenchymal transition, extracellular matrix remodeling, glucose and lipid metab

201 between peripheral and central inflammation, extracellular matrix remodeling, hippocampal development

202 ne metabolism, cell adhesion and chemotaxis, extracellular matrix remodeling, hypoxia and oxidative s

203 We associate this with reduced extracellular matrix remodeling, immune cell infiltratio

204 omic footprints related to proliferation and extracellular matrix remodeling, immune responses, and m

205 in multiple aspects of the disease including extracellular matrix remodeling, immunomodulation, neo-a

206 stasis, bioenergetic dysregulation, aberrant extracellular matrix remodeling, impaired satellite cell

207 immune responses, prohormone processing, and extracellular matrix remodeling important to bone develo

208 i by proinflammatory molecules may influence extracellular matrix remodeling in atherosclerosis by re

209 ental processes, valvular cell behavior, and extracellular matrix remodeling in congenital and acquir

210 ipocyte-derived fatty acids prevent fibrotic extracellular matrix remodeling in fibroblasts during th

211 GF-beta immunoreactivity in conjunction with extracellular matrix remodeling in gamma-irradiated mous

212 as well as myofibroblast differentiation and extracellular matrix remodeling in obstructed kidneys, t

213 Proteomic profiling reveals extracellular matrix remodeling in old mice, including i

214 d on previous studies implicating changes in extracellular matrix remodeling in other, related optic

215 tion in TM cells and offer new insights into extracellular matrix remodeling in the aqueous outflow p

216 t be caused by biophysical processes such as extracellular matrix remodeling in the case of mesenchym

217 ing, respectively, suggesting a new role for extracellular matrix remodeling in the establishment of

218 Overall, our findings identify a role for extracellular matrix remodeling in the functional modula

219 Ps) is responsible for multiple processes of extracellular matrix remodeling in the healthy body but

220 dentified Sept4 as an important regulator of extracellular matrix remodeling in the heart.

221 y be a quantitative biomarker of subclinical extracellular matrix remodeling in the lungs of communit

222 g of the ocular globe and is associated with extracellular matrix remodeling in the posterior sclera.

223 trated the role of PMN-MDSCs in collagen and extracellular matrix remodeling in the premetastatic com

224 Extracellular matrix remodeling in the pulmonary arteria

225 g caveolin 1 (Cav1WT) demonstrated increased extracellular matrix remodeling in vitro compared to Cav

226 suggest that changes in a key determinant of extracellular matrix remodeling, in addition to changes

227 ll invasion by producing factors involved in extracellular matrix remodeling, increasing T cell recru

228 Additionally, extracellular matrix remodeling, inflammation, cytotoxic

229 sponses to insults such as oxidative stress, extracellular matrix remodeling, inflammation, or cell d

230 ic and failing hearts that are indicative of extracellular matrix remodeling, insulin resistance and

231 Extracellular matrix remodeling involves the complex int

232 of decellularized extracts indicated partial extracellular matrix remodeling involving enhanced secre

233 d gene expression in the heart that included extracellular matrix remodeling, ion handling, and metab

234 The fibrotic extracellular matrix remodeling is mediated by matrix me

235 sregulated reparative processes and abnormal extracellular matrix remodeling leading to cartilage deg

236 erative bronchiolitis: neutrophil influx and extracellular matrix remodeling leading to luminal obstr

237 P signaling promotes hypertrophic growth and extracellular matrix remodeling, leading to the developm

238 Atrial extracellular matrix remodeling manifested by the select

239 xtracellular matrix components, mediators of extracellular matrix remodeling, matricellular proteins,

240 f alphaSMA and upregulated the expression of extracellular matrix-remodeling matrix metalloproteinase

241 Thus, targeting extracellular matrix remodeling may present a therapeuti

242 udying pathways involving growth factors and extracellular matrix remodeling may yield additional ins

243 Collectively, these data uncover a novel extracellular matrix remodeling mechanism required for p

244 tent and expression of genes associated with extracellular matrix remodeling, mitochondrial energy me

245 rdant pathways between COPD and IPF included extracellular matrix remodeling, Mitogen-activated prote

246 -C); 2) inflammation (interleukin-1beta); 3) extracellular matrix remodeling (MMP1, MMP3, TIMP1, uPA,

247 nvolved in inflammation (Il1b, Il6, Tnf) and extracellular matrix remodeling (Mmp13, Timp1).

248 VEGF), inflammatory response (IL-1beta), and extracellular matrix remodeling (MMP3 and 9) were measur

249 t the potential modulation of cell adhesion, extracellular matrix remodeling, motility, metabolism, s

250 that induces tissue alert, characterized by extracellular matrix remodeling, myeloid cell infiltrati

251 cells revealed a gene signature dominated by extracellular matrix remodeling, notably affecting STMN3

252 fined cell culture system recapitulating the extracellular matrix remodeling observed in vivo, we sho

253 Extracellular matrix remodeling occurs during developmen

254 Significant extracellular matrix remodeling occurs in the posterior

255 otential therapeutic approach to prevent the extracellular matrix remodeling of adipose tissue in typ

256 tion of certain anti-glaucoma drugs involves extracellular matrix remodeling of ocular ciliary muscle

257 be required for all MMP functions including extracellular matrix remodeling of the tracheal system,

258 with a focus on angiogenesis, inflammation, extracellular matrix remodeling, osteoporosis, sarcopeni

259 ation in transforming growth factor-beta and extracellular matrix remodeling (periostin) pathways.

260 Pancreas TRMs had an extracellular matrix remodeling phenotype that was impor

261 to a unique stem cell marker gene-positive, extracellular matrix-remodeling, "pioneer" cell phenotyp

262 Cardiomyocyte hypertrophy and extracellular matrix remodeling, primarily mediated by i

263 both a cell bound and soluble proteinase for extracellular matrix remodeling processes.

264 enzymes is comprised of critically important extracellular matrix remodeling proteases whose activity

265 Some extracellular matrix remodeling proteins showed a higher

266 ic glomeruli revealed that proliferation and extracellular matrix remodeling represented the first st

267 myotubes and lowered Young's modulus through extracellular matrix remodeling, resulting in 43% strong

268 (TAAs) develop secondary to abnormal aortic extracellular matrix remodeling, resulting in a weakened

269 rian cancer tumorigenesis and metastasis via extracellular matrix remodeling, revealing a role for pr

270 ardiomyocyte survival, debris clearance, and extracellular matrix remodeling/scar resolution without

271 tures that imply functional tasks, including extracellular matrix remodeling, stress response, and sh

272 ring cell necrosis and molecules involved in extracellular matrix remodeling such as hyaluronan, bigl

273 e, we identify metabolic enzymes involved in extracellular matrix remodeling that are upregulated dur

274 -MMP as a master regulator of the pathologic extracellular matrix remodeling that characterizes rheum

275 may be a marker for an initial phase of the extracellular matrix remodeling that occurs during the d

276 These findings reveal mechanisms for extracellular matrix remodeling that promote heart regen

277 several disease markers for hypertrophy and extracellular matrix remodeling that were upregulated in

278 In addition to the well-recognized role in extracellular matrix remodeling, the tissue inhibitor of

279 nase (MT-MMPs) family are dual regulators of extracellular matrix remodeling through direct degradati

280 aling within the adventitial layer, and ECM (extracellular matrix) remodeling throughout the vein wal

281 of numerous genes including those regulating extracellular matrix remodeling (Timp3, Adamts9) and cel

282 ndrogenesis, and is a key regulator coupling extracellular matrix remodeling to angiogenesis at the g

283 f RUNX transcription factors, which mediates extracellular matrix remodeling to promote metastasis an

284 njectable biomaterials can induce beneficial extracellular matrix remodeling to stimulate tissue repa

285 omes release signaling molecules that induce extracellular matrix remodeling to support long-lasting

286 pithelial cancers and has been implicated in extracellular matrix remodeling, tumor growth, and metas

287 teinase 1 (MT1-MMP) plays a critical role in extracellular matrix remodeling under both physiological

288 al a novel mechanism of myocyte-orchestrated extracellular matrix remodeling via PDE5/cyclic guanosin

289 nhibitor of matrix metalloproteinase-1; this extracellular matrix remodeling was also significantly a

290 Extracellular matrix remodeling was cell-type specific,

291 Extracellular matrix remodeling was defined by detection

292 An extensive extracellular matrix remodeling was supported by changes

293 th factor and cytokine processing as well as extracellular matrix remodeling, we evaluated the role o

294 and those involved with immune function and extracellular matrix remodeling were upregulated.

295 nstitutes a unique mechanism, independent of extracellular matrix remodeling, whereby a proteolytical

296 can result in pathological events, involving extracellular matrix remodeling, which can facilitate ca

297 osage influenced gene expression involved in extracellular matrix remodeling, which is critical for a

298 Canonical MED19 triggered genes related to extracellular matrix remodeling while suppressing those

299 o the greatest extent were those involved in extracellular matrix remodeling with both UVA1 (P=5.5e-7