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1 ransfusion, sickle cell disease, sepsis, and extracorporeal circulation.
2 n which coronary perfusion was controlled by extracorporeal circulation.
3 vehicle control immediately before starting extracorporeal circulation.
4 heir effects on the inflammatory response to extracorporeal circulation.
5 IL-10 (P=0.02) and peak IL-6 (P=0.02) after extracorporeal circulation.
6 eukin-10 (IL-10)] cytokines during and after extracorporeal circulation.
7 d at 5 time points before, during, and after extracorporeal circulation.
8 systemic heparinization in a rabbit model of extracorporeal circulation.
9 en cannula which was connected to an HRD via extracorporeal circulation.
10 with an exaggerated inflammatory response to extracorporeal circulation.
11 rombin formation or activity during in vitro extracorporeal circulation.
12 ombin formation and activity during in vitro extracorporeal circulation.
13 nosed in 2012 in 2 heart surgery patients on extracorporeal circulation.
14 L-10 rose significantly after institution of extracorporeal circulation and remained elevated through
15 ragile infants, extended the capabilities of extracorporeal circulation, and have brought new and inn
16 thrombosis, to inhibit contact activation in extracorporeal circulation, and to treat the swelling di
17 -hour hemodialysis session, during which the extracorporeal circulation blood flow was maintained bet
20 10) or an oscillation group (anesthesia and extracorporeal circulation for 20 hr with artificial PaO
21 andomized to a control group (anesthesia and extracorporeal circulation for 20 hr with constant PaO2,
22 to the abdominal organs after death using an extracorporeal circulation for a limited period before o
23 n reducing fibrinolysis and blood loss after extracorporeal circulation; however, the effects of anti
24 e care unit) after cardiac surgery involving extracorporeal circulation in a series of 187 consecutiv
27 moral artery and vein were cannulated and an extracorporeal circulation loop with 2 ultrasonic bubble
29 re closely mimics the in vivo situation, but extracorporeal circulation may alter the physiologic int
30 as an index of the inflammatory response to extracorporeal circulation, may be useful in identifying
34 nts, these animals were placed on venovenous extracorporeal circulation through a 1-m control circuit
35 d and left untreated for 8 mins, after which extracorporeal circulation was started and its flow adju
36 ergoing coronary artery bypass grafting with extracorporeal circulation were randomly assigned in a d