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1 ibro-obliteration of the intrahepatic and/or extrahepatic bile ducts.
2 sulting in dramatic and rapid enlargement of extrahepatic bile ducts.
3 pithelial malignant neoplasm of the liver or extrahepatic bile ducts.
4 mechanistically linked to obstruction of the extrahepatic bile ducts.
5 he inflammatory and fibrosing obstruction of extrahepatic bile ducts.
6 ter than 90% mortality due to obstruction of extrahepatic bile ducts.
7 en benign and malign lesions in intraluminal extrahepatic bile ducts.
8 by age at surgery and anatomy of the atretic extrahepatic bile ducts.
9 flammation and fibrosis of the intra- and/or extrahepatic bile ducts.
10 Sar among blood, hepatocytes, and intra- and extrahepatic bile ducts.
12 SC were managed with either resection of the extrahepatic bile ducts and long-term transhepatic stent
13 ancers, which arise from the intrahepatic or extrahepatic bile ducts and the gallbladder, generally h
14 gallbladder, 588 intrahepatic bile duct, 753 extrahepatic bile duct, and 458 ampulla of Vater cancer
15 h biliary tract cancer (237 gallbladder, 127 extrahepatic bile duct, and 47 ampulla of Vater), 895 wi
17 However, no associations were observed for extrahepatic bile duct cancer (EHBDC) or ampulla of Vate
18 gkin lymphoma (P < .001) and gallbladder and extrahepatic bile duct cancer (P = .01) was observed.
19 eased risk of gallbladder, intrahepatic, and extrahepatic bile duct cancer compared with the general
20 higher: 100% for gallbladder cancer, 97% of extrahepatic bile duct cancer, 91% of ampula of Vater ca
22 the body and tail of pancreas, cancer of the extrahepatic bile duct, cancer of the gallbladder, and c
23 d cholangiocytes, that line intrahepatic and extrahepatic bile ducts, contribute substantially to bil
27 mouse has revealed that gallbladder (GB) and extrahepatic bile duct (EHBD) development is exquisitely
29 derived from ductal tissue located outside (extrahepatic bile ducts; EHBDs) or inside the liver (int
30 of birth, microdissected the gallbladder and extrahepatic bile ducts en bloc 3, 7, and 14 days later,
31 and active caspase-3 staining in livers and extrahepatic bile ducts from Balb/c mice infected with R
32 breast, colorectum, endometrium, oesophagus, extrahepatic bile duct, gallbladder, head and neck, kidn
34 epatic bile duct in 25 (100%), the recipient extrahepatic bile duct in 17 of 18 (94%), and the anasto
35 c bile ducts in 23 (92%) patients, the donor extrahepatic bile duct in 25 (100%), the recipient extra
36 m an association between age and size of the extrahepatic bile duct in an asymptomatic adult populati
40 eriductular fibrosis of the intrahepatic and extrahepatic bile ducts leading to strictures, bacterial
41 inflammatory process that affects intra- and extrahepatic bile ducts, leading to fibrosis and obliter
42 tal stents compared with patients with other extrahepatic bile duct malignant diseases and patients t
43 cholangiocarcinoma (n = 11) or by secondary extrahepatic bile duct malignant tumors (n = 11) were tr
44 ary atresia is a neonatal liver disease with extrahepatic bile duct obstruction and progressive liver
45 ered or partially covered) for palliation of extrahepatic bile duct obstruction initially is more exp
46 related to total bilirubin, indicating that extrahepatic bile duct obstruction leads to down-regulat
48 primed CD8(+) cells preferentially homed to extrahepatic bile ducts of neonatal mice and invaded the
49 role of T lymphocytes in the destruction of extrahepatic bile ducts of neonatal mice using an experi
52 f CD4(+) and CD8(+) T cells within liver and extrahepatic bile duct remnant tissues, indicating the p
53 Vbeta repertoire of T cells from the liver, extrahepatic bile duct remnants, and peripheral blood of
56 progressive fibroinflammatory obstruction of extrahepatic bile ducts that presents as neonatal choles
57 truction of segments or the entire length of extrahepatic bile ducts, the timely pursuit of hepatopor
58 sverse and anteroposterior dimensions of the extrahepatic bile duct were measured proximally at the p
60 epithelial cells lining the intrahepatic and extrahepatic bile ducts, which are central to the pathog
61 gs, spleen, liver, lymph nodes, pancreas and extrahepatic bile duct with potential for recurrence and
62 from biliary epithelial cells of intra- and extrahepatic bile ducts with dismal prognosis and few no
63 epithelial and subepithelial compartments of extrahepatic bile ducts, with onset within 3 days and pe