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1 s and specimen source category (pulmonary vs extrapulmonary).
2 ight atrium versus 12 in the left atrium; 15 extrapulmonary).
3  and 3071 (9.6%) were combined pulmonary and extrapulmonary.
4 losis complex; 29 were pulmonary, and 8 were extrapulmonary.
5      Comorbid conditions can be pulmonary or extrapulmonary.
6 t of pulmonary abnormalities, as well as for extrapulmonary abnormalities.
7 eolar collapse were similar in pulmonary and extrapulmonary acute lung injury groups.
8 ary acute lung injury) or intraperitoneally (extrapulmonary acute lung injury).
9 oneally (20 mg/kg in 0.5 mL of saline/mouse, extrapulmonary acute lung injury).
10                                           In extrapulmonary acute lung injury, CPAP-30 and STEP-30/30
11                                Compared with extrapulmonary acute lung injury, mesenchymal stem cell
12 and STEP-30/30 yielded endothelial injury in extrapulmonary acute lung injury.
13 l, and remodeling properties in pulmonary or extrapulmonary acute lung injury.
14 ficantly higher than PEEPPL in subjects with extrapulmonary acute respiratory distress syndrome (p =
15  respiratory distress syndrome compared with extrapulmonary acute respiratory distress syndrome but p
16 led ventilation and variable ventilation; in extrapulmonary acute respiratory distress syndrome, howe
17 ress syndrome, n = 12) or intraperitoneally (extrapulmonary acute respiratory distress syndrome, n =
18                                           In extrapulmonary acute respiratory distress syndrome, only
19  preserved E-cadherin in lung tissue only in extrapulmonary acute respiratory distress syndrome, thus
20 te respiratory distress syndrome, but not in extrapulmonary acute respiratory distress syndrome, vari
21 ry distress syndrome donors on pulmonary and extrapulmonary acute respiratory distress syndrome.
22 ty-seven and 84 patients had a pulmonary and extrapulmonary acute respiratory distress syndrome.
23  higher physiologic dead space compared with extrapulmonary acute respiratory distress syndrome.
24 al ventilation in experimental pulmonary and extrapulmonary acute respiratory distress syndrome.
25 nary acute respiratory distress syndrome and extrapulmonary acute respiratory distress syndrome.
26 ity is regulated by subsets of pulmonary and extrapulmonary afferent nerve fibers, which continuously
27 nglion neurones project C-fibres to both the extrapulmonary airways (larynx, trachea and bronchus) an
28 ion to basal cells, specifying cells for the extrapulmonary airways.
29    COPD is a heterogeneous disease with both extrapulmonary and pulmonary components.
30 Intrapatient diversity was found both at the extrapulmonary and respiratory sites, meaning that this
31 k in nonambulatory patients or in those with extrapulmonary and smear-negative tuberculosis.
32 there were 36 cases of pulmonary, 9 cases of extrapulmonary, and 1 case of central nervous system (CN
33 ely pulmonary, 5085 (15.9%) were exclusively extrapulmonary, and 3071 (9.6%) were combined pulmonary
34 aminophen diminished NPSH in nasal, thoracic extrapulmonary, and lung tissues; it also induced the ox
35 4.2]) or disease that was both pulmonary and extrapulmonary (aPR, 2.4 [95% CI, 2.1-2.9]) were associa
36 cts with direct (pulmonary) versus indirect (extrapulmonary) ARDS.Conclusions: Clinical outcomes in A
37 ed bacterial burdens in the lungs, increased extrapulmonary bacterial dissemination, and more severe
38 uced tissue necrosis, fibrosis and decreased extrapulmonary bacterial dissemination.
39  samples collected post mortem from lung and extrapulmonary biopsies of 44 subjects in KwaZulu-Natal,
40    Mechanism of pulmonary immunosuppression: extrapulmonary burn injury suppresses bacterial endotoxi
41 lone on day 3 in patients with infectious or extrapulmonary causes of acute respiratory distress synd
42 1) but not in patients with noninfectious or extrapulmonary causes of acute respiratory distress synd
43 ess syndrome but only at day 3 in those with extrapulmonary causes of acute respiratory distress synd
44    Additionally, the impact of pulmonary and extrapulmonary comorbidities on the ability to meet remi
45 dominal pressure was significantly higher in extrapulmonary compared with pulmonary acute respiratory
46       These syndromes have serious potential extrapulmonary complications (e.g., malignant renal tumo
47 tcome was a composite of major pulmonary and extrapulmonary complications occurring within the first
48 h chronic conditions, such as arthritis, and extrapulmonary complications, such as encephalitis.
49  administered, to a variety of pulmonary and extrapulmonary complications.
50 onary control) or intraperitoneally (0.5 mL, extrapulmonary control).
51 , but not of neutrophils, abrogated lung and extrapulmonary CRKP clearance in ablated mice, suggestin
52 arcoma, esophageal candidiasis, CMV disease, extrapulmonary cryptococcosis, toxoplasmic encephalitis,
53  of tuberculosis cases that were exclusively extrapulmonary differed by lineage: East Asian, 13.0%; E
54                    Forty of the patients had extrapulmonary disease (17 had disseminated disease, 11
55 us infection (aPR, 1.43; 95% CI, 1.15-1.77), extrapulmonary disease (aPR, 3.02; 95% CI, 2.60-3.52), a
56                                  Exclusively extrapulmonary disease (aPR, 3.7 [95% CI, 3.3-4.2]) or d
57 retroviral therapy), as well as treatment of extrapulmonary disease (central nervous system, pericard
58 roportionably affected by smear-negative and extrapulmonary disease and who are also most adversely a
59 ibraries with pooled sera from patients with extrapulmonary disease and with sera from patients with
60 nchioloalveolar carcinoma and no evidence of extrapulmonary disease received transplants of either on
61                  Subacute, hematogenous, and extrapulmonary disease tends to be more frequent in infa
62                            Culture negative, extrapulmonary disease was more common with TB diagnosed
63 ive human immunodeficiency virus status, and extrapulmonary disease were also significantly associate
64  pulmonary and constitutional symptoms, more extrapulmonary disease, and fewer cavitary lesions on ch
65 e to induce pathological lesions or to cause extrapulmonary disease, despite retaining its ability to
66 yte for active TB detection in pediatric and extrapulmonary disease.
67 lts or children presenting with pulmonary or extrapulmonary disease.
68 ectrum of dissemination was noted, including extrapulmonary disease.
69 001 and 30 May 2006, including 59 (45%) with extrapulmonary disease.
70 developed pulmonary TB, either alone or with extrapulmonary disease.
71 vis, which was independently associated with extrapulmonary disease.
72                                   While many extrapulmonary diseases are now linked to abnormalities
73         Cfb(-/-) mice demonstrated increased extrapulmonary dissemination and serum inflammatory mark
74 l infection, the organization of granulomas, extrapulmonary dissemination and vaccine-induced protect
75                                              Extrapulmonary dissemination appeared to be a function o
76                                              Extrapulmonary dissemination can lead to infection of th
77  CatB was dispensable for lung infection and extrapulmonary dissemination in vivo.
78                    It has been implicated in extrapulmonary dissemination of M. tuberculosis from the
79                                              Extrapulmonary dissemination of mycoplasmas with coloniz
80 ntrapulmonary L. pneumophila infections with extrapulmonary dissemination of the bacteria to the sple
81                                              Extrapulmonary dissemination resulted in acquisition of
82                                              Extrapulmonary dissemination was almost exclusively note
83 . pneumoniae was inhibited by lovastatin and extrapulmonary dissemination was enhanced, both reversib
84 imals showed reduced pulmonary pathology and extrapulmonary dissemination, and protection correlated
85 f progression to disease, various extents of extrapulmonary dissemination, and various degrees of cav
86                         While not preventing extrapulmonary dissemination, the coadministered T cells
87 ction can result in eradication, latency, or extrapulmonary dissemination.
88 nterfere with cryptococcal neurotropism upon extrapulmonary dissemination.
89 nimal model to study cavity pathogenesis and extrapulmonary dissemination.
90 n treating clinicians for the possibility of extrapulmonary dissemination.
91  Klebsiella pneumoniae (KP) and assessed for extrapulmonary dissemination.Measurements and Main Resul
92 that the most likely candidate transducer of extrapulmonary effects of iNO was nitrite.
93 ncreasing epidemiological evidence points to extrapulmonary effects, including positive associations
94 the dissemination of P. carinii organisms in extrapulmonary (EP) tissues.
95 jects with latent (LTBI), pulmonary (PTB) or extrapulmonary (EPTB) tuberculosis remains unclear.
96                                     Elevated extrapulmonary extracellular matrix turnover was associa
97                        Finally, a variety of extrapulmonary factors may impact upon airway disease, i
98  (QS/QT), assessing the impact of intra- and extrapulmonary factors on the relationship and thus the
99 airways and surrounding lung parenchyma, and extrapulmonary factors.
100                        Background Incidental extrapulmonary findings are commonly detected on chest C
101                       Important quantifiable extrapulmonary findings include coronary artery calcific
102 ress syndrome patients, the pulmonary versus extrapulmonary form in terms of respiratory mechanics, l
103 irect or indirect lung insult (pulmonary and extrapulmonary form).
104 lymph nodes represent reactive adenitis, and extrapulmonary forms of tuberculosis (including lymphati
105                   Conclusions: We identified extrapulmonary galectin-3 as an important mediator that
106 hus imparts opposing effects upon intra- and extrapulmonary host defense by inducing tissue-divergent
107 is serum to infected C3H-SCID mice prevented extrapulmonary infection and disease, while the severity
108             Concern over early postoperative extrapulmonary infection may suggest a benefit of initia
109 ho develop chronic coccidioidal pneumonia or extrapulmonary infection often have complicated courses
110 iliense had Clone 1, including pulmonary and extrapulmonary infections, compared to 11/64 (17%) in th
111 side the airways and lungs may contribute to extrapulmonary inflammatory complications in subsets of
112  ACE2 in as many as 72 tissues suggests that extrapulmonary invasion and damage is likely, which inde
113 r HIV status, cases were more likely to have extrapulmonary involvement (47.6% versus 11.6%, p = 0.05
114 vs 144 microL), and were more likely to have extrapulmonary involvement (75% vs 18%, p=0.03) and conc
115  correlated with severity of lung disease or extrapulmonary involvement and was found in one patient
116                                     Overall, extrapulmonary involvement was a risk factor for shorter
117 ng transplantation (LT) due to concerns that extrapulmonary involvement will yield worse outcomes.
118                       A group with prominent extrapulmonary involvement, and low serum MIF, IL-10 and
119                                              Extrapulmonary loci of virus replication seem possible.
120  tract even after a nasal challenge, whereas extrapulmonary lymphoid responses enhanced responses in
121 te-like ability of CD4(+) T cells to contain extrapulmonary M. tuberculosis dissemination at very ear
122 eted macaques surprisingly led to very early extrapulmonary M. tuberculosis dissemination, whereas CD
123 he electronic charts of 149 patients with an extrapulmonary malignant neoplasm and a solitary pulmona
124             Acute cardiac injury is a common extrapulmonary manifestation of COVID-19 with potential
125                        Diarrhea was the only extrapulmonary manifestation.
126 s but also encourages clinicians to consider extrapulmonary manifestations in their management of pat
127                                              Extrapulmonary manifestations included hepatic, kidney,
128 lated pathways might all contribute to these extrapulmonary manifestations of COVID-19.
129 n-ICU setting without organ failure or major extrapulmonary manifestations of COVID-19.
130                      Most patients (92%) had extrapulmonary manifestations, including shock, acute ki
131 symptoms, comorbid conditions, pulmonary and extrapulmonary manifestations, measures of severity of i
132 es in propagating pulmonary fibrosis and the extrapulmonary manifestations, which uniquely characteri
133 age lung disease due to SSc who have limited extrapulmonary manifestations.
134 athologies, including asthma, and a range of extrapulmonary manifestations.
135 ory pathology, it can also result in several extrapulmonary manifestations.
136                               No evidence of extrapulmonary MERS-CoV antigens were detected, includin
137 o intervene in a wide range of pulmonary and extrapulmonary metastatic disease.
138 fied culture positive pediatric (N = 19) and extrapulmonary (N = 24) patients with high accuracy (ROA
139 NA), respectively, in pulmonary (n = 18) and extrapulmonary (n = 4) lesions from 22 women with LAM (1
140 ted on whether tuberculosis was pulmonary or extrapulmonary (n=57 421).
141 nds on the histologic characteristics of the extrapulmonary neoplasm and the patient's smoking histor
142 the nodule were correlated with those of the extrapulmonary neoplasm and with patient age and smoking
143 rimary lung tumours and lung metastasis from extrapulmonary neoplasms by contributing to inflammation
144 ults with otherwise unexplained disseminated/extrapulmonary Nocardia infections, anti-GM-CSF autoanti
145       Plasma from patients with disseminated/extrapulmonary nocardiosis and healthy controls was scre
146 ted with mediastinal adenopathy or suspected extrapulmonary nonnodal metastases (P <.05).
147 ered because of this outbreak include adding extrapulmonary NTM to ADH's reportable disease list and
148  delineated the molecular characteristics of extrapulmonary NTM.
149 veral important sequelae (both pulmonary and extrapulmonary) occur in these adult patients including
150 poxemic respiratory failure and considerable extrapulmonary organ dysfunction and is associated with
151 tionship between fibrogenesis, pulmonary and extrapulmonary organ dysfunction, and outcome during the
152 requency of invasive mechanical ventilation, extrapulmonary organ dysfunction, and substantial in-hos
153 re (< 29, 29-30, > 30 cm H2O), and number of extrapulmonary organ failure (< 2, 2, > 2) stratified ac
154 disease progression, including pulmonary and extrapulmonary organ failure and death.
155 ient's age, PaO2/FIO2, plateau pressure, and extrapulmonary organ failure provides prognostic enrichm
156 , PaO2/FIO2, plateau pressure, and number of extrapulmonary organ failures to predict ICU outcome at
157          Lower tidal volumes (Vts) attenuate extrapulmonary organ injury in other disease states and
158 ycobacterial responses in both pulmonary and extrapulmonary organ systems of mice, which may be impor
159  post-acute sequelae involving pulmonary and extrapulmonary organ systems-referred to as long COVID.
160 creased mycobacterial loads in pulmonary and extrapulmonary organ systems.
161                           Here we review the extrapulmonary organ-specific pathophysiology, presentat
162 y improve lung function but could jeopardize extrapulmonary-organ perfusion.
163 rculosis infection burdens in lung lobes and extrapulmonary organs than did the control groups receiv
164 e, increased L. pneumophila dissemination to extrapulmonary organs, and impaired glucose homeostasis.
165 he respiratory tract, virus dissemination to extrapulmonary organs, lymphopenia, significantly elevat
166 also isolated from the brains, but not other extrapulmonary organs, of infected animals.
167 lity to control M. tuberculosis infection in extrapulmonary organs.
168 ith the ability of VN/1203 to disseminate to extrapulmonary organs.
169 induce damage to 28S rRNA in kidney or other extrapulmonary organs.
170 s is assumed to be intrapulmonary, but their extrapulmonary origin and especially derivation from bon
171 spiratory disorders, and are associated with extrapulmonary pathologies.
172  a range of airway inflammation and lung and extrapulmonary pathologies.
173 survival interval, more severe pulmonary and extrapulmonary pathology, and a higher bacterial burden
174  nonspecific immune response and the rate of extrapulmonary phagocytosis.
175            Spoligotyping of 54 pulmonary and extrapulmonary positive tissues from 30 subjects showed
176 nd normal lungs to predicted full inflation (extrapulmonary restriction); or (2) increased capillary
177 onary vein isolation plus linear ablation of extrapulmonary rotor domains.
178 sputum samples, blood cultures, and relevant extrapulmonary samples.
179                                Children with extrapulmonary sepsis with acute respiratory distress sy
180           We hypothesized that children with extrapulmonary sepsis with versus without acute respirat
181 ges, and it can also result in other serious extrapulmonary sequelae.
182                                Patients with extrapulmonary shunting, clear chest radiographs, and ad
183 ar and 10-year follow-up and the presence of extrapulmonary significant incidental findings were pred
184 ance of the models for prediction of ACM and extrapulmonary significant incidental findings.
185  patients and was frequently associated with extrapulmonary sites of infection.
186 elease platelets in the lungs originate from extrapulmonary sites such as the bone marrow; we observe
187  by Pneumocystis in the lung and possibly at extrapulmonary sites via circulating fungal components.
188 dicated that dissemination from the lungs to extrapulmonary sites was as frequent as between lung sit
189 ine-treated animals, and tumor recurrence in extrapulmonary sites was seen only in the cyclosporine-t
190 nd 10%-15% of cases of reactivation occur at extrapulmonary sites without active pulmonary tuberculos
191 asmal colonization of spleens and lesions in extrapulmonary sites, particularly spleens, hearts, and
192 d correlates with the spread of the virus to extrapulmonary sites.
193 le defect of 2E-TU-4 was in dissemination to extrapulmonary sites.
194 c Ag-specific immunity or chemoattraction at extrapulmonary sites.
195  reducing the number of yeast in the lung or extrapulmonary sites.
196 ective T cell immunity in both the lungs and extrapulmonary sites.
197 f all ages and can affect both pulmonary and extrapulmonary sites.
198                                              Extrapulmonary (skin or bone) disease, probably resultin
199 rodromal respiratory symptoms and fever, and extrapulmonary (skin) manifestations.
200 rodromal respiratory symptoms and fever, and extrapulmonary (skin) manifestations.
201  small cell lung cancer (SCLC) cell lines, 3 extrapulmonary small cell cancer (ExPuSC) cell lines, an
202 ung cancers, NSCLC, four mesotheliomas, five extrapulmonary small cell cancers) were analysed for PTE
203  in one patient each with platinum-resistant extrapulmonary small-cell and fluoropyrimidine- and irin
204 e human lung might be similarly derived from extrapulmonary sources, we examined lung specimens from
205 eater proportion of pulmonary specimens than extrapulmonary specimens (IPC C(T) > 34: 6% (47/731) ver
206 h C(T) values in pulmonary specimens but not extrapulmonary specimens (Spearman's coefficient 0.5043
207    Pulmonary specimens had greater load than extrapulmonary specimens [TTPs (interquartile range) of
208 n Southeast Asia and collected pulmonary and extrapulmonary specimens to evaluate the prevalence of m
209 3% of the pulmonary specimens and 50% of the extrapulmonary specimens were smear positive.
210  specimens was 60% for pulmonary and 75% for extrapulmonary specimens, while the IS6110 LDT sensitivi
211 sis isolates in smear-positive pulmonary and extrapulmonary specimens, while the sensitivity of the d
212  MTB/RIF C(T) is a poor surrogate of load in extrapulmonary specimens.
213 ary specimens but an increased likelihood in extrapulmonary specimens.
214 nts, including 89 pulmonary specimens and 23 extrapulmonary specimens.
215                                  Viremia and extrapulmonary spread of SARS CoV to liver and spleen, w
216                                              Extrapulmonary spread was not observed in either mice or
217 clusion A fully automated AI model indicated extrapulmonary structures at risk on chest CT scans and
218 orbidity, mainly from pulmonary involvement, extrapulmonary symptoms are also major componen ts of th
219 s a wide range of airway diseases as well as extrapulmonary symptoms.
220      COVID-19 is associated with significant extrapulmonary symptoms.
221          Carefully selected patients without extrapulmonary systemic disease experience similar survi
222 F assay for pulmonary tuberculosis (PTB) and extrapulmonary TB (EPTB) has not been investigated in Ir
223                    Identifying patients with extrapulmonary TB (EPTB) remains challenging because mic
224 lture-positive pulmonary TB (PTB; 91.3%) and extrapulmonary TB (EPTB; 92.3%), and the sensitivities o
225 + cell counts and decreased the incidence of extrapulmonary TB and genital ulcers in HIV-negative pat
226 ng seasonality in clustered TB and clustered extrapulmonary TB cases but not in clustered pulmonary T
227 ificities for the diagnosis of pulmonary and extrapulmonary TB cases reported in these six studies ar
228 ealed barely significant seasonality only in extrapulmonary TB cases.
229 sion was increased 2.3-fold in patients with extrapulmonary TB compared with patients with purely pul
230  enables the detection of both pulmonary and extrapulmonary TB from easily accessible urine and/or bl
231 ntial proportion of patients presenting with extrapulmonary TB have urogenital TB (UG-TB), which can
232 lmonary TB in 52 persons, 26 observations of extrapulmonary TB in 25 persons, and 3 observations of C
233 agnosis of tuberculosis (TB) in children and extrapulmonary TB in adults continues to be a challenge.
234 f paucibacillary tuberculosis (TB) including extrapulmonary TB is a significant challenge, particular
235 y, diagnosis of smear-negative pulmonary and extrapulmonary TB remains challenging in such settings.
236 en aged 1 to 16 years detected pulmonary and extrapulmonary TB with high sensitivity versus culture a
237  had pulmonary TB, 5 (36%) had pulmonary and extrapulmonary TB, and 9/13 (69%) were smear positive.
238 d pulmonary TB, 5/14 (36%) had pulmonary and extrapulmonary TB, and 9/13 (69%) were smear-positive.
239 ose unable to produce sputum samples or with extrapulmonary TB, household contacts, and children-shou
240 al 18-24 month regimen) for patients without extrapulmonary TB, pregnancy, a previous second-line TB
241 ation of MODS into diagnostic algorithms for extrapulmonary TB.
242 study supports the clinical use of iNO as an extrapulmonary therapeutic to improve organ function fol
243 ced growth characteristics in eggs, extended extrapulmonary tissue tropism, and pathogenicity in mice
244 ture of the host response to Coccidioides in extrapulmonary tissue.
245 ontrast, G-CSF mRNA was not increased in the extrapulmonary tissues examined (liver, spleen, and kidn
246                          Direct infection of extrapulmonary tissues has been postulated, and using se
247  in cigarette smoke mediates CFTR defects in extrapulmonary tissues in smokers.
248 servations in humans support the notion that extrapulmonary tissues may be infected hematogenously by
249 th higher bacterial burdens in pulmonary and extrapulmonary tissues, development of more extensive hi
250 navirus SARS-CoV-2, is expressed in multiple extrapulmonary tissues, direct viral tissue damage is a
251 y involve the persistence of latent virus in extrapulmonary tissues, similar to what has been recentl
252 colysis) also underlies the abnormalities in extrapulmonary tissues, suggesting a global metabolic di
253 h increased cytokine and chemokine levels in extrapulmonary tissues.
254 oncentrations (80 ppm) inhibits IR injury in extrapulmonary tissues.
255  amount of the radioactivity was detected in extrapulmonary tissues.
256 crease in the fungal burden in pulmonary and extrapulmonary tissues.
257 lps contain pneumococcal burden in lungs and extrapulmonary tissues.
258 to the systemic circulation, suggesting that extrapulmonary toxicity may be caused indirectly by lung
259 ipants; aOR 0.81, 0.70-0.94) but not against extrapulmonary tuberculosis (106 [0.3%] in 40 318 vaccin
260 ited States, the proportion of patients with extrapulmonary tuberculosis (EPTB) has increased relativ
261 I, 1.79-341.1]; P = .02) and the presence of extrapulmonary tuberculosis (OR, 37.8 [95% CI, 2.78-513.
262 on according to recommendations provided for extrapulmonary tuberculosis and affected organs.
263 erculosis and pulmonary embolism, or between extrapulmonary tuberculosis and deep vein thrombosis.
264 were found significantly more likely to have extrapulmonary tuberculosis and negative tuberculin skin
265 is approach may be a useful method to detect extrapulmonary tuberculosis and the risk of death in imm
266  strain, it was isolated from a patient with extrapulmonary tuberculosis and vaccination with a subun
267 losis infection, pulmonary tuberculosis, and extrapulmonary tuberculosis are provided.
268 curacy of blood RNA biomarker signatures for extrapulmonary tuberculosis are scarce.
269 boy in North Dakota who was screened because extrapulmonary tuberculosis had been diagnosed in his fe
270 ew smear-negative samples from patients with extrapulmonary tuberculosis in our study, additional sim
271  (CNSTB) is the most severe manifestation of extrapulmonary tuberculosis infection, but the mechanism
272 ng people with kidney failure, 924 (32%) had extrapulmonary tuberculosis only, and nearly 40% died: 2
273 negative, pediatric and adult, pulmonary and extrapulmonary tuberculosis patients.
274  East Asian lineage, the odds of exclusively extrapulmonary tuberculosis relative to exclusively pulm
275  and correct diagnosis of both pulmonary and extrapulmonary tuberculosis remains a challenge because
276 ., San Diego, Calif.) for rapid diagnosis of extrapulmonary tuberculosis was evaluated by testing 178
277                                  The risk of extrapulmonary tuberculosis was higher among HIV-infecte
278 ren had pulmonary tuberculosis, 24 (41%) had extrapulmonary tuberculosis with or without pulmonary in
279 irs that can reactivate infection, producing extrapulmonary tuberculosis without lung involvement.
280 orld Health Organization stage 4 (other than extrapulmonary tuberculosis) and after 2 months of tuber
281 infection (eg, pulmonary tuberculosis versus extrapulmonary tuberculosis).
282         We excluded studies of patients with extrapulmonary tuberculosis, and studies in which mortal
283 erculosis is one of the most common forms of extrapulmonary tuberculosis, it is a disease entity that
284 l tuberculosis, smear-negative tuberculosis, extrapulmonary tuberculosis, multidrug-resistant tubercu
285 the endpoint review committee), pulmonary or extrapulmonary tuberculosis, or any bacterial infectious
286                                           In extrapulmonary tuberculosis, the most common site of inf
287            35 (45%) of these 78 patients had extrapulmonary tuberculosis.
288 s lymphadenitis is the main manifestation of extrapulmonary tuberculosis.
289 mphadenitis (TBL) is the most common form of extrapulmonary tuberculosis.
290 ective efficacy of BCG against pulmonary and extrapulmonary tuberculosis.
291 aediatric cohort, including all six cases of extrapulmonary tuberculosis.
292 , resulting in overt pulmonary, pleural, and extrapulmonary tuberculosis.
293                                              Extrapulmonary tumors (MCA-205 s.c. and intracranial) th
294 NEU1 and NEU3 proteins to both pulmonary and extrapulmonary vascular endothelia.
295          Together with pulmonary veins, many extrapulmonary vein areas may be the source of initiatio
296  arrhythmia with no pulmonary veins or other extrapulmonary vein site reconnection.
297 rus production and more severe pathology and extrapulmonary virus spread in chickens.
298 H9N2 virus with the G1-like M gene conferred extrapulmonary virus spread in chickens.
299 ia, with subsequent alveolar involvement and extrapulmonary virus spread to the brain.
300  but did not cause lethal disease or exhibit extrapulmonary virus spread.

 
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