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1 ding frontotemporal dementia, pyramidal, and extrapyramidal signs.
2 function, and three of them developed subtle extrapyramidal signs.
3 ough a subgroup developed moderate to severe extrapyramidal signs.
4 All subjects developed extrapyramidal signs.
5 duced, although there was a mild increase in extrapyramidal signs; 112 patients met the criteria for
6 ogeneous group of disorders with progressive extrapyramidal signs and neurological deterioration, cha
7 nce of myoclonus, seizures, pyramidal signs, extrapyramidal signs, and cerebellar signs) from all ind
8 ter psychiatric symptoms, cerebellar ataxia, extrapyramidal signs, and extensive calcifications on br
9 as well as the development of myoclonus and extrapyramidal signs are consistent manifestations of di
10 eimer's disease (AD) is often accompanied by extrapyramidal signs attributed to nigrostriatal dysfunc
11 ertension, diabetes mellitus, heart disease, extrapyramidal signs, depression, psychosis, aggression,
15 e of frontal lobe release signs, presence of extrapyramidal signs, gait disturbance, history of falls
16 typical cases, svPPA-tau showed significant extrapyramidal signs, greater executive impairment, and
21 disease associated with age and severity of extrapyramidal signs is related primarily to their combi
22 threads was increased in AD (with or without extrapyramidal signs) nigra compared to control tissue,
23 and combined effects of age and severity of extrapyramidal signs on the risk of incident dementia in
24 llitus, heart disease, incident strokes, and extrapyramidal signs, only conventional antipsychotic us
26 nts included abnormal involuntary movements, extrapyramidal signs, psychiatric symptoms, and medical
28 amination scores was slower, the presence of extrapyramidal signs was decreased, and the development