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1 isorders comprising cerebellar ataxia and/or extrapyramidal symptoms.
2 sedation, akathisia (for aripiprazole), and extrapyramidal symptoms.
3 dverse events at the injection site and more extrapyramidal symptoms.
4 performance and shows reduced liability for extrapyramidal symptoms.
5 toms without marked positive, depressive, or extrapyramidal symptoms.
6 oncentration correlated with the severity of extrapyramidal symptoms.
7 renia (PNS), and minimal positive/depressive/extrapyramidal symptoms.
8 rates of dysphoria, depressive symptoms, and extrapyramidal symptoms.
9 t is clinically effective with a low risk of extrapyramidal symptoms.
10 mptoms of schizophrenia; quality of life; or extrapyramidal symptoms.
11 impson-Angus Rating Scale was used to assess extrapyramidal symptoms.
12 onventional antipsychotic agents in terms of extrapyramidal symptoms.
13 e apparently unrelated subjects with similar extrapyramidal symptoms.
14 events and the use of medications related to extrapyramidal symptoms.
15 There was minimal risk of extrapyramidal symptoms.
16 e risperidone and olanzapine groups reported extrapyramidal symptoms (24% and 20%, respectively).
21 Safety and tolerability evaluations included extrapyramidal symptoms and effects on weight, prolactin
22 otic agents in producing significantly fewer extrapyramidal symptoms and having a lower risk of tardi
23 that has associated chronic cocaine use with extrapyramidal symptoms and striatal dopaminergic deplet
24 ated patients had a significant reduction in extrapyramidal symptoms and subjective measures of stiff
26 in the domains of symptoms, quality of life, extrapyramidal symptoms, and a synthetic measure of mult
27 ant even after lifetime medication exposure, extrapyramidal symptoms, and abnormal involuntary moveme
30 athy, clubfoot, absent deep tendon reflexes, extrapyramidal symptoms, and persistently deficient myel
32 ncluding weight gain, metabolic dysfunction, extrapyramidal symptoms, and tardive dyskinesia), especi
35 ol-treated patients experienced worsening of extrapyramidal symptoms, as indicated by several measure
36 nternational Neuropsychiatric Interview, and extrapyramidal symptom assessments indicating normal to
37 % of treated patients develop characteristic extrapyramidal symptoms caused by haloperidol-induced to
38 Safety was assessed using adverse events, Extrapyramidal Symptom (EPS) rating scales, laboratory v
39 or antipsychotic drugs which will not induce extrapyramidal symptoms (EPS) and tardive dyskinesias (T
40 ng there is a potential risk of exacerbating extrapyramidal symptoms (EPS) if H3R antagonists were us
41 regard to both the antipsychotic action and extrapyramidal symptoms (EPS) of antipsychotic drugs (AP
42 scular events, as well as metabolic effects, extrapyramidal symptoms, falls, cognitive worsening, car
43 th anti-NMDA receptor antibodies both showed extrapyramidal symptoms following initiation of treatmen
44 and is necessary to reduce the expression of extrapyramidal symptoms induced by chronic haloperidol t
45 Safety measures included adverse events, extrapyramidal symptoms, laboratory assessments, and sui
46 safe and effective antipsychotics devoid of extrapyramidal symptoms liability, sedation, and catalep
48 tolerated and associated with a low rate of extrapyramidal symptoms; neither weight gain nor clinica
49 H] = 87), stroke (NNH = 53 for risperidone), extrapyramidal symptoms (NNH = 10 for olanzapine; NNH =
50 se of atypical features, including exclusive extrapyramidal symptoms, normal eye movements, and norma
52 es carrying a single parkin mutation without extrapyramidal symptoms or signs also had psychiatric sy
54 ignificant cognitive impairment, increase in extrapyramidal symptoms, or central anticholinergic effe
57 lactic benztropine) in compliance, symptoms, extrapyramidal symptoms, or overall quality of life, and
58 Although olanzapine is associated with fewer extrapyramidal symptoms, other side effects may offset t
59 rrected QT-interval prolongation (p = 0.16), extrapyramidal symptoms (p = 0.31), excessive sedation (
61 to explain the therapeutic efficacy and low extrapyramidal symptom profile of atypical antipsychotic
63 elated to 1) change in negative symptoms, 2) extrapyramidal symptom profile, 3) effect on prolactin l
74 ter TTR (SMD=-0.27) and a lower incidence of extrapyramidal symptoms (RR=0.31, NNH=7) compared with h
75 hisia Scale, and Modified Simpson-Angus [for Extrapyramidal Symptoms] Scale) and electromechanical as
76 ssessed by recording adverse events and with extrapyramidal symptom scales and electrocardiograms at
77 luded spontaneously reported adverse events, extrapyramidal symptom scores, serum prolactin concentra
80 iapine was well tolerated and did not induce extrapyramidal symptoms, sustained elevations of prolact
81 experienced statistically significantly more extrapyramidal symptoms than haloperidol-treated multipl
83 at improvement in positive, negative, and/or extrapyramidal symptoms was associated with mood improve
86 hereas the incidence of QTc prolongation and extrapyramidal symptoms was similar between groups, more
87 ebo group (p = .60), and a global measure of extrapyramidal symptoms was similar between treatment gr
91 f somnolence, weight gain, restlessness, and extrapyramidal symptoms were similar in the two groups.
94 owever, haloperidol carries a higher rate of extrapyramidal symptoms, whereas olanzapine is associate