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1 nd undergo further differentiation into both extravillous and syncytiotrophoblast.
2  measured the general effects of LPS on both extravillous and villous trophoblast physiology, and the
3                              Using the human extravillous CTB cell line SGHPL-4, we examined the effe
4 d expression of stage-specific antigens that extravillous CTBs normally upregulate as they exit the p
5 s cytotrophoblast cells and chorion membrane extravillous cytotrophoblast cells contained mRNAs encod
6 NA and protein were expressed in villous and extravillous cytotrophoblast cells up to week 35 of gest
7 ost-mitotic syncytiotrophoblast and invasive extravillous cytotrophoblast cells.
8 eas m/sG2 protein was present exclusively in extravillous cytotrophoblast cells.
9 a disorder of pregnancy associated with poor extravillous cytotrophoblast invasion and above-normal r
10  placenta as well as syncytiotrophoblast and extravillous cytotrophoblast of normal third-trimester a
11                                     Invasive extravillous cytotrophoblast of the human placenta expre
12 (NK) cell inhibitory ligand expressed on the extravillous cytotrophoblast of the human placenta.
13 lass I MHC molecule selectively expressed on extravillous cytotrophoblast; this cell type does not ex
14                                              Extravillous cytotrophoblasts (CTBs) fail to differentia
15 blast, neither eNOS or iNOS was expressed by extravillous cytotrophoblasts at any time during invasio
16                 During early human pregnancy extravillous cytotrophoblasts invade the uterus and also
17                 During early human pregnancy extravillous cytotrophoblasts invade the uterus and spir
18                     The expression of NOS on extravillous cytotrophoblasts was studied in placental b
19 moting migration of natural killer cells and extravillous cytotrophoblasts.
20  the syncytiotrophoblast and the villous and extravillous cytotrophoblasts.
21 F did not alter proliferation, but initiated extravillous differentiation, with decreased alpha6 inte
22 ) is a gestational neoplasm derived from the extravillous (intermediate) trophoblast of the implantat
23 isplayed stronger LIN28B immunoreactivity in extravillous (invasive) cytotrophoblasts and syncytial s
24 om a transporting epithelium to an invasive, extravillous phenotype that expresses a distinct reperto
25 nvolves cytotrophoblast differentiation into extravillous trophoblast (EVT) and syncytiotrophoblast (
26 toderm and its derivatives: cytotrophoblast, extravillous trophoblast (EVT) and syncytiotrophoblast (
27 ytiotrophoblast (STB) in chorionic villi and extravillous trophoblast (EVT) at the implantation site.
28 by poor placentation, consequent on aberrant extravillous trophoblast (EVT) cell function during plac
29 il to complete faithful differentiation into extravillous trophoblast (EVT) cells and instead show a
30 and preeclampsia, is the failure of invading extravillous trophoblast (EVT) cells to remodel the mate
31                                uNK cells and extravillous trophoblast (EVT) cells were isolated from
32 tiate into specialized cell types, including extravillous trophoblast (EVT) cells.
33 ost and replaced by fibrinoid, incorporating extravillous trophoblast (EVT) cells.
34                                        Human extravillous trophoblast (EVT) invades the decidua via i
35 a, hTSCs can differentiate into cells of the extravillous trophoblast (EVT) lineage or the multinucle
36 from villous cytotrophoblast (VCT) to either extravillous trophoblast (EVT) or syncytiotrophoblast (S
37 ferentiated to syncytiotrophoblast (SCT) and extravillous trophoblast (EVT) was a two-dimensional (2D
38 hen into both syncytiotrophoblast (STB)- and extravillous trophoblast (EVT)-like cells, and showed th
39               We determined that transformed extravillous trophoblast (HTR-8/SVneo) cells were suscep
40    Disrupting signaling interactions between extravillous trophoblast and endometrial stromal cells c
41 CM thereafter could be differentiated to the extravillous trophoblast and syncytiotrophoblast lineage
42                                              Extravillous trophoblast cell (EVT) invasion of decidua
43 ive trophoblast chromatin accessibility with extravillous trophoblast cell accessibility.
44 ograms within the placental villous core and extravillous trophoblast cell column architecture while
45 red to as the junctional zone in rat and the extravillous trophoblast cell column in human.
46 wledge of transcriptional regulation driving extravillous trophoblast cell development is limited.
47 FAP2C, SNAI1, and EPAS1 in the regulation of extravillous trophoblast cell development.
48 ctors and regulatory mechanisms critical for extravillous trophoblast cell development.
49  that primary human trophoblast cells and an extravillous trophoblast cell line (HTR8), from first an
50                                          The extravillous trophoblast cell lineage is a key feature o
51 ell transcripts is conserved in the invasive extravillous trophoblast cell lineage of the human place
52 rk and provide a framework for understanding extravillous trophoblast cell specification in trophobla
53 s identified as an upstream regulator of key extravillous trophoblast cell transcription factors, inc
54 al placentation is driven by highly invasive extravillous trophoblast cells (EVT).
55                                              Extravillous trophoblast cells (EVTs) derived from place
56 ing their ability to interact with placental extravillous trophoblast cells and their potential role
57 trophoblast lineages, and robust invasion of extravillous trophoblast cells by day 14.
58                          Shallow invasion by extravillous trophoblast cells into the uterine wall red
59 temporally expressed only in the distal-most extravillous trophoblast cells, which represent a migrat
60 hoblast stem cells and their transition into extravillous trophoblast cells.
61 into multinucleated syncytiotrophoblasts and extravillous trophoblast cells.
62 B, FOS-like (FOSL) 1, and FOSL2) proteins in extravillous trophoblast cells.
63  of first-trimester chorionic villi enhanced extravillous trophoblast differentiation and invasive ac
64 r-connected regulatory mechanisms modulating extravillous trophoblast differentiation, providing a fr
65 rofile during human trophoblast stem cell to extravillous trophoblast differentiation, we define stag
66                                              Extravillous trophoblast expression was determined by im
67 on towards cells with syncytiotrophoblast or extravillous trophoblast features.
68 K cells have been demonstrated to facilitate extravillous trophoblast invasion into maternal decidua
69                                              Extravillous trophoblast invasion is a fundamental compo
70                It is associated with shallow extravillous trophoblast invasion of the decidua, leadin
71 f decidual macrophages implicated in shallow extravillous trophoblast invasion of the decidua.
72 last-independent remodeling and the start of extravillous trophoblast invasion, were compared to late
73 of pre-implantation maternal immune cells on extravillous trophoblast invasion.
74  promoting development of progenitors of the extravillous trophoblast lineage in the human placenta.
75 is exclusively detected in precursors of the extravillous trophoblast lineage, forming cell columns a
76 entiation of trophoblast stem cells into the extravillous trophoblast lineage.
77 TE sublineages, supported by the presence of extravillous trophoblast markers in the polar sublineage
78 decidualized stromal cells as a regulator of extravillous trophoblast migration.
79 es to multinucleated syncytiotrophoblast and extravillous trophoblast populations, revealing conserve
80 eptor promotes proliferation and survival of extravillous trophoblast progenitors.
81 and their action mechanisms modulating human extravillous trophoblast specification have been unknown
82 e, showing SUPYN localization in villous and extravillous trophoblast subtypes, the decidua and even
83 trast, B7-H2 and B7-H3 were prominent on the extravillous trophoblast throughout gestation.
84 l studies to determine the susceptibility of extravillous trophoblast to other viruses, and the mecha
85 2 bases with (peri)nuclear expression in the extravillous trophoblast using strand-specific RT-PCR co
86 d selectivity for the galectins expressed in extravillous trophoblast were validated in solid phase a
87  not HO-1 was detected on all populations of extravillous trophoblast, but expression of HO-2 or HO-1
88 ctivin/nodal signaling leads to formation of extravillous trophoblast, whereas loss of activin/nodal
89 pies the regulatory elements of the inactive extravillous trophoblast-active genes during the early s
90 tage transcription factors directly activate extravillous trophoblast-active genes, including themsel
91  and beta-hCG, and increased invasion of the extravillous trophoblast.
92 d increased expression of HLA-G, a marker of extravillous trophoblast.
93 lly, these cells differentiate into invasive extravillous trophoblast.
94 decreases with differentiation into HLA-G(+) extravillous trophoblast.
95 cental bed but only TGF-beta2 was present in extravillous trophoblast.
96 ted in the cytotrophoblast and intermediate (extravillous) trophoblast of normal and molar placentas,
97 e (MAPK) in 84% of cases, whereas the normal extravillous trophoblastic cells did not.
98 t, neither compound had any effect on normal extravillous trophoblastic cells or JEG-3 cells.
99 ed to choriocarcinoma JEG-3 cells and normal extravillous trophoblastic cells.
100 ibits the expression of SRC-3, which impairs extravillous trophoblastic invasion and migration by dir
101 s the cells to differentiate into functional extravillous trophoblasts (EVT) and syncytiotrophoblasts
102 Invading human leukocyte antigen-G+ (HLA-G+) extravillous trophoblasts (EVT) are rare cells that are
103 oxic decidual natural killer cells (dNK) and extravillous trophoblasts (EVT) at the maternal-fetal in
104 en (HLA) class I expression pattern of human extravillous trophoblasts (EVT) endows them with unique
105       During pregnancy, semiallogeneic fetal extravillous trophoblasts (EVT) invade the uterine mucos
106            During pregnancy, invading HLA-G+ extravillous trophoblasts (EVT) play a key role in place
107 igen (HLA) molecule exclusively expressed in extravillous trophoblasts (EVT), is a crucial factor in
108 urrounding maternal cells and invading fetal extravillous trophoblasts (EVT).
109 arly gestation syncytiotrophoblasts (ST) and extravillous trophoblasts (EVT).
110 blasts (CTs), and their differentiation into extravillous trophoblasts (EVTs) as our models, we revea
111 otrophoblasts in floating chorionic villi or extravillous trophoblasts (EVTs) at the anchoring villi.
112 tected in anchoring villi and choriodecidual extravillous trophoblasts (EVTs) during pregnancy.
113   Here, we reveal that ATOH8 is critical for extravillous trophoblasts (EVTs) formation while being d
114                                              Extravillous trophoblasts (EVTs) have the potential to p
115 d remodeling of maternal uterine arteries by extravillous trophoblasts (EVTs) in the first trimester
116 hat activation of PAR1 expressed by invasive extravillous trophoblasts (EVTs) influences human placen
117 ning in the first trimester, fetally derived extravillous trophoblasts (EVTs) invade the uterus and r
118                                              Extravillous trophoblasts (EVTs) were derived that expre
119 ts of progenitor cytotrophoblasts (CTBs) and extravillous trophoblasts (EVTs) with similar gene expre
120 ocesses replicated by invasive trophoblasts (extravillous trophoblasts (EVTs)) during early placentat
121                                           In extravillous trophoblasts (EVTs), B. abortus and B. suis
122  pivotal role in the development of invasive extravillous trophoblasts (EVTs), cells that are essenti
123 lasts (CTs), syncytiotrophoblasts (STs), and extravillous trophoblasts (EVTs), composing the placenta
124      Defects in the developmental program of extravillous trophoblasts (EVTs), migrating from placent
125 stem cells (TSCs) can be differentiated into extravillous trophoblasts (EVTs), syncytiotrophoblasts (
126 s (TSCs) into syncytiotrophoblasts (STs) and extravillous trophoblasts (EVTs), we reveal dynamic reco
127 ted syncytiotrophoblasts (STBs) and invasive extravillous trophoblasts (EVTs).
128 on (EMT) as they differentiate into invasive extravillous trophoblasts (EVTs).
129 o initiate remodeling before colonization by extravillous trophoblasts (EVTs); however, the trigger f
130                                   Developing extravillous trophoblasts accumulate heparin-binding EGF
131 ii) the maternal decidua, where mononuclear, extravillous trophoblasts anchor the villous region to t
132 ed, its role in the functional regulation of extravillous trophoblasts and the development of PE rema
133         In severe preeclampsia, PZP-positive extravillous trophoblasts are adjacent to extracellular
134                                Fetal-derived extravillous trophoblasts come in direct contact with ma
135 iological expression restricted to placental extravillous trophoblasts contributes to maternal tolera
136                                     Invasive extravillous trophoblasts derived from cytotrophoblast p
137 enta and placental bed but not by villous or extravillous trophoblasts in normal or pathological samp
138 hemistry analysis, PZP is found primarily in extravillous trophoblasts in the placenta.
139 l immune and endothelial cells juxtaposed to extravillous trophoblasts in the uterine implantation si
140 e to model the invasion of specialized fetal extravillous trophoblasts into the maternal uterus.
141 n mother and fetus, progenitors develop into extravillous trophoblasts invading the maternal uterus a
142  In the pregnancy complication preeclampsia, extravillous trophoblasts invasion and vessel remodeling
143                 These cells are analogous to extravillous trophoblasts of the human placenta.
144 lasts can terminally differentiate to either extravillous trophoblasts or syncytiotrophoblasts.
145                      During human pregnancy, extravillous trophoblasts play crucial roles in placenta
146 ne protease highly expressed in the invasive extravillous trophoblasts that invade decidua.
147 and selectively transfer it via nanotubes to extravillous trophoblasts to kill intracellular Listeria
148 strate in vivo-like directional migration of extravillous trophoblasts towards a microengineered mate
149 an MHC class Ib protein that is expressed on extravillous trophoblasts), LILRB1 on CD14(+) macrophage
150 , 197 commonly expressed genes with placenta extravillous trophoblasts, 128 with cytotrophoblasts and
151 d invasion and maternal vascular remodeling (extravillous trophoblasts, EVTs).
152 expression profiles of term intravillous and extravillous trophoblasts, including the transcriptome o
153 n of MIF expression in syncytiotrophoblasts, extravillous trophoblasts, IVB mononuclear cells, and am
154 s of the latter either develop into invasive extravillous trophoblasts, remodeling the uterine vascul
155 aled similarities between ruminant and human extravillous trophoblasts, suggesting conserved EMT acro
156 d the expression of CD1d on both villous and extravillous trophoblasts, the fetal cells that invade t
157 otrophoblast clusters and a small cluster of extravillous trophoblasts, which closely correspond to t
158                            JEG-3 cells model extravillous trophoblasts, which predominate during the
159  conditions differentiated human TSCs toward extravillous trophoblasts.
160 lar smooth muscle, and decreased invasion by extravillous trophoblasts.
161 cross trophoblasts, particularly in invasive extravillous trophoblasts.
162 s in response to HCMV-infected primary fetal extravillous trophoblasts.
163 n preeclamptic placentas, in first-trimester extravillous trophoblasts.
164  families decreased the invasion capacity of extravillous trophoblasts.
165 mors are thought to arise from intermediate (extravillous) trophoblasts based on histopathological st

 
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