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1  and beta-hCG, and increased invasion of the extravillous trophoblast.
2 d increased expression of HLA-G, a marker of extravillous trophoblast.
3 lly, these cells differentiate into invasive extravillous trophoblast.
4 decreases with differentiation into HLA-G(+) extravillous trophoblast.
5 cental bed but only TGF-beta2 was present in extravillous trophoblast.
6 lar smooth muscle, and decreased invasion by extravillous trophoblasts.
7 cross trophoblasts, particularly in invasive extravillous trophoblasts.
8 s in response to HCMV-infected primary fetal extravillous trophoblasts.
9 n preeclamptic placentas, in first-trimester extravillous trophoblasts.
10  families decreased the invasion capacity of extravillous trophoblasts.
11  conditions differentiated human TSCs toward extravillous trophoblasts.
12 , 197 commonly expressed genes with placenta extravillous trophoblasts, 128 with cytotrophoblasts and
13                                   Developing extravillous trophoblasts accumulate heparin-binding EGF
14 pies the regulatory elements of the inactive extravillous trophoblast-active genes during the early s
15 tage transcription factors directly activate extravillous trophoblast-active genes, including themsel
16 ii) the maternal decidua, where mononuclear, extravillous trophoblasts anchor the villous region to t
17    Disrupting signaling interactions between extravillous trophoblast and endometrial stromal cells c
18 CM thereafter could be differentiated to the extravillous trophoblast and syncytiotrophoblast lineage
19 ed, its role in the functional regulation of extravillous trophoblasts and the development of PE rema
20         In severe preeclampsia, PZP-positive extravillous trophoblasts are adjacent to extracellular
21 mors are thought to arise from intermediate (extravillous) trophoblasts based on histopathological st
22  not HO-1 was detected on all populations of extravillous trophoblast, but expression of HO-2 or HO-1
23                                              Extravillous trophoblast cell (EVT) invasion of decidua
24 ive trophoblast chromatin accessibility with extravillous trophoblast cell accessibility.
25 ograms within the placental villous core and extravillous trophoblast cell column architecture while
26 red to as the junctional zone in rat and the extravillous trophoblast cell column in human.
27 wledge of transcriptional regulation driving extravillous trophoblast cell development is limited.
28 FAP2C, SNAI1, and EPAS1 in the regulation of extravillous trophoblast cell development.
29 ctors and regulatory mechanisms critical for extravillous trophoblast cell development.
30  that primary human trophoblast cells and an extravillous trophoblast cell line (HTR8), from first an
31                                          The extravillous trophoblast cell lineage is a key feature o
32 ell transcripts is conserved in the invasive extravillous trophoblast cell lineage of the human place
33 rk and provide a framework for understanding extravillous trophoblast cell specification in trophobla
34 s identified as an upstream regulator of key extravillous trophoblast cell transcription factors, inc
35 al placentation is driven by highly invasive extravillous trophoblast cells (EVT).
36                                              Extravillous trophoblast cells (EVTs) derived from place
37 ing their ability to interact with placental extravillous trophoblast cells and their potential role
38 trophoblast lineages, and robust invasion of extravillous trophoblast cells by day 14.
39                          Shallow invasion by extravillous trophoblast cells into the uterine wall red
40 temporally expressed only in the distal-most extravillous trophoblast cells, which represent a migrat
41 into multinucleated syncytiotrophoblasts and extravillous trophoblast cells.
42 B, FOS-like (FOSL) 1, and FOSL2) proteins in extravillous trophoblast cells.
43 hoblast stem cells and their transition into extravillous trophoblast cells.
44                                Fetal-derived extravillous trophoblasts come in direct contact with ma
45 iological expression restricted to placental extravillous trophoblasts contributes to maternal tolera
46                                     Invasive extravillous trophoblasts derived from cytotrophoblast p
47  of first-trimester chorionic villi enhanced extravillous trophoblast differentiation and invasive ac
48 r-connected regulatory mechanisms modulating extravillous trophoblast differentiation, providing a fr
49 rofile during human trophoblast stem cell to extravillous trophoblast differentiation, we define stag
50 toderm and its derivatives: cytotrophoblast, extravillous trophoblast (EVT) and syncytiotrophoblast (
51 nvolves cytotrophoblast differentiation into extravillous trophoblast (EVT) and syncytiotrophoblast (
52 ytiotrophoblast (STB) in chorionic villi and extravillous trophoblast (EVT) at the implantation site.
53 by poor placentation, consequent on aberrant extravillous trophoblast (EVT) cell function during plac
54 il to complete faithful differentiation into extravillous trophoblast (EVT) cells and instead show a
55 and preeclampsia, is the failure of invading extravillous trophoblast (EVT) cells to remodel the mate
56                                uNK cells and extravillous trophoblast (EVT) cells were isolated from
57 tiate into specialized cell types, including extravillous trophoblast (EVT) cells.
58 ost and replaced by fibrinoid, incorporating extravillous trophoblast (EVT) cells.
59                                        Human extravillous trophoblast (EVT) invades the decidua via i
60 a, hTSCs can differentiate into cells of the extravillous trophoblast (EVT) lineage or the multinucle
61 from villous cytotrophoblast (VCT) to either extravillous trophoblast (EVT) or syncytiotrophoblast (S
62 ferentiated to syncytiotrophoblast (SCT) and extravillous trophoblast (EVT) was a two-dimensional (2D
63 hen into both syncytiotrophoblast (STB)- and extravillous trophoblast (EVT)-like cells, and showed th
64 s the cells to differentiate into functional extravillous trophoblasts (EVT) and syncytiotrophoblasts
65 Invading human leukocyte antigen-G+ (HLA-G+) extravillous trophoblasts (EVT) are rare cells that are
66 oxic decidual natural killer cells (dNK) and extravillous trophoblasts (EVT) at the maternal-fetal in
67 en (HLA) class I expression pattern of human extravillous trophoblasts (EVT) endows them with unique
68       During pregnancy, semiallogeneic fetal extravillous trophoblasts (EVT) invade the uterine mucos
69            During pregnancy, invading HLA-G+ extravillous trophoblasts (EVT) play a key role in place
70 igen (HLA) molecule exclusively expressed in extravillous trophoblasts (EVT), is a crucial factor in
71 urrounding maternal cells and invading fetal extravillous trophoblasts (EVT).
72 arly gestation syncytiotrophoblasts (ST) and extravillous trophoblasts (EVT).
73 blasts (CTs), and their differentiation into extravillous trophoblasts (EVTs) as our models, we revea
74 otrophoblasts in floating chorionic villi or extravillous trophoblasts (EVTs) at the anchoring villi.
75 tected in anchoring villi and choriodecidual extravillous trophoblasts (EVTs) during pregnancy.
76   Here, we reveal that ATOH8 is critical for extravillous trophoblasts (EVTs) formation while being d
77                                              Extravillous trophoblasts (EVTs) have the potential to p
78 d remodeling of maternal uterine arteries by extravillous trophoblasts (EVTs) in the first trimester
79 hat activation of PAR1 expressed by invasive extravillous trophoblasts (EVTs) influences human placen
80 ning in the first trimester, fetally derived extravillous trophoblasts (EVTs) invade the uterus and r
81                                              Extravillous trophoblasts (EVTs) were derived that expre
82 ts of progenitor cytotrophoblasts (CTBs) and extravillous trophoblasts (EVTs) with similar gene expre
83 ocesses replicated by invasive trophoblasts (extravillous trophoblasts (EVTs)) during early placentat
84                                           In extravillous trophoblasts (EVTs), B. abortus and B. suis
85  pivotal role in the development of invasive extravillous trophoblasts (EVTs), cells that are essenti
86 lasts (CTs), syncytiotrophoblasts (STs), and extravillous trophoblasts (EVTs), composing the placenta
87      Defects in the developmental program of extravillous trophoblasts (EVTs), migrating from placent
88 stem cells (TSCs) can be differentiated into extravillous trophoblasts (EVTs), syncytiotrophoblasts (
89 s (TSCs) into syncytiotrophoblasts (STs) and extravillous trophoblasts (EVTs), we reveal dynamic reco
90 on (EMT) as they differentiate into invasive extravillous trophoblasts (EVTs).
91 ted syncytiotrophoblasts (STBs) and invasive extravillous trophoblasts (EVTs).
92 o initiate remodeling before colonization by extravillous trophoblasts (EVTs); however, the trigger f
93 d invasion and maternal vascular remodeling (extravillous trophoblasts, EVTs).
94                                              Extravillous trophoblast expression was determined by im
95 on towards cells with syncytiotrophoblast or extravillous trophoblast features.
96               We determined that transformed extravillous trophoblast (HTR-8/SVneo) cells were suscep
97 enta and placental bed but not by villous or extravillous trophoblasts in normal or pathological samp
98 hemistry analysis, PZP is found primarily in extravillous trophoblasts in the placenta.
99 l immune and endothelial cells juxtaposed to extravillous trophoblasts in the uterine implantation si
100 expression profiles of term intravillous and extravillous trophoblasts, including the transcriptome o
101 e to model the invasion of specialized fetal extravillous trophoblasts into the maternal uterus.
102 n mother and fetus, progenitors develop into extravillous trophoblasts invading the maternal uterus a
103 K cells have been demonstrated to facilitate extravillous trophoblast invasion into maternal decidua
104                                              Extravillous trophoblast invasion is a fundamental compo
105                It is associated with shallow extravillous trophoblast invasion of the decidua, leadin
106 f decidual macrophages implicated in shallow extravillous trophoblast invasion of the decidua.
107 last-independent remodeling and the start of extravillous trophoblast invasion, were compared to late
108 of pre-implantation maternal immune cells on extravillous trophoblast invasion.
109  In the pregnancy complication preeclampsia, extravillous trophoblasts invasion and vessel remodeling
110 n of MIF expression in syncytiotrophoblasts, extravillous trophoblasts, IVB mononuclear cells, and am
111 an MHC class Ib protein that is expressed on extravillous trophoblasts), LILRB1 on CD14(+) macrophage
112  promoting development of progenitors of the extravillous trophoblast lineage in the human placenta.
113 is exclusively detected in precursors of the extravillous trophoblast lineage, forming cell columns a
114 entiation of trophoblast stem cells into the extravillous trophoblast lineage.
115 TE sublineages, supported by the presence of extravillous trophoblast markers in the polar sublineage
116 decidualized stromal cells as a regulator of extravillous trophoblast migration.
117                 These cells are analogous to extravillous trophoblasts of the human placenta.
118 ted in the cytotrophoblast and intermediate (extravillous) trophoblast of normal and molar placentas,
119 lasts can terminally differentiate to either extravillous trophoblasts or syncytiotrophoblasts.
120                      During human pregnancy, extravillous trophoblasts play crucial roles in placenta
121 es to multinucleated syncytiotrophoblast and extravillous trophoblast populations, revealing conserve
122 eptor promotes proliferation and survival of extravillous trophoblast progenitors.
123 s of the latter either develop into invasive extravillous trophoblasts, remodeling the uterine vascul
124 and their action mechanisms modulating human extravillous trophoblast specification have been unknown
125 e, showing SUPYN localization in villous and extravillous trophoblast subtypes, the decidua and even
126 aled similarities between ruminant and human extravillous trophoblasts, suggesting conserved EMT acro
127 ne protease highly expressed in the invasive extravillous trophoblasts that invade decidua.
128 d the expression of CD1d on both villous and extravillous trophoblasts, the fetal cells that invade t
129 trast, B7-H2 and B7-H3 were prominent on the extravillous trophoblast throughout gestation.
130 l studies to determine the susceptibility of extravillous trophoblast to other viruses, and the mecha
131 and selectively transfer it via nanotubes to extravillous trophoblasts to kill intracellular Listeria
132 strate in vivo-like directional migration of extravillous trophoblasts towards a microengineered mate
133 2 bases with (peri)nuclear expression in the extravillous trophoblast using strand-specific RT-PCR co
134 d selectivity for the galectins expressed in extravillous trophoblast were validated in solid phase a
135 ctivin/nodal signaling leads to formation of extravillous trophoblast, whereas loss of activin/nodal
136 otrophoblast clusters and a small cluster of extravillous trophoblasts, which closely correspond to t
137                            JEG-3 cells model extravillous trophoblasts, which predominate during the

 
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