ライフサイエンスコーパス: eye movement

1 arameters such as hand movement, licking, or eye movement.

2 he neural circuit controlling smooth pursuit eye movement.

3 spite the sudden disruptions created by each eye movement.

4 tively influences visual perception after an eye movement.

5 o the other because of a horizontal saccadic eye movement.

6 n shift covertly, decoupled from programming eye movements.

7 drifts remain to be viewed as largely random eye movements.

8 ural conditions, they make frequent saccadic eye movements.

9 at the hippocampus coordinates memory-guided eye movements.

10 ally involved in the neural control of these eye movements.

11 animal's conscious percept was inferred from eye movements.

12 lectrical stimulation of CDt evokes saccadic eye movements.

13 al environment to enable efficient binocular eye movements.

14 implicated in the transition from vision to eye movements.

15 uiring fast (exploratory) and slow (pursuit) eye movements.

16 ntages but also guides rapid actions such as eye movements.

17 lying the planning and execution of saccadic eye movements.

18 der specific conditions, including simulated eye movements.

19 K) paradigm and recordings of their saccadic eye movements.

20 ing high phi as a continuous stimulus across eye movements.

21 visually guided saccades and smooth-pursuit eye movements.

22 e field (FEF) carries such a CD for saccadic eye movements.

23 generated during smooth, symmetric vergence eye movements.

24 gaze but in mice relies on combined head and eye movements.

25 rm similar motion integration without making eye movements.

26 perform accurate vergence and accommodation eye movements.

27 luding automated detection of slow waves and eye movements.

28 representations and the learning of accurate eye movements.

29 ht-loss and has been shown to affect natural eye-movements.

30 ata efficient and relying more critically on eye-movements.

31 gnificantly higher V(E) throughout non-rapid eye movement (20.1 vs. -27.7 mL/min respectively, P < 0.

32 llocated to where we can potentially make an eye movement [3].

33 lomotor range that is accessible by saccadic eye movements [5,6].

34 We tested the hypothesis that important eye movement abnormalities in cerebellar disorders (i.e.

35 call of direction learning in smooth pursuit eye movements across multiple timescales.

36 related to Z- modules, whereas compensatory eye movement adaptation, linked to Z+ modules, is intact

37 nsient.SIGNIFICANCE STATEMENT Smooth pursuit eye movements allow us to track moving objects.

38 eveloped to provide continuous monitoring of eye-movements, allowing insight into the physiological p

39 motoneurons in fixations and slow and phasic eye movements, although their discharge properties indic

40 Nystagmus is a disorder of uncontrolled eye movement and can occur as an isolated trait (idiopat

41 Eye movement and MEG recordings revealed how participant

42 ughout the night, including during non-rapid eye movement and rapid eye movement sleep, to report the

43 viewers actively sample the environment with eye movements and also obtain a low-resolution preview o

44 What is the link between these eye movements and attention?

45 ccur both under conditions of smooth pursuit eye movements and constant fixation, and to be consisten

46 measured visual sensitivity before saccadic eye movements and during fixation at locations either wi

47 His brain activity, eye movements and hand/foot movements were recorded.

48 Here, we simultaneously tracked eye movements and hippocampal field potentials while neu

49 are slower and longer lasting than saccadic eye movements and it has been suggested that initiating

50 ake into account the sensory consequences of eye movements and map the fleeting positions of objects

51 These findings reveal a key role of eye movements and suggest that distinct insula and OFC a

52 how close the correlation is between imagery eye movements and the eye movements while looking at the

53 , their lack of disparity-dependent vergence eye movements and wide neuronal representation suggests

54 elationship between slower and less accurate eye movements and worse glaucoma severity.

55 ance extinction, or whether it is limited to eye movements, and [2] the effectiveness of such an inte

56 ked behavioral responses, vestibular-induced eye movements, and hair-cell activity as assessed with F

57 and humans, one such CD keeps track of rapid eye movements, and in monkeys, a circuit carrying this C

58 ng exclusive extrapyramidal symptoms, normal eye movements, and normal alpha-fetoprotein levels in so

59 he rapid and reproducible nature of saccadic eye movements, and the key role that they play in primat

60 Visually guided eye movements are accommodated by different parts of the

61 Our results show that rapid binocular eye movements are adapted to the statistics of the 3D en

62 Here we show that eye movements are also inhibited before predictable audi

63 There is growing evidence that these eye movements are associated with covert shifts of atten

64 We found that the majority of eye movements are compensatory for head movements, there

65 lts are relevant to the debate about whether eye movements are derived from separate saccadic and ver

66 Eye movements are disrupted in many neurodegenerative di

67 hifts of spatial attention in the absence of eye movements are elicited by preceding activation in th

68 Eye movements are inhibited prior to the onset of tempor

69 ver, when mice are free to move their heads, eye movements are more complex and often non-conjugate,

70 Signals related to eye movements are present in much of the primate brain,

71 spatial resolution, possible influences from eye movements are rarely considered.

72 le, how do we grasp an unstable object while eye movements are simultaneously changing its retinal lo

73 e compatible with the rate at which saccadic eye movements are typically observed in natural viewing.

74 rget speed and direction, as well as pursuit eye movements, are significantly impaired at 0.015% BAC,

75 efficient coding theory, which proposes that eye movements as well as stimulus encoding are jointly a

76 apse with photoreceptors, showed oscillating eye movements at a frequency of 4-7 Hz. nob ON direction

77 Little is known about eye movement behavior on phones, despite their pervasive

78 informs the visual system about the upcoming eye movement, behavioral studies investigating the time

79 his work we first quantify the similarity of eye movements between recalling an image and encoding th

80 e found that the pattern of small fixational eye movements called microsaccades changes around behavi

81 During fixation, humans make small eye movements called microsaccades, and inhibiting micro

82 stigation on whether comparing such pairs of eye movements can be used for computational image retrie

83 Here we show that such eye movements can play an important role in visual learn

84 For example, eye movements cause irrelevant retinal signals that coul

85 ems continuous and detailed despite saccadic eye movements changing retinal input several times per s

86 with glaucoma had significant differences in eye movements compared to healthy subjects, with a relat

87 l binocular vision while performing vergence eye movements compared to sustained gaze fixation within

88 study to show quantitatively that binocular eye movements conform to 3D scene statistics, thereby en

89 This circuit provides guidance for eye movements, contributes to stable visual perception,

90 culomotor vermis, a key area associated with eye movement control.

91 Such eye movements correlate strongly with the spatial layout

92 the time required for subjects to produce an eye movement could be predicted from the statistics of t

93 We tested whether eye movements could be used to infer subjects' beliefs a

94 During head restraint, mice make rapid eye movements coupled between the eyes, similar to conju

95 t dscaml1 mutants have a host of oculomotor (eye movement) deficits.

96 These metrics included rapid eye movement duration, features of the electroencephalog

97 PDx and is likely related to factors such as eye movement during OCT capture.

98 o examine the speed and accuracy of saccadic eye movements during a novel eye tracking threshold visu

99 To investigate, we monitored eye movements during a repetitive banknote authenticatio

100 not attributable to differential patterns of eye movements during adaptation.

101 We also show that eye movements during decision-making are predictive of t

102 that computational image retrieval based on eye movements during spontaneous imagery is feasible.

103 tudied how visual information was sampled by eye-movements during this process called fear generaliza

104 ly relevant location and prepared a response eye movement either toward or away from this location.

105 ontrolled using optogenetic stimulation, the eye movements elicited were well-described by a linear p

106 We used eye movement (EM) monitoring during a partial-cue recogn

107 processes both affect, and are affected by, eye movements (EMs).

108 y from kinematic features, 70% accuracy from eye movement features and 78% accuracy from combined fea

109 Machine learning models trained on eye movement features were able to identify abnormalitie

110 avioral evidence using monkey smooth pursuit eye movements for four principles of cerebellar learning

111 ng is necessary to generate the compensatory eye movements found experimentally during naturalistic s

112 ng horizontal vergence, we recorded vergence eye movements from ten observers in response to four typ

113 Smooth pursuit eye movements have been investigated as a diagnostic too

114 cal studies of the transition from vision to eye movements have measured the activity of one neuron a

115 ereas visually based corrections for ongoing eye movements have stronger effects and are likely most

116 ed with grid cell-like firing in response to eye movements, i.e., in visual space.

117 th improved task performance, and inhibiting eye movements in humans impaired navigation precision.

118 ween the eyes, similar to conjugate saccadic eye movements in humans.

119 ghlight similarities and differences between eye movements in mice and humans.

120 ral behavior, we measured head and bilateral eye movements in mice performing prey capture, an etholo

121 nveys oculomotor CD associated with saccadic eye movements in nonhuman primates.

122 ) threat-related emotional stimuli can guide eye movements in the absence of visual awareness; (2) th

123 Here we examine the role of eye movements in the most common assessment of visual ac

124 These findings relate eye movements in the mouse to other species, and provide

125 This study examined eye-movements in a sample of 202 participants (42 with d

126 We demonstrate the feasibility of capturing eye movement information using an inexpensive and widely

127 can be improved with a cognitively demanding eye-movement intervention.

128 n suggested that initiating a smooth pursuit eye movement involves an obligatory "open-loop" interval

129 r with (overt) or without (covert) foveating eye movements is critical to primate behavior.

130 Characterizing eye movements is important for diagnosis and may be usef

131 tal nystagmus, involuntary oscillating small eye movements, is commonly thought to originate from abe

132 as reached the brain, well in advance of the eye movement itself.

133 by overall looking time, social preference, eye movement latencies, or number of fixations.

134 VTA neurons elicited long-lasting non-rapid-eye-movement-like sleep resembling sedation.

135 Smooth pursuit eye movements maintain the line of sight on smoothly mov

136 cortical areas, subcortical areas mediating eye movements may be recruited with temporal attention.

137 Our findings show that a set of eye-movement measures can be used to provide sensitive a

138 ch is achieved within the canonical cortical eye movement network.SIGNIFICANCE STATEMENT The ability

139 Using fMRI, we replicated the core cortical eye-movement network for saccade generation (frontal eye

140 nses at powers 3-20 mW compared to non-rapid eye movement (non-REM) sleep and wakefulness (each P < 0

141 lectrical events that occur during non-rapid-eye-movement (non-REM) sleep(1-8) and whose disruption i

142 POA Tac1 neurons obliterates both non-rapid eye movement (NREM) and rapid eye movement (REM) sleep,

143 ed region in early visual areas in non-rapid eye movement (NREM) and REM sleep.

144 rylation site, S551, showed longer non-rapid eye movement (NREM) sleep and increased NREMS delta dens

145 HD-EEG source-localization, during non-rapid eye movement (NREM) sleep and rapid eye movement (REM) s

146 nia was accompanied by the onset of nonrapid eye movement (NREM) sleep beta oscillations that were sy

147 id transitions to wakefulness from non-rapid eye movement (NREM) sleep but did not affect REM-wakeful

148 sociated with consciousness during non-rapid eye movement (NREM) sleep following parietal TMS.

149 Non-rapid eye movement (NREM) sleep is supposed to play a key role

150 whether learning is facilitated by non-rapid eye movement (NREM) sleep or by REM sleep, whether it re

151 ral PAG (vlPAG) powerfully promote non-rapid eye movement (NREM) sleep partly through their projectio

152 nd OSA (n = 129) groups during the non-rapid eye movement (NREM) sleep period, after controlling for

153 The slow waves of non-rapid eye movement (NREM) sleep reflect experience-dependent p

154 the brain correlates with decreased nonrapid eye movement (NREM) sleep slow wave activity.

155 that initial baseline measures of non-rapid eye movement (NREM) sleep slow-wave activity (SWA) and s

156 ods elicits rapid transitions from non-rapid eye movement (NREM) sleep to wakefulness and produces su

157 ed how neural reactivations during non-rapid eye movement (NREM) sleep were causally linked to consol

158 typically considered a hallmark of nonrapid eye movement (NREM) sleep, but recent work in mice has s

159 LS, with strongest coupling during non-rapid eye movement (NREM) sleep, followed by waking immobility

160 During non-rapid eye movement (NREM) sleep, neuronal populations in the m

161 ally considered to be a hallmark of nonrapid eye movement (NREM) sleep, recent work in mice has shown

162 the arousal threshold (AT) during non-rapid eye movement (NREM) sleep.

163 s were presented to one nostril in non-rapid eye movement (NREM) sleep.

164 diated information transfer during non-rapid eye movement (NREM) sleep.

165 entified pIII neurons that promote non-rapid eye movement (NREM) sleep.

166 the arousal threshold (AT) during non-rapid eye movement (NREM) sleep.

167 memory consolidation processes in non-rapid eye movement (NREM) sleep.

168 us, the brainstem is essential for non-rapid eye movement (NREM) sleep.

169 In contrast, non-rapid eye movement (NREM) slow-wave oscillations offer an amel

170 In non-rapid eye movement (NREM) stage 3 sleep (N3), phase-locked pin

171 of PTSD in the awake state, during non-rapid eye movement (NREM) stage N2 sleep, and in a hybrid BCM

172 om hippocampus to neocortex during non-rapid-eye-movement (NREM) sleep.

173 To this aim, we first tracked eye movements of male and female observers during face r

174 ts remain stable, even though smooth-pursuit eye movements often distort optic flow.

175 est when stimuli are presented closer to the eye movement onset time.

176 We asked if an involuntary eye movement, optokinetic nystagmus (OKN), could serve a

177 Slow-wave sleep and rapid eye movement (or paradoxical) sleep have been found in m

178 The relationship between eye movement parameters and severity of glaucoma was exa

179 isual field assessment and determine whether eye movement parameters may improve ability to detect gl

180 However, in a multivariable model, eye movement parameters were not of additional benefit i

181 ome statistically significant effects in the eye movement parameters.

182 Key eye-movement parameters were computed, including saccade

183 ive neural responses and their idiosyncratic eye-movement patterns during identity processing, which

184 Eye movements provide a functional signature of how huma

185 The smooth eye movement region of the frontal eye fields (FEF(SEM))

186 disorders, blood pressure, urate, and rapid eye movement (REM) behaviour disorder scores.

187 ese ADRB1(+) neurons are active during rapid eye movement (REM) sleep and wakefulness.

188 Waking and rapid eye movement (REM) sleep are characterized by ongoing ir

189 timeline, prevalence, and survival of rapid eye movement (REM) sleep behavior disorder (RBD) in pati

190 o increases wakefulness and suppresses rapid-eye movement (REM) sleep in mice and rats and reduces ca

191 Although rapid eye movement (REM) sleep is also associated with diminis

192 The occurrence of dreaming during rapid eye movement (REM) sleep prompts interest in the role of

193 Notably, rapid eye movement (REM) sleep regulates emotional memory, and

194 Rapid eye movement (REM) sleep serves an important function fo

195 ward provided during training enhanced rapid eye movement (REM) sleep time, increased oscillatory act

196 While rapid eye movement (REM) sleep was marked by decreased hippoca

197 the diagnostic utility of quantitative rapid eye movement (REM) sleep without atonia analysis in the

198 During rapid eye movement (REM) sleep, behavioral unresponsiveness co

199 on-rapid eye movement (NREM) sleep and rapid eye movement (REM) sleep, in six medication-refractory f

200 both non-rapid eye movement (NREM) and rapid eye movement (REM) sleep, strongly consolidating the wak

201 ught to determine which species 'have' rapid eye movement (REM) sleep.

202 active only during sleep, particularly rapid eye movement (REM) sleep.

203 calp electroencephalogram (EEG) during rapid eye movement (REM) sleep.

204 s indicate that they are both wake and rapid eye movement (REM)-sleep active.

205 employs to achieve visual continuity across eye movements remains unclear.

206 Both types of eye movements require the mapping of retinal information

207 Our results show the potential for scaling eye movement research by orders-of-magnitude to thousand

208 ers, we replicate key findings from previous eye movement research on oculomotor tasks and saliency a

209 button press (humans) or a speeded saccadic eye movement response (humans and monkeys).

210 ing the location of a visual stimulus for an eye movement response.

211 These combined saccade-vergence eye movements result in disjunctive saccades with a verg

212 phasic firing pattern for different types of eye movement (saccades, vestibulo-ocular reflex, vergenc

213 asp movements are often accompanied by rapid eye movements (saccades) that displace the desired objec

214 7.7 mL/min respectively, P < 0.05) and rapid eye movement sleep (16.5 vs 23.4 mL/min, P < 0.05).

215 and the duration of stages 1 and 2 and rapid eye movement sleep (all P < 0.001), whereas slow-wave sl

216 ing sleep-deprived wakefulness and non-rapid eye movement sleep (experiment 2, n = 37).

217 , defining oscillations of stage 2 non-rapid eye movement sleep (N2), mediate memory consolidation.

218 Deep non-rapid eye movement sleep (NREM) and general anesthesia with pr

219 acid elicited robust increases in non-rapid-eye movement sleep (NREMS) and decreases in body tempera

220 e spent significantly less time in non-rapid eye movement sleep (NREMS) during the light phase while

221 nding to specific behaviors, including rapid eye movement sleep (REM sleep), a sleep phase when the b

222 ep including slow wave sleep (SWS) and rapid eye movement sleep (REM), raising the question of why an

223 while spending more time in NREMS and rapid eye movement sleep (REMS) during the dark phase.

224 movement sleep (stages N2 and N3) and rapid eye movement sleep (stage R) were selected from the firs

225 Sections during non-rapid eye movement sleep (stages N2 and N3) and rapid eye move

226 es become less synchronized during non-rapid eye movement sleep after sleep deprivation at the networ

227 r and collectively, and across waking, rapid eye movement sleep and non-rapid eye movement sleep, we

228 further highlight the prospect of non-rapid eye movement sleep as a therapeutic target for meaningfu

229 Isolated (or idiopathic) rapid eye movement sleep behavior disorder (iRBD) is associate

230 Rapid eye movement sleep behavior disorder (RBD) is a prodroma

231 ts was performed in PD (n = 1,575) and rapid eye movement sleep behavior disorder (RBD) patients (n =

232 ingulate, and parietal metabolism; and rapid eye movement sleep behavior disorder (RBD) with bilatera

233 activity, sex, constipation, possible rapid eye movement sleep behavior disorder (RBD), and smoking

234 as insomnia, obstructive sleep apnea, rapid eye movement sleep behavior disorder, and circadian rhyt

235 ry can include prodromal features (eg, rapid eye movement sleep behavior disorder, hyposmia, constipa

236 s disease and patients with idiopathic rapid eye movement sleep behaviour disorder (iRBD) exempt of P

237 Traumatic brain injury (TBI) and rapid eye movement sleep behavioural disorder (RBD) are risk f

238 The rapid eye movement sleep behavioural disorder (RBD) population

239 cohorts consisting of individuals with rapid eye movement sleep behavioural disorder, Parkinson's dis

240 astroglial calcium signals during non-rapid eye movement sleep change in proportion to sleep need.

241 oms, non-motor manifestations (such as rapid eye movement sleep disorder, anosmia, constipation and d

242 pikes emerged predominantly during non-rapid eye movement sleep in 24-hour vEEG of Syngap1(+/-) mice.

243 During non-rapid eye movement sleep, MD firing rate decreased around spin

244 of slow-wave sleep and paradoxical or rapid eye movement sleep, respectively.

245 ding during non-rapid eye movement and rapid eye movement sleep, to report their thoughts in that mom

246 king, rapid eye movement sleep and non-rapid eye movement sleep, we found preserved patterns of spike

247 or attentive waking and paradoxical or rapid eye movement sleep.

248 d CSF dynamics that appears during non-rapid eye movement sleep.

249 or attentive waking and paradoxical or rapid eye movement sleep.

250 e RMTg plays an essential role for non-rapid eye movement sleep.

251 ip with the slow wave component of non-rapid eye-movement sleep (NR) arousals.

252 ticobasal syndrome (n = 1), idiopathic rapid-eye-movement sleep behavior disorder (n = 1), and behavi

253 ct of orthostatic hypotension (OH) and rapid-eye-movement sleep behavioural disorder (RBD) on surviva

254 wake-promoting effects with decreased rapid-eye-movement sleep in orexin-B saporin lesioned rats sup

255 for the spike index in NR stage 2 and rapid eye-movement sleep.

256 re characterized by a larger amount of rapid eyes movement sleep with dominant theta waves without at

257 leep, but also a disinhibition of REM (rapid eye movement) sleep, demonstrated as a shortening of REM

258 how that humans finely tune their fixational eye movements so that they greatly contribute to normal

259 Small ballistic eye movements, so called microsaccades, occur even while

260 served signatures of exploratory behavior in eye movements, such as quicker, more entropic fixations

261 mbing fibers when monkeys were engaged in an eye movement task.

262 r information about the direction of planned eye movements than superficial neurons.

263 We observed eye movements that appeared to continuously track the go

264 The eye movements that did occur coinciding with the present

265 compensate for the reduced vigour of smooth eye movements that occurs with the ingestion of low-dose

266 Saccades are rapid eye movements that orient the visual axis toward objects

267 nlike the role of the OMV in the guidance of eye movements, the contribution of the adjoining vermal

268 of vision, most notably through the study of eye movements, the development of signal detection theor

269 l eye fields (FEF) while initiating vergence eye movements, the inward and outward rotation of the ey

270 that the CD cue triggers horizontal vergence eye movements, the role of the IOVD cue has only recentl

271 The sequence of events leading to an eye movement to a target begins the moment visual inform

272 ively sample their environment with saccadic eye movements to bring relevant information into high-ac

273 lculation or one using an internal signal of eye movements to compensate for their effects.

274 Humans and other primates rely on eye movements to explore visual scenes and to track movi

275 For example, smooth pursuit eye movements to follow a moving target are slower and l

276 bserved very short-latency changes in smooth eye movements to minimize such errors.

277 ur results reveal that mice combine head and eye movements to sample their environment and highlight

278 uce an optokinetic response (OKR) control of eye movements to stabilize vision.

279 eys while they covertly attended or prepared eye movements to visual stimuli.

280 tion was applied using a permutation test to eye-movement transitions at two granularity levels: betw

281 concurrently measuring cortical activity and eye movements using EEG and eye tracking while observers

282 tionship between OKN gain (ratio of tracking eye-movement velocity to stimulus velocity) and MSEs (-

283 In the case of eye movements, visual and motor signals coexist in indiv

284 The second type of eye movements was conjugate and coupled to head yaw rota

285 By analysing eye movements, we found that toddlers took longer to set

286 Even in head-restrained mice, eye movements were invariably associated with attempted

287 Eye movements were measured to identify differences in c

288 inverted stimuli (<=4 degrees ) while their eye movements were monitored.

289 Eye-movements were recorded using a remote eye tracker.

290 tural images, jittered to emulate fixational eye movements, were accurately predicted by the subunit

291 tation of reward increases speed of saccadic eye movements, whereas expectation of effort decreases t

292 ver, visual perception is tightly coupled to eye movements, which are necessarily sequential.

293 The first type comprised non-conjugate eye movements, which compensate for head tilt changes to

294 ion is between imagery eye movements and the eye movements while looking at the original image is unc

295 rded single neurons in the human pre-SMA and eye movements while subjects performed goal-directed vis

296 ct the task-relevant stimulus for a saccadic eye movement, while inhibiting saccades to task-irreleva

297 and at different distances, we must generate eye movements with appropriate versional and vergence co

298 fixate pattern is similar to humans who use eye movements (with or without head movement) to rapidly

299 tinuum in the modulations given by different eye movements, with oculomotor transitions primarily act

300 l integration between perception and action: eye movements work synergistically with the spatio-tempo