コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 n a cohort of rabbits before and after trace eyeblink conditioning.
2 neurons have been shown ex vivo, after trace eyeblink conditioning.
3 bit a parallel pattern of timing deficits in eyeblink conditioning.
4 role in establishing cross modal savings of eyeblink conditioning.
5 hich is consistent with the requirements for eyeblink conditioning.
6 hereas the hippocampus is critical for trace eyeblink conditioning.
7 in hippocampal neurons after learning trace eyeblink conditioning.
8 s (IpN) neurons over the course of Pavlovian eyeblink conditioning.
9 cy auditory CS pathway that is necessary for eyeblink conditioning.
10 s delay and half were tested in 500-ms trace eyeblink conditioning.
11 role, if any, of cerebellar cortex in trace eyeblink conditioning.
12 exhibited equivalent levels of differential eyeblink conditioning.
13 learning-specific cerebellar plasticity and eyeblink conditioning.
14 ns of the medial septum significantly retard eyeblink conditioning.
15 sal paradigm was examined in human classical eyeblink conditioning.
16 hippocampal-dependent learning task of trace eyeblink conditioning.
17 ice displayed defective spatial learning and eyeblink conditioning.
18 acquisition of hippocampally dependent trace eyeblink conditioning.
19 bellar interpositus nucleus during classical eyeblink conditioning.
20 generation (pcd) mutant mice are impaired in eyeblink conditioning.
21 ion of mRNAs in brains of rabbits undergoing eyeblink conditioning.
22 factor deficit, severe ataxia, and impaired eyeblink conditioning.
23 quisition in trace, but not delay, classical eyeblink conditioning.
24 quired CRs during 10 days of classical delay eyeblink conditioning.
25 ts who had previously shown equivalent delay eyeblink conditioning.
26 formation of plastic changes responsible for eyeblink conditioning.
27 erable interest in the neurobiology of human eyeblink conditioning.
28 ring in Purkinje cells, might be involved in eyeblink conditioning.
29 at various times after acquisition of trace eyeblink conditioning.
30 llum block both acquisition and retention of eyeblink conditioning.
31 tex and deep nuclei are important for normal eyeblink conditioning.
32 mber test, and impaired cerebellar-dependent eyeblink conditioning.
33 rtex (MC) and its possible role in classical eyeblink conditioning.
34 lls, resulting in an impaired acquisition of eyeblink conditioning.
35 ed to form associative memories during delay eyeblink conditioning.
36 associative learning task, called Pavlovian eyeblink conditioning.
37 d reinforcement value, within the context of eyeblink conditioning.
38 ulus (CS-US) time intervals during classical eyeblink conditioning.
39 ff that served as the instructive signal for eyeblink conditioning.
40 in rat pups while they were trained on trace eyeblink conditioning.
41 memory retention for whisker-signaled trace eyeblink conditioning.
42 tructive stimuli during cerebellar-dependent eyeblink conditioning.
43 sis on old arguments and new perspectives on eyeblink conditioning.
44 memory, including cerebellum-dependent delay eyeblink conditioning.
45 ntation of temporal information in classical eyeblink conditioning.
46 ultaneous feature-negative discrimination in eyeblink conditioning.
47 ural substrates for standard delay classical eyeblink conditioning.
48 underlie behavioral cross-modal transfer in eyeblink conditioning.
49 ex for its role in forebrain-dependent trace eyeblink conditioning.
50 mals were trained 24 hr later with classical eyeblink conditioning.
51 t been systematically demonstrated in rodent eyeblink conditioning.
52 were behaviorally naive or trained on trace eyeblink conditioning.
53 rebellar learning was investigated using rat eyeblink conditioning.
54 ulus input to the cerebellum during auditory eyeblink conditioning.
55 T) and CaMKIV KO mice were tested with delay eyeblink conditioning.
56 aint and intermittent tailshock on classical eyeblink conditioning 24 h after stressor cessation.
57 nt effects of luvadaxistat at either dose in eyeblink conditioning, a cerebellar-dependent learning m
58 ut to Purkinje cells, and a deficit in delay eyeblink conditioning, a cerebellum-dependent form of le
60 we provide evidence that the development of eyeblink conditioning, a form of associative learning th
61 vivo: VGF and the IEGs increased after trace eyeblink conditioning, a hippocampal-dependent learning
62 nces, we trained freely moving rats in trace eyeblink conditioning, a hippocampally dependent task in
64 roles of the cerebellar cortex and nuclei in eyeblink conditioning, a novel mouse model with Purkinje
65 the acquisition rate of cerebellum-dependent eyeblink conditioning, a type of associative motor learn
69 se findings contribute to evidence of robust eyeblink conditioning abnormalities in schizophrenia and
70 ty of CB1KO mice accounts for their impaired eyeblink conditioning across both animals and trials.
72 ess a severe learning deficit in associative eyeblink conditioning after a stressful life event, but
73 tched healthy controls by means of classical eyeblink conditioning and blink reflex recovery cycle be
74 well as the eyeblink CR, is acquired during eyeblink conditioning and influences the development of
75 their young adult counterparts in both trace eyeblink conditioning and Morris water maze learning.
76 bellar cortex in normal acquisition of delay eyeblink conditioning and MWM and raise questions about
79 t both GC and PC signaling are essential for eyeblink conditioning and vestibulo-ocular reflex (VOR)
80 granule-cell-specific CB1KOs exhibit normal eyeblink conditioning, and both global and granule-cell-
81 ve, which is a component of the circuitry of eyeblink conditioning, and is also essential for motor p
82 lts suggest that, even during a simple delay eyeblink conditioning, animals learn about different asp
83 tial cerebellar brain circuits for Pavlovian eyeblink conditioning appeared relatively complete by 20
85 for the interpositus nucleus to learn delay eyeblink conditioning as the ISI departs from an optimal
86 ssful experiences include classical fear and eyeblink conditioning, as well as processes related to l
87 Thus, pcd mice are partially impaired in eyeblink conditioning because of a deficiency in learnin
88 r mice exhibit attenuated anterior-dependent eyeblink conditioning, but faster nodular-dependent comp
89 ppocampus plays a critical role during trace eyeblink conditioning, but there is no evidence to date
90 of hippocampus-dependent tasks such as trace eyeblink conditioning by aging subjects may be caused by
91 rhinal cortex plays a role in discriminative eyeblink conditioning by resolving ambiguity in discrimi
93 s, Pavlovian fear conditioning and Pavlovian eyeblink conditioning, by describing studies using mutan
94 been debate about whether differential delay eyeblink conditioning can be acquired without awareness
95 prenatal or perinatal physiological insults, eyeblink conditioning can provide a well-studied method
96 conducted on a cohort of rabbits undergoing eyeblink conditioning can reveal functional brain connec
98 hippocampus may encode different features of eyeblink conditioning during discrimination and reversal
105 he technique is described and acquisition of eyeblink conditioning (EBC) with stimulation of a single
106 d trimester, also show deficits in classical eyeblink conditioning (EBC), a cerebellar-dependent asso
107 PNNs in the mouse DCN are diminished during eyeblink conditioning (EBC), a form of associative motor
108 causes SCA6-like symptoms, i.e., deficits in eyeblink conditioning (EBC), ataxia, and PC degeneration
109 ar reflex (VOR) gain adaptation but impaired eyeblink conditioning (EBC), which relies on the ability
121 l cerebellar cortex and deep nuclei to delay eyeblink conditioning have been debated and are difficul
122 llum and impairments in cerebellar-dependent eyeblink conditioning have been observed in ADHD, prompt
123 llum and impairments in cerebellar-dependent eyeblink conditioning have been observed in attention-de
125 e removal of the medial septum retards delay eyeblink conditioning in a manner similar to the disrupt
126 In this study, we demonstrate that pavlovian eyeblink conditioning in adult mice can induce robust ax
129 by previous lesion and recording studies of eyeblink conditioning in animals and humans and suggest
131 found impairment in the acquisition of delay eyeblink conditioning in comparison with their wild-type
138 an acute stressful event enhances classical eyeblink conditioning in male rats, but severely impairs
139 e to an acute stressful event enhances trace eyeblink conditioning in male rats, even when rats begin
140 Acute stress exposure enhances classical eyeblink conditioning in male rats, whereas exposure to
141 Acute inescapable stress enhances classical eyeblink conditioning in male rats, whereas the same str
142 y over multiple days of cerebellum-dependent eyeblink conditioning in mice, that granule cell populat
147 rts the development of procedures to conduct eyeblink conditioning in preweanling lambs and demonstra
148 nt were studied on Pavlovian delay and trace eyeblink conditioning in rabbits (Oryctolagus cuniculus)
149 the acquisition and performance of classical eyeblink conditioning in rabbits using a delay paradigm.
150 wo hallmark features of cerebellar-dependent eyeblink conditioning in rabbits: (1) gradual acquisitio
151 to show that the developmental emergence of eyeblink conditioning in rats is associated with the mat
159 strain, Wistar-Kyoto (WKY) rats, to compare eyeblink conditioning in strains that are exclusively hy
160 ul event is reported to facilitate classical eyeblink conditioning in the male rat (Rattus norvegicus
163 mpal slices 24 hr after acquisition of trace eyeblink conditioning in young adult and aging rabbits.
165 gated in both trace and delay discrimination eyeblink conditioning in young and aging participants, i
166 ngs with a 200-ms trace interval resulted in eyeblink conditioning in younger animals than previously
167 ly and remotely acquired memory in rat trace eyeblink conditioning, in which a stimulus-free interval
168 plasticity mechanisms may also contribute to eyeblink conditioning including LTP, excitability, and e
169 of the sensory input pathways necessary for eyeblink conditioning indicate that the cerebellum regul
170 e impairment in the acquisition of classical eyeblink conditioning, indicating cerebellar malfunction
171 verely impaired acquisition and retention of eyeblink conditioning, indicating that the amygdala cont
179 standard model of the mechanisms underlying eyeblink conditioning is that there two synaptic plastic
183 We have shown that, in cerebellar-dependent eyeblink conditioning, male WKHAs emit eyeblink CRs with
185 yeblink CR to equal levels, suggest that rat eyeblink conditioning may provide a useful behavioral mo
188 that the hippocampus is active during trace eyeblink conditioning or is differentially responsive to
190 terns in the region during blocks of a trace eyeblink conditioning paradigm performed in two environm
191 gus cuniculus), whose performance in a delay eyeblink conditioning paradigm was compared with that of
192 sker stimulation as a CS in the well studied eyeblink conditioning paradigm will facilitate character
193 Adult male rats were trained using the trace eyeblink conditioning paradigm, an associative learning
194 nisms are being systematically examined with eyeblink conditioning paradigms in nonprimate mammalian
196 BLA, respectively) was recorded during delay eyeblink conditioning, Pavlovian fear conditioning, and
197 ust results on a battery of tests, including eyeblink conditioning, prepulse inhibition of acoustic s
198 and rabbits has been shown to support trace eyeblink conditioning, presumably by providing an input
200 These methods will permit application of eyeblink conditioning procedures in the analysis of func
201 present study utilized previously determined eyeblink conditioning procedures that effectively decoup
205 , breast feeding, poison-avoidance learning, eyeblink conditioning, sexual conditioning, fear conditi
206 spinach-enriched lab chow diet learned delay eyeblink conditioning significantly faster than old anim
209 strain, Wistar, were trained on a long-delay eyeblink conditioning task in which a tone conditioned s
210 hat learning the hippocampus-dependent trace eyeblink conditioning task induces enhanced inhibition o
211 week later with paired stimuli using a trace eyeblink conditioning task or exposed to the same number
212 iency impairs cerebellar learning in a delay eyeblink conditioning task, a common test of cerebellar
217 ociated temporal lobe regions play a role in eyeblink conditioning that becomes essential in more com
220 d animals did not exhibit faster learning in eyeblink conditioning, the peak timing of their conditio
221 gic system is demonstrated to be involved in eyeblink conditioning, this experiment was undertaken to
222 e responsive to shock from an early age, but eyeblink conditioning to a tone-conditioned stimulus (co
223 ously from multiple tetrodes during auditory eyeblink conditioning to examine the relative timing of
224 (fMRI) in parallel with both delay and trace eyeblink conditioning to image the learning-related func
225 r the learning of a tactile variant of trace eyeblink conditioning (TTEBC) and undergoes distinct map
226 dy examined the role of cerebellar cortex in eyeblink conditioning under conditioned stimulus?uncondi
227 during classical discrimination and reversal eyeblink conditioning using 2 tones as the conditioned s
231 e of the perirhinal cortex in discriminative eyeblink conditioning was examined by means of feature-p
233 -ms CS, 500-ms trace interval, 1,250-ms ISI) eyeblink conditioning was examined in 5-month-old human
234 lus (CS) pathway that is necessary for delay eyeblink conditioning was investigated with induced lesi
236 rkinje cell degeneration, and standard delay eyeblink conditioning was performed in the conditional k
237 ecific effects, the acquisition of classical eyeblink conditioning was potentiated or depressed by th
241 hanisms underlying excitatory and inhibitory eyeblink conditioning were compared using muscimol inact
243 correlates of latent inhibition (LI) during eyeblink conditioning were studied in 2 experiments.
245 ats as young as 12 days old show associative eyeblink conditioning when pontine stimulation is used i
246 ncies is not required for differential delay eyeblink conditioning when simple conditioned stimuli ar
247 is exemplified in cerebellar-dependent delay eyeblink conditioning, where arbitrary cues such as a to
248 the LEC had no effect on retrieval in delay eyeblink conditioning, where two stimuli were presented
249 rebellum is involved in both delay and trace eyeblink conditioning whereas the hippocampus is critica
250 stressful event did not exhibit facilitated eyeblink conditioning, whereas those infused with the ve
251 We used the behavioral paradigm of trace eyeblink conditioning, which is a hippocampus-dependent
252 l, TRPC3 loss-of-function mice show impaired eyeblink conditioning, which is related to Z- modules, w
253 occur robustly in both eyelids of rats given eyeblink conditioning, which is similar to previous find
254 t, the BLA exhibited minimal activity during eyeblink conditioning, while demonstrating pronounced in
255 ained with a temporal learning task of trace eyeblink conditioning, while the other half were not tra
256 , all rats underwent 10 days of 350 ms delay eyeblink conditioning with a tone conditioned stimulus (
257 bellar interpositus nucleus during classical eyeblink conditioning with a tone conditioned stimulus a
258 llowed by twenty 100-trial sessions of delay eyeblink conditioning with a tone CS and then five sessi
259 aminergic projections and retarded Pavlovian eyeblink conditioning with low-salient conditional stimu
260 ion were measured before and after classical eyeblink conditioning with paired pontine stimulation (c
262 and the lateral pontine nucleus on classical eyeblink conditioning with tone or lateral reticular nuc
263 ge differences in cerebellum-dependent delay eyeblink conditioning, with 24-month mice showing impair