ライフサイエンスコーパス: facial nerve

1 itch evoked by electrical stimulation of the facial nerve.

2 uality depends primarily on the input of the facial nerve.

3 aneous rami of spinal nerves, but not by the facial nerve.

4 rlapping with the area of termination of the facial nerve.

5 ists for neurovascular contact involving the facial nerve.

6 lar compression of the proximal intracranial facial nerve.

7 resection, or radiation of a tumor near the facial nerve.

8 ves no motor or sensory innervation from the facial nerve.

9 d associative stimulation protocol using the facial nerve.

10 ese most likely represent motoneurons of the facial nerve.

11 well as the motor root of the trigeminal and facial nerves.

12 celerates the functional recovery of injured facial nerves.

13 ) in Schwann cells as well as in sciatic and facial nerves.

14 ry neurons located lateral and dorsal to the facial nerve-a region we termed the parafacial zone (PZ)

15 This is in contrast to 31.2% of facial nerve afferent endings in the nst which make syna

16 ll infiltration in the distal portion of the facial nerve after axotomy in mice.

17 were located ventrally to the nucleus of the facial nerve and extended from the caudal part of the nu

18 to originate from the recurrent ramus of the facial nerve and from dorsal rami of the spinal cord, de

19 gene deletion only results in defects in the facial nerve and not the glossopharyngeal and vagus nerv

20 r sectioning the ipsilateral branches of the facial nerve and resecting the superior cervical ganglia

21 of segmentation of the inner ear, ossicles, facial nerve and sigmoid sinus.

22 er that is characterized by paralysis of the facial nerves and variable other congenital anomalies.

23 ner ear; 0.85 for the ossicles; 0.75 for the facial nerve; and 0.86 for the sigmoid sinus.

24 rating distance A (p = 0.003) and horizontal facial nerve angle (p = 0.017) explained 44.0-59.9% of t

25 st every 1-degree increase in the horizontal facial nerve angle was associated with an 18.1% increase

26 the anatomy of the temporal branches of the facial nerve are depicted.

27 nto the facial muscle of adult rats prior to facial nerve avulsion.

28 In contrast, facial nerve axotomy in C.B-17 (-/-) severe combined imm

29 Facial nerve axotomy increased the total cofilin abundan

30 ermined that the mSOD1 molecular response to facial nerve axotomy is phenotypically regenerative and

31 n the current investigation, we utilized the facial nerve axotomy model and a presymptomatic amyotrop

32 FMN loss after axotomy, we superimposed the facial nerve axotomy model on presymptomatic mSOD1 mice

33 /- mice showed increased rate of death after facial nerve axotomy, a response documented for SOD1-/-

34 After a right facial nerve axotomy, facial motoneuron (FMN) survival i

35 Following facial nerve axotomy, nerve function is not fully restor

36 tect transected axon cell-autonomously after facial nerve axotomy.

37 Peripheral facial nerve (Bell) palsy has been reported and widely s

38 One complication is damage to the facial nerve branches, which can result in brow ptosis a

39 lence of neurovascular contact involving the facial nerve can be as high as 51% in patients asymptoma

40 Injury to the facial nerve can result in facial paralysis with major f

41 o surgery, and certainly after injury to the facial nerve, clinicians should consider the patient's g

42 The rat facial nerve (CN VII) controls the orbicularis oculi (OO

43 genic injury to the temporal branches of the facial nerve (CN VII) is the leading postoperative compl

44 e hypertonicity of muscles innervated by the facial nerve, commonly attributed to the aberrant regene

45 the treatment of adult mice with LiCl after facial nerve crush injury stimulated the expression of m

46 We have used the adult mouse facial nerve crush model and adult-onset conditional dis

47 without CNTFRalpha, even when challenged by facial nerve crush or the injection-associated trauma, t

48 However, the incidence of facial nerve damage after TAB is unknown.

49 There was no correlation with facial nerve damage and use of blood thinners, biopsy re

50 Postoperative facial nerve damage was found in 12 patients (16.0%) and

51 There is a 16.0% incidence of postoperative facial nerve damage with TABs, which recovers fully in o

52 Incidence of postoperative facial nerve damage, other complications, and rates of f

53 performed for any potential correlation with facial nerve damage.

54 brow were less likely to have postoperative facial nerve damage.

55 pathway on regeneration across a 5-mm rodent facial nerve defect.

56 ngeal nerve and was not sufficient to rescue facial nerve defects, suggesting that FGF8 is functional

57 xin conveyed SEMA3/neuropilin signals during facial nerve development, we combined an expression anal

58 ally redundant with other RTK ligands during facial nerve development.

59 ns, PLXNA1 and PLXNA2 were not essential for facial nerve development.

60 brow were more likely to have postoperative facial nerve dysfunction.

61 t, share the same migratory behaviour to the facial nerve exit points and express the same markers as

62 internal carotid arteries) or osteichthyans (facial nerve exiting through jugular canal, endolymphati

63 means of pulsed magnetic stimulation of the facial nerve for the purpose of increasing cerebral bloo

64 providing normal viability as well as proper facial nerve formation even in the Hoxb1 mutant backgrou

65 the variation observed in the breadth of the facial nerve-free zone near the TMJ.

66 Facial nerve function, swallow, and voice quality were n

67 sults in optimal preservation of hearing and facial nerve function.

68 resection and those with worse postoperative facial nerve function.

69 hiomeric motor neurons of the trigeminal and facial nerves generate spontaneous [Ca2+]i transients th

70 imaging of axial cryosections of human cross-facial nerve grafts demonstrated enhanced resolution of

71 ilateral weakness in the distribution of the facial nerve has been reported.

72 The ZMB contains axons from the facial nerve; however, myelination within the barbel its

73 We noted, nonetheless, a specific facial nerve hypomorphism in hemizygous Hoxb1(A1/-) mice

74 and for the rehabilitation of patients with facial nerve impairments.

75 tine magnetic resonance imaging (MRI) of the facial nerve in patients with suspected idiopathic perip

76 tion of the sciatic nerve in rodents and the facial nerve in rabbits.

77 rning algorithm to predict the likelihood of facial nerve injury following microsurgical resection of

78 of motor versus sensory nerve grafting after facial nerve injury has not been previously investigated

79 responses in vivo with or without peripheral facial nerve injury in male and female mice.

80 nerve injury is disabling for patients, and facial nerve injury is particularly debilitating due to

81 In the hamster facial nerve injury paradigm, we have established that a

82 racy, 90% sensitivity and 90% specificity in facial nerve injury prognostication.

83 When validated via prospective assessment of facial nerve injury risk, this model demonstrated 84% ac

84 bgaleal hemorrhage, intracranial hemorrhage, facial nerve injury, and brachial plexus injury (BPI).

85 ation, defined as House-Brackmann (HB) I vs. facial nerve injury, defined as HB II-VI, as determined

86 generation in a clinically relevant model of facial nerve injury.

87 towards the brainstem as prognosticators of facial nerve injury.

88 Facial nerve innervated taste buds (FITBs) are thought t

89 In catfish, the facial nerve innervates taste buds distributed over the

90 Given that the facial nerve is a pure motor nerve, we presume that the

91 deformity of the susceptible portion of the facial nerve is highly associated with the symptomatic s

92 yngeal and vagus nerves, suggesting that the facial nerve is most sensitive to perturbations in RTK s

93 Magnetic resonance imaging of the facial nerve led to a correction of the diagnosis in 8 p

94 ha-internexin protein expression after three facial nerve lesion paradigms: crush, transection, and r

95 lled forelimb training in conjunction with a facial nerve lesion, cholinergic mechanisms were require

96 blink as a functional recovery parameter of facial nerve lesion.

97 uggest the added value of the routine use of facial nerve MRI in suspected cases of BP.

98 ed by local infiltration around the lids and facial nerve (n = 6), topical anesthesia (n = 5), and ge

99 ), among whom 106 (94.6%) showed evidence of facial nerve neuritis on the affected side (hypersignal

100 sates at the level of the caudal pole of the facial nerve nucleus in the rostral medulla oblongata.

101 Taste and tactile fibers in the facial nerve of catfish innervate extraoral taste buds a

102 d macrophage accumulation was reduced in the facial nerve of CD38 KO mice after axotomy.

103 nerve innervation to fungiform papillae, the facial nerve of developing animals was labeled with the

104 g to decipher predictive factors relevant to facial nerve outcomes following microsurgical resection

105 As facial nerve outcomes remain challenging to prognosticat

106 t of the cochlear basal turn and the mastoid facial nerve (p = 0.040).

107 als to the posterior auricular branch of the facial nerve (PA neurons).

108 es), lateral canthal coloboma (3 cases), and facial nerve palsy (1 case).

109 Facial nerve palsy (FNP) in children presents treatment

110 One monkey, with prior facial nerve palsy and a very steep amplitude versus pea

111 id, excluding one monkey who was affected by facial nerve palsy and was analyzed separately.

112 re, analysis of the number of admissions for facial nerve palsy during the same period in preceding y

113 Thirty-seven patients were admitted for facial nerve palsy during the study period, 22 (59.5%) o

114 son with the overall number of patients with facial nerve palsy in preceding years was performed.

115 Facial nerve palsy is a potentially devastating conditio

116 Appropriate management of facial nerve palsy is dependent on a multitude of factor

117 Patients admitted for facial nerve palsy were matched by age, sex, and date of

118 ith this early effect were aberrant tearing, facial nerve palsy, ear malformations, and autism.

119 he underlying anatomy and pathophysiology of facial nerve palsy, while also exploring different treat

120 aring contralateral to an eyelid weakened by facial nerve palsy.

121 e among patients with acute-onset peripheral facial nerve palsy.

122 nation with the BNT162b2 vaccine and risk of facial nerve palsy.

123 patient's morbidity, especially the risk of facial nerve palsy.

124 the complications associated with permanent facial nerve palsy.

125 ght strategies in the surgical management of facial nerve palsy.

126 gical procedures for patients with permanent facial nerve palsy.

127 eys and, more dramatically, in a monkey with facial nerve palsy.

128 sions in 23 percent, arthritis in 6 percent, facial-nerve palsy in 3 percent, aseptic meningitis in 2

129 rogens enhance both functional recovery from facial nerve paralysis and the rate of regeneration in t

130 rove understanding and clinical treatment of facial nerve paralysis, mitigating facial asymmetry, abe

131 None of the patients had permanent facial nerve paralysis.

132 unilateral vision loss (3 malignancies), and facial nerve paresis (5 malignancies).

133 g experiments for the study of the autonomic facial nerve pathway in birds in terms of both its anato

134 rm the somatic motor component of the VIIth (facial) nerve, possibly through a failure to specify the

135 ised machine learning classifiers to predict facial nerve preservation, defined as House-Brackmann (H

136 cally relevant animal models for research in facial nerve reconstruction is challenging.

137 the sural nerve can be successfully used for facial nerve reconstruction research in a clinically rel

138 ine sural nerve as a potential candidate for facial nerve reconstruction, and performed its histologi

139 ve damage, other complications, and rates of facial nerve recovery were evaluated.

140 changes, PTEN deletion positively regulated facial nerve regeneration and recovery of whisker moveme

141 Testosterone propionate (TP), augments facial nerve regeneration in the adult hamster.

142 eltaNLS-GFP) stimulated axonal sprouting and facial nerve regeneration in vivo.

143 ferents are segregated in the trigeminal and facial nerves, respectively.

144 ode at the facial motor nerve and found that facial nerve spiking is functionally associated with the

145 (LAI) afferent inhibition of face M1, while facial nerve stimulation evoked LAI but not SAI.

146 In contrast, facial nerve stimulation produced significant LAI (P < 0

147 By contrast, movements evoked by facial nerve stimulation showed no such frequency-depend

148 The effect of facial nerve stimulation was found to be dependent on st

149 enuated the cerebral vasodilator response to facial nerve stimulation when applied locally to the cor

150 evelopment of a non-invasive pulsed magnetic facial nerve stimulator that will increase CBF as a trea

151 contact along the intracranial course of the facial nerve, the culprit vessel, and the severity of co

152 xamined motor versus sensory grafting of the facial nerve to investigate effect of pathway on regener

153 s 4 and 5 underwent surgical severing of the facial nerve (to cause complete paralysis of the OOM).

154 t neurons, located dorsal to the genu of the facial nerve, to the cerebellar flocculus and ventral pa

155 drotestosterone on FMNs of P7 hamsters after facial nerve transection at the SMF.

156 adult male hamsters were subjected to right facial nerve transection at the stylomastoid foramen.

157 We performed a facial nerve transection axotomy in both mSOD1 subgroups

158 and synapse recovery following extracranial facial nerve transection in mice.

159 Upon facial nerve transection, damaged CD200-deficient neuron

160 After facial nerve transection, neither compartment alone is s

161 for mediating plasticity associated with the facial nerve transection.

162 forms of plasticity within the motor cortex: facial nerve transections evoke reorganization of cortic

163 r contact of the intracranial segment of the facial nerve typically occurs at one point, involving th

164 comparison to the buccal branch (BB) of the facial nerve using cadaver and anesthetized sheep.

165 graded as contact alone (vessel touching the facial nerve) versus deformity (indentation or deviation

166 ng the first and second transplantation, the facial nerve was coapted at the level of the branches.

167 the distribution of temporal branches of the facial nerve was examined in 20 cadaveric donors.

168 ation of neurovascular compression along the facial nerve was recorded.

169 eduction in complications, such as immediate facial nerve weakness and Frey syndrome.

170 d in 7 of 27 articles and included transient facial nerve weakness, persistent blue dye staining of t

171 ) and results of contrast enhancement of the facial nerve were described.

172 rm the somatic motor component of the VIIth (facial) nerve which controls the muscles of facial expre

173 Interestingly, sectioning the facial nerve, which abolished whisker movements, did not

174 hat carry a risk of iatrogenic injury to the facial nerve, which can significantly impact patients' q

175 RP/PLXN signalling in the development of the facial nerve, which contains axons from two motor neuron

176 Fibers of the facial nerve, which innervate all external taste buds, r

177 ctive appearance of the internal genu of the facial nerve, which is lacking in birds.

178 ry sulci, olfactory bulbs and oculomotor and facial nerves, which support underlying abnormalities in

179 suspected BP underwent an MRI of the entire facial nerve with a double-blind reading of all images.

180 is an acute and idiopathic paralysis of the facial nerve, with an estimated incidence ranging from 1

181 focal magnetic field was directed toward the facial nerve within the temporal bone by placing a 6.5 c