ライフサイエンスコーパス: facial nucleus

1 for transmitting information from MI to the facial nucleus.

2 ensory inputs to vibrissa motoneurons in the facial nucleus.

3 d fine varicose nerve fiber terminals in the facial nucleus.

4 l trigeminal nucleus, and motoneurons of the facial nucleus.

5 nsely packed cell group ventrolateral to the facial nucleus.

6 shows significant dysregulation in the mSOD1 facial nucleus.

7 end dorsolaterally to partially encircle the facial nucleus.

8 inergic motor neurons located in the lateral facial nucleus.

9 ical structures (0.47 ug . g(-1) +/- 0.10 in facial nucleus, 0.39 ug . g(-1) +/- 0.10 in choroid plex

10 m, contains neurons that project directly to facial nucleus (7n) motoneurons to coordinate orofacial

11 s tractus solitarii, lateral medulla, medial facial nucleus, A5 area, lateral vestibular nucleus, and

12 cting the brainstem at the caudal end of the facial nucleus abolished active expirations, while rhyth

13 or nuclei that mediate expression of the CR (facial nucleus and accessory abducens) were reversibly i

14 solateral to the rostral-most portion of the facial nucleus and caudal superior olive, where they int

15 RVM and caudal pons were found medial to the facial nucleus and lateral to the pyramid in a column di

16 brain exhibited near-complete absence of the facial nucleus and superior olive along with shortening

17 ttes at 100-200 microm intervals between the facial nucleus and the calamus scriptorius.

18 spinal and principal trigeminal nuclei, the facial nucleus, and the lateral reticular nucleus.

19 that the perioral muscles innervated by the facial nucleus are rhythmically coordinated during lipsm

20 The projection to the facial nucleus arises from nearby reticular neurons, whe

21 internuclear interneurons projecting to the facial nucleus, as well as those neurons of the parvocel

22 e in alpha-internexin mRNA expression in the facial nucleus at 7 and 14 d after injury.

23 of the vagus, motor trigeminal nucleus, and facial nucleus, but not in most forebrain cholinergic ce

24 did not reveal any direct projections to the facial nucleus, but retrograde tracer injections in the

25 Within the facial nucleus, calcineurin-containing motoneurons were

26 Injections of retrograde tracers into the facial nucleus consistently labeled neurons in the hypog

27 that, during disease progression, the mSOD1 facial nucleus displays target disconnection-induced gen

28 he GHSR gene was confirmed in neurons of the facial nucleus (FacN, 7), the dorsal vagal complex (DVC)

29 entified by their antidromic activation from facial nucleus (FN) or red nucleus (RN).

30 hile their accessory abducens nucleus (ACC), facial nucleus (FN), and surrounding reticular formation

31 he coordination of muscles innervated by the facial nucleus has not been carefully compared between c

32 ctions from trigeminal sensory nuclei to the facial nucleus have been described, but the pathway wher

33 n neuronal death and glial activation in the facial nucleus in the brain stem, and on axon degenerati

34 ating active expiration located close to the facial nucleus in the region of the retrotrapezoid nucle

35 e hypoglossal nucleus of animals with severe facial nucleus involvement.

36 der motor neurons were found in dorsolateral facial nucleus ipsilaterally.

37 noradrenergic cell group and overlapping the facial nucleus lateral subnuclei and para-facial zones.

38 he case of Adv.RSV-GDNF, a greater number of facial nucleus motoneurons survived than were transduced

39 fibers and send monosynaptic connections to facial nucleus motoneurons that directly innervate vibri

40 In the facial nucleus, neuron number consistently declined (48%

41 d Adv.RSV-GDNF, a significant portion of the facial nucleus neurons was protected, 16.5, 18.2, and 53

42 xpressed in the olfactory bulb, red nucleus, facial nucleus, pontine nucleus, oculomotor nucleus, sub

43 The facial nucleus received input from all face representati

44 sis of the superior olive, dysgenesis of the facial nucleus, reduced numbers of Purkinje neurons, hyp

45 siologically identified wFMNs in the lateral facial nucleus resulted in dense, bilateral labeling thr

46 eus, but retrograde tracer injections in the facial nucleus revealed some labeled neurons in MI corte

47 ot eliminate the severe phenotype in the FPG facial nucleus, suggesting that the FPG phenotype is the

48 d documented corticofugal projections to the facial nucleus, surrounding pontine reticular formation,

49 lateral tegmental field (LTF) extending from facial nucleus to calamus scriptorius.

50 Cells in the nearby facial nucleus upregulated c-fos in the same number of c

51 ne and medullary), motor trigeminal nucleus, facial nucleus, vestibular nucleus, dorsal motor nucleus

52 oject to the trigeminal motor nucleus (Vmo), facial nucleus (VII) and hypoglossal nucleus (XII) are a

53 some studies point to the caudal tip of the facial nucleus (VIIc) as the oscillator's core, others s

54 ns (EBs) have been found at the level of the facial nucleus (VIIn), and 500 mum caudally, within the

55 nimals, the musculotopic organization of the facial nucleus was defined by injecting fluorescent retr

56 t of fluorogold from the whisker pads to the facial nucleus was seen only in motoneurons that lacked

57 The nuclear volume of the facial nucleus was smaller in the SOD-1 mutant mice (45.

58 o musculotopically defined subsectors of the facial nucleus was studied from the face representation

59 the levels of CX3CR1 and fractalkine in the facial nucleus were evident.

60 vectors were retrogradely transported to the facial nucleus where the NFs or beta-gal were expressed.

61 showed reduced microglial activation in the facial nucleus, whereas the levels of neuronal death wer

62 All treatments spared the facial nucleus, which forms still later.

63 rated in the intermediate subdivision of the facial nucleus, with a strong ipsilateral prevalence.