ライフサイエンスコーパス: facial palsy

1 re than did antibiotic-treated patients with facial palsy.

2 gnosis of Lyme borreliosis in cases of acute facial palsy.

3 ce in the cohort of patients without HFS and facial palsy.

4 as 51% in patients asymptomatic for HFS and facial palsy.

5 educed the burden of long-term disability in facial palsy.

6 ly had a sensorimotor variant 8/11 (73%) and facial palsy 7/11 (64%).

7 Two cases of facial palsy and 1 acute myeloid leukemia occurred.

8 e (GST)-Arp, as did 59% of the patients with facial palsy and 68% of those with Lyme arthritis.

9 burgdorferi infection in patients with acute facial palsy and a positive enzyme immunoassay result.

10 underlie changes in blinking associated with facial palsy and may play a role in the development of d

11 velopment of large datasets of patients with facial palsy and the translation of basic science eviden

12 c criteria (MDC) (congenital, nonprogressive facial palsy, and abduction deficit) and genetic testing

13 ommands, gaze palsy, abnormal visual fields, facial palsy, arm drift, leg drift, and abnormal languag

14 f life of adults ( 18 years) with peripheral facial palsy can be estimated using clinician measures o

15 Facial palsy (FP) can lead to significant psychological

16 e neuropsychiatric disorders, e.g. stroke or facial palsy, had a physical basis requiring the attenti

17 The resulting phenotype includes bilateral facial palsy, hearing loss, and strabismus and correlate

18 's palsy (BP), are the most common causes of facial palsy in borrelia-endemic areas and are clinicall

19 ck of large databases tracking patients with facial palsy, inherent selection bias, and the wide rang

20 Lyme neuroborreliosis peripheral facial palsy (LNB PFP) and idiopathic PFP, Bell's palsy

21 because they had abduction deficits without facial palsy or facial palsy with full ocular motility.

22 onnecticut, region who had erythema migrans, facial palsy, or Lyme arthritis 10-20 years ago and 30 u

23 ng, static and dynamic validated scoring for facial palsy patients, and complications.

24 aise (39%), paresthesias (32.5%), peripheral facial palsy (PFP) (36.4%), meningeal signs (19.5%), and

25 atients with suspected idiopathic peripheral facial palsy (PFP) (ie, Bell palsy [BP]).

26 se with a history of hemifacial spasm (HFS), facial palsy, traumatic brain injury, intracranial tumou

27 he rates of granulation tissue, otalgia, and facial palsy were 90.9%, 31.8%, and 9.1%, respectively.

28 ed quality of life in adults with peripheral facial palsy were included.

29 Moreover, patients with facial palsy who did not receive antibiotics for acute n

30 were apparent primarily among patients with facial palsy who did not receive antibiotics for acute n

31 However, patients with facial palsy, who frequently had more widespread nervous

32 us syndrome, especially in patients who have facial palsy with full ductions.

33 d abduction deficits without facial palsy or facial palsy with full ocular motility.